ライフサイエンスコーパス: faecium

1 for 45 of 46 cultures with E. faecalis or E. faecium.

2 uence of esp on the transport behavior of E. faecium.

3 in the effects of esp on the retention of E. faecium.

4 aureus and vancomycin-resistant Enterococcus faecium.

5 ureus, and vancomycin-resistant Enterococcus faecium.

6 vancomycin-resistant strains of Enterococcus faecium.

7 avancin have different modes of action in E. faecium.

8 cedes invasive infection due to Enterococcus faecium.

9 ween vancomycin-resistant E. faecalis and E. faecium.

10 ree) were also tested for the presence of E. faecium.

11 ge of quinupristin-dalfopristin-resistant E. faecium.

12 in-dalfopristin resistance in human fecal E. faecium.

13 29 cultures, all of which were Enterococcus faecium.

14 ntified in nonstool isolates of Enterococcus faecium.

15 tified in a clinical isolate of Enterococcus faecium.

16 , Streptococcus pneumoniae, and Enterococcus faecium.

17 perhaps from a human vancomycin-resistant E. faecium.

18 sed by E. faecalis and 37% were caused by E. faecium.

19 not be eradicated by prolonged feeding on E. faecium.

20 e Clostridium sp. 7 2 43FAA and Enterococcus faecium.

21 ia such as Escherichia coli and Enterococcus faecium.

22 synthetic urine also containing Enterococcus faecium.

23 aureus and vancomycin-resistant Enterococcus faecium.

24 licating a role for CcpA in the growth of E. faecium.

25 casei, Lactococcus lactis, and Enterococcus faecium.

26 nized with vancomycin-resistant Enterococcus faecium.

27 E strains (vancomycin-resistant Enterococcus faecium [0], methicillin-resistant Staphylococcus aureus

28 , Escherichia coli (11.8%), and Enterococcus faecium (11.4%).

29 Enterococcus faecalis (48%), Enterococcus faecium (14%), Enterococcus mundtii (13%), and Enterococ

30 ococcus faecalis, 8 isolates of Enterococcus faecium, 2 isolates of Enterococcus hirae, 7 isolates of

31 Enterococcus faecium 664.1H1 is multiply antibiotic resistant and mer

32 upport for the tetracycline resistance of E. faecium 664.1H1 was characterized.

33 this gene is located on the chromosome of E. faecium 664.1H1, on a novel conjugative transposon.

34 63%, 63%, 63%, and 56%, respectively, for E. faecium, 87%, 83%, 98%, and 80%, respectively, for E. fa

35 the vancomycin-binding site) in Enterococcus faecium, a leading antibiotic-resistant pathogen.

36 ses are nanomolar inhibitors of Enterococcus faecium AAC(6')-Ii.

37 ococci are Enterococcus faecalis, whereas E. faecium accounts for 5 to 10% of isolates.

38 ally and in combination against Enterococcus faecium, Acinetobacter baumannii and Klebsiella pneumoni

39 A collagen-binding adhesin of Enterococcus faecium, Acm, was identified.

40 the clinical importance of multiresistant E. faecium across different continents.

41 Interestingly, a high titer of E. faecium also accumulates in the nematode gut, but does n

42 to 2002, we evaluated the percentages of E. faecium among all enterococcal isolates and the percenta

43 Both the percentage of E. faecium among the enterococci and the proportion of vanc

44 eria were MDR, including 95% of Enterococcus faecium and 55% of Enterobacteriaceae; 82% of deep SSIs

45 t Acm has contributed to the emergence of E. faecium and CC17 in nosocomial infections.

46 identifying patients colonized with both E. faecium and E. faecalis, a feature useful for infection

47 s of the recovery of vancomycin-resistant E. faecium and E. faecalis, the sensitivity and PPV were as

48 ferentiate vancomycin-resistant Enterococcus faecium and Enterococcus faecalis (VRE).

49 epidermidis and vanA or vanB in Enterococcus faecium and Enterococcus faecalis) by the BC-GP assay al

50 When overexpressed in E. faecium and Escherichia coli, the native and recombinant

51 w-spectrum antibacterial activity against E. faecium and exhibited metabolic stability with low intri

52 cluding human pathogens such as Enterococcus faecium and Listeria monocytogenes.

53 rity) were vancomycin-resistant Enterococcus faecium and meticillin-resistant Staphylococcus aureus.

54 th the exception of 2 isolates, Enterococcus faecium and Nocardia exalbida, all the other 725 (99.7%)

55 For E. faecium and other enterococci, the combination of FS and

56 l impact on colonization and virulence in E. faecium and possibly other Gram-positive bacterial speci

57 e and drug-resistant strains of Enterococcus faecium and Staphylococcus aureus.

58 of E. faecalis strains (unlike Enterococcus faecium and streptococci) have a single pilus locus.

59 vanA and vanB], 93 vancomycin-susceptible E. faecium) and epidemiological data were collected.

60 iae, Enterococcus faecalis, and Enterococcus faecium) and three associated genetic resistance determi

61 us 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 Staphylococcus aureus isolates using (i

62 imipenem was 91% for E. faecalis, 98% for E. faecium, and 87% for other enterococci.

63 us aureus, vancomycin-resistant Enterococcus faecium, and beta-lactam-resistant Klebsiella pneumoniae

64 ccurate detection of Staphylococcus spp., E. faecium, and E. faecalis and its ability to ascertain me

65 ptide in Enterococcus faecalis, Enterococcus faecium, and Enterococcus hirae.

66 three additional C. coli, one additional E. faecium, and one C. jejuni also developed resistance whe

67 ccus species, 633 strains of E. faecalis, E. faecium, and other enterococci isolated from blood cultu

68 -level vancomycin resistance in Enterococcus faecium, and resistance to extended-spectrum cephalospor

69 er/platinum combination against Enterococcus faecium, and silver/copper combination against Acinetoba

70 rsed MRSA, vancomycin-resistant Enterococcus faecium, and Staphylococcus epidermidis biofilms.

71 in infection-derived clinical isolates of E. faecium, and suggest that Acm is the primary adhesin res

72 antibiotic-resistant hospital-associated E. faecium are often replaced by clade B strains once patie

73 their involvement has been implicated in E. faecium as well.

74 Genomes were also available for E. faecium associated with bloodstream infections in 15 pat

75 sion library with sera from patients with E. faecium-associated endocarditis.

76 walls of vancomycin-susceptible Enterococcus faecium (ATTC 49624).

77 es of Enterococcus faecalis and Enterococcus faecium bacteremia and 9 cases of Streptococcus pneumoni

78 i surface protein (esp) found in Enterococci faecium, Bacteroides HF183, adenoviruses (AVs), and poly

79 country, indicating frequent movement of E. faecium between regions that rarely share hospital patie

80 collagen, and this protein competed with E. faecium binding to immobilized CI.

81 ycin MICs of 3-4 microg/mL in the initial E. faecium blood isolate predicted microbiological failure

82 ited Kingdom identified 342 patients with E. faecium bloodstream infection over 7 years.

83 ibiotic for multidrug-resistant Enterococcus faecium bloodstream infections (BSIs).

84 took whole-genome sequencing (WGS) of 495 E. faecium bloodstream isolates from 2001-2011 in the Unite

85 t study that included adult patients with E. faecium BSI for whom initial isolates, follow-up blood c

86 We postulate that patients with E. faecium BSIs exhibiting daptomycin MICs of 3-4 microg/mL

87 (VM) errors (4/14) with enterococci (all E. faecium) but none (0/22) with staphylococci.

88 lfopristin resistance was absent in human E. faecium, but 56% of conventional poultry isolates were q

89 llin-imipenem tested with E. faecalis and E. faecium by BMD was >/=94% but was </=90% for other enter

90 ntigen, SagA, was identified in Enterococcus faecium by screening an E. faecium genomic expression li

91 illin- and vancomycin-resistant Enterococcus faecium C68 in a mouse model.

92 ization by vancomycin-resistant Enterococcus faecium C68 in a mouse model.

93 ), at doses that promoted colonization by E. faecium C68.

94 For E. faecium, categorical agreement was poor between the refe

95 ly US AMP isolates were clonally related, E. faecium CC17 isolates have been circulating in the Unite

96 We suggest that actively dividing E. faecium cells have three zones of unique peptidoglycan p

97 d significantly enhance the attachment of E. faecium cells onto the surface of silica sands and thus

98 of colony-forming units per gram) by the E. faecium clades at any time point (P > .05).

99 purified from culture supernatants of an E. faecium clinical isolate, was found to match the N-termi

100 This gene is highly enriched in E. faecium clinical isolates and absent in commensal isolat

101 These results indicate that E. faecium clinical isolates express transmissible factors

102 sistant Enterococci faecalis and Enterococci faecium clinical isolates revealed that the nature and r

103 rom three CI-binding vancomycin-resistant E. faecium clinical isolates showed 100% identity, analysis

104 alysis revealed that CVM1869 was a canine E. faecium clone that had acquired Tn1546, perhaps from a h

105 e demonstrated a significant reduction in E. faecium collagen adherence by affinity-purified anti-Acm

106 deleted from the chromosome of Enterococcus faecium D344R in all possible combinations in order to i

107 of a large genomic region from Enterococcus faecium D344R in which the sequence from "joint" regions

108 lin-resistant, Tn916-containing Enterococcus faecium D344R.

109 conjugants derived from matings that used E. faecium D344SRF as a recipient strain colonized mouse ga

110 Correspondingly, E. faecalis, but not E. faecium, degrades the glycan on RNase B during growth.

111 eficient Lactococcus lactis and Enterococcus faecium did not enhance biofilm formation as was observe

112 ive staphylococci, Enterococcus faecalis, E. faecium, E. avium, E. durans, E. casseliflavus, and E. g

113 luding the species Enterococcus faecalis, E. faecium, E. casseliflavus, and E. gallinarum.

114 . faecalis, E. mundtii, E. casseliflavus, E. faecium, E. hirae, E. avium, and E. durans, respectively

115 e identified 2 gene clusters in Enterococcus faecium each encoding a homolog of Gls24 (Gls33 and Gls2

116 Enterococcus faecium encodes a triple-specific aaPGS (RakPGS) that ut

117 four distinct surface carbohydrates from E. faecium endocarditis isolate Tx16, shown previously to b

118 ffinity-purified anti-Acm antibodies from E. faecium endocarditis patient sera, suggesting that Acm m

119 antibodies to Acm were present in all 14 E. faecium endocarditis patient sera.

120 on of VanA- and VanB-containing Enterococcus faecium (ENFM) and Enterococcus faecalis (ENFS) isolates

121 aused by Staphylococcus aureus, Enterococcus faecium, Enterococcus faecalis, Klebsiella pneumoniae, P

122 ncluding Enterococcus faecalis, Enterococcus faecium, Enterococcus gallinaraum, and Enterococcus dura

123 ously characterized aaPGSs, the Enterococcus faecium enzyme used an expanded repertoire of amino acid

124 Strains of Enterococcus faecium express a cell wall-anchored protein, Acm, which

125 fibronectin-binding protein of Enterococcus faecium (fnm), a homologue of Streptococcus pneumoniae p

126 of morphine-withdrawn mice were Enterococcus faecium followed by Klebsiella pneumoniae.

127 e and grew vancomycin-resistant Enterococcus faecium from the cerebrospinal fluid.

128 erred DNA into a second recipient strain (E. faecium GE-1), which also colonized mice in significantl

129 ates, are part of the hospital-associated E. faecium genogroup referred to as clonal complex 17 (CC17

130 The Enterococcus faecium genogroup, referred to as clonal complex 17 (CC1

131 d in Enterococcus faecium by screening an E. faecium genomic expression library with sera from patien

132 sting that the sagA gene is essential for E. faecium growth and may be involved in cell wall metaboli

133 E. faecium has been categorized as belonging to three clade

134 uggest that hydrogen peroxide produced by E. faecium has cytotoxic effects and highlight the utility

135 solation of multidrug-resistant Enterococcus faecium has dramatically reduced the therapeutic alterna

136 Enterococcus faecium has recently emerged as an important cause of no

137 ansferase from a human urinary isolate of E. faecium, has been determined as an apoenzyme and in comp

138 /mL, show almost all vancomycin-resistant E. faecium have AUICs <125.

139 Antimicrobial-resistant E. coli and E. faecium have become significant pathogens in oncology.

140 promote gastrointestinal colonization by E. faecium have not been identified.

141 ncidence of multidrug-resistant Enterococcus faecium hospital infections has been steadily increasing

142 portant for the pathogenesis of Enterococcus faecium in a rat infective endocarditis model.

143 opristin, a drug for vancomycin-resistant E. faecium in humans.

144 and dissemination of hospital-associated E. faecium in the UK&I and provide evidence for WGS as an i

145 s, Bifidobacterium bifidum and Streptococcus faecium) in capsules, twice a day for 30 days.

146 to effectively kill both E. faecalis and E. faecium (including vancomycin-resistant strains), as wel

147 ge of enterococci that were identified as E. faecium increased from 12.7 to 22.2% (P < 0.001) and the

148 nd the proportion of vancomycin-resistant E. faecium increased significantly over this 10-year period

149 the potential for streptogramin-resistant E. faecium infection in humans.

150 ts of a glycoconjugate vaccine to prevent E. faecium infection.

151 Enterococcus faecalis and Enterococcus faecium infections are increasingly difficult to treat d

152 articular, vancomycin-resistant Enterococcus faecium infections have been increasing in frequency, re

153 hirty-seven of 41 sera from patients with E. faecium infections showed reactivity with recombinant Ac

154 tions; and vancomycin-resistant Enterococcus faecium infections, including cases with concurrent bact

155 eatment of vancomycin-resistant Enterococcus faecium infections.

156 ration approval for the treatment of VRE (E. faecium) infections, namely, linezolid and quinupristin/

157 Enterococcus faecium is a common cause of nosocomial infections, of w

158 pportunistic bacterial pathogen Enterococcus faecium is a leading source of nosocomial infections, it

159 Enterococcus faecium is a multidrug-resistant opportunist causing dif

160 E. faecium is able to produce hydrogen peroxide by using gl

161 Enterococcus faecium is an important cause of hospital-associated inf

162 s, non-susceptibility to DAP by Enterococcus faecium is correlated frequently with a mutation in LiaR

163 triazolo[1,5-a]pyrimidines active against E. faecium is reported herein.

164 secreted antigen A (SagA) from Enterococcus faecium is sufficient to protect Caenorhabditis elegans

165 ization by antibiotic-resistant Enterococcus faecium is the first step in a process that can lead to

166 ion of transpeptidation by oritavancin in E. faecium is the result of the large number of secondary b

167 ity of a colonization-proficient clinical E. faecium isolate (C68) to transfer colonizing ability to

168 ncomycin heteroresistance in an Enterococcus faecium isolate from a patient.

169 One E. faecium isolate, CVM1869, displayed high-level resistanc

170 he first report of a vancomycin-resistant E. faecium isolated from a companion animal in the United S

171 Here we analyzed 90 E. faecium isolates (99% acm(+)) and found that the Acm pro

172 E. faecium isolates (n=577) from diverse sources were scree

173 aI restriction patterns from 62 Enterococcus faecium isolates and the other containing SmaI restricti

174 utic alternatives because the majority of E. faecium isolates are resistant to ampicillin and vancomy

175 53 clinical and geographically diverse US E. faecium isolates dating from 1971 to 1994, we determined

176 United Kingdom and Ireland (UK&I) and 11 E. faecium isolates from a reference collection.

177 in quinupristin-dalfopristin in Enterococcus faecium isolates from chickens on the Eastern Seaboard,

178 vatE was present in 38% of E. faecium isolates from patients and none from vegetarians

179 stin resistance was found in 51 to 78% of E. faecium isolates from the food production environment.

180 e found primarily in vancomycin-resistant E. faecium isolates in nonstool cultures obtained from pati

181 revious studies showed that some clinical E. faecium isolates produce a cell wall-anchored collagen a

182 Twenty-two percent of E. faecium isolates showed reduced susceptibility to quinup

183 es were frequently present in 30 clinical E. faecium isolates studied; one of these, acm, has been st

184 erococcal isolates and the percentages of E. faecium isolates that were vancomycin resistant.

185 terococcus faecalis, and 88% of Enterococcus faecium isolates were interpreted as susceptible by Etes

186 VR E. faecium isolates were less genetically diverse than vanc

187 All Enterococcus faecalis and Enterococcus faecium isolates were observed to be nonmotile in all me

188 stant Enterococcus faecalis and Enterococcus faecium isolates with a G2576U rRNA mutation.

189 The increasing frequency of Enterococcus faecium isolates with multidrug resistance is a serious

190 occus faecalis isolates, 30 of 58 (51.7%) E. faecium isolates, 1 of 1 E. raffinosus isolate, 0 of 4 E

191 -dalfopristin resistance were assessed in E. faecium isolates, and resistance genes were identified b

192 ium populations, we have now assessed 433 E. faecium isolates, including 264 isolates from human clin

193 pite the detection of acm in 32 out of 32 E. faecium isolates, only 11 of these (all clinical isolate

194 R E. faecalis isolates and none of the VR E. faecium isolates.

195 in most E. faecalis but not in Enterococcus faecium isolates.

196 ecium strain TX82 and all other sequenced E. faecium isolates.

197 n resistance and 1 for nitrofurantoin, in E. faecium isolates.

198 for the reference method, especially for E. faecium isolates.

199 ecies, we found that both E. faecalis and E. faecium kill C. elegans eggs and hatchlings, although on

200 determined the structure of the activated E. faecium LiaR protein at 3.2A resolution and, in combinat

201 A comparison of the E. faecalis LiaR, E. faecium LiaR, and the LiaR homolog from Staphylococcus a

202 nization levels by strains from different E. faecium lineages: clade B, part of the healthy human mic

203 cereus, Enterococcus faecalis, Enterococcus faecium, Listeria monocytogenes, Streptococcus pneumonia

204 resistant, vancomycin-resistant Enterococcus faecium (LRVRE).

205 faecalis (green colonies) from Enterococcus faecium (mauve colonies) on the basis of chromogenic sub

206 Using E. faecium-mediated killing of the nematode worm Caenorhabd

207 c strain overproducing catalase prevented E. faecium-mediated killing.

208 cs against vancomycin-resistant Enterococcus faecium, methicillin-resistant Staphylococcus aureus, an

209 g/mL), and vancomycin-resistant Enterococcus faecium (MIC(50) 2.9 +/- 0.8 microg/mL).

210 ion caused by a DAP-susceptible Enterococcus faecium (minimum inhibitory concentration, 3 microg/mL)

211 c-Lys could lyse the ampicillin-resistant E. faecium mutant with 3-->3 L-Lys(3)-D-Asn-L-Lys(3) bridge

212 Isolated species included Enterococcus faecium (n = 13), Enterococcus faecalis (n = 7), Enteroc

213 hylococcus aureus (n = 14), and Enterococcus faecium (n = 14).

214 olid were highly active against Enterococcus faecium (n = 267) globally (100% and 98% susceptible, re

215 pathogens: vancomycin-resistant Enterococcus faecium (n=19), methicillin-resistant Staphylococcus aur

216 The frequency of E. faecium occurrence increased from 6% in the dry period t

217 gene in 81 cultures containing Enterococcus faecium or E. faecalis was 100% sensitive and specific.

218 ients presented here had either Enterococcus faecium or Enterococcus faecalis bacteremia caused by bo

219 as either vancomycin-resistant Enterococcus faecium or Enterococcus faecalis, based on distinct colo

220 eus, but not Bacillus subtilis, Enterococcus faecium, or Streptococcus pyogenes, kill adult C. elegan

221 s consideration of vancomycin-susceptible E. faecium Our findings reveal the evolution and disseminat

222 he first factor shown to be important for E. faecium pathogenesis.

223 S permease BepA is directly implicated in E. faecium pathogenesis.

224 a secondary binding interaction with the E. faecium peptidoglycan.

225 d-Ala-d-Ala binding pocket still binds to E. faecium peptidoglycan.

226 rated platinum and gold against Enterococcus faecium, platinum against Klebsiella pneumoniae and plat

227 lence of the other 14 genes among various E. faecium populations, we have now assessed 433 E. faecium

228 indicator of toxicity, we determined that E. faecium produces hydrogen peroxide at levels that cause

229 we identify and characterize 6 Enterococcus faecium proteins containing the WxL domain which, by rev

230 in-dalfopristin (Q-D)-resistant Enterococcus faecium (QDREF) isolates were isolated from humans, turk

231 [CI], 6.20-8.76) and vancomycin-resistant E. faecium (rate ratio, 2.27, 95% CI, 2.03-2.53).

232 Enterococcus faecium recently evolved from a generally avirulent comm

233 nsfer colonizing ability to noncolonizing E. faecium recipient strains.

234 The past few years have seen Enterococcus faecium resistance to vancomycin increase from 10% of st

235 nd 3.1 x 10(1) CFU/mL or g of E. coli and E. faecium, respectively) sprout and water samples tested p

236 Deletion of ahyD in E. faecium resulted in increased formation of Ala-PG and Ly

237 significant ESKAPE pathogens: E nterococcus faecium, S taphylococcus aureus, K lebsiella pneumoniae,

238 The E. faecium sagA gene, like the S. mutans homologue, is loca

239 We have shown previously that Enterococcus faecium SagA has broad-spectrum binding to extracellular

240 In conclusion, the extracelluar E. faecium SagA protein is apparently essential for growth,

241 ST6 (n = 3), and ST185 (n = 1), which are E. faecium sequence types belonging to clonal complex 5 (CC

242 emic clones ( E. coli ST131 and Enterococcus faecium ST17) were identified for the first time by mult

243 Enterococcus faecium strain 9631355 was isolated from animal sources

244 coccus pneumoniae pavA, in the genomes of E. faecium strain TX82 and all other sequenced E. faecium i

245 one Campylobacter coli and one Enterococcus faecium strain, while these strains plus three additiona

246 For penicillin, three of five E. faecium strains but none of five Enterococcus faecalis s

247 We previously showed that some E. faecium strains express a cell wall-anchored collagen ad

248 These data suggest that specific E. faecium strains may be enriched in determinants that mak

249 onal acm gene is necessary for binding of E. faecium strains to CI.

250 olates and (10(0) CFU/mL) E. faecalis and E. faecium strains were detected within 4 and 8 h of pre-en

251 We previously showed that E. faecium strains with daptomycin minimum inhibitory conce

252 Overall, our results suggested that E. faecium strains with esp could display lower mobility wi

253 e used cautiously against DAP-susceptible E. faecium strains with minimum inhibitory concentrations >

254 mobility within saturated sand packs than E. faecium strains without esp.

255 e virulence gene associated with epidemic E. faecium strains.

256 es to intestinal colonization in clinical E. faecium strains.

257 esistance development in four out of five E. faecium strains; however, increased resistance was obser

258 ococcus, Enterococcus faecalis, Enterococcus faecium, Streptococcus agalactiae, Escherichia coli, Kle

259 ree collagen-binding clinical isolates of E. faecium tested, but in none of the strains with a non-fu

260 Rectal or fecal samples for E. faecium testing were obtained from 567 newly admitted ho

261 o 22.2% (P < 0.001) and the proportion of E. faecium that was vancomycin resistant increased from 28.

262 is and 4 isolates of vancomycin-resistant E. faecium that were not recovered by BEAV.

263 also show that CcpA affects the growth of E. faecium, that an intact ccpA gene is important for full

264 ry adhesin responsible for the ability of E. faecium to bind collagen.

265 ver, all the Emp pilins are important for E. faecium to cause infection in the urinary tract.

266 tive Acm is sufficient for the binding of E. faecium to CI.

267 TS, significantly impaired the ability of E. faecium to colonize the murine intestinal tract during a

268 nstrate that Fnm affects the adherence of E. faecium to fibronectin and is important in the pathogene

269 acquisition has favored the adaptation of E. faecium to nosocomial environments and/or patients.

270 ance of the nosocomial pathogen Enterococcus faecium to the group A component of natural and semisynt

271 ed surveillance and control of nosocomial E. faecium transmission and infection.

272 We identified E. faecium transposon insertion mutants with altered C. ele

273 urified against these subsegments reduced E. faecium TX2535 strain collagen adherence up to 73 and 50

274 ld decrease (P < 0.05) in the adhesion of E. faecium TX82 to fibronectin.

275 onpiliated DeltaempABC::cat derivative of E. faecium TX82 was attenuated in biofilm formation and in

276 e no reports experimentally demonstrating E. faecium virulence determinants.

277 oniae (6), vancomycin-resistant Enterococcus faecium (VRE FCM) (16), vancomycin-susceptible Enterococ

278 typing 45 vancomycin-resistant Enterococcus faecium (VRE) isolates.

279 us (MRSA), vancomycin-resistant Enterococcus faecium (VRE), and beta-lactam-resistant Klebsiella pneu

280 us (MRSA), vancomycin-resistant Enterococcus faecium (VRE), Escherichia coli SMS-3-5, and Pseudomonas

281 (MRSA) and vancomycin-resistant Enteroccocus faecium (VRE).

282 Vancomycin-resistant Enterococcus faecium (VREF) was isolated from six pigs but not from h

283 (MRSA) and vancomycin-resistant Enterococcus faecium (VREF) with MIC values of 1.4 and 2.2 mug mL(-1)

284 Vancomycin-resistant Enterococcus faecium (VREfm) is a leading cause of nosocomial infecti

285 Vancomycin-resistant Enterococcus faecium (VREfm) is an important cause of healthcare-asso

286 stant Enterococcus faecalis and Enterococcus faecium, VRESelect, was compared to bile esculin azide a

287 llin-imipenem tested with E. faecalis and E. faecium was >/=98% and was 92% for other enterococci; CA

288 E. faecium was isolated from 105 patients, 65 vegetarians,

289 of VR isolates, belonging to esp-positive E. faecium, was revealed.

290 esis of Gram-positive bacteria, including E. faecium We previously demonstrated that a nonpiliated De

291 ction with antibiotic-resistant Enterococcus faecium We used a mouse GIT colonization model to test d

292 cy of quinupristin-dalfopristin-resistant E. faecium, we used selective medium to culture samples fro

293 hen 10(3) or 10(6) cfu/mL of VR Enterococcus faecium were added to the CFCE culture, the VRE were eli

294 ecalis and 24 blood isolates of Enterococcus faecium were compared.

295 ighly related vanA-positive and -negative E. faecium, which implies that control of vancomycin-resist

296 encoded immediately adjacent to rakPGS in E. faecium, which is responsible for the hydrolysis of aa-P

297 he disparity in the transport behavior of E. faecium with and without esp could limit the effectivene

298 ycin use in poultry selects for Enterococcus faecium with cross-resistance to quinupristin-dalfoprist

299 lica sands and thus lower the mobility of E. faecium within sand packs.

300 to investigate the transport of Enterococcus faecium within saturated quartz sands.