ライフサイエンスコーパス: fasting

1 ation (unlike their rapid degradation during fasting).

2 thy individuals, collected before and during fasting.

3 drop in body temperature that occurs during fasting.

4 ted with failure to inactivate mTORC1 during fasting.

5 c glycogen levels during the daytime or upon fasting.

6 nd Akt2) were unaffected by extended morning fasting.

7 e hypothalamus is upregulated in response to fasting.

8 is required to maintain normoglycemia during fasting.

9 d is essential for survival during prolonged fasting.

10 ction, especially during states of prolonged fasting.

11 ically cooperate to restore homeostasis upon fasting.

12 of the food cue, we assessed activity during fasting.

13 isassembly and degradation on denervation or fasting.

14 halamic expression of Ctbp2 and Nbeal1 after fasting.

15 verexpressing hepatic Ppp1r3b upon long-term fasting (12-36 h) were protected from blood ketone-body

16 ll secretory responses were determined under fasting, 230 mg/dL, and 340 mg/dL hyperglycemia clamp co

17 and few have investigated stress effects in fasting-adapted species.

18 Fasting Adipo-IR was increased twofold in obese subjects

19 iated with a progressive increase in FFA and fasting Adipo-IR.

20 Fasting adiponectin (P = 0.04) and ghrelin (P = 0.03) in

21 Here we report that fasting alone robustly inhibits the initiation and rever

22 American Diabetes Association criteria from fasting and 2-h glucose measurements.

23 lated with diabetes prevalence and levels of fasting and 2-hour glucose (each P < 0.008).

24 In this LDL-C range, 19% of fasting and 30% of nonfasting patients had differences >

25 de-rich lipoproteins (TRLs) were isolated at fasting and 4-6 h following test meals rich in SFA, unsa

26 In this triglyceride range, 73% of fasting and 81% of nonfasting patients had >/=10 mg/dL d

27 n and blood coagulation to devise whole-body fasting and bleeding regimens to prevent rupture.

28 xhibited partial nuclear localization during fasting and cAMP/cAMP-dependent protein kinase signaling

29 The results show that both short-term fasting and chronic caloric restriction significantly re

30 hepatic gluconeogenesis, both upon prolonged fasting and during T2D.

31 peptide YY or ghrelin did not differ between fasting and fed patients or between the high and low gas

32 n lipid metabolism, and impaired response to fasting and feeding contributes to steatosis and nonalco

33 and temporal variation in spring polar bear fasting and food web productivity suggests that polar be

34 e-fed with a high-protein diet after 6 hours fasting and gavaged a (15)NH4Cl tracer.

35 thm, whose amplitude depends on both feeding-fasting and light-dark cycles.

36 lucose flux were examined in ZDF rats during fasting and near-normal postprandial insulinemia and gly

37 In both fasting and nonfasting samples, accuracy was higher with

38 cebo (PLA) were rectally administered during fasting and postprandial conditions (oral glucose load).

39 and maximum tolerated volume), satiety, and fasting and postprandial gastric volumes at 16 weeks.

40 in terms of volume to fullness, satiety, or fasting and postprandial gastric volumes at week 16.

41 addition, all three SCFA mixtures increased fasting and postprandial plasma peptide YY (PYY) concent

42 (MR) imaging in children and young adults in fasting and postprandial states.

43 Fasting and postprandial urine samples were analyzed usi

44 KATPHI is characterized by fasting and protein-induced hypoglycemia that is unrespo

45 that plasma uridine levels are regulated by fasting and refeeding in mice, rats, and humans.

46 ell dysfunction and is associated with lower fasting and stimulated gastric inhibitory polypeptide (G

47 he ARC (ARC(AgRP) neurons) are stimulated by fasting and, once activated, they rapidly (within minute

48 energy states: both energy deficiency (acute fasting) and excessive energy storage (high-fat diet-ind

49 nous LPA levels, reduced PEPCK levels during fasting, and decreased hepatic gluconeogenesis in respon

50 dence on the health benefits of intermittent fasting, and discuss physiological mechanisms by which i

51 critically ill patients, during feeding and fasting, and to search for a correlation with gastric em

52 promote roaming behaviors characteristic of fasting animals.

53 position (dual-energy X-ray absorptiometry), fasting appetite ratings (visual analog scales), eating

54 ute (1 meal) and daily (24-h) EI and between fasting appetite ratings and certain eating behavior tra

55 ia the gluconeogenic pathway, in response to fasting, are critical.

56 TE was performed after overnight fasting (baseline values) and 15, 30, 45, 60, 90, and 12

57 d in significantly but only slightly lowered fasting blood glucose (-0.14 mmol/L; 95% CI: -0.24, -0.0

58 ed with a higher risk of developing elevated fasting blood glucose (HR = 1.33, 95% CI: 1.14, 1.56).

59 lycemic control (random blood glucose [RBG], fasting blood glucose [FBG], and glycated hemoglobin [Hb

60 The main outcomes analyzed were fasting blood glucose and insulin as well as fasting tri

61 d its components-abdominal obesity, elevated fasting blood glucose concentration, low high-density li

62 We found that treatment with ApoA-IV lowered fasting blood glucose in both WT and diabetic KKAy mice

63 ence interval (CI): 1.06, 1.52), an elevated fasting blood glucose level (HR = 1.20, 95% CI: 1.03, 1.

64 Lower fasting blood glucose levels, higher insulin, and lower

65 Our findings for metabolic syndrome and high fasting blood glucose remained significant for PM2.5 lev

66 terol, total cholesterol, triglycerides, and fasting blood glucose) and identified several cis-eGenes

67 Body weight, organ mass, fasting blood glucose, energy expenditure, cardiac geome

68 The mice also had elevated fasting blood glucose, fatty liver, and insulin resistan

69 PCSK9 genetic variants with LDL cholesterol, fasting blood glucose, HbA1c, fasting insulin, bodyweigh

70 There was no effect on fasting blood insulin or blood lipids.The evidence sugge

71 lood pressure, waist circumference (WC), and fasting blood sample (total cholesterol, high-density li

72 glycosylated hemoglobin were measured from a fasting blood sample, and 2-hour glucose was measured af

73 Fasting blood samples were collected and plasma was anal

74 Non-fasting blood samples were collected at the ages of 1, 1

75 Fasting blood samples were collected to estimate lipid p

76 tic side effects, vital signs were obtained, fasting blood samples were collected, and the Adherence

77 Fasting blood samples were taken from a subgroup (placeb

78 ciated with the risk of high blood pressure, fasting blood sugar, and triglycerides, with marginal si

79 Blood pressure, non-fasting blood sugar, body mass index and abdominal girth

80 Fasting blood sugar, serum insulin, fasting serum lipids

81 antitative food frequency questionnaires and fasting blood, anthropometric and blood pressure measure

82 At the last follow-up visit, median fasting C-peptide was 0.43 (0.19-0.93) ng/mL; median ins

83 s duration, glycosylated hemoglobin A1c, and fasting C-peptide.

84 S) have identified >175 loci associated with fasting cholesterol levels, including total cholesterol

85 In human type 1 diabetes pancreatic islets, fasting conditions reduce PKA and mTOR activity and indu

86 eficient adipocytes under postabsorptive and fasting conditions, resulting from impaired signal trans

87 Under fasting conditions, the Them2/PC-TP complex directs satu

88 odulated by nutritional status: exercise and fasting decrease ACSL6 mRNA, whereas acute lipid ingesti

89 rmore, glucose injections alone reversed the fasting-dependent protection against DXR in mice, indica

90 acements, and newly popularized intermittent fasting diets.

91 ue insulin sensitivity with extended morning fasting do not necessarily require changes in insulin si

92 acterize the molecular underpinnings of this fasting effect, we comparatively analyzed the NLRP3 infl

93 is of pediatric patients with pre-B-ALL, and fasting effectively inhibited B-ALL growth in a human xe

94 Fasting elicits transcriptional programs in hepatocytes

95 ial lipid oxidation was normalized, and with fasting, endogenous glucose production (EGP) increased b

96 We demonstrate that in the absence of MRAP2, fasting fails to activate agouti-related protein neurons

97 med using PubMed and the terms "intermittent fasting," "fasting," "time-restricted feeding," and "foo

98 lic inflexibility' is characterized by lower fasting fat oxidation (higher fasting RQ) and/or an impa

99 All three SCFA mixtures increased fasting fat oxidation (P < 0.01), whilst resting energy

100 Fasting for >8 h, as previously required for lipid profi

101 cially pronounced for most volunteers during fasting for 3 h after exercise, with strong variation be

102 tide YY (PYY) concentrations, and attenuated fasting free glycerol concentrations versus PLA (P < 0.0

103 ven intragastrically to assess its effect on fasting gastrointestinal motility and hunger ratings, mo

104 ined as having blood pressure <120/80 mm Hg, fasting glucose <100 mg/dl, glycosylated hemoglobin <5.7

105 and untreated blood pressure <140/90 mm Hg, fasting glucose <126 mg/dl, total cholesterol <240 mg/dl

106 d diabetes was defined as elevated levels of fasting glucose (>/=7.0 mmol/L [>/=126 mg/dL]) and hemog

107 olesterol showed associations with increased fasting glucose (0.09 mmol/L, 95% CI 0.02 to 0.15), body

108 Allele C at rs3093059 was associated with fasting glucose (beta = 0.20, P = 0.045) and G at rs1205

109 were assessed across subgroups defined upon fasting glucose (FG) and body mass index (BMI).

110 beta-cell function in subjects with impaired fasting glucose (IFG).

111 arter mile) was associated with increases in fasting glucose (mean = 0.22 mg/dL, 95% confidence inter

112 Eight subjects with normal fasting glucose (NFG) and eight subjects with IFG receiv

113 PPP1R3B was associated with higher levels of fasting glucose (P = 7.70 x 10-7) and fasting insulin (P

114 F, we detected an interaction effect between fasting glucose and fasting triglycerides with rs9939609

115 Laboratory status was improved, for example, fasting glucose and glycated hemoglobin decreased from 6

116 Baseline fasting glucose and rs9939609 interacted on weight chang

117 y, we investigated the interaction effect of fasting glucose and triglyceride levels with rs9939609 i

118 O) criteria to define GDM: >/=7.0 mmol/L for fasting glucose and/or >/=7.8 mmol/L for 2-h post-glucos

119 Lower FG fasting glucose at BR was more commonly associated with

120 Fasting glucose at re-assessment was also predictive of

121 rol seem to be protective against increasing fasting glucose but not against type 2 diabetes.

122 The C-statistics were 0.662 for ADA fasting glucose clinical concentration categories and 0.

123 ent chronic kidney disease was 0.636 for ADA fasting glucose concentration clinical categories and 0.

124 erotic cardiovascular disease, 0.701 for ADA fasting glucose concentration clinical categories and 0.

125 ripheral arterial disease, and 0.683 for ADA fasting glucose concentration clinical categories and 0.

126 [9%] of 10 844 people; 8.4-9.5), and the WHO fasting glucose concentration cutoff (1213 [11%] of 10 8

127 Prediabetes defined using the ADA fasting glucose concentration cutoff (prevalence 4112 [3

128 The definition of prediabetes using the ADA fasting glucose concentration cutoff was more sensitive

129 were also associated with circulating higher fasting glucose concentration, bodyweight, and waist-to-

130 s different prediabetes definitions based on fasting glucose concentration, HbA1c, and 2 h glucose co

131 al disease, and all-cause mortality than did fasting glucose concentration-based definitions (all p<0

132 hance both glucagon and insulin secretion at fasting glucose concentrations and that FFAR1 and enhanc

133 mulates secretion of glucagon and insulin at fasting glucose concentrations.

134 circulating metabolites that correlate with fasting glucose in the Erasmus Rucphen Family (ERF) stud

135 SLC5A1 showed a significant association with fasting glucose in the expected opposing direction.

136 We defined diabetes as a fasting glucose level >/=126 mg/L (or >/=200 mg/L for th

137 ntly of this, with a steeper increase of the fasting glucose level (beta=131; 95% CI 38-225) during f

138 efit for cardiometabolic outcomes, including fasting glucose level.

139 ody mass index, systolic blood pressure, and fasting glucose level.

140 we show that hepatic GCN5L1 ablation reduces fasting glucose levels and blunts hepatic gluconeogenesi

141 0.3 vs. 2.2 +/- 0.44 kg/m(2) in persons with fasting glucose levels below and above the median, respe

142 Higher fasting glucose levels may amplify obesity-risk in FTO c

143 Variants associated with type 2 diabetes and fasting glucose levels reside in introns of ADCY5, a gen

144 asting glucose, with a mean +/- SD change in fasting glucose of -1.1 +/- 8.4 mg/dL compared with an i

145 the inclusion criteria (NAFLD with impaired fasting glucose or impaired glucose tolerance) and were

146 R3527Q variant and impaired fasting glucose, fasting glucose or insulin, or oral glucose tolerance te

147 ssociated with the clinical glycemic markers fasting glucose or the HbA1c, and vice versa.

148 , systolic and diastolic blood pressure, and fasting glucose were measured.

149 m glycemic changes; and the association with fasting glucose were significantly modified by postpartu

150 There were no changes in weight, fasting glucose, 2-h glucose and insulin, haemoglobin A1

151 ospective cohort, concentrations of 11 PFAS, fasting glucose, and lipids were measured in maternal mi

152 otein- and total cholesterol, triglycerides, fasting glucose, body mass index, waist circumference, h

153 ociation between R3527Q variant and impaired fasting glucose, fasting glucose or insulin, or oral glu

154 The changes in fasting glucose, fasting insulin, insulin resistance (ho

155 CB0313.1 improved diabetic markers (fasting glucose, glucose tolerance, insulin tolerance, G

156 tent variable for glycemia (diabetes status, fasting glucose, glycated hemoglobin (HbA1c), fructosami

157 In unadjusted GEE analyses, for a given fasting glucose, HbA1c values were statistically signifi

158 unspecified subtypes of MDD with changes of fasting glucose, high-density lipoprotein-cholesterol, t

159 or more cardiometabolic abnormalities (high fasting glucose, low high-density lipoprotein cholestero

160 rticipants taking KCl had stable or improved fasting glucose, with a mean +/- SD change in fasting gl

161 The fasting glucose-raising allele near PDX1, a known key in

162 Thirty type 2 diabetes or fasting glucose-raising alleles were associated with a m

163 pite weight gain, KCl prevented worsening of fasting glucose.

164 ted to provide 90% power and a difference in fasting glycemia of 0.25 mmol/L.

165 Eight patients could not be fed (fasting group).

166 tional sample of 1 545 634 patients (959 153 fasting >/=10-12 hours; 586 481 nonfasting) from the sec

167 that undergo seasonal cycles of feeding and fasting have adaptations that maintain integrity of orga

168 Dietary restriction regimens, including fasting, have been considered for the prevention and tre

169 independent effects by which leptin reverses fasting hyperglycemia and ketoacidosis in a rodent model

170 elevated hepatic gluconeogenic activity and fasting hyperglycemia.

171 Clinically, GSDIa is characterized by fasting hypoglycaemia and hepatic glycogen and triglycer

172 Winter fasting in hibernators shifts the microbiota to favor ta

173 Fasting increased in the Beaufort Sea between 1983-1999

174 Fasting increases plasma uridine levels, and this increa

175 ean individuals, 6 weeks of extended morning fasting increases the expression of genes involved in li

176 organizes liver chromatin, exposing numerous fasting-induced enhancers.

177 ficiency of the Tgr5 gene in mice alleviated fasting-induced hepatic lipid accumulation.

178 Double knockout mice were protected against fasting-induced hepatic steatosis (a model of enhanced e

179 in determining bile acid composition and in fasting-induced hepatic steatosis through a novel mechan

180 trols and, as a result, delayed the onset of fasting-induced hypoglycemia.

181 Compound 38 temporarily reduces fasting-induced refeeding of mice, thereby emulating the

182 resistant to acetaminophen hepatotoxicity or fasting-induced steatosis.

183 Interestingly, fasting induction of fibroblast growth factor 21 in live

184 development and maintenance of ALL, and that fasting inhibits ALL development by upregulation of LEPR

185 ociations with HbA1c (0.03%, -0.01 to 0.08), fasting insulin (0.00%, -0.06 to 0.07), and BMI (0.11 kg

186 of MARV with analysis of triglycerides (TG), fasting insulin (FI) and waist-to-hip ratio (WHR) in 4,7

187 patients with type 2 diabetes, a decrease in fasting insulin (MD -7 microU/ml, 95% CI -11.5, -2.5) wa

188 Type I had the highest values of fasting insulin (median = 12.98 mU/mL) and HOMA IR value

189 els of fasting glucose (P = 7.70 x 10-7) and fasting insulin (P = 4.79 x 10-6), but these association

190 0.01 to -0.12; P=0.01) lower log-transformed fasting insulin (pmol/L) and 21% lower odds (95% confide

191 gy compared with >12.5% of energy) had lower fasting insulin (ratio of geometric means: 0.82; 95% CI:

192 rs35929 modified the association of uMg with fasting insulin and fat mass in a general population.

193 (FM), systolic and diastolic blood pressure, fasting insulin and glucose, and homeostasis model asses

194 etween maternal protein intake and offspring fasting insulin and homeostasis model assessment of insu

195 ns of plasma leptin, resistin, fed-state and fasting insulin and increased expression of adipogenic t

196 igher genetically determined log-transformed fasting insulin level was associated with higher CHD ris

197 d with insulin resistance phenotypes (higher fasting insulin levels adjusted for BMI, lower HDL chole

198 Mice fed a HFD displayed increased fasting insulin levels, hepatosteatosis and major change

199 of 609 whites and 339 blacks who had BMI and fasting insulin measured twice in childhood (mean age =

200 trol of brain activity during deep sleep and fasting insulin secretion rate.

201 und significant heritability for measures of fasting insulin sensitivity and beta-cell function, for

202 s model assessment of insulin resistance and fasting insulin) through a systematic review and meta-an

203 protein cholesterol, triglycerides, glucose, fasting insulin) were measured with the use of standard

204 dipose DI was calculated as ATIS: (1/GlyRa x fasting insulin) x first-phase insulin secretion.

205 had normalized glucose tolerance, decreased fasting insulin, and decreased adiposity.

206 L cholesterol, fasting blood glucose, HbA1c, fasting insulin, bodyweight, waist-to-hip ratio, BMI, an

207 me body weight as chow fed control mice, the fasting insulin, glucose, and hepatic triglyceride level

208 The changes in fasting glucose, fasting insulin, insulin resistance (homeostasis model a

209 Body weight, food intake, adiposity index, fasting insulin, triglycerides and cholesterol levels we

210 Upon fasting, insulin is lowered to remove ARF1 and kinesin f

211 Twenty-four hours of fasting is known to blunt activation of the human NLRP3

212 orrelate with altered terminal GABA release, fasting is likely to cause a decrease in GABA release fr

213 Every year, Ramadan fasting is practiced by many Muslim individuals.

214 ltered SCAT insulin sensitivity with morning fasting is unlikely to be explained by signalling proxim

215 l mean changes for random, nonfasting versus fasting levels are +26 mg/dl for triglycerides, -8 mg/dl

216 In obesity fasting levels of both glucagon and insulin are elevated

217 In these subjects fasting levels of the free fatty acid palmitate are rais

218 e of 3416 US adults aged 40 to 75 years with fasting lipid data and triglyceride levels of 400 mg/dL

219 Fasting lipid fractions (triglycerides [TGs], high-densi

220 files, carriers in both cohorts had improved fasting lipid profiles.

221 d in WD + PDX mice inversely correlated with fasting lipids and downregulated genes Dgat1, Cd36 and F

222 Individuals with elevated fasting low-density lipoprotein cholesterol levels were

223 suring chromatin accessibility revealed that fasting massively reorganizes liver chromatin, exposing

224 altered nutritional landscape during winter fasting may provide insights into protective mechanisms

225 g patterns: skipping breakfast, intermittent fasting, meal frequency (number of daily eating occasion

226 cose by 1.1-fold in fed mice and 2.5-fold in fasting mice; lactate is made primarily from glucose but

227 ysiological mechanisms by which intermittent fasting might lead to improved health outcomes.

228 ion and that the anti-inflammatory effect of fasting-mimetic diets may be mediated, in part, through

229 In mice, a 4-day fasting mimicking diet (FMD) induces a stepwise expressi

230 evels of HbA1c at any given concentration of fasting or 2-hour glucose compared with participants wit

231 on between SCT and HbA1c for given levels of fasting or 2-hour glucose levels among African Americans

232 on of SCT with HbA1c levels, controlling for fasting or 2-hour glucose measures.

233 Here, we show that fasting or glucose restriction (GR) regulate PKA and AMP

234 ed by reducing circulating glucose levels by fasting or insulin.

235 take, but did not decrease food intake after fasting or salt intake following salt depletion; inactiv

236 tric pressure by a median of 6.9 mmHg during fasting (P = .002) and by 9.0 mmHg after the meal (P = .

237 bA1c difference by SCT was greater at higher fasting (P = .02 for interaction) and 2-hour (P = .03) g

238 Concomitantly, a longer fasting period with breakfast skipping also increased th

239 We measured baseline fasting plasma concentrations of 23 metals and used cond

240 The main outcome measures were diabetes (fasting plasma glucose >/= 126 mg/dL or taking medicatio

241 nately, tests such as hemoglobin A1c (HbA1c)/fasting plasma glucose (FPG) alone fail to diagnose or m

242 sporter inhibition with empagliflozin on the fasting plasma glucose (FPG) concentration and beta-cell

243 ne HbA1c of 6.5% (48 mmol/mol) or greater or fasting plasma glucose (FPG) of 7.0 mmol/L or greater.

244 nd postpartum weight reduction on changes in fasting plasma glucose (P-interaction = 0.03).

245 rly women in Denmark, we found that impaired fasting plasma glucose concentration was associated with

246 to <40 mg/dL, which is well below the normal fasting plasma glucose concentration.

247 s 7.5+/-1.2% in each group, whereas the mean fasting plasma glucose level was significantly lower in

248 Fasting plasma glucose levels (Hedges g = 0.20; 95% CI,

249 Case-control studies reporting on fasting plasma glucose levels, plasma glucose levels aft

250 , and mice lacking hepatic Ppp1r3b had lower fasting plasma glucose than controls.

251 Waist circumference, body mass index, fasting plasma glucose, glycohemoglobin, low-density lip

252 or elevated triglycerides, 18.95% for raised fasting plasma glucose.

253 quency allele associated with an increase in fasting plasma insulin level and risk of type 2 diabetes

254 s g = 0.61; 95% CI, 0.16 to 1.05; P = .007), fasting plasma insulin levels (Hedges g = 0.41; 95% CI,

255 levels after an oral glucose tolerance test, fasting plasma insulin levels, insulin resistance, and h

256 did not observe any differences in fecal or fasting plasma SCFA concentrations or in systemic concen

257 In particular, we detect associations with fasting plasma trimethylamine N-oxide (TMAO) levels, a g

258 17.70; betaPFOA=15.93; 95% CI: 6.78, 25.08), fasting proinsulin (betaPFOS=1.37 pM; 95% CI: 0.50, 2.25

259 Fasting proinsulin-to-C-peptide and proinsulin secretory

260 The combination of fasting prospective food consumption ratings and RMR was

261 ricted feeding," and "food timing." Modified fasting regimens appear to promote weight loss and may i

262 Intermittent fasting regimens are hypothesized to influence metabolic

263 ew is to provide an overview of intermittent fasting regimens, summarize the evidence on the health b

264 transcriptional activators that mediate the fasting response.

265 s, we implicated four key TFs regulating the fasting response: glucocorticoid receptor (GR), cAMP res

266 mic responses involving appetite-stimulating fasting-responsive hypothalamic neurons expressing agout

267 rized by lower fasting fat oxidation (higher fasting RQ) and/or an impaired ability to oxidize carboh

268 We also measured fasting serum chromogranin A, neuron-specific enolase, g

269 er participants based on pre-supplementation fasting serum in the Alpha-Tocopherol, Beta-Carotene Can

270 improved insulin tolerance, higher levels of fasting serum insulin, and lower pancreatic insulin cont

271 ovel associations between gut microbiota and fasting serum levels of a number of metabolites, includi

272 Fasting blood sugar, serum insulin, fasting serum lipids and serum alanine aminotransferase

273 food-frequency questionnaire and provided a fasting serum sample before study randomization (1985-19

274 gas chromatography in end-of-feeding-period fasting serum samples and expressed in both relative and

275 Eight hours of fasting significantly increased the expression of two cr

276 ers underwent phase-contrast MR imaging in a fasting state and again after a standardized meal.

277 ne and at the end of each 4-wk period in the fasting state for metabolomics analysis by using liquid

278 ndicate that oral glucose consumption in the fasting state is followed by increased glucose levels in

279 ntral motility and hunger ratings during the fasting state, possibly because of a decrease in motilin

280 th neurotransmitters in both the feeding and fasting states enables increased behavioral adaptability

281 lation of glucose homeostasis during fed and fasting states.

282 Fasting status (>/=7 days) was estimated using serum ure

283 y to assess for the first time the impact of fasting status on novel LDL-C accuracy.

284 aracteristics, preprocedural medications and fasting status, current or underlying health risks, and

285 el >/=126 mg/L (or >/=200 mg/L for those not fasting), the use of insulin or oral hypoglycemic agents

286 , during the rapid muscle atrophy induced by fasting, the desmin cytoskeleton and the attached Z-band

287 Upon prolonged fasting, the impaired lipolytic response resulted in abn

288 During fasting, the KO mice had a defect in fatty acid oxidatio

289 ubMed and the terms "intermittent fasting," "fasting," "time-restricted feeding," and "food timing."

290 es of chronic disease, religious texts allow fasting to be broken.

291 nce-based modern care changes from overnight fasting to carbohydrate drinks 2 hours before surgery, m

292 three other cardiovascular risk factors, and fasting total cholesterol concentrations of 6.5 mmol/L o

293 dipocyte GR is indispensable for the feeding-fasting transition but also promotes adiposity and assoc

294 Fasting triglycerides above the median increased the per

295 teraction effect between fasting glucose and fasting triglycerides with rs9939609 on BMI (p = 0.0005

296 fasting blood glucose and insulin as well as fasting triglycerides, blood lipoproteins, HbA1c, and bo

297 We conclude that morning fasting up-regulates lipid turnover genes in SCAT of lea

298 Fasting venous ammonia levels were estimated daily from

299 ailure, stroke, or retinopathy, or prevalent fasting versus postprandial hyperglycaemia, could also b

300 mmunosorbent assay) were measured once in 10 fasting volunteers (controls) and daily from admission u

301 his buffering function is most visible after fasting, when liver triglyceride increases manyfold but

302 ss quiescent behavior and promote roaming in fasting worms, whereas 5-HT produced by the NSM neurons