ライフサイエンスコーパス: facial

1 ion was presence of sinus tract at buccal or facial abscess of apical portion of implant, and subsequ

2 anatomically-based tools, FACS and DogFACS (Facial Action Coding Systems), to quantify and compare h

3 We found that dogs displayed distinctive facial actions depending on the category of stimuli.

4 nd older who were consulting for any type of facial aesthetic treatment were studied.

5 lotomy were impaired in recognizing negative facial affect expressions but not positive or neutral fa

6 Here we report on a nationwide analysis of facial allograft donor surgery experience and long-term

7 The desired effects include increased facial and body hair, increased lean mass and strength,

8 Taste information is conveyed via the facial and glossopharyngeal nerves, while general mucosa

9 as unveiled the elegant choreography of this facial and head motion during tactile and olfactory expl

10 but the loss was evenly distributed between facial and lingual aspects of the extraction site.

11 ublished their famous article, "Imitation of facial and manual gestures by human neonates." Their cen

12 nd weakness associated with mild-to-moderate facial and neck weakness, significant neonatal-onset res

13 e study also suggests more prominent bulbar, facial and respiratory involvement in individuals positi

14 Main primary gustatory centers (facial and vagal lobes) received sensory projections fro

15 lobes) received sensory projections from the facial and vagal nerves, respectively.

16 e Immunodeficiency, Centromeric instability, Facial anomalies (ICF) syndrome is a chromatin disorder

17 Immunodeficiency, Centromere instability and Facial anomalies (ICF) syndrome, which is associated wit

18 Immunodeficiency, Centromere instability and Facial anomalies syndrome cases (ICF1).

19 et dystonia, often associated with a typical facial appearance and characteristic brain magnetic reso

20 spectrum disorder may have a characteristic facial appearance in addition to neurodevelopmental impa

21 First impressions based on facial appearance predict many important social outcomes

22 d facial macules and 12 control photodamaged facial areas were included in the study.

23 The duration of the treated facial attacks (mean: 26.6 h; SD: 10.1 h) was significan

24 orter than that of the previous 88 untreated facial attacks in the same women (mean: 64.1 h; SD: 28.0

25 ven women were treated with pdC1-INH for 143 facial attacks.

26 evidence of the effects of facial ratios on facial attractiveness and suggested that there are notab

27 are notable gender differences in perceiving facial attractiveness as induced by facial proportions.

28 change) was correlated with ratings of men's facial attractiveness.

29 facial ratios that were best correlated with facial attractiveness.

30 owever, little is known about preference for facial averageness in monkeys.

31 volutionary implications, the texture of the facial bones of the new taxon, and other derived tyranno

32 ures of the mandible, temporal bone or other facial bones.

33 enoid bone connects the cranium vault to the facial bones.

34 is characterized by the malformation of the facial bones.

35 1) is important to regulate the migration of facial branchiomotor (FBM) neurons in the hindbrain.

36 n a population of cranial motor neurons, the facial branchiomotor neurons (FBMNs).

37 ngly, we propose that enlarged neurovascular facial canals shouldn't be used to exclusively support a

38 ll understood whether there is a gradient of facial characteristics of children who did not receive a

39 knockout mutants (Mllt10-KO) exhibit midline facial cleft.

40 play of genetics and environment involved in facial clefting.

41 e that the more global beta-sheet/beta-sheet facial complementarity is a critical determinant for amy

42 sses and all patients gained improvements in facial contour, speech and swallow.

43 uded prolonged use of eye make- up and other facial cosmetic products.

44 sample design with neutral, happy, and angry facial cues embedded in realistic background scenes.

45 text encoding in the presence of threatening facial cues.

46 PWWP2A in Xenopus results in severe cranial facial defects, arising from neural crest cell different

47 ndividuals who exhibited growth restriction, facial deformities, and a history of bacterial and viral

48 annotated for 1) reported associations with facial development, 2) multiple occurrence of variants,

49 mutation of UTX, which supports appropriate facial development.

50 x etiology that reflects a failure of normal facial development.

51 Oral-facial-digital (OFD) syndromes are rare heterogeneous di

52 LT1) mutations were associated with the oral-facial-digital syndrome (OFD), an autosomal recessive ci

53 , we show that families presenting with Oral-Facial-Digital syndrome type 6 (OFD6) have likely pathog

54 rlie the clinically complex ciliopathy, Oral-Facial-Digital syndrome Type I (OFD Type I).

55 stosis, cranioectodermal dysplasia, and oral-facial-digital syndrome, altogether demonstrating that p

56 TMEM107 were previously connected with oral-facial-digital syndrome, Meckel-Gruber syndrome, and Jou

57 He had generalized muscle weakness, facial discomfort, recurrent episodes of carpopedal spas

58 pearance secondary to proptosis, ptosis, and facial disfigurement, leading to social embarrassment an

59 l prodrome, faciobrachial dystonic spells or facial dyskinesias, and mesial temporal sclerosis abnorm

60 bility, congenital anomalies, characteristic facial dysmorphic features, and low cholesterol levels s

61 ectual disability, speech delay, ataxia, and facial dysmorphism and carrying a deleterious EBF3 varia

62 Minor non-specific facial dysmorphism was also noted in some individuals, i

63 ognizable syndrome with developmental delay, facial dysmorphism, and camptodactyly.

64 ndividuals exhibit cerebellar ataxia, subtle facial dysmorphism, strabismus, and vesicoureteric reflu

65 luding skeletal and organ anomalies and mild facial dysmorphism.

66 rized by short stature, shortened limbs, and facial dysmorphism.

67 es (mild "molar tooth sign"), typical cranio-facial dysmorphisms (hypertelorism, depressed nasal brid

68 d by growth retardation, spine malformation, facial dysmorphisms, and developmental delays.

69 ety disorders), hypotonia, broad-based gait, facial dysmorphisms, and periods of fever and vomiting.

70 ability, expressive language impairment, and facial dysmorphisms.

71 evance: Half of the patients with periocular facial dystonias used alleviating maneuvers.

72 Participants completed a facial emotion and identity discrimination task prior to

73 IFC or sham stimulation prior to completing facial emotion and identity perception tasks.

74 Facial emotion perception plays a key role in interperso

75 ultiple aspects of social cognition, such as facial emotion recognition, theory-of-mind ability, and

76 erformed those receiving sham stimulation on facial emotion, but not identity, perception tasks.

77 Imitation and observation of actions and facial emotional expressions activates the human fronto-

78 tability and anxiety, on neural responses to facial emotions during functional magnetic resonance ima

79 may provide an immediate, natural-appearing facial enhancement outcome.

80 h an ultimate diagnosis most consistent with facial EPD.

81 escribe 3 women who presented with prolonged facial erosions after cosmetic resurfacing procedures, s

82 ation and therapeutic options for nonhealing facial erosions occurring after ablative procedures (car

83 entials (nociceptive cortical activity), and facial expression (behavior) were acquired in individual

84 ding changes in volition/motivation, posture/facial expression and derealization/depersonalization.

85 Facial expression and eye gaze provide a shared signal a

86 ponent displays either an angry or a neutral facial expression at the beginning of each trial and del

87 d the dimension of valence within the single facial expression category of surprise.

88 s unclear to what extent there exists common facial expression in species more phylogenetically dista

89 nd emotion ambiguity (the uncertainty that a facial expression is categorized as fearful or happy).

90 onth-old infants, implicating this system in facial expression processing from a very young age.

91 ing facial proportions, but the same neutral facial expression, baldhead and skin tone, as stimuli.

92 y viewed as being specialized for processing facial expression.

93 fically, while the perception of incongruent facial expressions activates somatosensory-related repre

94 pecific "preparatory" system learns aversive facial expressions and autonomic responses such as skin

95 dala processes both the degree of emotion in facial expressions and the categorical ambiguity of the

96 Facial expressions are a central part of human communica

97 Because facial expressions are indeed created by moving one's fa

98 roducing facial expressions, suggesting that facial expressions are not just inflexible and involunta

99 study, we aimed to test whether domestic dog facial expressions are subject to audience effects and/

100 rimate lineage, also have the ability to use facial expressions as a means of gaining social informat

101 the extent to which mothers mirrored infant facial expressions at two months postpartum predicted in

102 ere, we demonstrate the utility of surprised facial expressions because exemplars within this emotion

103 mans have revealed that the motor control of facial expressions has a distributed neural representati

104 Historically, animal facial expressions have been considered inflexible and i

105 r system during observation and execution of facial expressions in nine-month-old infants, implicatin

106 quantify and compare human and domestic dog facial expressions in response to emotionally-competent

107 Processing facial expressions is an essential component of social i

108 enteen healthy participants (8 females) with facial expressions of anger, disgust, fear, happiness, s

109 Here we investigated whether facial expressions of different basic emotions modulate

110 ccurred, infants were less likely to display facial expressions of distaste initially when eating the

111 nts from both cultures visually discriminate facial expressions of emotion by relying on culturally d

112 The commonality of facial expressions of emotion has been studied in differ

113 and 7-month old infants were presented with facial expressions of happiness, anger, and fear.

114 and anger, and no impairment in recognizing facial expressions of surprise or happiness.

115 Viewing facial expressions often evokes facial responses in the

116 ted amygdala reactivity to fearful and angry facial expressions using functional magnetic resonance i

117 r social information conveyed by conspecific facial expressions using the framework of optimal foragi

118 ey watched videos of conspecifics displaying facial expressions with direct or averted gaze.

119 fect expressions but not positive or neutral facial expressions, and impaired in Stroop cognitive con

120 is a key structure for processing emotional facial expressions, but it remains unclear what aspects

121 oscience mainly focused on the processing of facial expressions, overlooking the exploration of emoti

122 of unknown conspecifics portraying different facial expressions, showing appropriate behavioural and

123 n covert vigilance and avoidance of aversive facial expressions, social anxiety appears to confer a s

124 the human's attentional state when producing facial expressions, suggesting that facial expressions a

125 ear (happy, angry) and ambiguous (surprised) facial expressions, then re-rated similar stimuli after

126 a responses tracked the valence of surprised facial expressions, unconfounded by arousal.

127 ammals, since dogs do not display human-like facial expressions.

128 Most mammalian species produce facial expressions.

129 passive viewing of dynamic angry and neutral facial expressions.

130 uralistic angry, fearful, happy, and neutral facial expressions.

131 ained visuocortical facilitation to aversive facial expressions.

132 l vocalizations (e.g., crying), and (silent) facial expressions.

133 amygdala and hypothalamus responses to angry facial expressions.

134 ramolecular structures, in contrast with the facial (fac) coordination common to smaller and higher-s

135 r, preference and anti-preference of extreme facial features (e.g., very large/small inter-eye distan

136 gorization in which occasionally conflicting facial features are resolved through competition between

137 ments previous approaches to quantifying the facial features associated with specific body characteri

138 igin of extant apes, and that hylobatid-like facial features evolved multiple times during catarrhine

139 quantitatively, several tuning properties to facial features found in the middle patch of face proces

140 d that monkeys fixated the illusory internal facial features in a pattern consistent with how they vi

141 We report a genome-wide association scan for facial features in approximately 6,000 Latin Americans.

142 we hypothesize that the coding principle of facial features in the middle patch of face processing i

143 lts indicate that the perception of illusory facial features on inanimate objects is driven by a broa

144 lia is the compelling illusion of perceiving facial features on inanimate objects, such as the illuso

145 types that correspond to naturally perceived facial features remains largely a mystery.

146 These patients have distinctive facial features similar to those with mutations in CCBE1

147 These subjects share a set of common facial features that include a prominent maxilla and upp

148 particular, tuning to only a small number of facial features that were often related to geometrically

149 However, the facial features used to do so are not always clear-cut b

150 When facial features were shown at typical (rather than rever

151 ly, congenital heart defects, and dysmorphic facial features with hypertelorism, synophrys, macroglos

152 ted by mild to severe cutis laxa, dysmorphic facial features, and cardiopulmonary involvement identif

153 racterized by growth retardation, dysmorphic facial features, brachydactyly with carpal-tarsal fusion

154 stasis, ocular abnormalities, characteristic facial features, heart defects, and vertebral malformati

155 by lymphedema, lymphangiectasia and distinct facial features.

156 oloboma, sensorineural deafness, and typical facial features.

157 er into multiple social categories from mere facial features.

158 growth problems as well as common dysmorphic facial features.

159 rtening, genital hypoplasia, and distinctive facial features.

160 ncy to stereotype individuals based on their facial features.

161 ifficulties, deafness, and subtle dysmorphic facial features.

162 We tested cue integration of facial form and motion in an identity categorization tas

163 In dogs, facial form is stunningly diverse.

164 Through deconvolution of facial forms, we identified quantitative trait loci that

165 marks enabled us to intuitively characterize facial geometry at a fine level of detail through curvat

166 act), growth retardation, and a recognizable facial gestalt (interrupted wavy eyebrows, dense eyelash

167 terine growth restriction, lipoatrophy and a facial gestalt involving a triangular face, deep set eye

168 hypothesis that maternal mirroring of infant facial gestures is central to the development of a neura

169 between visual and motor representations of facial gestures, which increases infant neural sensitivi

170 believed to couple self- and other-generated facial gestures.

171 Decellularization was successful in all facial grafts within 12 days revealing acellular scaffol

172 ill enable further engineering of postmortem facial grafts, thereby offering new perspectives in comp

173 p hair (shape, colour, greying, balding) and facial hair (beard thickness, monobrow, eyebrow thicknes

174 several months of starting therapy, although facial hair and alopecia continue to develop after 1 yea

175 -androgen therapy include decreased body and facial hair, decreased muscle mass, breast growth, and r

176 d had lifelong sparse scalp hair with normal facial hair.

177 osis and severe midface hypoplasia, body and facial hypertrichosis, microphthalmia, short stature, an

178 gth as 4+ out of 5) that was associated with facial hypomimia and a rigid akinetic syndrome only in t

179 l features present in inverted or unspecific facial identities are not sufficient to produce the adap

180 In a first condition, (1) two facial identities are presented in alternation at a rate

181 g assumption that face cells encode specific facial identities, confirmed by engineering faces with d

182 nce, following 10-s adaptation to one of the facial identities; this results in a significant identit

183 Here, we demonstrate that facial identity and expression can be processed in the s

184 tity becomes discriminable from an unadapted facial identity at a neural population level.

185 n from low- to high-level representations of facial identity in human face-selective cortex and demon

186 hese results indicate that representation of facial identity is localized to face patches, but activi

187 Here, we reveal the brain's code for facial identity.

188 al lobe play key roles in the recognition of facial identity.

189 eft V1) yielded successful classification of facial identity.

190 testosterone, as well as in response to non-facial images pre-rated as either sexually arousing or t

191 broadening the evidence for automaticity of facial impressions.

192 aplotype are profound, accounting for 36% of facial length variation in the dogs we tested.

193 Facial lipoatrophy (FLA) is associated with human immuno

194 were topographically organized, those of the facial lobe mainly ending medially to those of the vagal

195 Descending facial lobe projections to the medial funicular nucleus

196 ing the Y1226/7 deletion formed a functional facial lymphatic network enabling them to develop normal

197 rast, flt4 null larvae displayed hypoplastic facial lymphatics and severe lymphedema.

198 xty-one patients with 63 ambiguous pigmented facial macules and 12 control photodamaged facial areas

199 nfocal microscopy (RCM), ambiguous pigmented facial macules and establish a correlation between RCM,

200 Pigmented facial macules on photodamaged skin are a clinical, derm

201 A prospective study of ambiguous pigmented facial macules on photodamaged skin was conducted in a t

202 croscopy enhanced the diagnosis of pigmented facial macules with 91.7% sensitivity and 86.8% specific

203 Nonsyndromic cleft palate only (nsCPO) is a facial malformation that has a livebirth prevalence of 1

204 s and 3 patients required eyelid surgery for facial malignancies outside the periocular zone.

205 We identified a mosaic of features including facial, mandibular and dental morphology that aligns the

206 L/min compared with a standard nonocclusive facial mask at the same clinically set FiO2 (20 min/step

207 Compared with the facial mask, HFNC significantly improved oxygenation (P

208 s, including shorter snout, expansion of the facial midline, cleft lip, extensive exencephaly, and mi

209 Neurons in the trigeminal (Mo5), facial (Mo7), ambiguus (Amb), and hypoglossal (Mo12) mot

210 High-resolution, three-dimensional facial models have been acquired on a cohort of 952 twin

211 DH10 is essential during the early stages of facial morphogenesis for the formation of a functional n

212 he margin reflex distances 1 and 2 and other facial morphometric data.

213 Facial motoneurons that drive intrinsic muscles to protr

214 g nervous system, with some populations-like facial motor neurons-exhibiting greater resilience to ab

215 tly, do dogs display specific discriminatory facial movements in response to different categories of

216 However, dogs produced different facial movements to humans in comparable states of emoti

217 Secondly, do dogs display similar facial movements to humans when reacting in emotionally

218 Dogs produced significantly more facial movements when the human was attentive than when

219 limited in number, volitional movements (eg, facial muscle activity, head movements, shoulder shrugs)

220 lts indicated no selective activation of the facial muscles for the expressions in 4-month-old infant

221 pressions are indeed created by moving one's facial muscles, it is logical to assume that our visual

222 We have used the adult mouse facial nerve crush model and adult-onset conditional dis

223 sates at the level of the caudal pole of the facial nerve nucleus in the rostral medulla oblongata.

224 ment of ocular sequelae due to nonperiocular facial NMSC.

225 s optogenetic silencing specifically reduced facial nociception.

226 gery was traumatic brain injury, followed by facial or orbital fracture, long bone fracture, and ches

227 d with CRS cases who reported smell loss and facial pain and/or pressure and had the weakest associat

228 /pressure; smell loss without pain/pressure; facial pain and/or pressure without smell loss; and both

229 , high fever and purulent nasal discharge or facial pain lasting for at least 3 consecutive days, or

230 h trigeminal stimulation triggers paroxysmal facial pain, affects defensive peripersonal space (DPPS)

231 ataxia, diplopia, cognitive impairment, and facial paraesthesia did not discriminate CLIPPERS from n

232 e syndrome, 50% were found to have bilateral facial paralysis on examination.

233 hich resulted in separate representations of facial parts and object parts.

234 poglossal (Mo12) motor nuclei innervate jaw, facial, pharynx/larynx/esophagus, and tongue muscles, re

235 ardiac myopathy but none has the skeletal or facial phenotype seen in patients with Uruguay syndrome.

236 ther, these features comprise a recognizable facial phenotype.

237 Furthermore, the construction of facial phenotypes that correspond to naturally perceived

238 Sickness status presented by odor and facial photograph resulted in increased neural activatio

239 Facial photographs and body odor samples were taken from

240 Facial photographs annotated with automated measurements

241 Wrinkle area was quantified digitally using facial photographs of 3,831 northwestern Europeans (51-9

242 roles in representing opposite directions of facial preference.

243 ed to the finding that positive and negative facial preferences are represented by different brain re

244 involves sequential growth and fusion of the facial prominences.

245 yet realistic face images, which had varying facial proportions, but the same neutral facial expressi

246 rceiving facial attractiveness as induced by facial proportions.

247 active faces result from their similarity to facial prototypes, the categorical central tendencies of

248 neurophysiologic evidence of the effects of facial ratios on facial attractiveness and suggested tha

249 analysis was used to learn the most relevant facial ratios that were best correlated with facial attr

250 These spontaneous facial reactions (SFRs) are believed to play an importan

251 7-month-old infants, evidence for selective facial reactions was found especially for happy (leading

252 Furthermore, attention to various facial regions was differentially modulated depending on

253 nfants developing an attractive, female-like facial representation that guides children's attention t

254 Viewing facial expressions often evokes facial responses in the observer.

255 s to identify the molecular underpinnings of facial retrusion is limited to association of a missense

256 Facial retrusion, the proximodistal shortening of the sn

257 Complex acellular facial scaffolds were obtained, preserving simultaneousl

258 ), deep rhytides on the face (20%), multiple facial scars (20%), verruca vulgaris on the face (20%),

259 most importantly maintained excellent allene facial selectivities regardless of the substrate stereoc

260 The facial selectivity of their interactions with axial liga

261 and argue instead that an enhanced degree of facial sensitivity may have been linked with any number

262 Focusing on facial shape, we resolved a quantitative trait locus on

263 utionary mechanism underlying adaptations of facial shape.

264 e trait loci that are responsible for canine facial shapes and sizes.

265 trast to the behavior of chondrocytes during facial skeletal development.

266 SMOC2 disruption affects the facial skeleton in a dose-dependent manner.

267 Patterning of the facial skeleton involves the precise deployment of thous

268 = 661; 50.5% women, median age 63.1 years), facial skin aging features (perceived age, wrinkling, pi

269 ates of cataract in left eyes and left-sided facial skin cancer.

270 Rosacea, a common facial skin disorder, has a poorly understood pathogenes

271 ctivation from the intracranial dura but not facial skin or cornea.

272 sap1 expression was detected on at least one facial skin site of 17 healthy human volunteers.

273 patients have marked difficulties to exploit facial social cues elicited by a humanoid robot to modul

274 mbic network's increased response to neutral facial stimuli as a marker of the extended psychosis phe

275 duce the adaptation effect found for upright facial stimuli, which appears to truly reflect identity-

276 nvolving the eyelid, orbit, periorbital, and facial structures (orbital-periorbital plexiform neurofi

277 o visualize and quantitate Xenopus heart and facial structures establishing normative data.

278 aryngeal arches, which give rise to most mid-facial structures.

279 places thousands of landmarks throughout the facial surface and automatically establishes point-wise

280 -gaze fear) and ambiguous (direct-gaze fear) facial threat cues via selective engagement of M and P p

281 ehavior by altering the neural processing of facial threat signals.

282 Nevertheless, heritability estimates of facial traits have often been surprisingly low or diffic

283 ficiency, such as lipoatrophy, lumpectomy or facial trauma, is a formidable challenge in reconstructi

284 around the world, while the Tasmanian devil facial tumor disease (DFTD) is much younger and geograph

285 Devil facial tumour disease (DFTD) has swept across nearly the

286 Devil facial tumour disease (DFTD) is a transmissible cancer d

287 extinction due to a contagious cancer, devil facial tumour disease (DFTD).

288 i) affected by a transmissible cancer, devil facial tumour disease.

289 Current knowledge of the impact of facial vascularized composite allograft (VCA) procuremen

290 Facial VCA procurement does not appear to adversely affe

291 There were 10 facial VCA procurements in organ donors between December

292 Mean operative duration for facial VCA recovery was 6.9 hours (range, 4-13.25 hours)

293 circuitry for the differential processing of facial versus bodily pain remains unknown.

294 The condition is primarily characterized by facial volume loss that affects the contours of the chee

295 MK-CFZS as a congenital myopathy with marked facial weakness and additional clinical and pathologic f

296 ations such as ptosis and ophthalmoplegia or facial weakness, and links myasthenic disorders with dys

297 Visualization and tracking of the facial whiskers is required in an increasing number of r

298 flammation persisted until withdrawal of the facial wipes thought to contain the inciting agent, thou

299 e investigated sex-specific determinants for facial wrinkles.

300 Facial wrinkling is one of the most notable signs of ski