ライフサイエンスコーパス: fasciitis

1 tening invasive disease, such as necrotizing fasciitis.

2 of diseases from pharyngitis to necrotizing fasciitis.

3 s; average, 49.9 years) with chronic plantar fasciitis.

4 fibrils but a severely destructive monocytic fasciitis.

5 for previous cases of C albicans necrotizing fasciitis.

6 d invasive infections, including necrotizing fasciitis.

7 e model of group A streptococcal necrotizing fasciitis.

8 d organ failure, with or without necrotizing fasciitis.

9 ction in a murine model of human necrotizing fasciitis.

10 valuable in assessing suspected necrotizing fasciitis.

11 (ABC), and the related benign lesion nodular fasciitis.

12 e in a nonhuman primate model of necrotizing fasciitis.

13 ed virulence in a mouse model of necrotizing fasciitis.

14 ococcal toxic shock syndrome and necrotizing fasciitis.

15 sses ranging from pharyngitis to necrotizing fasciitis.

16 an primate experimental model of necrotizing fasciitis.

17 such as toxic shock syndrome and necrotizing fasciitis.

18 ce phenotype in a mouse model of necrotizing fasciitis.

19 g invasive infections, including necrotizing fasciitis.

20 compare them with patients with necrotizing fasciitis.

21 animal models of pharyngitis and necrotizing fasciitis.

22 human primates with experimental necrotizing fasciitis.

23 ed virulence in a mouse model of necrotizing fasciitis.

24 literature on this rare cause of necrotizing fasciitis.

25 ce phenotype in a mouse model of necrotizing fasciitis.

26 e-threatening infections such as necrotizing fasciitis.

27 sed with caution in the diagnosis of plantar fasciitis.

28 rimate models of pharyngitis and necrotizing fasciitis.

29 s anginosus constellatus causing necrotizing fasciitis.

30 mary isolate from a patient with necrotizing fasciitis.

31 ess virulent in a mouse model of necrotizing fasciitis.

32 ning invasive infections such as necrotizing fasciitis.

33 ns is an extremely rare cause of necrotizing fasciitis.

34 severely ill than patients with necrotizing fasciitis.

35 irulence factors in our model of necrotizing fasciitis.

36 host interactions underlying GAS necrotizing fasciitis.

37 e-threatening infections such as necrotizing fasciitis.

38 he molecular pathogenesis of GAS necrotizing fasciitis.

39 A) is a rarely reported cause of necrotizing fasciitis.

40 ), and subsequent development of necrotizing fasciitis (1 of 8).

41 e were performed on 28 patients with plantar fasciitis; 17 had spondylarthropathy (SpA)-associated di

42 comorbidities than patients with necrotizing fasciitis (20 [87.0%] vs 17 [54.8%]; P = .02), especiall

43 with cellulitis vs patients with necrotizing fasciitis (3 [2-5] vs 5 [3-11]; P = .01), while median (

44 unger and had a higher degree of necrotizing fasciitis (56% vs 14%).

45 h toxic shock syndrome (55%) and necrotizing fasciitis (58%).

46 This review highlights three areas: plantar fasciitis, Achilles tendinitis, and carpal tunnel syndro

47 rep throat) to severely invasive necrotizing fasciitis (also known as the flesh-eating syndrome).

48 Necrotizing fasciitis, also known as the flesh-eating disease, is a

49 deviation, 46.3 years +/- 8.7) with plantar fasciitis and 50 feet of 50 asymptomatic volunteers (27

50 in a high clinical suspicion for necrotizing fasciitis and distinguish it from more common forms of c

51 riage strain in a mouse model of necrotizing fasciitis and had enhanced growth ex vivo in human blood

52 Necrotizing fasciitis and myonecrosis caused by invasive infection w

53 eptococcal toxic shock syndrome, necrotizing fasciitis and myositis, and the post-infection sequelae

54 individuals with TRAPS is due to a monocytic fasciitis and not to myositis.

55 he role of SpyCEP in S. pyogenes necrotizing fasciitis and respiratory tract infection in mice using

56 fibroblasts from patients with eosinophilic fasciitis and scleromyxedema were studied as well as PBM

57 vere invasive infections such as necrotizing fasciitis and streptococcal toxic shock syndrome.

58 a variety of diseases, including necrotizing fasciitis and toxic shock syndrome, were analyzed.

59 sive human infections, including necrotizing fasciitis and toxic shock syndrome.

60 e infections in humans including necrotizing fasciitis and toxic shock syndrome.

61 h invasive infections, including necrotizing fasciitis and toxic shock syndrome.

62 etigo to severe outcomes such as necrotizing fasciitis and toxic shock syndrome.

63 infections worldwide, including necrotizing fasciitis and toxic shock syndrome.

64 o invasive conditions, including necrotizing fasciitis and toxic shock syndrome.

65 n infections to life-threatening necrotizing fasciitis and toxic shock syndrome.

66 ve infections, including sepsis, necrotizing fasciitis and toxic shock.

67 specific association between GAS necrotising fasciitis and trauma.

68 that causes both invasive (e.g., necrotizing fasciitis) and noninvasive (e.g., pharyngitis) diseases.

69 kness, clostridial gas gangrene, necrotizing fasciitis, and acute crush injury.

70 carditis, necrotizing pneumonia, necrotizing fasciitis, and septicemia.

71 coccal bacteremia, myositis, and necrotizing fasciitis are presented and compared with those of Strep

72 The diagnosis and treatment of plantar fasciitis are reviewed; nonsurgical treatments remain th

73 toxic shock syndrome (STSS) and necrotizing fasciitis are the 2 most severe invasive manifestations

74 itis, impetigo, toxic shock, and necrotizing fasciitis, as well as the postinfection sequelae rheumat

75 as been a very uncommon cause of necrotizing fasciitis, but we have recently noted an alarming number

76 ed protease activity and reduced necrotizing fasciitis capacity.

77 ificantly underrepresented among necrotizing fasciitis cases has a unique frameshift mutation that tr

78 Necrotizing fasciitis caused by C albicans is extremely rare in the

79 Necrotizing fasciitis caused by community-associated MRSA is an emer

80 coccal toxic shock syndrome, and necrotizing fasciitis) caused by six distinct M types of GAS serocon

81 lysis of primary clinical samples of nodular fasciitis confirmed the activation of a Jak1-STAT3 gene

82 developed into rapidly spreading necrotizing fasciitis despite antimicrobial therapy and surgical deb

83 ing a cascade of events, resulting in severe fasciitis, destruction of tissues, and subsequent rheuma

84 two cases of fatal monomicrobial necrotizing fasciitis due to Acinetobacter baumannii, an unusual fin

85 Necrotising fasciitis due to invasive group A streptococcus (iGAS) i

86 Necrotizing fasciitis due to Vibrio vulnificus may result in overwhe

87 Importance: Eosinophilic fasciitis (EF) is a connective tissue disorder in which

88 om pharyngitis (strep throat) to necrotizing fasciitis (flesh-eating disease).

89 sease, including pharyngitis and necrotizing fasciitis (flesh-eating syndrome).

90 associated with decreased human necrotizing fasciitis ("flesh-eating disease").

91 ssue provides a clinical overview of plantar fasciitis focusing on prevention, diagnosis, treatment,

92 cent reports have cited cases of necrotising fasciitis following non-combat-related injuries or in th

93 sherman with rapidly progressive necrotizing fasciitis from Photobacterium (Vibrio) damsela infection

94 Necrotizing fasciitis has conventionally been associated with the st

95 Historically, necrotising fasciitis has often, but not exclusively, been associate

96 ds to show how the management of necrotising fasciitis has progressed in parallel with prevailing sci

97 recapitulate key features of ABC and nodular fasciitis; however, the identity of USP6's relevant subs

98 or ICU admission in 23 patients, necrotizing fasciitis in 31 patients, and other diagnoses in 47 pati

99 we present a case of multifocal necrotizing fasciitis in a healthy adult patient, secondary to Haemo

100 port a case of Vibrio vulnificus necrotizing fasciitis in a patient with previously undiagnosed chron

101 an unusually severe case of MRSA necrotizing fasciitis in a previously undiagnosed AIDS patient.

102 r group A Streptococcus to cause necrotizing fasciitis in mice and nonhuman primates.

103 gh index of suspicion for fungal necrotizing fasciitis in the setting of wound infection and merits a

104 Current therapeutic regimens for necrotizing fasciitis include surgical debridement and treatment wit

105 Real-time sonoelastography can show plantar fasciitis, increase diagnostic performance of B-mode US,

106 We describe a rare case of necrotizing fasciitis involving Candida albicans, an organism that h

107 Necrotizing fasciitis is a life-threatening infection requiring urge

108 rome (strep TSS) with associated necrotizing fasciitis is a rapidly progressive process that kills 30

109 Periocular necrotizing fasciitis is a rare but potentially devastating disease,

110 Necrotising fasciitis is a rare, but potentially fatal, soft-tissue

111 potential devastation caused by necrotizing fasciitis is also reviewed, since this diagnosis is easi

112 tinent to S. marcescens-mediated necrotizing fasciitis is also reviewed.

113 Retroperitoneal necrotizing fasciitis is an uncommon soft tissue infection that is o

114 of magnetic insoles for the pain of plantar fasciitis is lacking.

115 Necrotizing fasciitis is often confused for cellulitis at initial pr

116 re suggests that survival of retroperitoneal fasciitis is possible with prompt debridement and antibi

117 e, epidemic toxic oil syndrome, eosinophilic fasciitis, localized forms of scleroderma, keloid, and t

118 een in these 5 cases, periocular necrotizing fasciitis may cause severe visual loss more often than p

119 icantly decreased virulence in a necrotizing fasciitis model of infection.

120 n in group A streptococcal (GAS) necrotizing fasciitis/myonecrosis often necessitates extensive debri

121 [SD] age, 57.2 [17.7] years) or necrotizing fasciitis (n = 31; mean [SD] age, 54.3 [13.5]) were incl

122 l and intraoperative findings of necrotizing fasciitis, necrotizing myositis, or both.

123 ty in vivo and recapitulated the necrotizing fasciitis-negative phenotype of the DeltamtsR mutant str

124 th invasive infections including necrotizing fasciitis (NF) and express a potent DNase (Sda1).

125 toxic shock syndrome (STSS) and necrotizing fasciitis (NF) cases.

126 Necrotizing fasciitis (NF) caused by flesh-eating bacteria is associ

127 Shock frequently complicates necrotizing fasciitis (NF) caused by group A Streptococcus (GAS) or

128 toxic shock syndrome (STSS) and necrotizing fasciitis (NF) express numerous virulence factors, inclu

129 Necrotizing fasciitis (NF) is a rapidly progressive, life-threatenin

130 r from a wound in a patient with necrotizing fasciitis (NF) or streptococcal toxic shock syndrome (ST

131 spectrum of infections including necrotizing fasciitis (NF).

132 We report the first case of necrotizing fasciitis of the chest wall due to infection with S. mar

133 re infections that can result in necrotizing fasciitis or even death in otherwise healthy adults outs

134 erosis was defined when cutaneous sclerosis, fasciitis, or joint contracture was first documented in

135 te recovered from a patient with necrotizing fasciitis, or with isogenic gene replacement mutants def

136 to address whether CLI improves necrotizing fasciitis outcome by modulating virulence factors of CLI

137 ouse infectious disease model of necrotizing fasciitis, PAD4(-/-) mice are more susceptible to bacter

138 nt species of bacteria can cause necrotising fasciitis, perhaps the most widely known is group A stre

139 ollected from a patient with GAS necrotizing fasciitis post-IVIG infusions markedly inhibited the mit

140 Fasciitis precedes and frequently accompanies muscle nec

141 Chromosomal translocations in nodular fasciitis result in USP6 overexpression, leading to tran

142 We report 5 cases of periocular necrotizing fasciitis resulting in severe vision loss, 3 of which re

143 usative agent of pharyngitis and necrotizing fasciitis, secretes the potent cysteine protease SpeB.

144 eptococcal toxic shock syndrome, necrotizing fasciitis, septic shock, or GAS cellulitis with shock) w

145 empirical treatment of suspected necrotizing fasciitis should include antibiotics predictably active

146 and patients with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%] vs 4 [12.9%

147 a murine model of streptococcal necrotizing fasciitis, the activated partial thromboplastin times we

148 ts of the clinical management of necrotising fasciitis; these treatment approaches all originate from

149 on admission than patients with necrotizing fasciitis, they have more chronic comorbidities and most

150 Fasciitis was observed in 18 of 30 biopsies (60%).

151 ients with pathologically proved necrotizing fasciitis were reviewed retrospectively for fascial thic

152 cluding toxic shock syndrome and necrotizing fasciitis, which are both associated with significant mo

153 cteria can result in devastating necrotizing fasciitis, which can progress to septicemia.

154 sions of linear scleroderma and eosinophilic fasciitis, which can result in considerable morbidity.

155 oon as possible to patients with necrotizing fasciitis, while our in vitro studies emphasize that a h

156 rly diagnosis of retroperitoneal necrotizing fasciitis, wide and repeated debridement, broad-spectrum

157 sembling both systemic sclerosis and diffuse fasciitis, with severe loss of motion and flexion contra

158 xic shock syndrome cases and one necrotizing fasciitis without shock, were treated with i.v. infusion