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1 for transmitting information from MI to the facial nucleus.
2 ensory inputs to vibrissa motoneurons in the facial nucleus.
3 shows significant dysregulation in the mSOD1 facial nucleus.
4 d fine varicose nerve fiber terminals in the facial nucleus.
5 inergic motor neurons located in the lateral facial nucleus.
6 l trigeminal nucleus, and motoneurons of the facial nucleus.
7 s tractus solitarii, lateral medulla, medial facial nucleus, A5 area, lateral vestibular nucleus, and
8 cting the brainstem at the caudal end of the facial nucleus abolished active expirations, while rhyth
9 or nuclei that mediate expression of the CR (facial nucleus and accessory abducens) were reversibly i
10 solateral to the rostral-most portion of the facial nucleus and caudal superior olive, where they int
11 RVM and caudal pons were found medial to the facial nucleus and lateral to the pyramid in a column di
12 brain exhibited near-complete absence of the facial nucleus and superior olive along with shortening
15 that the perioral muscles innervated by the facial nucleus are rhythmically coordinated during lipsm
17 internuclear interneurons projecting to the facial nucleus, as well as those neurons of the parvocel
19 of the vagus, motor trigeminal nucleus, and facial nucleus, but not in most forebrain cholinergic ce
20 did not reveal any direct projections to the facial nucleus, but retrograde tracer injections in the
22 Injections of retrograde tracers into the facial nucleus consistently labeled neurons in the hypog
23 that, during disease progression, the mSOD1 facial nucleus displays target disconnection-induced gen
24 he GHSR gene was confirmed in neurons of the facial nucleus (FacN, 7), the dorsal vagal complex (DVC)
26 hile their accessory abducens nucleus (ACC), facial nucleus (FN), and surrounding reticular formation
27 he coordination of muscles innervated by the facial nucleus has not been carefully compared between c
28 ctions from trigeminal sensory nuclei to the facial nucleus have been described, but the pathway wher
29 ating active expiration located close to the facial nucleus in the region of the retrotrapezoid nucle
32 noradrenergic cell group and overlapping the facial nucleus lateral subnuclei and para-facial zones.
33 he case of Adv.RSV-GDNF, a greater number of facial nucleus motoneurons survived than were transduced
34 fibers and send monosynaptic connections to facial nucleus motoneurons that directly innervate vibri
36 d Adv.RSV-GDNF, a significant portion of the facial nucleus neurons was protected, 16.5, 18.2, and 53
37 xpressed in the olfactory bulb, red nucleus, facial nucleus, pontine nucleus, oculomotor nucleus, sub
39 sis of the superior olive, dysgenesis of the facial nucleus, reduced numbers of Purkinje neurons, hyp
40 siologically identified wFMNs in the lateral facial nucleus resulted in dense, bilateral labeling thr
41 eus, but retrograde tracer injections in the facial nucleus revealed some labeled neurons in MI corte
42 ot eliminate the severe phenotype in the FPG facial nucleus, suggesting that the FPG phenotype is the
43 d documented corticofugal projections to the facial nucleus, surrounding pontine reticular formation,
46 ne and medullary), motor trigeminal nucleus, facial nucleus, vestibular nucleus, dorsal motor nucleus
47 oject to the trigeminal motor nucleus (Vmo), facial nucleus (VII) and hypoglossal nucleus (XII) are a
48 ns (EBs) have been found at the level of the facial nucleus (VIIn), and 500 mum caudally, within the
49 nimals, the musculotopic organization of the facial nucleus was defined by injecting fluorescent retr
50 t of fluorogold from the whisker pads to the facial nucleus was seen only in motoneurons that lacked
52 o musculotopically defined subsectors of the facial nucleus was studied from the face representation
54 vectors were retrogradely transported to the facial nucleus where the NFs or beta-gal were expressed.
56 rated in the intermediate subdivision of the facial nucleus, with a strong ipsilateral prevalence.
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