ライフサイエンスコーパス: fastidiosa

1 egy for the control of diseases caused by X. fastidiosa.

2 antivirulence effects of XfDSF itself in X. fastidiosa.

3 ted decreased symptoms after infection by X. fastidiosa.

4 ysis of Xfas53, a temperate phage of Xylella fastidiosa.

5 ve regulation of DSF synthesis by RpfF in X. fastidiosa.

6 aused by the Gram-negative bacterium Xylella fastidiosa.

7 ransmit the bacterial plant pathogen Xylella fastidiosa.

8 A similar protein has been found in Xylella fastidiosa, a bacterium that infects grapes, citrus and

9 Here we show that OMVs produced by Xylella fastidiosa, a xylem-colonizing plant pathogenic bacteriu

10 Cell-cell signaling in Xylella fastidiosa, a xylem-colonizing plant pathogenic bacteriu

11 Two XLB, Xylella fastidiosa and Pseudomonas syzygii, are transmitted by s

12 ll Xanthomonas species as well as in Xylella fastidiosa and the human pathogen, Stenotrophomonas malt

13 ell into the active site of Ohr from Xylella fastidiosa and were efficiently reduced by the recombina

14 IV pilus dependent for infection of both X. fastidiosa and Xanthomonas.

15 only xylem-feeding specialists vectors of X. fastidiosa (and probably P. syzygii), when many leafhopp

16 f X. campestris were replaced by those of X. fastidiosa, and the contribution of each gene to the ind

17 plants and vectoring to new host plants, X. fastidiosa apparently coordinates these traits in a popu

18 ylem network, the downward spread of Xylella fastidiosa bacteria in grape stems was modeled, and reve

19 X. fastidiosa biocontrol strain EB92-1 is infectious to gra

20 Citrus Tristeza Virus (CTV) and iii) Xylella fastidiosa, both causing great economic loss worldwide.

21 approach to characterize the secretome of X. fastidiosa, both in vitro and in planta, and identified

22 OMV production thus is a strategy used by X. fastidiosa cells to adjust attachment to surfaces in its

23 or-dependent quorum-sensing system, and a X. fastidiosa DeltarpfF mutant in which quorum signaling wa

24 The mutant in X. fastidiosa exhibited reduced cell length, hypermotility

25 ysaccharides, are important in regulating X. fastidiosa gene expression and mediating vector transmis

26 Diseases caused by Xylella fastidiosa have attained great importance worldwide as t

27 sA as one of the pathogenicity factors of X. fastidiosa in grapevines that leads to leaf scorching an

28 temic spread of the causal bacterium Xylella fastidiosa in infected vines.

29 Growth of X. fastidiosa in microfluidic chambers under flow condition

30 Despite the systemic spread of X. fastidiosa in PD-susceptible grapevines, the pathogen co

31 X. fastidiosa induces diseases of grapevines, citrus, coffe

32 Xylella fastidiosa infects a wide range of plant hosts and cause

33 Xylella fastidiosa is a bacterial plant pathogen that infects nu

34 Xylella fastidiosa is a Gram-negative plant-pathogenic bacterium

35 Xylella fastidiosa is a xylem-limited nonflagellated bacterium t

36 Xylella fastidiosa is a xylem-limited plant-pathogenic bacterium

37 lent response in grapevines observed when X. fastidiosa is disrupted for production of PrtA, and that

38 e strains from two distinct subspecies of X. fastidiosa is indicative of recent horizontal transfer,

39 The xylem-limited bacterium Xylella fastidiosa is the causal agent of several plant diseases

40 The Rpf system of X. fastidiosa is thus a novel example of a quorum-sensing s

41 Xylella fastidiosa, like related Xanthomonas species, employs an

42 dition of 50 to 100 microM Cu to standard X. fastidiosa media increases biofilm, while higher concent

43 ures of xylem vessels, we discovered that X. fastidiosa migrates via type IV-pilus-mediated twitching

44 ombination between a widespread family of X. fastidiosa P2-related prophage elements and a podophage

45 ntegrity and the systemic presence of the X. fastidiosa pathogen was confirmed.

46 detection of surface proteins of CTV and X. fastidiosa phytopathogens.

47 egrading enzymes likely to be produced by X. fastidiosa (polygalactuoronase and endo-1,4- beta -gluca

48 ing enzyme impacts on PMP and how a small X. fastidiosa population, introduced into grapevines by ins

49 Xylella fastidiosa possesses both type I and type IV pili at the

50 , XfDSF-dependent signaling required both X. fastidiosa proteins RpfF and RpfC.

51 P31758 are the mostly closely related non-X. fastidiosa proteins to most of the Trb proteins encoded

52 e history of advances in research on Xylella fastidiosa provides excellent examples of how paradigms

53 X. fastidiosa rpfC mutants hyperexpress rpfF and overproduc

54 A mutated X. fastidiosa RpfF protein with two substitutions of glutam

55 es previously identified as important for X. fastidiosa's pathogenicity in plants.

56 ed medium resulted in dramatic changes in X. fastidiosa's phenotype and gene-expression profile.

57 es of DPP-7 were found in genomes of Xylella fastidiosa, Shewanella putrefaciens, and P. gingivalis.

58 rization of the first virulent phages for X. fastidiosa, siphophages Sano and Salvo and podophages Pr

59 Xfas53 was isolated from supernatants of X. fastidiosa strain 53 and forms plaques on the sequenced

60 to that of pXFAS01 from X. fastidiosa subsp. fastidiosa strain M23; the two plasmids vary at only 6 n

61 s of ~4 x 10(-12) ml cell(-1) min(-1) for X. fastidiosa strain Temecula 1 and ~5 x 10(-10) to 7 x 10(

62 ltiple prophage elements of the sequenced X. fastidiosa strains.

63 almost identical to that of pXFAS01 from X. fastidiosa subsp. fastidiosa strain M23; the two plasmid

64 5) was present in the Riv5 strain of Xylella fastidiosa subsp. multiplex isolated from ornamental plu

65 omosomal and plasmid (pXF51) sequences of X. fastidiosa subsp. pauca strain 9a5c and more distant sim

66 es hxfA and hxfB to a DeltarpfF strain of X. fastidiosa, suggesting that RpfF is involved in XfDSF se

67 campestris, Xanthomonas oryzae, and Xylella fastidiosa T2S also occurs in nonpathogenic bacteria, fa

68 e sizes, thus facilitating the ability of X. fastidiosa to cross the PMs, was tested.

69 subsequent to the introduction of subspecies fastidiosa to the United States in the late 19(th) centu

70 gely on the ability of the bacterium Xylella fastidiosa to use cell wall-degrading enzymes (CWDEs) to

71 The frequency of attachment of X. fastidiosa to xylem vessels was 20-fold lower in the pre

72 of stems infected with the bacterium Xylella fastidiosa was compared with the PMP of healthy stems.

73 Furthermore, vector transmission of X. fastidiosa was induced in the presence of both polysacch

74 es of the phytopathogenic bacterium, Xylella fastidiosa, which causes serious disease in plants, incl

75 olysaccharides mediate gene regulation in X. fastidiosa, which results in phenotypic changes required

76 of an important bacterial pathogen, Xylella fastidiosa, within artificial plant xylem.

77 Xylella fastidiosa (Xf) causes wilt disease in plants and is res

78 ation of the xylem-limited bacterium Xylella fastidiosa (Xf) within xylem vessels is the sole factor

79 ease (PD) of grapevines is caused by Xylella fastidiosa (Xf), a xylem-limited gamma-proteobacterium t

80 to protect against the gram-negative Xylella fastidiosa (Xf), which causes diseases in multiple plant

81 ented by its own DSF, the DSF produced by X. fastidiosa (XfDSF) did not restore expression of the XfD

82 As in X. fastidiosa, XfDSF-dependent signaling required both X. f

83 of this chimera to target the Xf subspecies fastidiosa (Xff), which causes Pierce disease in grapevi