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3 ce of brainstem afferents terminating in the fastigial cerebellar nucleus and/or ventrolateral reticu
5 nsitivity in the vestibular (VN) and rostral fastigial nuclei (FN) using high-frequency (2 Hz) oscill
7 ostral contralateral anterior interposed and fastigial nuclei, but did not label cells within the ves
8 tor output neurons in the medial cerebellar (fastigial) nuclei are in fact glycinergic, not glutamate
10 s executed during inactivation of the caudal fastigial nucleus (cFN) on one side of the cerebellum.
12 at, electrical stimulation of the cerebellar fastigial nucleus (FN) for 1 h reduces the volume of foc
13 Electrical stimulation of the cerebellar fastigial nucleus (FN) in spontaneously hypertensive (SH
15 cited, would, like stimulation of cerebellar fastigial nucleus (FN), produce central neurogenic neuro
18 rea) to rostral [vasopressor] regions of the fastigial nucleus and ventral reticular formation were r
21 ulations via their projections to the caudal fastigial nucleus, and uncovered a simple-spike populati
22 f reference frames for motion in the rostral fastigial nucleus, in contrast to cells in the vestibula
23 ing that the posterior vermis and the caudal fastigial nucleus, to which it projects, provide a signa
24 by electrical stimulation of the cerebellar fastigial nucleus, we found that prohibitin is upregulat
25 l of the deep cerebellar nuclei, the rostral fastigial nucleus, were compared with those in the brain
30 nd selective neuronal loss within the mature fastigial pathway of the deep cerebellar nuclei (DCN), a
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