ライフサイエンスコーパス: failure to thrive

1 pmental delay, progressive microcephaly, and failure to thrive).

2 l heart disease, skeletal abnormalities, and failure to thrive.

3 rized by neonatal-onset watery diarrhoea and failure to thrive.

4 rrent fevers, interstitial lung disease, and failure to thrive.

5 ein-losing enteropathy, hypoalbuminemia, and failure to thrive.

6 lor, and lethargy; chronic FPIES can lead to failure to thrive.

7 ading to irreversible kidney dysfunction and failure to thrive.

8 to deteriorated eczema, severe diarrhea and failure to thrive.

9 ated with severe neurological regression and failure to thrive.

10 , moderate to severe developmental delay and failure to thrive.

11 pecially if accompanied by systemic signs of failure to thrive.

12 These defects result primarily in failure to thrive.

13 trolyte imbalance, recurrent infections, and failure to thrive.

14 y P14, when they began exhibiting ataxia and failure to thrive.

15 us cause severe Cbl deficiency and the noted failure to thrive.

16 al retardation, conductive hearing loss, and failure to thrive.

17 2 yr of age, who presented with vomiting or failure to thrive.

18 mes that conspire to create the phenotype of failure to thrive.

19 Classical symptoms of CS patients include failure to thrive and a severe neuropathology characteri

20 This multisystem disorder causes failure to thrive and accelerated atherosclerosis leadin

21 ions and gastrointestinal disorders, such as failure to thrive and delayed gastric emptying, together

22 gastrointestinal (GI) disease because of the failure to thrive and early death from malnutrition in i

23 mice lacking alpha but not beta suffer from failure to thrive and early mortality.

24 alities, hypotonia, intellectual disability, failure to thrive and feeding problems.

25 y different; one presented in childhood with failure to thrive and hypertrophic cardiomyopathy, and t

26 as severe psychomotor retardation, seizures, failure to thrive and intolerance to wheat and dairy pro

27 beginning at postnatal day 5 with subsequent failure to thrive and median survival of 35 d.

28 usually presents in the newborn period with failure to thrive and metabolic crisis leading to coma o

29 This paper examines the medical etymology of failure to thrive and proposes a rational approach to ev

30 ile hypotonia, and irritability, followed by failure to thrive and short stature.

31 ons are common and include chronic diarrhea, failure to thrive, and abdominal distention; however, ex

32 ce, a substantial attenuation in the initial failure to thrive, and an increase in life span.

33 resulting in sparse hair, male infertility, failure to thrive, and hydrocephaly, the anemia is the f

34 We describe PAP, failure to thrive, and increased GM-CSF levels in two si

35 n and thereby result in persistent acidosis, failure to thrive, and nephrocalcinosis.

36 delirium, dehydration, falls and fractures, failure to thrive, and pressure ulcers.

37 al mutilation, cerebral leukoencephalopathy, failure to thrive, and recurrent metabolic acidosis with

38 d Mut(ko/ki) mice survive post-weaning, show failure to thrive, and show increased methylmalonic acid

39 sy including infantile spasms, irritability, failure to thrive, and stereotypic hand movements.

40 ep apnea in infants has been associated with failure to thrive, behavioral deficits, and sudden infan

41 r, neutrophilic dermatitis/panniculitis, and failure to thrive, but without obvious primary immunodef

42 perimental group, 11 of 11 mice demonstrated failure to thrive by day 13; 5 deaths occurred between d

43 multiple organ systems, encompassing severe failure to thrive, cardiac anomalies including hypertrop

44 f VEGF is associated with (1) a high rate of failure to thrive/death and (2) formation of endothelial

45 We describe a new case with failure to thrive, developmental delay, lactic acidosis

46 A2 who exhibited global developmental delay, failure to thrive, dilated cardiomyopathy and epilepsy,

47 uterine growth retardation, dehydration, and failure to thrive due to a lack of normal insulin secret

48 spectrum disorder, developmental regression, failure-to-thrive, exercise intolerance/fatigue) was ass

49 Elevated sweat chloride levels, failure to thrive (FTT), and lung disease are characteri

50 Infected mice had a relative failure to thrive, gaining weight significantly more slo

51 idge-Ropers syndrome (BRS), characterized by failure to thrive, global developmental delay, feeding p

52 F1 gene in a 2-year-old girl presenting with failure to thrive, global neurodevelopmental regression,

53 premature aging, including low birth weight, failure to thrive, graying and loss of hair, reduced ski

54 Despite this heterogeneity, failure to thrive has had its own international Classifi

55 r that presents during infancy, resulting in failure to thrive, hepatomegaly, and hepatic failure, an

56 Tat expression in the brain caused failure to thrive, hunched gesture, tremor, ataxia, and

57 nine XSCID share similar features, such as a failure to thrive, hypogammaglobulinemia, an absent T ce

58 use an early onset multisystem disorder with failure to thrive, immunodeficiency and neurological sym

59 peatedly cited as prevalent in patients with failure to thrive: impaired physical functioning, malnut

60 PWS is characterized by severe hypotonia, a failure to thrive in infancy and, on emerging from infan

61 onatal respiratory depression, hypotonia and failure to thrive in infancy, followed by hyperphagia an

62 racterized by severe fat malabsorption and a failure to thrive in infancy.

63 role in inflammation and contributes to the failure to thrive in this mouse model of CF.

64 ter of these infants meet the definition of 'failure to thrive' in the first year of life.

65 The term "failure to thrive" is frequently used to describe older

66 sorder resulting in seizures, hypotonia, and failure to thrive, is due to inherited loss-of-function

67 cal form of celiac disease, characterized by failure to thrive, is still the most frequent presentati

68 uited for investigating nutritionally based "failure-to-thrive" issues, particularly regarding the lo

69 ylation imprints at Ndn and Mkrn3 and suffer failure to thrive leading to a fully penetrant neonatal

70 the clinical presentation of CD can include failure to thrive, malnutrition, and distension in juven

71 en with SMS and include infantile hypotonia, failure to thrive, mental retardation, autistic features

72 on of patients with PYCR2 mutations included failure to thrive, microcephaly, craniofacial dysmorphis

73 ons and intra-abdominal abscess (n = 75) and failure to thrive (n = 38).

74 ment were cerebrovascular accident (n = 80), failure to thrive (n = 71), other central nervous system

75 ), rectal haemorrhage (n=1), dyspnoea (n=1), failure to thrive (n=1), and interstitial lung disease (

76 endently increased for comorbid diagnoses of failure to thrive, neurodevelopmental delay, cardiopulmo

77 mb2(-/-) mice are responsible for the severe failure to thrive phenotype, and that renal replacement

78 ia, hypokalemic alkalosis, low birth weight, failure to thrive, poor growth, and in many cases nephro

79 mmon problem which can manifest as vomiting, failure to thrive, recurrent pneumonias, asthma, sinusit

80 PTLS is characterized by hypotonia, failure to thrive, reduced body weight, intellectual dis

81 onates had severe malabsorptive diarrhea and failure to thrive, required prolonged parenteral nutriti

82 ive fumaric aciduria in early childhood with failure to thrive, seizures, developmental delay, mental

83 th -3.4 SDS; weight -2.1 SDS) presented with failure to thrive, short stature, severe hypocalcemia an

84 age of one with severe infantile-onset IBD, failure to thrive, skin rash, and perirectal abscesses r

85 t a valid diagnostic entity, but encompasses failure to thrive, stunting secondary to chronic malnutr

86 the mouse p locus that is associated with a failure-to-thrive syndrome, in addition to diminished pi

87 Live-born FX(-/-) mice exhibit postnatal failure to thrive that is suppressed with a fucose-suppl

88 op a neurological phenotype characterized by failure to thrive, tremor, and gait ataxia.

89 ma, hypotrichosis, severe nail dystrophy and failure to thrive, two heterozygous mutations in ABCA12

90 ma, hypotrichosis, severe nail dystrophy and failure to thrive, two heterozygous mutations in ABCA12

91 s characterized by severe hypercalcemia with failure to thrive, vomiting, dehydration, and nephrocalc

92 We describe three infants, two with failure to thrive, who had dehydration and diarrhea with

93 re normal at birth but exhibited progressive failure to thrive, whole-body wasting, and ataxia and di