ライフサイエンスコーパス: fasting hypoglycemia

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1 as a result, CD36Tg mice were protected from fasting hypoglycemia.

2 of glucose-6-phosphatase resulting in severe fasting hypoglycemia.

3 interference (RNAi) to the liver, experience fasting hypoglycemia.

4 and fasting conditions was achieved without fasting hypoglycemia.

5 ion suppresses gluconeogenesis, resulting in fasting hypoglycemia.

6 In parallel, the VAMP8 null mice also had fasting hypoglycemia (84 +/- 11 vs. 115 +/- 4) and enhan

7 They also have chronic fasting hypoglycemia and a reduced rise in blood glucose

8 dren with unexplained, recurrent, and severe fasting hypoglycemia and asymmetrical growth.

9 As in the humans with HI/HA, H454Y mice had fasting hypoglycemia, but plasma insulin concentrations

10 Metabolic evaluation revealed fasting hypoglycemia, dicarboxylic aciduria, and reduced

11 The fasting hypoglycemia in Foxa3(-/-) mice could not be att

12 Pathological fasting hypoglycemia in humans is usually explained by e

13 ings indicate that exendin-(9-39) normalizes fasting hypoglycemia in SUR-1(-/-) mice via a direct eff

14 9) suppresses insulin secretion and corrects fasting hypoglycemia in SUR-1(-/-) mice.

15 blation of FoxO genes causes more pronounced fasting hypoglycemia, increased glucose tolerance, and e

16 s characterized by reduced glycogen storage, fasting hypoglycemia, increased insulin sensitivity and

17 y insensitive to glucocorticoids, exhibiting fasting hypoglycemia, increased insulin sensitivity desp

18 DH sensitivity to GTP inhibition, leading to fasting hypoglycemia, leucine hypersensitivity, and prot

19 The finding of fasting hypoglycemia raises important clinical questions

20 Acute ethanol intoxication promotes fasting hypoglycemia through an increase in hepatic NADH

21 Fasting hypoglycemia was associated with normal levels o

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