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1 function mutations in the gene encoding this facilitative glucose transporter.
2 nd as dehydroascorbic acid (DHA) through the facilitative glucose transporters.
3 rbic acid is taken up by these cells through facilitative glucose transporters.
4 oxidized form, dehydroascorbic acid, through facilitative glucose transporters.
5 e movement of glucose into cells through the facilitative glucose transporters.
6 ascorbic acid (DHA), enters mitochondria via facilitative glucose transporter 1 (Glut1) and accumulat
7         Here, we show that expression of the facilitative glucose transporter 1 (GLUT1) is induced by
8         Altered glucose reabsorption via the facilitative glucose transporter 2 (GLUT2) during diabet
9                                          The facilitative glucose transporter-3 (GLUT 3) and hexokina
10 y bind and inactivate the insulin-responsive facilitative glucose transporter 4 (GLUT4).
11 suppresses the glucose transporter-1 (GLUT1) facilitative glucose transporter 49-66% in preimplantati
12 ion matched the sequence for the human GLUT3 facilitative glucose transporter, a high-velocity-high-a
13    Subcellular targeting and the activity of facilitative glucose transporters are likely to be regul
14 u and increased transport of DHA through the facilitative glucose transporters at the cell membrane.
15 is novel protein to members of the mammalian facilitative glucose transporter family (GLUT), we refer
16 Mutations in SLC2A10/GLUT10, a member of the facilitative glucose transporter family, are associated
17 tence of additional members of the mammalian facilitative glucose transporter family.
18 ate channel of this and other members of the facilitative glucose transporter family.
19 g the exocytic trafficking rate of the GLUT4 facilitative glucose transporter from intracellular stor
20       Insulin stimulates the movement of the facilitative glucose transporter glucose transporter-4 (
21 d muscle cells by regulating delivery of the facilitative glucose transporter, glucose transporter is
22 ve correlation between the expression of the facilitative glucose transporter Glut-1 and FDG accumula
23 ated extracellular glucose concentrations on facilitative glucose transporter (GLUT) expression in ra
24  mainly by insufficient expression levels of facilitative glucose transporter (GLUT)1 in up to 50% of
25 GLUT5, a fructose-transporting member of the facilitative glucose transporter (GLUT, SLC2) family, is
26 ucose efflux from tubules to interstitium by facilitative glucose transporters (GLUT).
27                 We report that a decrease in facilitative glucose transporter (GLUT1) expression and
28                        The human erythrocyte facilitative glucose transporter (Glut1) is predicted to
29 sly, we demonstrated a critical role for the facilitative glucose transporter, Glut1, in the regulati
30                                          The facilitative glucose transporter, GLUT1, is expressed on
31  interaction with purified human erythrocyte facilitative glucose transporter, GLUT1.
32 -dependent activation and recruitment of the facilitative glucose transporter GLUT2 to the brush-bord
33 s oocyte expression, we investigated whether facilitative glucose transporters GLUT2 and GLUT5-12 tra
34                  Our hypothesis was that the facilitative glucose transporter, GLUT2, could act as a
35 e in fat and muscle is mediated by the major facilitative glucose transporter Glut4.
36  the regulation of glucose transport via the facilitative glucose transporter (GLUT4) and glycogen sy
37 ese hamster ovary cells transfected with the facilitative glucose transporter, GLUT4, we identified t
38                            The expression of facilitative glucose transporters (Gluts) 1, 3, and 4 wa
39 lucose transporters in the body, the passive facilitative glucose transporters (GLUTs) and the second
40 ross mammalian cell membranes is mediated by facilitative glucose transporters (GLUTs) embedded in li
41 ium-coupled glucose transporters (SGLTs) and facilitative glucose transporters (GLUTs) in glucose hom
42  in most cells, vitamin C enters through the facilitative glucose transporters (GLUTs) in the form of
43    Glucose transporter 3 (GLUT3) is the main facilitative glucose transporter in neurons.
44 in B, indicating the direct participation of facilitative glucose transporters in the transport of ox
45  glucose permeation pathway within the GLUT1 facilitative glucose transporter is hypothesized to be f
46                                    The GLUT4 facilitative glucose transporter is recruited to the pla
47                  The trafficking of GLUT4, a facilitative glucose transporter, is examined in transfe
48                                   The murine facilitative glucose transporter isoform 3 (Glut 3) is d
49                                   The murine facilitative glucose transporter isoform 3 is developmen
50                                              Facilitative glucose transporter isoform 4 (GLUT4) in ra
51                                    The GLUT4 facilitative glucose transporter mediates insulin-depend
52 orbic acid than neutrophils, related to more facilitative glucose transporters on the monocyte cell m
53 pparent K(m) for substrate transport through facilitative glucose transporters on the plasma membrane
54 the total amount of GLUT4 protein or related facilitative glucose transporters present in skeletal mu
55 ake is highly sensitive to the levels of the facilitative glucose transporter protein, GLUT4.
56 ips between FDG uptake and the expression of facilitative glucose transporters, the sizes of populati
57      Both the GLUT1 and GLUT3 high-affinity, facilitative glucose transporters were expressed in GT1(

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