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1 ithin 18 hours after the administration of a factor Xa inhibitor.
2 Otamixaban is an intravenous direct factor Xa inhibitor.
3 y potent, selective, and orally bioavailable factor Xa inhibitor.
4 gned to reverse the anticoagulant effects of factor Xa inhibitors.
5 atheter ablation in AF patients treated with factor Xa inhibitors.
6 available in the United States for the oral factor Xa inhibitors.
7 n amide that is found in previously reported factor Xa inhibitors.
8 nt a novel class of potent dual thrombin and factor Xa inhibitors.
9 y potent, selective, and orally bioavailable factor Xa inhibitors.
11 on with anticoagulants (thrombin inhibitors, factor Xa inhibitors) act synergistically in inhibiting
15 erruption, or discontinuation of direct oral factor Xa inhibitor) and the remaining eight (14%) event
16 se of anticoagulants (vitamin K antagonists, factor Xa inhibitors, and direct thrombin inhibitors) fo
21 s), including direct thrombin inhibitors and factor Xa inhibitors, are emerging options for thrombopr
23 xtended-duration thromboprophylaxis with the factor Xa inhibitor betrixaban to reduce the risk of str
24 he carbonyl group of an amide found in prior factor Xa inhibitors but also maintained a hydrogen bond
25 Bleeding is a complication of treatment with factor Xa inhibitors, but there are no specific agents f
28 onfatal pulmonary embolism, was reduced with factor Xa inhibitors compared with LMWH (4 fewer events
31 ived data from phase 3 trials of direct oral factor Xa inhibitors done at University Hospital Carl Gu
34 rombolysis in Myocardial Infarction 48), the factor Xa inhibitor edoxaban was noninferior to warfarin
37 eral anticoagulant that combines an indirect factor Xa inhibitor (fondaparinux analog) and a direct t
38 considerable interest in the development of factor Xa inhibitors for the intervention in thrombic di
39 been intense interest in the development of factor Xa inhibitors for the treatment of thrombotic dis
41 A fixed-dose regimen of rivaroxaban, an oral factor Xa inhibitor, has been shown to be as effective a
42 agulants (nOAC), which includes thrombin and factor Xa inhibitors, has been shown to be effective, bu
43 nhibitor, and rivaroxaban and apixaban, oral factor Xa inhibitors, have been found to be safe and eff
45 ctive, reversible, and direct small-molecule factor Xa inhibitor in patients with stable coronary dis
46 omplications during therapy with direct oral factor Xa inhibitors in a case series of women of reprod
47 ivaroxaban, apixaban, and edoxaban, the oral factor Xa inhibitors in the most advanced stages of deve
48 the foundation for further investigation of factor Xa inhibitors in the treatment of patients with c
56 oral anticoagulants, such as factor IIa and factor Xa inhibitors, may provide a novel treatment appr
57 in clinical trials suggest that direct oral factor Xa inhibitors might increase menstrual bleeding i
60 SAR107375), a novel potent dual thrombin and factor Xa inhibitor resulted from a rational optimizatio
63 thrombin inhibitor dabigatran etexilate and factor Xa inhibitors rivaroxaban and apixaban are new or
65 he effects of the DTI dabigatran, the direct factor Xa-inhibitor rivaroxaban, and of 2-O, 3-O desulfa
74 loped a novel series of potent and selective factor Xa inhibitors that employ a key 7-fluoroindazolyl
75 n a series of potent and orally bioavailable factor Xa inhibitors that ultimately led to the discover
76 of reproductive age treated with direct oral factor Xa inhibitors: the non-interventional Dresden NOA
77 of reproductive age who received direct oral factor Xa inhibitor therapy, of whom 57 had vaginal blee
79 pathway inhibitor (TFPI), and the selective factor Xa inhibitor, tick anticoagulant peptide (TAP), a
82 f an ongoing effort to prepare orally active factor Xa inhibitors using structure-based drug design t
86 luded because of the intake of a direct oral factor Xa inhibitor which has a strong impact on prothro
87 highly potent, selective, and orally active factor Xa inhibitor which was chosen for clinical develo
90 ther optimize the selectivity profile of our factor Xa inhibitors with a series of ortho- and/or para
91 combined treatment of aspirin or C921-78 (a factor Xa inhibitor) with CT50547 or 2-MeSAMP (a P2Y12 a
92 ts with acute major bleeding associated with factor Xa inhibitors, with effective hemostasis occurrin
93 mbinant tick anticoagulant peptide (rTAP), a factor Xa inhibitor, would reduce the thickness of neoin
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