ライフサイエンスコーパス: faecalis

1 imicrobial activity for P. gingivalis and E. faecalis.

2 tease-secreting enteric microbe Enterococcus faecalis.

3 stinal opportunistic bacterium, Enterococcus faecalis.

4 rial growth, mainly E. coli and Enterococcus faecalis.

5 regulator of antimicrobial resistance in E. faecalis.

6 oli, Klebsiella pneumoniae, and Enterococcus faecalis.

7 eloriated with antibiotics active against E. faecalis.

8 s was more effective against E. coli than E. faecalis.

9 C as well as PC from pathogenic Enterococcus faecalis.

10 nals modulated by the bacterium Enterococcus faecalis.

11 pportunistic bacterial pathogen Enterococcus faecalis.

12 t to the Gram-positive pathogen Enterococcus faecalis.

13 regulator of cephalosporin resistance in E. faecalis.

14 tetracycline-resistance plasmid pCF10 in E. faecalis.

15 ment were notable for growth of Enterococcus faecalis.

16 lts with vancomycin-susceptible Enterococcus faecalis.

17 level aminoglycoside resistance status of E. faecalis.

18 ignificantly more H(2)O(2) than wild-type E. faecalis.

19 , and EbpC) have not been investigated in E. faecalis.

20 important virulence factor for Enterococcus faecalis.

21 system, a major regulator of virulence in E. faecalis.

22 y found in clinical isolates of Enterococcus faecalis.

23 express the bacteriocin activity against E. faecalis.

24 ls, yeast, Escherichia coli and Enterococcus faecalis.

25 deficient (Il10(-/-)) mice with Enterococcus faecalis.

26 microorganism in institution A: Enterococcus faecalis, 18 cultures [51.4%]; institution B: Staphyloco

27 M) (16), vancomycin-susceptible Enterococcus faecalis (3), Aerococcus viridans (2), Bacillus (6), Cor

28 Enterococcus faecalis (48%), Enterococcus faecium (14%), Enterococcus

29 or testing daptomycin versus 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 Staphylococcus

30 Enterococcus faecalis, a Gram-positive bacterium, and Candida albican

31 Enterococcus faecalis, a leading cause of hospital-acquired infection

32 EF1143 from Enterococcus faecalis, a life-threatening pathogen that is resistant

33 The genome of Enterococcus faecalis, a low-GC Gram-positive opportunistic pathogen,

34 against the oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptococcus mutans,

35 O-1, were first developed using Enterococcus faecalis, an organism that has previously been shown to

36 re observed for daptomycin in isolates of E. faecalis and 2 ME, 1 for high-level gentamicin resistanc

37 apeutic and preventive strategies against E. faecalis and beyond.

38 lymicrobial infection, we discovered that E. faecalis and C. albicans negatively impact each other's

39 genic E. coli isolates and (10(0) CFU/mL) E. faecalis and E. faecium strains were detected within 4 a

40 ach with less than 28 SNP differences, of E. faecalis and E. hormaechei.

41 ilm and faecal samples included Enterococcus faecalis and Enterobacter hormaechei.

42 ectly identified 14/15 cases of Enterococcus faecalis and Enterococcus faecium bacteremia and 9 cases

43 sand (IOCS) to remove two FIB (Enterococcus faecalis and Escherichia coli) suspended in synthetic st

44 mM), the toxicity of 10 mg L(-1) PCP for E. faecalis and fresh anaerobic sludge was detected in 10 m

45 differences among seven chlorophenols to E. faecalis and fresh mixed anaerobic sludge were elucidate

46 n NICU organisms including K. oxytoca and E. faecalis and increases in common adult organisms includi

47 n of Staphylococcus spp., E. faecium, and E. faecalis and its ability to ascertain mecA, vanA, and va

48 ing of how (p)ppGpp promotes virulence in E. faecalis and other bacterial pathogens is still lacking.

49 g overproduction of PrgB-like adhesins in E. faecalis and other clinically-important Gram-positive sp

50 to characterize the infection dynamics of E faecalis and show that infected wounds result in 2 diffe

51 The Gram-positive bacterium Enterococcus faecalis and the fungus Candida albicans are both found

52 teria Staphylococcus aureus and Enterococcus faecalis and two Gram-negative bacteria Escherichia coli

53 ith host-protective properties (Enterococcus faecalis) and a pathogen (Staphylococcus aureus) within

54 a (Photorhabdus luminescens and Enterococcus faecalis) and two nonpathogenic bacteria (Escherichia co

55 f Staphylococcus aureus, 90% of Enterococcus faecalis, and 88% of Enterococcus faecium isolates were

56 SA), Listeria monocytogenes and Enterococcus faecalis, and against the Gram-negative bacteria Escheri

57 87%, 83%, 98%, and 80%, respectively, for E. faecalis, and all 100% for S. aureus.

58 including Bacillus subtilis and Enterococcus faecalis, and drug-sensitive and drug-resistant strains

59 acement assays using PGTs from S. aureus, E. faecalis, and E. coli.

60 idis, Streptococcus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and three associated

61 e facultative anaerobic strain, Enterococcus faecalis, and fresh mixed anaerobic sludge, with or with

62 o inhibitory activity against periodontal E. faecalis, and may be clinically useful in treatment of p

63 heir bactericidal activity against E coli, E faecalis, and S typhimurium, whereas exposure to CO or o

64 .00% for Staphylococcus aureus, Enterococcus faecalis, and streptococci, respectively.

65 perbugs." Bacteria of the genus Enterococcus faecalis are highly antibiotic-resistant nosocomial path

66 is isolates underscores the importance of E. faecalis as a reservoir of VGs in the fresh water aquati

67 We undertook whole genome sequencing of E. faecalis associated with bloodstream infection in the UK

68 tionic human beta-defensins interact with E. faecalis at discrete septal foci, and this exposure disr

69 R/ESI-MS again found genetic evidence for E. faecalis at levels comparable to the pretreatment levels

70 n detect superoxide produced by Enterococcus faecalis at rates that are too low for detection by CW E

71 and two AR bacterial strains (E. coli and E. faecalis, at 10(5) CFU mL(-1)) were spiked in real WW.

72 CC 29213 (0.03 to 0.12 mug/ml), Enterococcus faecalis ATCC 29212 (0.03 to 0.12 mug/ml), and Streptoco

73 aureus ATCC 29213 (broth only), Enterococcus faecalis ATCC 29212 (broth only), Streptococcus pneumoni

74 reus ATCC 29213, 0.016 to 0.12 mug/ml for E. faecalis ATCC 29212, 0.008 to 0.03 mug/ml for S. pneumon

75 ococcus epidermidis ATCC 12228, Enterococcus faecalis ATCC 29212, and Escherichia coli DH5alpha, onto

76 ding Pseudomonas putida KT2440, Enterococcus faecalis ATCC 29212, Salmonella Typhimurium ATCC 14028,

77 Monomicrobial E. faecalis bacteremia (hazard ratio [HR], 3.60; 95% confid

78 either Enterococcus faecium or Enterococcus faecalis bacteremia caused by both vancomycin-resistant

79 ic gentamicin for uncomplicated Enterococcus faecalis bacteremia in children.

80 paring clinical outcomes of children with E. faecalis bacteremia without endocarditis receiving ampic

81 Monomicrobial E. faecalis bacteremia, community acquisition, prosthetic h

82 ests that for children with uncomplicated E. faecalis bacteremia, the addition of low-dose gentamicin

83 tudy is the first to report heterogeneous E. faecalis bacteremia.

84 we included 647 consecutive patients with E. faecalis bacteremia.

85 rditis (IE) among patients with Enterococcus faecalis bacteremia.

86 Newly isolated Enterococci faecalis bacterial strains AIM06 (DSM100702) and SR14 (D

87 We report here the identification of the E. faecalis bacteriocin, EntV, produced from the entV (ef10

88 ulture-positive endophthalmitis caused by E. faecalis between January 1, 2002, and December 31, 2012,

89 omal genetic determinants responsible for E. faecalis biofilm-mediated infection, we used a rabbit mo

90 ation of a laboratory strain of Enterococcus faecalis, but depressed photoinactivation of sewage-sour

91 wed complete bacteriocin activity against E. faecalis, but neither BacL1 nor BacA protein alone showe

92 faecalis, but resulted in greater survival of E. coli in

93 anB in Enterococcus faecium and Enterococcus faecalis) by the BC-GP assay also was assessed.

94 ing disruption of intestinal homeostasis, E. faecalis can overgrow, cross the intestinal barrier, and

95 investigations demonstrate that GelE from E. faecalis can regulate enteric epithelial permeability vi

96 The bacterium Enterococcus faecalis cannot synthesize heme but can acquire it from

97 lating expression of proteins involved in E. faecalis carbohydrate uptake and utilization.

98 ts from the type II-A system of Enterococcus faecalis Cas1 and Cas2 during spacer integration.

99 Enterococcus faecalis Cas1-Cas2 selectively binds to a splayed 30-bas

100 Compared to the wild type, the E. faecalis ccpA mutant had an impaired ability to adhere t

101 The Enterococcus faecalis cell wall-anchored protein Ace is an important

102 of the peptide pheromone cCF10, Enterococcus faecalis cells carrying pCF10 produce three surface adhe

103 s present, results in the lysis of viable E. faecalis cells.

104 Pure cultures of 47 subgingival E. faecalis clinical isolates were each inoculated onto spe

105 eriocin 41 (Bac41) is produced by certain E. faecalis clinical isolates, and it is active against oth

106 vitro activity against human subgingival E. faecalis clinical isolates, and would likely be ineffect

107 E. faecalis co-exists with Escherichia coli and other patho

108 -) mice attenuated infection and promoted E. faecalis colonization resistance by restoring the divers

109 prior to urinary catheterization enhanced E. faecalis colonization, suggesting that implant-mediated

110 dentification of genetic determinants for E. faecalis commensal and pathogenic interactions with M. s

111 mine utilization (eut) locus of Enterococcus faecalis, containing at least 19 genes distributed over

112 nting colonization by multidrug-resistant E. faecalis could therefore be a valuable approach towards

113 aneous mutants that allowed growth of the E. faecalis DeltafabI strain on fatty acid-free medium.

114 ally regulate cephalosporin resistance in E. faecalis, dependent on the kinase activity of IreK.

115 gram-positive bacteria, such as Enterococcus faecalis, disulfide bonds are formed in secreted bacteri

116 nd epithelial translocation of pathogenic E. faecalis during severe microbial dysbiosis and was amelo

117 crobial resistance for bacteria including E. faecalis, E. coli, K. pneumoniae and S. aureus.

118 s subcellular compartmentalization in the E. faecalis Ebp pilus system.

119 egionella pneumophila (Lp), and Enterococcus faecalis (Ef) by using anti-infective, antivirulence, an

120 one-responsive plasmid pCF10 of Enterococcus faecalis encodes a putative cell wall hydrolase, PrgK, a

121 of ace (adhesin to collagen of Enterococcus faecalis), encoding a virulence factor in endocarditis a

122 t EfbA is an important factor involved in E. faecalis endocarditis and that rEfbA immunization is eff

123 ection is not a random sample of cases of E. faecalis endocarditis, these results indicate that nonen

124 aomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O157:H7, Salmone

125 type of bacterium in the order: Enterococcus faecalis, Escherichia coli O157:H7, and Escherichia coli

126 Periodontal E. faecalis exhibited substantial in vitro resistance to te

127 To investigate E. faecalis factors required for commensalism, we identifie

128 resisting immune clearance, contributes to E faecalis fitness.

129 osition in vancomycin-resistant Enterococcus faecalis following the growth in presence of vancomycin

130 even Escherichia coli and three Enterococcus faecalis ) for all ten patient samples.

131 hia coli, Bacillus subtilis and Enterococcus faecalis, from the guts of the desert woodrat (Neotoma l

132 ind putative promoter segments of several E. faecalis genes in an NADH-responsive manner, indicating

133 To identify and characterize E. faecalis genes that are key to intestinal colonization,

134 required for commensalism, we identified E. faecalis genes that are upregulated in the gut of M. sex

135 Fibrobacteres phylum and 12 genera in the E. faecalis group and antibiotics group were lower than tha

136 12 genera were uniquely identified in the E. faecalis group on d 14 and 28.

137 ary treatments: the basal diet group, the E. faecalis group, and the antibiotic group on d 0, 14, and

138 Bacterial abundance and diversity in the E. faecalis group, bacterial diversity in the antibiotic gr

139 Here we show that E. faecalis harbouring pPD1 replaces indigenous enterococci

140 omone-responsive, conjugative plasmids of E. faecalis have retained Prg-like surface functions over e

141 PCR/ESI-MS detected genetic evidence of E. faecalis in all CSF samples, but the level of detection

142 nisms Staphylococcus aureus and Enterococcus faecalis in comparison with known analogues.

143 in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the elucidation of GAC biosynthe

144 ctivity against the cell wall fraction of E. faecalis in the absence of BacA.

145 e absence of the silicone tubing implant, E. faecalis induced only minimal inflammation and was rapid

146 investigate the role of this receptor in E. faecalis-induced permeability.

147 This antiapoptotic activity in E. faecalis-infected cells was dependent on the activation

148 (ADP-ribose) polymerase were inhibited in E. faecalis-infected cells, indicating that E. faecalis pro

149 ected from the peritoneal lavage fluid of E. faecalis-infected mice showed reduced levels of apoptosi

150 The pathogenesis of E. faecalis infection relies in part on its capacity to col

151 E. faecalis infections are associated with a high mortality

152 ous animal models have been used to mimic E. faecalis infections, but none of them is considered idea

153 The pathogenesis of many E. faecalis infections, including endocarditis and catheter

154 tial targets for interventions to prevent E. faecalis infections.

155 (AG) combinations for treating Enterococcus faecalis infective endocarditis (EFIE).

156 mpare outcomes in patients with Enterococcus faecalis infective endocarditis treated in the years bef

157 nts of cholate and lysozyme resistance in E. faecalis, IreK was the only one found to be required for

158 Enterococcus faecalis is a commensal and pathogen of humans and insec

159 Enterococcus faecalis is a commensal bacterium found in the gastroint

160 Enterococcus faecalis is a commensal bacterium of the human intestine

161 Since Enterococcus faecalis is a natural heme auxotroph and cause of bloods

162 from the Gram-positive pathogen Enterococcus faecalis is a sequence-specific RNA-binding protein.

163 In Manduca sexta, E. faecalis is an infrequent member of the commensal gut co

164 The Gram-positive bacterium Enterococcus faecalis is both a colonizer of the gastrointestinal tra

165 Enterococcus faecalis is both a common commensal of the human gastroi

166 Enterococcus faecalis is frequently associated with polymicrobial inf

167 mechanisms of cephalosporin resistance in E. faecalis is lacking.

168 Enterococcus faecalis is one of the most frequently isolated bacteria

169 Enterococcus faecalis is part of the human intestinal microbiome and

170 red for pathogenesis, GIT colonization by E. faecalis is poorly understood.

171 Expansion of intestinal Enterococcus faecalis is sufficient to exacerbate ethanol-induced liv

172 Enterococcus faecalis is unusual in that it encodes two unrelated eno

173 the in vitro antibiotic susceptibility of E. faecalis isolated from periodontitis patients in the Uni

174 Enterococcus faecalis isolates (n = 1,112) were highly susceptible (>

175 U gene pairs are widely distributed among E. faecalis isolates and other enterococcal and staphylococ

176 This revealed that 6 E. faecalis isolates behaved in a commensal manner with no

177 eages were predominant among endocarditis E. faecalis isolates recovered during this time period.

178 The virulence of 28 E. faecalis isolates representing 24 multilocus sequence ty

179 Eighty-one endocarditis-derived Enterococcus faecalis isolates that were collected from individual pa

180 e presence of multiple VGs in most of the E. faecalis isolates underscores the importance of E. faeca

181 All E. faecalis isolates were judged to be S by the reference m

182 ed to be a strain-independent trait among E. faecalis isolates.

183 ivation mechanism may be active against Ent. faecalis, it is not for the sewage-source organisms.

184 gelatinase-and-cytolysin-negative strain E. faecalis JH2-2.

185 species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Klebsiella pneumoniae, and

186 l isolates of Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, and Pseudomonas aerugin

187 es indigenous enterococci and outcompetes E. faecalis lacking pPD1.

188 ned media (CCM) from E. faecalis V583 and E. faecalis lacking the gelatinase gene (gelE).

189 robiota (Lactobacillus reuteri, Enterococcus faecalis, Lactobacillus crispatus and Clostridium orbisc

190 In Enterococcus faecalis, lateral transfer of conjugative plasmids that

191 ecalis LDH2 </= Lactococcus lactis LDH2 < E. faecalis LDH1 < L. lactis LDH1 </= Streptococcus pyogene

192 P, Pi is an activator of S. pyogenes LDH, E. faecalis LDH1, and L. lactis LDH1 and LDH2 at pH 6.

193 itivity in the following order: Enterococcus faecalis LDH2 </= Lactococcus lactis LDH2 < E. faecalis

194 3) Addition of Pi inhibits E. faecalis LDH2, whereas in the absence of FBP, Pi is an a

195 A comparison of the E. faecalis LiaR, E. faecium LiaR, and the LiaR homolog fro

196 Enterococcus faecalis may contribute to periodontal breakdown in heav

197 mevalonate diphosphate decarboxylase from E. faecalis (MDDEF).

198 E. faecalis MN1 inhibited interleukin-8 production from hum

199 amic acid compounds naturally produced by E. faecalis MN1, may be useful in prevention of diseases th

200 3% of attached E. coli and 2% attached Ent. faecalis mobilized from the saturated sand column.

201 Thus, E. faecalis modulates its local environment by contributing

202 We have in E. faecalis monitored production of the enzyme polypeptide

203 Analyses of E. faecalis mutants exhibiting defects in antimicrobial res

204 , and daptomycin-nonsusceptible Enterococcus faecalis (n = 6).

205 sis), from daptomycin-resistant Enterococcus faecalis not only reversed resistance to 2 clinically av

206 Here, we expressed and purified the E. faecalis OG1RF EfbA and confirmed that this protein bind

207 Much of the negative effect of E. faecalis on C. albicans was due to the inhibition of C.

208 patterns, including heat-killed Enterococcus faecalis or CpG DNA, led to increased IkappaB cleavage,

209 esting results for 45 of 46 cultures with E. faecalis or E. faecium.

210 bial peptide activity developed either an E. faecalis or Pseudomonas aeruginosa urinary tract infecti

211 One of the hallmarks of E. faecalis pathogenesis is its unusual ability to tolerate

212 h is a novel, powerful model for studying E. faecalis pathogenesis, enabling us to dissect the mechan

213 iding new insights into the regulation of E. faecalis pathogenesis.

214 Enterococcus faecalis pCF10 transfers at high frequencies upon pherom

215 e of the catheter itself is essential for E. faecalis persistence in the bladder.

216 Further analysis revealed that active E. faecalis physiology was important for inhibition of host

217 ibody to the major component of Enterococcus faecalis pili, EbpC, labels polymerized pilus structures

218 r mobile genetic elements shapes in vitro E. faecalis populations.

219 t can be exploited to alter heterogeneous E. faecalis populations.

220 Multidrug-resistant Enterococcus faecalis possess numerous mobile elements that encode vi

221 robiota of MAT mothers, whereas Enterococcus faecalis predominated within the MAT infant GIT.

222 heptapeptide pheromone produced by native E. faecalis present in the fecal consortium.

223 avA-like fibronectin adhesin of Enterococcus faecalis previously shown to be important in experimenta

224 response induced by urinary implantation, E. faecalis produced biofilm and high bladder titers in the

225 , Keogh et al. (2016) show that Enterococcus faecalis promotes Escherichia coli biofilm formation in

226 faecalis-infected cells, indicating that E. faecalis protects macrophages from apoptosis by inhibiti

227 Secreted E. faecalis proteins induced permeability in epithelial cel

228 Secreted E. faecalis proteins induced permeability in the colonic ep

229 enteroides, Bacillus cereus and Enterococcus faecalis proving its antimicrobial action.

230 as Staphylococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebsiella pneumon

231 ications with Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphylococcus aureus

232 cocci diversity, 11 isolates of Enterococcus faecalis recovered from freshwater watersheds (environme

233 fied signatures suggest that ancestors of E. faecalis resided in extra-enteric habitats, challenging

234 ort chains in SCV S. aureus and wild-type E. faecalis results in reduced growth rate but provides res

235 logs from Bacillus subtilis and Enterococcus faecalis retain this ability.

236 To study the role of E. faecalis Rex, we purified EF2638 and evaluated its DNA b

237 st identified from the pathogen Enterococcus faecalis S613.

238 We studied the survival of Enterococcus faecalis, Salmonella spp., E. coli O157 and porcine parv

239 We show that E. faecalis significantly augments E. coli biofilm growth a

240 of Gram-positive facultative anaerobes (Ent. faecalis, Staph. aureus, and Strep. bovis).

241 Colonization with an E. faecalis strain carrying a conjugation-defective pPD1 mu

242 A carbapenem-resistant Alcaligenes faecalis strain was isolated from a surveillance swab of

243 protein named AfIP-1A/1B from an Alcaligenes faecalis strain.

244 e intestine, pPD1 is transferred to other E. faecalis strains by conjugation, enhancing their surviva

245 aecium strains but none of five Enterococcus faecalis strains consistently developed resistance at al

246 g exposure to CCM from parental or mutant E. faecalis strains indicated paracellular permeability.

247 The E. faecalis strains were from two sequence types (ST191 and

248 The false-positive E. faecalis strains were typed by Diversilab Rep-PCR (bioMe

249 isolates, and it is active against other E. faecalis strains.

250 638 represents an authentic Rex factor in E. faecalis that influences the production or detoxificatio

251 In this study, we show in Enterococcus faecalis that SecA and Sortase A, required for the attac

252 of the human bacterial pathogen Enterococcus faecalis The final enzyme in this pathway, mevalonate di

253 2) For L. plantarum, S. pyogenes, and E. faecalis, the effects of Pi are distinguishable from the

254 In Enterococcus faecalis, the regulatory nucleotides pppGpp and ppGpp, c

255 with the opportunistic pathogen Enterococcus faecalis through promotion of host-microbiota mutualism.

256 tudy, we investigated the contribution of E. faecalis to mixed-species infection when iron availabili

257 E. faecalis tolerates high concentrations of GIT antimicrob

258 Interestingly, we observe that E. faecalis transiently maintains CRISPR targets despite ac

259 Finally, a screen of an E. faecalis transposon mutant library identified other gene

260 earch demonstrated that dietary Enterococcus Faecalis UC-100 substituting antibiotics enhanced growth

261 microbiota in the distal gut of pigs fed E. faecalis UC-100 substituting antibiotics, this study ass

262 ed against Escherichia coli and Enterococcus faecalis urinary tract infection isolates.

263 ver, little is known about the mechanisms E. faecalis uses to colonize and compete for stable gastroi

264 concentrated conditioned media (CCM) from E. faecalis V583 and E. faecalis lacking the gelatinase gen

265 : (i) the accretion of mobile elements in E. faecalis V583 renders it incompatible with commensal str

266 Gelatinase (GelE) purified from E. faecalis V583 was used to confirm the ability of this pr

267 group (</=0.06 microg/mL), and Enterococcus faecalis (vancomycin susceptible, </=0.25 microg/mL).

268 irulence map that explains enhancement in E. faecalis virulence and contributes to a deeper comprehen

269 ciate CcpA with the production of a major E. faecalis virulence factor, providing new insights into t

270 Several factors contributing to E. faecalis virulence have been identified.

271 s, we hypothesized that (p)ppGpp mediates E. faecalis virulence through regulation of metal homeostas

272 nvestigation indicated that, unlike other E. faecalis virulence traits, phage03-like elements were fo

273 jor role in control of ace expression and E. faecalis virulence.

274 calcitrant vancomycin-resistant Enterococcus faecalis (VRE) and 2 patients with infectious crystallin

275 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial pathogen(1,2

276 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial pathogen1,2.

277 of vancomycin-resistant and -susceptible E. faecalis (VSEfs), which has important implications for i

278 ltures containing Enterococcus faecium or E. faecalis was 100% sensitive and specific.

279 D) determined for HAdV 41, Phi X 174, and E. faecalis was 35 GU/muL, 1 GU/muL, and 5 x 10(3) GU/muL (

280 nic host-microbe interaction of Enterococcus faecalis was explored using a Caenorhabditis elegans mod

281 Under sterile conditions, 1 muL Enterococcus faecalis was inoculated inside the implants, and abutmen

282 f solar exposure for AR E. coli, while AR E. faecalis was more resistant to the disinfection process

283 io of occurrence of ace and gelE genes in E. faecalis was much higher at 7.96 and 6.40 times, respect

284 nce of ace, gelE, efaA, and asa1 genes in E. faecalis were found to be much higher compared to the ot

285 alis and the endodontic species Enterococcus faecalis were grown to early log phase and inoculated on

286 AcpAs of Lactococcus lactis and Enterococcus faecalis were inactive.

287 ly and phylogenetically the environmental E. faecalis were indistinguishable from their enteric count

288 of attached E. coli and 3% of attached Ent. faecalis were mobilized from the drained sand column com

289 of attached E. coli and 8% of attached Ent. faecalis were released from IOCS columns during draining

290 teicoplanin-susceptible (VanB phenotype) E. faecalis were responsible for major and minor errors.

291 rcine gut in response to diets containing E. faecalis were similar to the response to which containin

292 E Clostridium perfringens, and Enterococcus faecalis, whereas the reverse was true for Clostridium b

293 1, Phi X 174) and the bacterium Enterococcus faecalis, which are relevant for water hygiene.

294 uired from vancomycin-resistant Enterococcus faecalis, which is known to alter cell wall structure an

295 ntibiotic resistance plasmid of Enterococcus faecalis, which negatively regulates conjugation of the

296 from multidrug-resistant (MDR) Enterococcus faecalis, which only possess an orphan CRISPR locus, ter

297 gle very major error was obtained against E. faecalis, while vancomycin-intermediate S. aureus (VISA)

298 Incubation of E. faecalis with heme increased growth and restored catalas

299 del of colonization of the mouse gut with E. faecalis, without disrupting the microbiota, to evaluate

300 y describe a mouse model for investigating E faecalis wound infection determinants, and suggest that