ライフサイエンスコーパス: fail to stimulate

1 Monomeric IgG2aj failed to stimulate.

2 ells bearing boVbetaBTB35, which SEC(bovine) failed to stimulate.

3 , HU-210, and Delta(9)-tetrahydrocannabinol, failed to stimulate [(35)S]GTP gamma S binding in CB(1)(

4 yrrolidine dicarboxylate and dihydrokainate) failed to stimulate [3H]D-aspartate efflux but did inhib

5 ctivation of Stat1 and Stat3 (up to 1 h) but fail to stimulate a second phase of response and do not

6 In contrast, POT1 failed to stimulate a bacterial 3'-5'-helicase.

7 anti-NK1.1 inhibited the lytic activity and failed to stimulate a calcium response in cells expressi

8 ound to HLA-DR1, but three of these peptides failed to stimulate a CD4(+) T lymphocyte line which rec

9 lpha-dihydrotestosterone, and dexamethasone, failed to stimulate a change in mRNA levels.

10 of Shigella to the T84-cell apical membrane failed to stimulate a directed PMN transepithelial migra

11 lation of NG2(+) glial cell in older animals failed to stimulate a similar repopulation response, pos

12 In contrast, infection with RESTV failed to stimulate a strong host response in infected m

13 umor cells) remain within brain tissue, thus failing to stimulate a systemic immune response.

14 use the mutant defective in DNA binding also fails to stimulate Abf1 ARS1 DNA-binding activity, our r

15 es, a catalytically defective mutant of SopB failed to stimulate actin cytoskeleton rearrangements an

16 We also found that the agonist pramipexole failed to stimulate activation of Akt in PC12-D2R cells,

17 aidic acid (a trans-isomer of linoleic acid) failed to stimulate adhesion to collagen IV, suggesting

18 CR/ABL mutants deficient in PI-3k activation failed to stimulate Akt kinase, a recently identified PI

19 Treatment of ki/ki platelets with thrombin failed to stimulate Akt phosphorylation, resulting in po

20 insulin could actually induce apoptosis and failed to stimulate Akt/GSK-3beta phosphorylation.

21 Human MSC fail to stimulate allogeneic PBMC or T-cell proliferatio

22 e presence of an anticodon-derived stem-loop failed to stimulate aminoacylation of the minihelix.

23 Although most of the suppressor mutants failed to stimulate amplification of genomes encoding th

24 highest activity and an 11mer linear CpG DNA failed to stimulate an immune response.

25 b was a genuine antagonist of CCR5, since it failed to stimulate an increase in intracellular calcium

26 the RPA mutant lacking the C terminus of p34 failed to stimulate an XPA-DNA interaction, and (ii) the

27 c Northwest of Canada and the United States, fails to stimulate an effective immune response in other

28 ed the ability to bind to RhoA, while K1321A failed to stimulate and to bind to RhoA.

29 but not SLP-76(-/-) platelets (P <.01), and failed to stimulate annexin V binding to either SLP-76(+

30 Immunization with Gal+/+ xenogeneic cells failed to stimulate anti-Gal antibody production in mixe

31 Murine MIP-1 beta, which fails to stimulate any biologic functions in vascular sm

32 Remarkably, BMP4 infusion failed to stimulate aortic NADPH oxidases, increase bloo

33 a and receptor activator of NF-kappaB ligand fail to stimulate AP-1 and NF-kappaB binding to DNA in c

34 es in which whole-cell electrophile flooding fails to stimulate ARE induction prior to causing cytoto

35 ed a mutated, nonfunctional insulin receptor failed to stimulate ARF activation.

36 Serum failed to stimulate association of 14-3-3 with these mut

37 that is defective in nucleolar localization, failed to stimulate ATF5 polyubiquitination and was unab

38 protein dimerization, since monomeric Gal-1 fails to stimulate axonal re-growth.

39 imulate macrophages and dendritic cells, but fail to stimulate B cells.

40 PET-CT in every volunteer, whereas ephedrine failed to stimulate BAT.

41 Furthermore, the NO donors failed to stimulate bile flow in mutant TR- rats in whic

42 ruitment of the MAPK p38delta (SAPK4), while failing to stimulate binding of JNK, the preferred kinas

43 nds nucleic acid; however, protein substrate fails to stimulate binding.

44 ability to transport AI-2 (lsr null mutant) failed to stimulate biofilm formation.

45 o 11,12-EET, because both 8,9- and 14,15-EET failed to stimulate BK channels.

46 defective EDR1 hepatoma cells, dexamethasone failed to stimulate C/EBP alpha and p21 protein expressi

47 atidylserine (dihexanoyl-phosphatidylserine) failed to stimulate C1P transfer.

48 Furthermore, activation of AHR by Way-169916 fails to stimulate canonical DRE-driven AHR-mediated CYP

49 d increase in caspase-3 activity, while IL-1 fails to stimulate caspase-3 activity.

50 The mutant with a replaced AP-1-binding site failed to stimulate CAT expression in an oxidation-sensi

51 t to outer surface protein A, B. burgdorferi failed to stimulate CD14-transfected Chinese hamster ova

52 and MRL lpr/lpr mice, while nonspecific APCs failed to stimulate CD4 T cells.

53 r histocompatibility complex (MHC) antigens, failed to stimulate CD4(+) and CD8(+) T-cell allorespons

54 49) also has RNA triphosphatase activity but fails to stimulate Ceg1 in vitro and is lethal when expr

55 dient but, unlike the wt protein, the mutant failed to stimulate cell migration across a model endoth

56 treatment with the moderate dose of estrogen failed to stimulate cell proliferation, and a decrease i

57 By contrast, cocaine fails to stimulate cell death processes reflecting parth

58 that activation of either chimeric receptor failed to stimulate cellular proliferation.

59 endogenous PKA activity, and phorbol esters failed to stimulate CFTR channels trapped into either th

60 tide (ASBNP) was synthesized and unlike BNP, failed to stimulate cGMP in vascular cells or vasorelax

61 to stimulate cytolytic activity in CTLs also failed to stimulate chemokine release.

62 While IgG1 anti-CD3 MAbs fail to stimulate chimpanzee T cells, IgG2a anti-CD3 MAb

63 C2 (Pro120Ser) fails to bind cholesterol and fails to stimulate cholesterol transfer from NPC1(NTD) t

64 Because the uncaging of Ca2+ fails to stimulate CICR in the absence of cAMP-elevating

65 l(-) current and a Ca(2+)-mobilizing agonist failed to stimulate Cl(-) efflux, requirements for fluid

66 Nuclear-specific overexpression of GSK-3beta failed to stimulate CM differentiation.

67 acterium-infected osteoblasts, peptidoglycan failed to stimulate CXCL10 secretion.

68 ith these residues mutated, GCAP1 completely failed to stimulate cyclase activity but still bound Ret

69 ic domain truncated after the GFFKR sequence failed to stimulate cyclin E/cdk2 activation or entry in

70 The D58G/D65G double mutation also failed to stimulate CYP2C19-catalyzed (S)-mephenytoin 4-

71 und to appropriate HLA class I molecules but failed to stimulate cytolytic activity in CTLs also fail

72 mparable degrees; however, unlike LPS, SMase failed to stimulate detectable interferon activity.

73 However, opsonized C. neoformans failed to stimulate detectable release of interleukin 10

74 FCS1/RFCS2 formed in solution; however, they failed to stimulate DNA synthesis by a cognate DNA polym

75 Alone, poly IC fails to stimulate DNA damage in islets isolated from PK

76 LEF-3 was added prior to the polymerase, it failed to stimulate DNApol replication on a singly prime

77 5-HT also fails to stimulate egg laying in ser-7(tm1325), ser-1(ok

78 HERF-1 null kidney tissue; however, dopamine failed to stimulate either cAMP accumulation or PKC acti

79 al ERK and p38 kinase activity, whereas FMLP failed to stimulate either mitogen-activated protein kin

80 Androgens failed to stimulate either the growth of ebp1 transfecta

81 We now present evidence that taxol fails to stimulate either apoptosis or phosphorylation o

82 metholone ejaculated, oxymetholone generally failed to stimulate ejaculation above the levels of the

83 Moreover, aldosterone fails to stimulate ENaC acutely, suggesting that Ang II

84 ermore, culture supernatants from such cells failed to stimulate endothelial cell growth in vitro.

85 uPAR-expressing cells failed to stimulate engulfment of viable cells, suggesti

86 athophysiology of the disease, BMP2 and BMP4 failed to stimulate eNOS phosphorylation in PAECs isolat

87 Furthermore, BMP2 and BMP4 failed to stimulate eNOS phosphorylation when BMPRII was

88 nt on serum constituents because ERR alpha 1 fails to stimulate eNOS promoter-dependent luciferase ac

89 but signaling-defective EphB1 point mutants failed to stimulate ephrin-B1 dependent attachment.

90 oupling to biological responses, Neuregulin4 fails to stimulate ErbB4 coupling to biological response

91 e CRT null mouse embryonic fibroblasts (MEF) fail to stimulate ERK phosphorylation in response to TSP

92 Direct activation of Epac1 failed to stimulate ERK activity in starved cells, sugge

93 pathway without inducing PTH1R endocytosis, failed to stimulate ERK1/2 activity.

94 -34) (10(-8) m), PTH-(1-31), or 8-bromo-cAMP failed to stimulate ERKs, whereas treatment with phorbol

95 ithin SSB's C terminus produce variants that fail to stimulate ExoI activity, whereas the SSB-Ct pept

96 transgenic flies, this mutant dCAMTA variant failed to stimulate expression of dFbxl4 and rescue the

97 Further, IGF-I failed to stimulate F-actin reorganization and phosphory

98 MC3-R and MC4-R antagonist SHU9119 not only failed to stimulate food intake at the same doses as HS0

99 bertal Tg glands, reduced ERalpha expression failed to stimulate formation of the ERalpha/IRS-1 compl

100 s well as angiotensin II, and phorbol ester, fail to stimulate forward Na+-H+ exchange in adult hyper

101 tly, variants detected at both epitopes also failed to stimulate fresh uncultured cells while index p

102 Insulin failed to stimulate FTase in cells expressing the chimer

103 Although FEN acts via beta(2)-ARs, it fails to stimulate G(i)-/NO signalling and preferentiall

104 h facilitates microsomal G6P uptake by G6PT, fails to stimulate G6P uptake in P(i)-loaded G6PT-proteo

105 r the other cyclase receptors, nevertheless, failed to stimulate GC-G expressed in transient or stabl

106 n and an inhibition of insulin secretion but fails to stimulate glucagon secretion.

107 torage because a kinase-inactive (M721) EGFR failed to stimulate glucose incorporation into glycogen

108 Peripheral injection of NMDA failed to stimulate GnRH/LH release in prepubertal or go

109 Moreover, human and mouse VAT failed to stimulate growth in soft of agar in cells defi

110 spho-CREB, JUN/FOS, GATA-1, Pit-1, and EKLF, failed to stimulate HAT activity.

111 e discovered a mutant form of IE1 (YL2) that fails to stimulate HCMV infection while retaining 30 to

112 In contrast, FacRPA1 fails to stimulate helicase activity to the same extent

113 ctivation of counterregulation, hypoglycemia failed to stimulate hepatic glycogen breakdown or activa

114 Significantly, partial hepatectomy failed to stimulate hepatocytes to proliferate in p21 tr

115 tion is specific because other RecQ homologs fail to stimulate hExo1.

116 on of T cells in spleen cell suspensions but failed to stimulate highly purified T cells unless these

117 In contrast, T. gondii infection failed to stimulate HIV-1 transcription in tissues of tw

118 ontaining the entire human enhancer homology failed to stimulate human renin promoter activity in tra

119 nsity to form helical structures in TFE also failed to stimulate I(SC).

120 e type 2 IL-4R and mimics many IL-4 effects, failed to stimulate IFN-gamma production and, in most ex

121 In PKR null cell lines, pIC failed to stimulate IKK activity compared to cells from

122 (dnTCF4) or HCT116 cells with silenced Snail failed to stimulate IL1beta production in macrophages, d

123 s the B7-CD28 costimulatory pathway, DC that failed to stimulate in primary MLR induced markedly augm

124 ecombinant antigens ESAT-6, MPB83, and MPB64 failed to stimulate in vivo DTH in cattle that had been

125 FN-gamma, for in IL-12-/- mice egg injection fails to stimulate increased production of either of the

126 ed from iPLA2beta-null mice, virus infection fails to stimulate iNOS mRNA accumulation and protein ex

127 pletion, leucine, not alpha-ketoisocaproate, failed to stimulate insulin release.

128 ce of nitrendipine and thapsigargin, glucose failed to stimulate insulin release.

129 m of the non-AM-ester of 8-pCPT-2'-O-Me-cAMP failed to stimulate insulin secretion and was a weak act

130 Pilocarpine also fails to stimulate insulin secretion and, instead, antag

131 Curiously, glycerol fails to stimulate insulin secretion, even though it has

132 persisting isolate LCMV clone 13 (CL13) also failed to stimulate interleukin-6 (IL-6) in macrophages.

133 evoid of ligand-dependent downregulation and failed to stimulate intracellular calcium release, cell

134 IP3 failed to stimulate intracellular or plasma membrane (PM

135 oA mutated so that it cannot be geranylated, failed to stimulate invasion.

136 S-1 (Ser-312) and ERK phosphorylation, IGF-I failed to stimulate IRS-1 (Tyr-612) or Akt phosphorylati

137 as reported that in vitro acetylation of p53 fails to stimulate its DNA binding to large DNA fragment

138 IK potently induced NF-kappaB activation, it failed to stimulate JNK activation.

139 uit Raf-1 to the membrane efficiently and so fail to stimulate kinase activity.

140 Compound 14 failed to stimulate locomotor activity in C57BL/6J mice

141 ddition to cultured RAP-deficient adipocytes failed to stimulate LPL secretion in the medium, suggest

142 ted with dexamethasone (10 microM) for 24 hr failed to stimulate luciferase activity, whereas cells c

143 Whilst ATP alone fails to stimulate LX ligation activity, addition of XLF

144 IFN-gamma itself failed to stimulate lymphocyte proliferation and lymphok

145 e tyrosine kinase Syk, both m1 and m2 mAChRs failed to stimulate MAPK kinase and MAPK.

146 In addition, flagellin-null mutants failed to stimulate matrilysin expression in cultured ce

147 Daily administration of recombinant Epo fails to stimulate melanoma growth in vivo, but the trea

148 timulate NF-kappa B-dependent transcription, failed to stimulate membrane translocation of PKC.

149 At 48 h, infected MPhis (I-MPhis) failed to stimulate MHC-II-restricted T cells but not MH

150 Although activated Rit fails to stimulate mitogen-activated protein kinase/extr

151 Moreover, epidermal growth factor (EGF) failed to stimulate MMP-9 expression, cell invasion, and

152 Although IFN-alpha2a failed to stimulate monocyte cytokine secretion, IFN-kap

153 Further, IGF-I failed to stimulate motility of the cells, in which S6K1

154 since a DEF-1 mutant lacking the GAP domain failed to stimulate motility.

155 based vaccines induced MUC1-specific Abs but failed to stimulate MUC1-specific T cells.

156 Insulin treatment also failed to stimulate muscle cytochrome C oxidase activity

157 stent with an insufficient APC function, HSC failed to stimulate naive OT-II TCR transgenic CD4(+) T

158 over, diabetic-derived Gr-1(+)CD11b(+) cells fail to stimulate neovascularization in vivo and have ab

159 Additionally, a GABA(B)R agonist failed to stimulate neutrophil superoxide burst.

160 or mutants lacking a functional death domain failed to stimulate NF-kappa B, while phosphatidylcholin

161 Lipopolysaccharide failed to stimulate NF-kappaB activation in rip1-/-MyD88

162 In contrast, Tax1 mutant M22, which fails to stimulate NF-kappa B-dependent transcription, f

163 Interestingly, HMGB1 failed to stimulate NFkappaB translocation to the nucleu

164 ctivity and increased surface expression but failed to stimulate NHE3 activity or increase surface ex

165 ha, interferon-gamma, and lipopolysaccharide fail to stimulate nitric oxide formation by RINm5F cells

166 AT(2) antagonist PD123319 (1 microm), Ang II failed to stimulate NO (0.1 +/- 0.1 fluorescence units/m

167 ansduced with dominant negative Akt1, Ang II failed to stimulate NO (0.1 +/- 0.2 fluorescence units/m

168 luorescence units/min (p < 0.001; n = 5) but failed to stimulate NO production in THALs isolated from

169 eted FGFR1 (TK-), which did not bind to RSK1 failed to stimulate nuclear RSK1 activity or RSK1 activa

170 In contrast, the kinase inhibitor Lapatinib fails to stimulate nuclear accumulation of the receptor

171 Ang II failed to stimulate O. in TALs from p47(phox) -/- mice (

172 pressing dominant-negative PKC alpha, Ang II failed to stimulate O2.

173 r-tyrosyl phosphorylation of FRS-2 alpha but fails to stimulate or potentiate either FGF-2-induced Er

174 were overexpressed in medium with salt, urea failed to stimulate osmotic stress response.

175 wever, overexpression of C/EBPalpha or c-Fos failed to stimulate osteoclastogenesis in the mutant cel

176 A mutant CBP lacking the N terminus failed to stimulate p53-dependent transactivation.

177 M/ATR kinases and the phosphorylation mutant fails to stimulate p53 ubiquitination.

178 This mutant failed to stimulate PAK and JNK activity but still induc

179 ilar approach, we demonstrate that cytokines fail to stimulate peroxynitrite generation by rat islets

180 5-HT fails to stimulate pharyngeal pumping and the firing of

181 Moreover, Tsc1 deletion failed to stimulate phospho-Ser-302 or other putative S6

182 established criteria for a PITP in vitro and fails to stimulate phosphoinositide production in vivo.

183 GH failed to stimulate phosphorylation or activation of Jun

184 of 1,25(OH)2D3 to isolated colonic membranes failed to stimulate PI hydrolysis, but required secoster

185 Integrin alpha2beta1 failed to stimulate PI3Kbeta in platelets from phospholi

186 uces expression of MSH in keratinocytes, but fails to stimulate pigmentation in the absence of functi

187 3-silenced (Sh-Pdia3) cells, 1,25(OH)(2)D(3) failed to stimulate PKC and PGE(2) responses; in Pdia3-o

188 Furthermore, ET-1 failed to stimulate PLA2 activity measured in the cytoso

189 the effects, as a mutant lacking this region failed to stimulate plasminogen activation, although a s

190 mediated inhibition of adenylyl cyclase, but failed to stimulate PLC activity as did wild-type SSTR2.

191 the mutagenesis-defective pol30-113 mutant, fails to stimulate Pol zeta(4) activity, providing an ex

192 Finally, TZDs failed to stimulate PPARgamma activation and adipocyte d

193 Heat-inactivated C. pneumoniae failed to stimulate production of these proteins by all

194 specific inhibition of EGFR or MEK, and GRP failed to stimulate proliferation in EGFR-deficient cell

195 DCs from MB49-bearing female mice fail to stimulate proliferative and IFN-gamma-producing

196 oocytes and embryos revealed that enhancers failed to stimulate promoters prior to formation of a tw

197 biosis associated with sensitization to food fails to stimulate protective tolerogenic pathways, lead

198 A "kinase-dead" EGFR failed to stimulate Rab5a function.

199 in RFS-1, which abolish filament remodeling, fail to stimulate RAD-51 strand exchange activity, demon

200 BRC-2 alone, lacking the DNA-binding domain, fail to stimulate RAD-51-mediated D-loop formation and d

201 A binding and tetramerization mutants of Rta fail to stimulate RBP-Jk DNA binding.

202 F. novicida and F. tularensis subspecies failed to stimulate reactive oxygen species production i

203 Consistent with this, Ang II failed to stimulate renal medullary prostaglandin E(2) a

204 Hyperglycemia failed to stimulate Rho-kinase activity and PAI-1 expres

205 hoGEF and LARG (leukemia-associated RhoGEF), fails to stimulate Rho-dependent transcriptional activat

206 e depletion also increases SGK1 activity but fails to stimulate ROMK channels and K secretion.

207 A) bound effectively to nuclear RSK1, but it failed to stimulate RSK1.

208 tivated ras mutant in Balb/c-3T3 fibroblasts failed to stimulate S phase entry in the absence of plas

209 sence of heregulin, elevation of cAMP levels failed to stimulate Schwann cell proliferation.

210 e previously shown that viable F. tularensis fails to stimulate secretion of proinflammatory cytokine

211 In macrophages from MyD88(-/-) mice, RBP4 fails to stimulate secretion of tumor necrosis factor, I

212 The ligand-coated 6-micron beads also fail to stimulate significant degranulation of RBL-2H3 c

213 show that maximally activated P2X1 receptors failed to stimulate significant aggregation but could am

214 The 0.1-mg simvastatin dose failed to stimulate significant bone growth.

215 Infusion of insulin alone or TGFalpha alone failed to stimulate significant DNA synthesis.

216 is incubated with serum and then washed also failed to stimulate significant TNF-alpha production by

217 ologous single-stranded DNA-binding proteins fail to stimulate similarly the helicase activity of BLM

218 1 were protected from CTL-mediated lysis and failed to stimulate specific memory T-cell responses to

219 Cytokines, however, failed to stimulate sphingomyelin metabolism.

220 However, the complex fails to stimulate splicing activity.

221 ogenic glycoprotein tumor Ags, such as MUC1, fail to stimulate strong immune responses.

222 p38 activity, and heterotypic cross-linking failed to stimulate synergistic activation of either ERK

223 ranscription 3 (STAT3) signaling because gAd failed to stimulate system A in cells in which STAT3 had

224 three NS3358-375 and one NS3505-521 variants failed to stimulate T cell proliferation, and two other

225 eptide was unable to bind I-Ak molecules and failed to stimulate T cells in the absence of intracellu

226 Immature DCs in healthy arteries failed to stimulate T cells, but DCs in PMR arteries cou

227 In MAPTA-loaded hepatocytes, CPT-cAMP failed to stimulate TC uptake, failed to increase cytoso

228 The gamma W72A mutant failed to stimulate TFIID-DNA binding (D-A complex) but

229 s II-restricted altered peptide ligands that fail to stimulate the circulating T lymphocyte repertoir

230 urons in all three areas in intact males but fail to stimulate the magnocellular division of the medi

231 nt antigens arise in HIV-1(+) patients which fail to stimulate the T cell antigen receptor of HLA cla

232 In the Uvhog1 mutant, NaCl treatment failed to stimulate the accumulation of sorbitol and gly

233 In contrast, anti-CD3 antibody failed to stimulate the binding of F-HA in Jurkat transf

234 -deficient cell line in which phorbol esters failed to stimulate the diffusion of integrin.

235 -dependent increase in intracellular calcium failed to stimulate the extracellular signal-regulated p

236 a-subunit gene expression 3-fold, whereas it failed to stimulate the gene cyclooxygenase-2, which was

237 Endothelin-3 failed to stimulate the incorporation of [3H]thymidine o

238 Unlike Tat, NF-kappaB failed to stimulate the integrated transcriptionally sil

239 to induce macrophage proteinase expression, failed to stimulate the phosphorylation of MAPK(erk1/2).

240 hed to fibronectin-coated dishes, calcitonin failed to stimulate the phosphorylation of paxillin and

241 tor, were added to cultured macrophages, but failed to stimulate the production of macrophage inflamm

242 However, activated Rac in non-adherent cells failed to stimulate the Rac effector PAK.

243 l activity of an NFAT-IL-2 reporter gene, it failed to stimulate the transcriptional or DNA-binding a

244 In addition, ZnG UvrA failed to stimulate the UvrB DNA damage-associated ATPas

245 nt with familial platelet disorder with AML, fails to stimulate the ELA2 promoter in vitro, and bone

246 dc2 in granule neurons, activity deprivation fails to stimulate the expression of E2F-target genes th

247 In the absence of p38 activity, EGF fails to stimulate the ubiquitin ligase Cbl or ubiquitin

248 t mammary (Rama) 27 fibroblasts, although it failed to stimulate their proliferation.

249 activity in antisense expressing clones, but failed to stimulate their proliferation.

250 However, the TrkC kinase insert forms fail to stimulate these events.

251 omes from miR-214-depleted endothelial cells failed to stimulate these processes.

252 These two beta-glucans failed to stimulate TNF-alpha in Dectin-1 (beta-glucan r

253 ut is unable to bind topoisomerase IIIalpha, fails to stimulate topoisomerase activity.

254 , since other single strand binding proteins failed to stimulate transfer.

255 e exponential replication over time, Schu S4 failed to stimulate transforming growth factor beta, int

256 2+) mobilization and ERK phosphorylation but failed to stimulate TrkA phosphorylation, NFAT activatio

257 omains from other receptors, EGFR and FGFR1, failed to stimulate TrkB phosphorylation.

258 lutathione S-transferase-TRP in solution but failed to stimulate TRP binding to DNA.

259 Agrin, however, fails to stimulate tyrosine phosphorylation of MuSK that

260 timulation of JAB1 siRNA-transfected RA FLSs failed to stimulate ubiquitination of TRAF2.

261 iophage T4 gene 32 protein (gp32) completely failed to stimulate WRN helicase to unwind long DNA dupl

262 receptor (IGF1R) but, in the absence of FSH, fails to stimulate YXXM phosphorylation of IRS1 (insulin