ライフサイエンスコーパス: eye movement

1 sual stimuli presented around the time of an eye movement.

2 es of visual neurons around the time of each eye movement.

3 al sensitivity is markedly reduced during an eye movement.

4 perceptual awareness and different types of eye movement.

5 fers from a normal, physiological divergence eye movement.

6 and is important for generating compensatory eye movements.

7 ct recognition) to the precision of saccadic eye movements.

8 adigm used to study the voluntary control of eye movements.

9 e was also observed to have slow and limited eye movements.

10 features shows the accuracy of 94% with five eye movements.

11 ational visual cues generated during pursuit eye movements.

12 ct is stationary, we view it with fixational eye movements.

13 can be established only by residual vertical eye movements.

14 ples underlying the generation of fixational eye movements.

15 reshapes feature selectivity contingent upon eye movements.

16 retinal displacement associated with saccade eye movements.

17 onhuman primates trained to execute saccadic eye movements.

18 there is no auditory accompaniment to visual eye movements.

19 ional velocities of either real or simulated eye movements.

20 ar saccade amplitudes and binocular vergence eye movements.

21 ion in favor of a motor explanation, namely, eye movements.

22 atypical restrictive strabismus and reduced eye movements.

23 one object to another, in a rapid series of eye movements.

24 visual cortex (V1) to regulate goal-directed eye movements.

25 ion is actively maximized through rotational eye movements.

26 n rate with behavioral costs of carrying out eye movements.

27 inates skilled voluntary movements including eye movements.

28 D modulation associated with visually guided eye movements.

29 ing visual neuron function in the context of eye movements.

30 related with eye position during spontaneous eye movements.

31 object moves, we view it with smooth pursuit eye movements.

32 that become active during different types of eye movements.

33 e planning of behaviorally relevant saccadic eye movements.

34 rms decisions comes from studies of saccadic eye movements.

35 ugate and vergence), vertical, and torsional eye movements.

36 ble, although we constantly perform saccadic eye movements?

37 lly update to new retinotopic locations with eye-movements?

38 ironments (29%), and often initiated by head/eye movements (60%).

39 detailed eyetracking investigations revealed eye movement abnormalities in 80% of patients with poste

40 Automatic eye movements accompanied each blink, and an aftereffect

41 sists gradual neuronal adjustments cause the eye movement again to land near the target.

42 ecific object detection classifiers to guide eye movements, aligns its fovea with regions of interest

43 of progressive balance, speech, swallowing, eye movement and cognitive impairment, ultimately leadin

44 e retina generally showed a higher degree of eye movement and higher head movement rate likely becaus

45 search by offering a unified account of both eye movement and manual response behaviour across the en

46 t is most prominent in lobules that regulate eye movement and process vestibular information.

47 s in freely moving mice, revealing increased eye movements and altered binocular coordination compare

48 hoice tasks in two distinct action contexts--eye movements and arm movements.

49 Eye movements and behavioural data demonstrated that pat

50 iscrete and fleeting, separated by expansive eye movements and discontinuous views of our spatial sur

51 y performing combined recordings of saccadic eye movements and fast event-related fMRI during a conti

52 imilar to that of wake, accompanied by rapid eye movements and muscle atonia.

53 isual information requires a coordination of eye movements and ongoing brain oscillations.

54 space-time function that best predicts both eye movements and perception of translating dot patterns

55 stinguish the retinal image shifts caused by eye movements and shifts due to movements of the visual

56 on of both retinal velocity and direction of eye movement, and we show that smooth eye movements modu

57 esents with congenital ptosis and restricted eye movements, and can be caused by heterozygous missens

58 increase in pupil-linked arousal, fixational eye movements, and fluctuations in bottom-up sensory pro

59 mphasized covert attention at the expense of eye movements, and others have focused on eye movements

60 the EL model is consistent with the choices, eye movements, and pupillary responses of subjects who c

61 In real-world search tasks, context guides eye movements, and task-irrelevant social stimuli may ca

62 even when decisions are communicated without eye movements, and that this interaction has a direction

63 biases, perceptual noise and inaccuracies in eye movements, and the central process of selecting fixa

64 characterized by hypotonia, ataxia, abnormal eye movements, and variable cognitive impairment.

65 ative about the underlying process, and that eye movements are an epiphenomenon that can be safely ig

66 Eye movements are an integral part of how we explore the

67 STATEMENT Outward-directed gaze-stabilizing eye movements are commanded by abducens motoneurons that

68 Visual perception and eye movements are considered to be tightly linked.

69 d that both rapid saccades and slow vergence eye movements are continuously recalibrated independentl

70 hin and across saccades to ensure that these eye movements are guided effectively by learned expectat

71 orimotor transformations leading to saccadic eye movements are implemented in the brain, less is know

72 Like every motor action, these eye movements are subject to noise and introduce instabi

73 ver, we revealed a strong modulation of slow eye movements around the R peak in the ECG.

74 he evolution of foveated visual systems with eye movements as a solution that preserves perceptual pe

75 ram (EEG), hippocampal theta activity, rapid eye movements, autonomic activation and loss of postural

76 tion between human and devices; for example, eye movement-based wheelchair control.

77 r the inter-specific variation in avian head/eye movement behavior.

78 physical stimulus constant by measuring the eye movement behaviour of a single group of neglect pati

79 tical retinal image displacements induced by eye movements, but the brain mechanisms underlying this

80 of the test that relies on the activation of eye movements by electrical stimulation of vestibular or

81 retinal and visual signals related to smooth eye movements can modulate the responses of neurons in a

82 When saccadic eye movements consistently fail to land on the intended

83 n regions involved in spatial processing and eye movement control.

84 The participants' eye movement data were recorded as they viewed the ECGs

85 elective fragmentation: R = 0.57, P < 0.001; eye movement density: R = 0.46, P < 0.01) in 32 polysomn

86 Eye movement desensitization and reprocessing (EMDR) is

87 Saccadic eye movements direct the high-resolution foveae of our r

88 ne retraction syndrome (DRS) is a congenital eye-movement disorder defined by limited outward gaze an

89 Our review links awareness to perceptual-eye movement dissociations and furthers our understandin

90 To prevent eye movement during the procedure, all 3 patients underw

91 found that FEF contributes to memory-guided eye movements during every epoch of the memory-guided sa

92 ss Scale, blink duration, and number of slow eye movements during postnight-shift drives compared wit

93 ng by measuring both diagnostic accuracy and eye movements during visual search.

94 ability in the reaction time and accuracy of eye-movements during a memory guided saccade task are re

95 es-oculomotor prosthetics-designed to modify eye movements dynamically by physical means in cases whe

96 ing low-frequency activity was predictive of eye movement dynamics tens of milliseconds in advance of

97 t help in designing drug therapies for human eye movement dysfunctions such as abducens nerve palsy.

98 generate different types of visually guided eye movements (e.g., saccades/smooth pursuit/vergence).

99 ish potential markers of visual expertise in eye movement (EM) patterns of early residents, advanced

100 During EMDR, patients make horizontal eye movements (EMs) while simultaneously recalling a tra

101 cortex of awake animals, small "fixational" eye movements (FEMs) inevitably introduce trial-to-trial

102 awake primates, however, small "fixational" eye movements (FEMs) introduce uncontrolled trial-to-tri

103 ptokinetic response (OKR) consists of smooth eye movements following global motion of the visual surr

104 We show that although humans adapt their eye movements for simpler tasks such as object following

105 study examines the consequences of saccadic eye movements for the internal representation of visual

106 Eye movements generated by simple brainstem circuits pro

107 s information about visual motion in pursuit eye movements guided by that cortical activity.

108 Such a corollary discharge circuit for eye movements has been identified in macaque monkey.

109 Avian saccadic head/eye movements have been shown to vary considerably betwe

110 phase and motion activity in slow fixational eye movements; i.e., retinal image slip caused by physio

111 with central vision loss should direct their eye movements in face identification tasks, which could

112 for reliable, high-resolution measurement of eye movements in freely moving mice, revealing increased

113 od, based on magnetic sensing, for measuring eye movements in head-fixed and freely moving mice.

114 fts of random textures matching saccade-like eye movements in mice elicit robust inhibitory inputs an

115 TATEMENT The mechanism by which humans adapt eye movements in response to central vision loss is stil

116 ns efficiently learn to adjust the timing of eye movements in response to environmental regularities

117 rain center involved in controlling head and eye movements in response to inputs from multiple sensor

118 Purkinje cells could effectively accelerate eye movements in the nasotemporal and temporonasal direc

119 Most eye movements in the real-world redirect the foveae to o

120 We investigated the effect of eye-movements in predictive feedback using functional br

121 ted to statistical modulations of fixational eye movements, in particular, the generation of microsac

122 ffects of a stationary mask on the reflexive eye movements induced by a moving stimulus.

123 e a novel approach for controlling for these eye-movement-induced effects.

124 By integrating this eye movement into blinks, the inevitable down times of v

125 tion to cognition; hence, the measurement of eye movements is an important tool in neuroscience resea

126 studies in sportsmen suggest that timing of eye movements is learned.

127 ound us as spatially stable despite frequent eye movements is one of the long-standing mysteries of n

128 A key structure for directing saccadic eye movements is the superior colliculus (SC).

129 iming, direction and targeting of individual eye movements, is strongly influenced by genetic factors

130 the spurious retinal motion generated by the eye movements, it is crucial that saccadic suppression a

131 ients with strabismus are able to make rapid eye movements, known as saccades, toward visual targets

132 Although visually evoked smooth eye movements, known as the optokinetic response (OKR),

133 In 10 of 13 subjects, the eye movement made after spontaneous loss of fusion was i

134 To characterize eye movements made by patients with intermittent exotrop

135 s is the optokinetic reflex (OKR), an innate eye movement mediated by the brainstem accessory optic s

136 ng of specific motoneuronal contributions to eye movements might help in designing drug therapies for

137 and erythema, conjunctival chemosis, pain on eye movement, minimal diplopia, the usual absence of pro

138 ion of eye movement, and we show that smooth eye movements modulate MT responses in a systematic, tem

139 Specifically, we show that smooth eye movements modulate the gain of responses of neurons

140 task-relevant spatial location while making eye-movements necessitates a rapid, saccade-synchronized

141 is a copy of the neuronal signal driving the eye movement, now referred to as a corollary discharge (

142 the transition to wakefulness from non-rapid eye movement (NREM) and rapid eye movement (REM) sleep.

143 d of two global behavioral states, non-rapid eye movement (NREM) and rapid eye movement (REM), charac

144 es: delta (0-3 Hz) activity during non-rapid eye movement (NREM) associated stages was greater than d

145 at 21:00 decreased the latency to non-rapid eye movement (NREM) sleep and increased the duration of

146 e, compared to wakefulness, during non-rapid eye movement (NREM) sleep TMS elicits a larger positive-

147 During non-rapid eye movement (NREM) sleep, cortical neurons alternate be

148 EEG delta (0.5-4 Hz) power during non-rapid eye movement (NREM) sleep, increased time spent in the N

149 During poststimulus nonrapid eye movement (NREM) sleep, LGN neuron overall spike-fiel

150 of stimuli was conserved in light non-rapid eye movement (NREM) sleep.

151 dominant consolidated trace during non-rapid eye movement (NREM) sleep.

152 s exhibited persistent reduction in nonrapid-eye-movement (NREM) and rapid-eye-movement (REM) sleep,

153 ow waves are a defining feature of non-rapid eye-movement (NREM) sleep and are thought to be importan

154 s and plausible mechanisms linking non-rapid-eye-movement (NREM) sleep disruption, Abeta, and AD; (ii

155 llations and sleep spindles during non-rapid-eye-movement (NREM) sleep has been proposed to support m

156 During non-rapid eye-movement (NREM) sleep, cortical and thalamic neurons

157 raction of the cardinal rhythms of non-rapid-eye-movement (NREM) sleep-the thalamo-cortical spindles,

158 ons in the prefrontal cortex during nonrapid-eye-movement (NREM) sleep.

159 iguration, head movement rate, and degree of eye movement of 29 bird species with a single fovea, con

160 on of the array was steered according to the eye movements of the participant as they followed a visu

161 ing spatial resolution and can program their eye movements optimally to maximize information acquisit

162 changes in pupil-linked arousal, fixational eye movements, or gamma-band responses were not necessar

163 Repetitive strain from eye movements over decades might in susceptible individu

164 oneurons are the major contributor to actual eye movements over the tested stimulus range.

165 es the hippocampus show alterations in their eye movement patterns and recent findings that the two s

166 tic factors do indeed contribute strongly to eye movement patterns, influencing both one's general te

167 d that Mayan and US infants utilize the same eye-movement patterns in which fixation duration and sac

168 which US participants diverge and engage in eye-movement patterns where fixation durations and sacca

169 ing: a distributed representation of learned eye-movement plans represented in domain-specific areas

170 Saccadic eye movements play a central role in primate vision.

171 onal symptoms; in particular, for functional eye movements, positive clinical signs such as convergen

172 Saccadic eye movements provide a valuable model to study the brai

173 Eye movements provide insights about a wide range of bra

174 leep fragmentation, abnormal short non-rapid eye movement - rapid eye movement (REM) cycling, REM sle

175 ently, adaptive increases in visually evoked eye movements rapidly restore oculomotor function in wil

176 n neuroscience to avoid misinterpretation of eye-movement-related artifacts as heart-evoked modulatio

177 retinal image motion with signals regarding eye movement relative to the scene.

178 orphological recovery.SIGNIFICANCE STATEMENT Eye movements rely on multiple neuronal circuits for app

179 1064 promotes sleep and increases both rapid eye movement (REM) and non-REM (NREM) sleep in rats at O

180 cturnal activity (siesta), a period of rapid eye movement (REM) and non-REM sleep, was absent in all

181 bnormal short non-rapid eye movement - rapid eye movement (REM) cycling, REM sleep reduction or loss,

182 ranslated into sustained inhibition of rapid eye movement (REM) in vivo.

183 tline key models for the regulation of rapid eye movement (REM) sleep and non-REM sleep, how mutual i

184 administration of glycine-induced non-rapid eye movement (REM) sleep and shortened NREM sleep latenc

185 IEDs also induce spindles during rapid-eye movement (REM) sleep and wakefulness-behavioral stat

186 Although quiet wake and rapid eye movement (REM) sleep are characterized by similar, l

187 The presence of probable rapid eye movement (REM) sleep behavior disorder was strongly

188 Rapid eye movement (REM) sleep behaviour disorder (RBD) is cha

189 Although wake and rapid eye movement (REM) sleep exhibit long timescales, these

190 Rapid eye movement (REM) sleep is a distinct brain state chara

191 Rapid eye movement (REM) sleep is a recurring part of the slee

192 Rapid eye movement (REM) sleep is an important component of th

193 sleep, whereas during the second half, rapid eye movement (REM) sleep is more predominant.

194 ts of this study suggest that baseline rapid eye movement (REM) sleep may serve a protective function

195 functions and underlying mechanisms of rapid eye movement (REM) sleep remain unclear.

196 bout equally during quiet wake and non-rapid eye movement (REM) sleep, and to a lesser degree during

197 g been implicated in the generation of rapid eye movement (REM) sleep, but the underlying circuit mec

198 Narcolepsy, a disorder of rapid eye movement (REM) sleep, is characterized by excessive

199 lf of the night, which is dominated by rapid eye movement (REM) sleep, led to better discrimination b

200 When dreaming during rapid eye movement (REM) sleep, we can perform complex motor b

201 stingly, IIS primarily occurred during rapid-eye movement (REM) sleep, which is notable because REM i

202 motor activity was related to time in rapid eye movement (REM) sleep.

203 from non-rapid eye movement (NREM) and rapid eye movement (REM) sleep.

204 high apnea and hypopnea indices during rapid eye movement (REM) sleep.

205 tes, non-rapid eye movement (NREM) and rapid eye movement (REM), characterized by quiescence and redu

206 g NREM states was lower than Awake and rapid eye movement (REM).

207 Recently, restless rapid-eye-movement (REM) sleep emerged as a robust signature o

208 ing of the mechanisms and functions of rapid-eye-movement (REM) sleep have occurred over the past dec

209 on in nonrapid-eye-movement (NREM) and rapid-eye-movement (REM) sleep, as well as increased sleep fra

210 lly, dreaming has been identified with rapid eye-movement (REM) sleep, characterized by wake-like, gl

211 ivity and behavioral states, including rapid eye-movement (REM) sleep.

212 At least for the eye movements reported here, a motion-from-form mechanis

213 Here we have studied eye movement representations in the SC of mice, a specie

214 months post initial presentation ptosis and eye movements returned normal and choroidal emboli absor

215 Examination of eye movements revealed that monkeys fixated the illusory

216 for sensory-motor latency in smooth pursuit eye movements reveals general principles of neural varia

217 to make the choice using one of two actions: eye movements (saccades) and arm movements (reaches).

218 However, this encoding of action for rapid eye movements (saccades) has remained unclear: Purkinje

219 Human observers make large rapid eye movements-saccades-to bring behaviorally relevant in

220 Eye movements serve to accumulate information from the v

221 re we demonstrate evidence for a new type of eye movement serving a distinct oculomotor demand, namel

222 imple foveated bottom-up saliency model with eye movements showed agreement in the selection of top s

223 , induced significant increases in non-rapid-eye movement sleep (NREMS) lasting for 4-10 h.

224 Sleep, and particularly rapid eye movement sleep (REM), has been implicated in the mod

225 echanisms that determine the amount of rapid eye movement sleep (REMS) and non-REMS (NREMS) remain un

226 ry brain vigilance states (waking, non-rapid eye movement sleep [NREM] and REM sleep) within an ultra

227 tive contribution of two sleep states, rapid eye movement sleep and slow-wave sleep, to offline memor

228 e (PD), in 15 subjects with idiopathic rapid eye movement sleep behavior disorder (iRBD) and compared

229 r (DAT) imaging to identify idiopathic rapid eye movement sleep behavior disorder (IRBD) patients at

230 ypotension, mild cognitive impairment, rapid eye movement sleep behavior disorder (RBD), depression,

231 0.65; mean follow-up, 2.88; P = .01), rapid eye movement sleep behavior disorder scores were signifi

232 mples from 56 patients with idiopathic rapid eye movement sleep behavior disorder, before and after t

233 sing standardized scales for hyposmia, rapid eye movement sleep behavior disorder, depression, autono

234 etwork dysfunction differentiated both rapid eye movement sleep behaviour disorder and Parkinson's di

235 vity, and for loss of tracer uptake in rapid eye movement sleep behaviour disorder and Parkinson's di

236 by addition of clinical scores (UPSIT, Rapid Eye Movement Sleep Behaviour Disorder Screening Question

237 Rapid eye movement sleep behaviour disorder was indistinguisha

238 In addition, eight patients with rapid eye movement sleep behaviour disorder, 10 with Parkinson

239 with polysomnographically-established rapid eye movement sleep behaviour disorder, 48 patients with

240 itivity (96%) and specificity (74% for rapid eye movement sleep behaviour disorder, 78% for Parkinson

241 es are present in so-called idiopathic rapid eye movement sleep behaviour disorder, a condition assoc

242 functions and were more likely to have rapid eye movement sleep behaviour disorder.

243 s for future neuroprotective trials in rapid eye movement sleep behaviour disorder.

244 t basal ganglia network dysfunction in rapid eye movement sleep behaviour disorder.

245 eatures of Parkinson's disease such as rapid eye movement sleep behavioural disorder and the postural

246 RO5256390 profoundly reduced rapid eye movement sleep in wild-type mice; these effects were

247 ed slow wave activity power during non-rapid eye movement sleep over widespread, bilateral scalp regi

248 frequent interictal spikes during non-rapid eye movement sleep predicted a reduced homeostatic decre

249 r age, higher SW density (nb/min of nonrapid eye movement sleep) was associated with higher CT in cor

250 phalographic (EEG) activity during non-rapid eye movement sleep, a highly heritable trait with finger

251 power spectrum, produced low-delta non-rapid eye movement sleep, and slightly increased wakefulness i

252 ness promotes slow-wave sleep, but not rapid eye movement sleep, during a period of low sleep pressur

253 l as a theta power (4-7Hz) decrease in rapid eye movement sleep, were associated with disease burden

254 ng mechanism may be the suppression of rapid eye movement sleep.

255 aracterized by increased percentage of rapid eye movement sleep.

256 keletal muscle paralysis characterizes rapid eye movement sleep.

257 promoted wakefulness and suppressed nonrapid eye movement sleep.

258 logram (EEG) signatures of stage 2 non-rapid eye movement sleep.

259 associated with time in slow-wave and rapid-eye-movement sleep after training.

260 In contrast, during rapid-eye-movement sleep, the neocortical tone is sustained ma

261 ctional differences between waking and rapid-eye-movement sleep.

262 Parkinson's Disease (SCOPA-AUT), REM (Rapid Eye Movement) Sleep Behavior Disorder Single-Question Sc

263 ween periods of presumed slow-wave and rapid-eye-movement-sleep/active-state, which were characterize

264 ther associated with the quality of nonrapid eye movement slow wave oscillations during recovery slee

265 Our studies show mice make saccadic eye movements spontaneously and in response to SC stimul

266 ing thalamocortical oscillation of non-rapid eye movement stage 2 sleep, correlate with IQ and are th

267 hile monkeys performed instructed and choice eye movement tasks, to determine time-specific contribut

268 nt occurs via a unique, pathological type of eye movement that differs from a normal, physiological d

269 surroundings by looking at things, but each eye movement that we make causes an abrupt shift of the

270 is syndrome, and the constellation of unique eye movements that accompany Joubert syndrome, are eluci

271 n those oculomotor structures interacts with eye movements that are decision irrelevant.

272 saccades and slower components of fixational eye movements that are part of the visual processing str

273 y sample the dynamic environment by variable eye movements that lead to inherent instability of the o

274 STATEMENT Saccades are the rapid, ballistic eye movements that we make approximately three times eve

275 During saccadic eye movements, the job of the nervous system is not to p

276 et of visual loss, the presence of pain with eye movements, the visual acuity, and the retention of c

277 ate the history of the neural integrator for eye movements, then further develop the idea of a neural

278 the spatiotemporal specificity of the evoked eye movements, thus facilitating the interpretation of c

279 requires participants to suppress a reactive eye movement to a visual target and to concurrently init

280 ies, we trained monkeys to make synchronized eye movements to a visual metronome.

281 he visual field (foveated vision) and deploy eye movements to actively sample regions of interests in

282 these regularities to control the timing of eye movements to detect behaviorally relevant events.

283 We used visually driven smooth eye movements to find the 3D space-time function that be

284 n visual motion estimates and smooth pursuit eye movements to measure stimulus-response correlations

285 ht and send this information to FEF to guide eye movements to those relevant stimuli.

286 le out possible confounds related to altered eye movement trajectories or order of presentation.

287 Humans perform saccadic eye movements two to three times per second.

288 This form of coupling between heartbeat and eye movements was substantiated by the additional findin

289 Although derived from eye movements, we find that the filter predicts perceptu

290 ude, nasally directed (ipsiversive) saccadic eye movements were evoked by microstimulation in anterio

291 anterior frontal sulcus, from which saccadic eye movements were evoked with electrical stimulation.

292 Saccadic eye movements were measured using infra-red oculography.

293 Eye movements were measured with electro-oculography (EO

294 brain responses were recorded with fMRI and eye movements were monitored simultaneously.

295 Eye movements were recorded with a Tobii TX300 (Tobii Te

296 ular muscles (responsible for the control of eye movement) were resistant to degeneration in endstage

297 tly, our study highlights the need to record eye movements when studying the influence of heartbeat i

298 LIP)] specifically biased choices made using eye movements, whereas lesions on the medial bank of the

299 of eye movements, and others have focused on eye movements while ignoring covert attention.

300 Our results suggest that participants select eye movements with the goal of maximizing information ab