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1 ently, allelic loss) occur in tumors in FAP (familial adenomatous polyposis).
2 ects had pathogenic variants associated with familial adenomatous polyposis.
3 sporadic tumors or in tumors associated with familial adenomatous polyposis.
4 the development of adenomas in subjects with familial adenomatous polyposis.
5 osis coli gene (APC) that is associated with familial adenomatous polyposis.
6 size 5 mm; range 1-13 mm) from patients with familial adenomatous polyposis.
7 fr) signaling on the Apc(Min) mouse model of familial adenomatous polyposis.
8 s in the treatment of ulcerative colitis and familial adenomatous polyposis.
9 on/activity in the Apc(Min/+) mouse model of familial adenomatous polyposis.
10 lastic role in the Apc(Min/+) mouse model of familial adenomatous polyposis.
11 (APC) allele can lead to the development of familial adenomatous polyposis.
12 rcinoma and causes regression of adenomas in familial adenomatous polyposis.
13 PAA), 1 had indeterminate colitis, and 8 had familial adenomatous polyposis.
14 mas, but do not have the florid phenotype of familial adenomatous polyposis.
15 bitor, on colorectal polyps in patients with familial adenomatous polyposis.
16 mber and size of adenomas in mouse models of familial adenomatous polyposis.
17 osses with ApcMin/+, a murine model of human familial adenomatous polyposis.
18 ired biopsies from IPAA patients with UC and familial adenomatous polyposis.
19 testinal tumor formation in a mouse model of familial adenomatous polyposis.
20 ous polyposis coli (APC) gene that underlies familial adenomatous polyposis.
21 urrence of colonic adenomas in patients with familial adenomatous polyposis.
22 the adenoma phenotype in this mouse model of familial adenomatous polyposis.
23 e number and size of polyps in patients with familial adenomatous polyposis.
24 ange, which are similar to those observed in familial adenomatous polyposis.
25 oli (APC) is associated with pathogenesis of familial adenomatous polyposis.
26 the leading causes of death in patients with familial adenomatous polyposis.
27 r interact with Apc, are important models of familial adenomatous polyposis.
28 al adenoma development in an animal model of familial adenomatous polyposis.
29 ally or in combination in an animal model of familial adenomatous polyposis.
30 Min mice provide a good model of human familial adenomatous polyposis.
31 Wnt pathway or APC gene mutations that cause familial adenomatous polyposis.
32 for the treatment of ulcerative colitis and familial adenomatous polyposis.
33 mphocyte depletion observed in this model of familial adenomatous polyposis.
34 that of patients with classic or attenuated familial adenomatous polyposis.
35 d several cancers such as retinoblastoma and familial adenomatous polyposis.
36 Diagnosis (chronic ulcerative colitis 97%, familial adenomatous polyposis 3%) and previous operativ
37 ugh studies of hereditary syndromes, such as familial adenomatous polyposis, a cancer predisposition
38 smaller median number of polyps than typical familial adenomatous polyposis, a wide variability in po
39 re also detected in APC(min) mouse and human familial adenomatous polyposis adenomas compared with no
41 hers syndrome and the phenotypic features of familial adenomatous polyposis aid significantly in synd
42 r protein, germline mutation of which causes familial adenomatous polyposis, an autosomal intestinal
46 ajor syndromes related to colorectal cancer, familial adenomatous polyposis and hereditary non-polypo
47 al dominant hereditary forms of the disease, familial adenomatous polyposis and hereditary nonpolypos
48 ion of preexisting adenomas in patients with familial adenomatous polyposis and significantly inhibit
50 lasia type 2, von Hippel-Lindau disease, and familial adenomatous polyposis are examples of syndromes
51 The loss of APC function is observed in most familial adenomatous polyposis-associated and sporadic c
53 years) who were genotypically affected with familial adenomatous polyposis but phenotypically unaffe
54 y of 41 subjects genotypically affected with familial adenomatous polyposis but phenotypically unaffe
55 benefit not only in patients diagnosed with familial adenomatous polyposis but, perhaps, in other pr
56 ectomy is required for ulcerative colitis or familial adenomatous polyposis, but long-term data on fu
57 al polyp in animals and in patients with the familial adenomatous polyposis by downregulating beta-ca
58 zygote Min/+ mouse model, like patients with familial adenomatous polyposis, carries a nonsense mutat
59 the inherited autosomal dominant condition, familial adenomatous polyposis, celecoxib proved efficac
61 ly recognized as a potential complication of familial adenomatous polyposis coli (APC) and attenuated
65 cancer, adenomas taken from individuals with familial adenomatous polyposis contain high levels of Ct
66 Colectomy specimens from 3 patients with familial adenomatous polyposis containing polypoid and n
68 ncer from human inherited syndromes, such as familial adenomatous polyposis (FAP) and hereditary non-
70 orders predisposing to colorectal cancer are familial adenomatous polyposis (FAP) and hereditary nonp
71 e patients who had undergone colectomies for familial adenomatous polyposis (FAP) and specimens from
72 rmines the type of the 'second hit', both in familial adenomatous polyposis (FAP) and sporadic colore
73 tween phenotypic expression in patients with familial adenomatous polyposis (FAP) and the site of mut
74 n the indications of MMVTx for patients with familial adenomatous polyposis (FAP) and the technical f
76 denomatous polyposis coli (APC) mutations or familial adenomatous polyposis (FAP) are born with norma
78 attern dramatically altered for the worse in familial adenomatous polyposis (FAP) because these patie
80 riginally identified as a candidate gene for familial adenomatous polyposis (FAP) but further study i
81 These tumors often occur in patients with familial adenomatous polyposis (FAP) coli who have germ
82 were ulcerative colitis (UC) in 37 patients, familial adenomatous polyposis (FAP) in 12 patients, and
83 incidence of thyroid cancer in patients with Familial adenomatous polyposis (FAP) in a prospective st
89 the familial cancer predispositions known as familial adenomatous polyposis (FAP) or Gardner syndrome
90 tudied methylation in ACF from patients with familial adenomatous polyposis (FAP) or sporadic colorec
91 tations in colonic and duodenal tumours from familial adenomatous polyposis (FAP) patients has shown
92 of the earliest colonic tissue alteration in familial adenomatous polyposis (FAP) patients, we presen
95 commercial availability of gene testing for familial adenomatous polyposis (FAP) represents an impor
96 a pancreatoblastoma in an adult patient with familial adenomatous polyposis (FAP) suggests that they
97 mucosa from an XO/XY mosaic individual with familial adenomatous polyposis (FAP) was examined direct
98 tions in APC are classically associated with familial adenomatous polyposis (FAP), a highly penetrant
99 in, germline mutation of which characterizes familial adenomatous polyposis (FAP), an autosomal intes
100 in Adenomatous polyposis coli (APC) underlie familial adenomatous polyposis (FAP), an inherited cance
101 l ulcerative colitis, indeterminate colitis, familial adenomatous polyposis (FAP), and a select group
102 ssor gene APC are associated with hereditary familial adenomatous polyposis (FAP), and somatic mutati
103 tary nonpolyposis colorectal cancer (HNPCC), familial adenomatous polyposis (FAP), and sporadic CRC.
105 arbor an inherited predisposition, including familial adenomatous polyposis (FAP), hereditary nonpoly
106 e frequently present in FGPs associated with familial adenomatous polyposis (FAP), raising the possib
107 m, combinatorial chemoprevention efficacy in familial adenomatous polyposis (FAP), signal of benefit
108 y (Family N) with symptoms characteristic of familial adenomatous polyposis (FAP), which is typically
109 way is a hallmark of many cancers, including familial adenomatous polyposis (FAP)-related desmoid tum
123 strategy to iPSCs derived from patients with familial adenomatous polyposis (FAP-iPSCs) harboring ger
126 history of polyp formation in patients with familial adenomatous polyposis has provided a benchmark
127 ereditary nonpolyposis colorectal cancer and familial adenomatous polyposis has provided insight into
129 ition, clinical studies of the human disease familial adenomatous polyposis have demonstrated the eff
130 olyposis coli gene, which is responsible for familial adenomatous polyposis, have been described in p
131 ystic Fibrosis, Duchenne Muscular Dystrophy, Familial Adenomatous Polyposis, Hereditary Non-polyposis
132 romes such as multiple endocrine neoplasias, familial adenomatous polyposis, hereditary nonpolyposis
135 Celecoxib is prescribed for the treatment of familial adenomatous polyposis in humans, and the CEA-ba
136 amilial adenomatous polyposis mimics typical familial adenomatous polyposis in some cases but in othe
137 This investigation shows that attenuated familial adenomatous polyposis in the kindreds examined
138 ents with sporadic colorectal carcinomas and familial adenomatous polyposis, in 18 sporadic HPs from
139 colon adenomas recovered from patients with familial adenomatous polyposis, including adenomas as sm
140 ereditary nonpolyposis colorectal cancer and familial adenomatous polyposis, indicates that mutations
145 testing on 810 individuals from 2 attenuated familial adenomatous polyposis kindreds harboring an ide
147 herited syndromes, including Lynch syndrome, familial adenomatous polyposis, MUTYH-associated polypos
148 ets in intestinal tissues from patients with familial adenomatous polyposis (n = 18) or sessile serra
149 adenomas (n = 18), adenomas associated with familial adenomatous polyposis (n = 7), hyperplastic pol
152 d IPAA procedures for ulcerative colitis and familial adenomatous polyposis, of which all but 2 were
153 Patients who underwent genetic tests for familial adenomatous polyposis often received inadequate
154 lear-cut inherited Mendelian traits, such as familial adenomatous polyposis or hereditary nonpolyposi
155 uished clinically from cases with attenuated familial adenomatous polyposis or MUTYH-associated polyp
156 ested, 83.0 percent had clinical features of familial adenomatous polyposis or were at risk for the d
158 ation of the premalignant crypt phenotype in familial adenomatous polyposis patients, the hypothesis
165 rectal adenomas in animals and patients with familial adenomatous polyposis suggest that COX-2 inhibi
166 r at increased frequency among patients with familial adenomatous polyposis, suggesting that alterati
170 in the majority of the 30% of patients with familial adenomatous polyposis that do not test positive
171 genetic diseases, three females (1.6 %) had familial adenomatous polyposis; three patients (1 male a
172 th mutation-negative Lynch syndrome, 16 with familial adenomatous polyposis, two with constitutional
173 polyps from a 16-year-old male patient with familial adenomatous polyposis was insufflated, submerge
174 sis sulindac sulfide, which is used to treat familial adenomatous polyposis, was selected to detect g
175 for patients with indeterminate colitis and familial adenomatous polyposis were inconclusive because
176 intestinal neoplasia mice (a model for human familial adenomatous polyposis) were crossed with cyclin
178 patients with inflammatory bowel disease or familial adenomatous polyposis who underwent IAPT by a s
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