ライフサイエンスコーパス: facial nerve

1 ves no motor or sensory innervation from the facial nerve.

2 ese most likely represent motoneurons of the facial nerve.

3 uality depends primarily on the input of the facial nerve.

4 aneous rami of spinal nerves, but not by the facial nerve.

5 rlapping with the area of termination of the facial nerve.

6 ) in Schwann cells as well as in sciatic and facial nerves.

7 well as the motor root of the trigeminal and facial nerves.

8 ry neurons located lateral and dorsal to the facial nerve-a region we termed the parafacial zone (PZ)

9 This is in contrast to 31.2% of facial nerve afferent endings in the nst which make syna

10 gene deletion only results in defects in the facial nerve and not the glossopharyngeal and vagus nerv

11 r sectioning the ipsilateral branches of the facial nerve and resecting the superior cervical ganglia

12 er that is characterized by paralysis of the facial nerves and variable other congenital anomalies.

13 nto the facial muscle of adult rats prior to facial nerve avulsion.

14 In contrast, facial nerve axotomy in C.B-17 (-/-) severe combined imm

15 Facial nerve axotomy increased the total cofilin abundan

16 ermined that the mSOD1 molecular response to facial nerve axotomy is phenotypically regenerative and

17 n the current investigation, we utilized the facial nerve axotomy model and a presymptomatic amyotrop

18 FMN loss after axotomy, we superimposed the facial nerve axotomy model on presymptomatic mSOD1 mice

19 /- mice showed increased rate of death after facial nerve axotomy, a response documented for SOD1-/-

20 After a right facial nerve axotomy, facial motoneuron (FMN) survival i

21 One complication is damage to the facial nerve branches, which can result in brow ptosis a

22 The rat facial nerve (CN VII) controls the orbicularis oculi (OO

23 the treatment of adult mice with LiCl after facial nerve crush injury stimulated the expression of m

24 We have used the adult mouse facial nerve crush model and adult-onset conditional dis

25 without CNTFRalpha, even when challenged by facial nerve crush or the injection-associated trauma, t

26 However, the incidence of facial nerve damage after TAB is unknown.

27 There was no correlation with facial nerve damage and use of blood thinners, biopsy re

28 Postoperative facial nerve damage was found in 12 patients (16.0%) and

29 There is a 16.0% incidence of postoperative facial nerve damage with TABs, which recovers fully in o

30 Incidence of postoperative facial nerve damage, other complications, and rates of f

31 performed for any potential correlation with facial nerve damage.

32 brow were less likely to have postoperative facial nerve damage.

33 ngeal nerve and was not sufficient to rescue facial nerve defects, suggesting that FGF8 is functional

34 xin conveyed SEMA3/neuropilin signals during facial nerve development, we combined an expression anal

35 ally redundant with other RTK ligands during facial nerve development.

36 ns, PLXNA1 and PLXNA2 were not essential for facial nerve development.

37 brow were more likely to have postoperative facial nerve dysfunction.

38 t, share the same migratory behaviour to the facial nerve exit points and express the same markers as

39 internal carotid arteries) or osteichthyans (facial nerve exiting through jugular canal, endolymphati

40 means of pulsed magnetic stimulation of the facial nerve for the purpose of increasing cerebral bloo

41 providing normal viability as well as proper facial nerve formation even in the Hoxb1 mutant backgrou

42 sults in optimal preservation of hearing and facial nerve function.

43 hiomeric motor neurons of the trigeminal and facial nerves generate spontaneous [Ca2+]i transients th

44 ilateral weakness in the distribution of the facial nerve has been reported.

45 The ZMB contains axons from the facial nerve; however, myelination within the barbel its

46 We noted, nonetheless, a specific facial nerve hypomorphism in hemizygous Hoxb1(A1/-) mice

47 and for the rehabilitation of patients with facial nerve impairments.

48 In the hamster facial nerve injury paradigm, we have established that a

49 Facial nerve innervated taste buds (FITBs) are thought t

50 In catfish, the facial nerve innervates taste buds distributed over the

51 yngeal and vagus nerves, suggesting that the facial nerve is most sensitive to perturbations in RTK s

52 ha-internexin protein expression after three facial nerve lesion paradigms: crush, transection, and r

53 lled forelimb training in conjunction with a facial nerve lesion, cholinergic mechanisms were require

54 blink as a functional recovery parameter of facial nerve lesion.

55 sates at the level of the caudal pole of the facial nerve nucleus in the rostral medulla oblongata.

56 Taste and tactile fibers in the facial nerve of catfish innervate extraoral taste buds a

57 nerve innervation to fungiform papillae, the facial nerve of developing animals was labeled with the

58 als to the posterior auricular branch of the facial nerve (PA neurons).

59 es), lateral canthal coloboma (3 cases), and facial nerve palsy (1 case).

60 One monkey, with prior facial nerve palsy and a very steep amplitude versus pea

61 id, excluding one monkey who was affected by facial nerve palsy and was analyzed separately.

62 Facial nerve palsy is a potentially devastating conditio

63 Appropriate management of facial nerve palsy is dependent on a multitude of factor

64 ith this early effect were aberrant tearing, facial nerve palsy, ear malformations, and autism.

65 he underlying anatomy and pathophysiology of facial nerve palsy, while also exploring different treat

66 patient's morbidity, especially the risk of facial nerve palsy.

67 the complications associated with permanent facial nerve palsy.

68 ght strategies in the surgical management of facial nerve palsy.

69 gical procedures for patients with permanent facial nerve palsy.

70 eys and, more dramatically, in a monkey with facial nerve palsy.

71 aring contralateral to an eyelid weakened by facial nerve palsy.

72 sions in 23 percent, arthritis in 6 percent, facial-nerve palsy in 3 percent, aseptic meningitis in 2

73 rogens enhance both functional recovery from facial nerve paralysis and the rate of regeneration in t

74 unilateral vision loss (3 malignancies), and facial nerve paresis (5 malignancies).

75 g experiments for the study of the autonomic facial nerve pathway in birds in terms of both its anato

76 rm the somatic motor component of the VIIth (facial) nerve, possibly through a failure to specify the

77 ve damage, other complications, and rates of facial nerve recovery were evaluated.

78 Testosterone propionate (TP), augments facial nerve regeneration in the adult hamster.

79 eltaNLS-GFP) stimulated axonal sprouting and facial nerve regeneration in vivo.

80 By contrast, movements evoked by facial nerve stimulation showed no such frequency-depend

81 The effect of facial nerve stimulation was found to be dependent on st

82 enuated the cerebral vasodilator response to facial nerve stimulation when applied locally to the cor

83 evelopment of a non-invasive pulsed magnetic facial nerve stimulator that will increase CBF as a trea

84 s 4 and 5 underwent surgical severing of the facial nerve (to cause complete paralysis of the OOM).

85 t neurons, located dorsal to the genu of the facial nerve, to the cerebellar flocculus and ventral pa

86 drotestosterone on FMNs of P7 hamsters after facial nerve transection at the SMF.

87 adult male hamsters were subjected to right facial nerve transection at the stylomastoid foramen.

88 We performed a facial nerve transection axotomy in both mSOD1 subgroups

89 and synapse recovery following extracranial facial nerve transection in mice.

90 Upon facial nerve transection, damaged CD200-deficient neuron

91 After facial nerve transection, neither compartment alone is s

92 for mediating plasticity associated with the facial nerve transection.

93 forms of plasticity within the motor cortex: facial nerve transections evoke reorganization of cortic

94 rm the somatic motor component of the VIIth (facial) nerve which controls the muscles of facial expre

95 Interestingly, sectioning the facial nerve, which abolished whisker movements, did not

96 RP/PLXN signalling in the development of the facial nerve, which contains axons from two motor neuron

97 Fibers of the facial nerve, which innervate all external taste buds, r

98 ctive appearance of the internal genu of the facial nerve, which is lacking in birds.

99 ry sulci, olfactory bulbs and oculomotor and facial nerves, which support underlying abnormalities in

100 is an acute and idiopathic paralysis of the facial nerve, with an estimated incidence ranging from 1

101 focal magnetic field was directed toward the facial nerve within the temporal bone by placing a 6.5 c