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1 an (fasudil and ripasudil) and one in China (fasudil).
2 nged with HDM extracts and then treated with fasudil.
3 icial effects of ANG-(1-7) were prevented by fasudil.
4 y pretreatment with the rho kinase inhibitor fasudil.
5 ogical intervention with the ROCK inhibitor, fasudil.
6 neurons or treatment with the ROCK inhibitor fasudil.
7 udies revealed that the Rho-kinase inhibitor fasudil (1 micromol/L) significantly blunted artery cont
8 te experiments, diabetic db/db mice received fasudil (10 mg x kg(-) x day(-) i.p.) or simvastatin (40
9 als were randomly assigned to treatment with fasudil (136 or 213 mg x kg(-1) x d(-1) in drinking wate
10 ome mice also were given the ROCK1 inhibitor fasudil; 2 weeks later, serum EVs were isolated and char
12 that was inhibited by treating animals with fasudil, a pharmacologic ROCK inhibitor, and that both R
14 In this study, we tested the hypothesis that fasudil, a Rho-kinase inhibitor, could attenuate Ang II-
17 downstream from RhoA, we treated cells with fasudil, a selective Rho kinase inhibitor, and found tha
26 Despite more occlusive vascular lesions, fasudil also markedly reduced right ventricular systolic
29 ought to evaluate the efficacy and safety of fasudil, an orally available rho kinase inhibitor, in pa
33 e presence of Rho-effector kinase inhibitors Fasudil and Y-27632, but ATP-induced IL-1 beta release w
36 mm ST-segment depression was increased with fasudil at both peak and trough compared with placebo (1
37 demonstrate that inhibition of Rho-kinase by fasudil attenuated Ang II-induced AAA through inhibition
39 human CCM endothelium, and they suggest that fasudil could ameliorate both CCM disease and vascular l
48 entrapment efficiency of optimized liposomal fasudil formulations was between 68.1+/-0.8% and 73.6+/-
49 tion of an investigational anti-PAH molecule fasudil (HA-1077), a Rho-kinase inhibitor, into liposoma
55 transduction pathway with the ROCK inhibitor fasudil induced myofibroblast apoptosis through a mechan
56 bition of the spatial rescue of alpha2C-ARs, fasudil inhibited alpha2-AR-mediated mobilization of cal
58 eased in lungs from both groups of rats, and fasudil (intravenous) reversed the increased phosphoryla
59 roof-of-principle that aerosolized liposomal fasudil is a feasible option for a non-invasive, control
61 fter intratracheal instillation of liposomal fasudil, mean pulmonary arterial pressure (MPAP) was red
65 lockade of the RhoA/ROCK pathway with either fasudil or simvastatin would ameliorate progression of d
67 ssigned to receive the Rho kinase inhibitor, fasudil, or placebo for 1 month each in a double-blind c
68 ndicate that the Rho-A/Rho kinase inhibitor, fasudil, plays a negative regulatory role in allergen-in
69 ation of the Rho-associated kinase inhibitor fasudil prevented renal fibrosis and restored expression
70 activity, using the clinically approved drug fasudil, prevented dendritic network disorganization, me
72 , non-placebo-controlled trials suggest that fasudil reduces myocardial ischemia in patients with sta
73 thelium-dependent vasodilation achieved with fasudil relative to placebo was inversely proportional t
74 , treatment with the approved ROCK inhibitor fasudil resulted in increased apoptosis and decreased vi
86 epared by hydration and extrusion method and fasudil was loaded by ammonium sulfate-induced transmemb
89 ffect of RhoA/ROCK inhibitors (C3-exoenzmye, fasudil, Y-27632, ibuprofen, siRhoA, and p21) in experim
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