ライフサイエンスコーパス: fat mass

1 ated endpoints (body mass index, weight, and fat mass).

2 an mass (in both sexes, after adjustment for fat mass).

3 han that of control subjects, as well as low fat mass.

4 sue factor, which positively correlated with fat mass.

5 female mice is due to the reduction in body fat mass.

6 f inflammatory markers despite their greater fat mass.

7 tivity in adipose tissue and negatively with fat mass.

8 th higher childhood total-body and abdominal fat mass.

9 th respect to age, body mass index, FFM, and fat mass.

10 vity, whereas SCD1 ratio was associated with fat mass.

11 and fetal growth without affecting maternal fat mass.

12 early childhood and particularly with higher fat mass.

13 36 +/- 4 kg/m2 who were matched for BMI and fat mass.

14 ght homeostat ("gravitostat") that regulates fat mass.

15 r adjustment for change in MVPA and baseline fat mass.

16 -protein weight-loss diet on body weight and fat mass.

17 ng behavior depending on the location of the fat mass.

18 r may influence long-term maternal abdominal fat mass.

19 terms to a 103-g (95% CI: 27, 179-g) higher fat mass.

20 ty levels, and reduced their body weight and fat mass.

21 nd the strongest association is for visceral fat mass.

22 ht in male mice was accompanied by decreased fat mass.

23 ngestion of a single flavor but no change in fat mass.

24 developed exaggerated obesity and increased fat mass.

25 analysis, adjusted for sex, age, height, and fat mass.

26 hed when values are normalised to whole-body fat mass.

27 greater accretion of lean mass, rather than fat mass.

28 smaller infant birth size and less abdominal fat mass.

29 1 [-0.01, -0.0]; P = 0.02) and gluteofemoral fat mass (-0.03 [-0.05, -0.02; P = 1.4 x 10(-6)) and wit

30 activity group gained the largest amount of fat mass (1.7 +/- 0.3 kg) after adjustment for change in

31 (in kg/m(2)): -0.24; 95% CI: -0.41, -0.07], fat mass (-1.10 kg; 95% CI: -1.77, -0.44), and waist cir

32 taneous fat mass (1650-1850 cm(3)), visceral fat mass (1350-1650 cm(3)), and total body weight (11-12

33 umference (11-13 cm), abdominal subcutaneous fat mass (1650-1850 cm(3)), visceral fat mass (1350-1650

34 ean mass (-0.7 +/- 0.1 kg), and appendicular fat mass (-2.6 +/- 0.2 kg) each decreased.

35 predicted GWG was associated with increased fat mass (24.0 g; 95% CI: 17.4, 30.5 g), fat-free mass (

36 kg and -2.8 +/- 2.8 kg; both P < 0.001) and fat mass (-3.2 +/- 3.1 kg and -2.5 +/- 2.4 kg; both P <

37 .6 +/- 2.2 kg/m), LBM (-2.5 +/- 8.7 kg), and fat mass (-3.4 +/- 5.8 kg) was evident from preoperative

38 l BMI was associated with increased neonatal fat mass (5.2 g; 95% CI: 3.5, 6.9 g), fat-free mass (7.7

39 body lean mass (-1.0 +/- 0.2 kg), whole-body fat mass (-6.9 +/- 0.5 kg), appendicular lean mass (-0.7

40 -fat diet-induced weight gain, in particular fat mass accumulation, improved glucose tolerance, and l

41 ttenuated HFD-induced body weight gain, body fat mass accumulation, increased energy expenditure, and

42 es of BMR, adjusted for fat-free mass (FFM), fat mass, age, and sex, were identified.

43 , Sf1Gck(-/-) mice displayed increased white fat mass and adipocyte size, reduced lean mass, impaired

44 t ages 60-64 years was associated with lower fat mass and android (abdominal):gynoid (hip) fat ratio

45 bers listed in them for the decile values of fat mass and appendicular skeletal muscle mass utilizing

46 ssion and adiposity concomitant to increased fat mass and BMI (haplotype AT>GC).

47 There was no relation between change in fat mass and change in EE.

48 Children treated for cancer have increased fat mass and decreased body cell mass, which are evident

49 composition measurements revealed increased fat mass and decreased lean mass.

50 Despite similar fat mass and energy balance, M(IL10) mice were protected

51 We have also shown that fat mass and fasting glucose concentrations were lower i

52 ndations was associated with higher neonatal fat mass and fat-free mass but not percentage of body fa

53 CR reduced fat mass and FFM by 114 and 159 g/d, respectively.

54 lementation results in lower body weight and fat mass and higher lean mass in animals and adult human

55 (iBAT) thermogenesis accompanied by reduced fat mass and improved glucose/insulin homoeostasis.

56 The treatment brought about decreases in fat mass and improvements in insulin sensitivity in both

57 eficient mice, primarily through the loss of fat mass and increase in basal metabolic rate with brown

58 se-deficient mice were smaller, with reduced fat mass and increased bone formation that was accompani

59 n myocardial lipid accumulation and systemic fat mass and inflammation and increased insulin-stimulat

60 The estimated causal RRs for the effects of fat mass and lean mass on asthma were 1.41 (95% CI 1.11-

61 Fat mass and lean mass were measured using dual-energy-x

62 rient and food profiles, may affect visceral fat mass and metabolic syndrome.

63 ion between the rs993609 variant of the FTO (fat mass and obesity associated) gene and body mass inde

64 Significance statement: Variations in the fat mass and obesity-associated (FTO) gene are associate

65 Variations in the fat mass and obesity-associated (FTO) gene are linked to

66 Polymorphisms in the fat mass and obesity-associated (FTO) gene have been ass

67 h body mass index, those in an intron of the fat mass and obesity-associated (FTO) gene have the larg

68 Although the effect of the fat mass and obesity-associated (FTO) gene on adiposity

69 The fat mass and obesity-associated (FTO) gene plays a pivot

70 Noncoding polymorphisms in the fat mass and obesity-associated (FTO) gene represent com

71 vious evidence suggests that variants in the fat mass and obesity-associated gene (FTO) affect adipos

72 ms through which common polymorphisms in the fat mass and obesity-associated gene (FTO) drive the dev

73 Fat mass and obesity-associated gene (FTO) is a member o

74 Mechanistically, R-2HG inhibits fat mass and obesity-associated protein (FTO) activity,

75 d that m(6)Am is selectively demethylated by fat mass and obesity-associated protein (FTO).

76 upies a similar region as the L1 loop of the fat mass and obesity-associated protein that is proposed

77 transferase-like 3) and the demethylase FTO (fat mass and obesity-associated protein).

78 the best characterized RNA demethylase, FTO (fat mass and obesity-associated) in memory.

79 es of GWG were independently associated with fat mass and percentage of body fat.

80 fatty acids (PUFAs) to a rodent diet reduces fat mass and prevents the development of obesity, but ev

81 We propose that manipulations that increase fat mass and reduce energy expenditure will be beneficia

82 mice had a rapid increase in body weight and fat mass and specifically showed an increased number of

83 red childhood anthropometric measures, total fat mass and the android:gynoid fat ratio by using dual-

84 ght were assessed at ages 5 and 7 years, and fat mass and waist circumference at age 7.

85 have recently been implicated in regulating fat mass and weight gain.

86 und that NOD mice had approximately 50% less fat mass and were 2-fold more insulin sensitive, as meas

87 android (P < 0.001), and gynoid (P < 0.001) fat masses and lower arm (P < 0.001) and leg (P = 0.001)

88 ned whether the disclosure of information on fat-mass and obesity-associated (FTO) genotype risk had

89 ts show increased food intake, enhanced body fat mass, and elevated plasma levels of triglycerides an

90 ived estimates of intracellular water (ICW), fat mass, and extracellular water (ECW) as the main pred

91 ght, waist circumference, hip circumference, fat mass, and fat-free mass were linearly related to inc

92 Adjustment for physical activity, diet, fat mass, and fatfree mass in addition to demographics a

93 and locomotor activity; reduced body weight, fat mass, and food intake; and improved glucose metaboli

94 proach to prevent body weight gain, decrease fat mass, and improve insulin sensitivity.

95 lele score was strongly associated with BMI, fat mass, and lean mass (all p-values<0.001) and with ch

96 zation to investigate causal effects of BMI, fat mass, and lean mass on current asthma at age 7(1/2)

97 mma2(RG) mice exhibited reduced body weight, fat mass, and liver steatosis when fed with a high fat d

98 clinically modest, benefits on body weight, fat mass, and markers of oxidative stress and inflammato

99 ated with systemic inflammation, greater leg fat mass, and patterns of mRNA expression consistent wit

100 ng effect was a lower body weight, decreased fat mass, and reduced leptin levels.

101 ng to elevated energy expenditure, decreased fat mass, and resistance to diet-induced obesity.

102 intensities of activity were associated with fat mass, and the latter was associated with lean mass.

103 ain, there are limited data on its impact on fat mass, and to our knowledge, the contribution of gene

104 Insulin concentrations and release are fat mass- and LDs-dependent respectively.

105 common obesity-risk variant rs9939609 in the fat mass- and obesity-associated (FTO) gene was recently

106 Variants in the fat mass- and obesity-associated gene (FTO) predisposing

107 Common variants of human fat mass- and obesity-associated gene Fto have been link

108 creased body weight ( approximately 16%) and fat mass ( approximately 64%) and show glucose intoleran

109 chanisms underpinning this redistribution of fat mass are unknown.

110 ference, as estimates of total and abdominal fat mass, are now accepted as predictors of the increasi

111 ss and resting metabolic rate (RMR), but not fat mass, are strong predictors of energy intake (EI).

112 fat mass ratio, and abdominal preperitoneal fat mass area (P< 0.05) but not with childhood BMI.

113 hildhood BMI and the abdominal preperitoneal fat mass area.

114 selves against water shortage is to increase fat mass as a means for providing metabolic water.

115 amma agonist, increases food intake and body/fat mass as side-effects.

116 t SOD1 mice possess increased bodyweight and fat mass, as well as decreased energy expenditure.

117 e associated with lower total and percentage fat mass at 1 mo.

118 Median fat mass at day 2 was 0.35 kg (interquartile range, 0.25

119 wever, this could be confounded by abdominal fat mass before pregnancy because it is unknown whether

120 ) and effects on liver weight and total body fat mass being essentially independent of mERalpha (<35%

121 of body weight and significant reductions of fat mass, blood glucose, and lipid levels in DIO mice.

122 omes were a change from baseline in absolute fat mass, body weight, plasma F2-isoprostane concentrati

123 ntake was not associated with body weight or fat mass, but was significantly associated with slightly

124 In this study, we tested whether the loss of fat mass by dietary restriction would remove the major s

125 easures of body weight and fat-free mass and fat mass by dual-energy X-ray absorptiometry.

126 id ratio, and preperitoneal and subcutaneous fat mass by physical examinations, dual-energy x-ray abs

127 norhabditis elegans results in animals whose fat mass can be modulated by exposure to light, despite

128 gained more body weight and had greater body fat mass compared to the control, and these differences

129 sity had higher baseline body mass index and fat mass compared with lower genetic risk peers, and the

130 feeding, Gpr119(-/-) mice exhibited reduced fat mass, decreased levels of circulating adipokines, im

131 increased lean mass and strength, decreased fat mass, deepening of the voice, increased sexual desir

132 ptiometry studies revealed increased central fat mass (Delta+40 g), accompanied by dyslipidemic (>30%

133 ferences at discharge between RUTF groups in fat mass [Delta = 0.3 kg (95% CI: -0.6, 1.6 kg);P= 0.341

134 muciniphila enhanced its capacity to reduce fat mass development, insulin resistance and dyslipidemi

135 137, 469 g) than breastfed infants, but not fat mass (difference: -42 g; 95% CI: -299, 215 g).Formul

136 We assessed body composition and fat mass distribution before and after body weight resto

137 of hepatic fat fraction (HFF) and abdominal fat mass distribution, along with lipid profile, insulin

138 I (in kg/m(2)) >/=30 or fat mass index (FMI; fat mass divided by the square of height) >/=5.2 (men) a

139 In conclusion, obesity and high abdominal fat mass doubles the risk of psoriasis, and long-term we

140 the characteristic distal loss of muscle and fat mass during the cachectic process is still not deepl

141 ssue exactly compensated for the decrease in fat mass during weight loss.

142 A low-calorie diet (LCD) reduces fat mass excess, improves insulin sensitivity, and alter

143 ater age at peak was associated with a lower fat mass, fat mass index, and fat-free mass index at age

144 d significantly with higher body mass index, fat mass, fat percentage, and waist circumference (all P

145 atterns were observed for obesity risk, body fat mass, fat percentage, fat mass index, and waist circ

146 ations between body mass index (in kg/m(2)), fat mass, fat-free mass, and RMR with acute (1 meal) and

147 Repeated measures included weight, fat mass, fat-free mass, midupper arm circumference, tri

148 Measurements of maternal fat mass (FM) are important for studies of maternal and

149 body weight (TBW), fat-free mass (FFM), and fat mass (FM) on left ventricular (LV) geometry and func

150 ing the extent to which they correspond with fat mass (FM) or fat-free mass (FFM) during infancy.This

151 Preterm infants have a higher fat mass (FM) percentage and a lower fat-free mass (FFM)

152 mass (LBM), skeletal muscle index (SMI), and fat mass (FM) were determined pre-treatment, preoperativ

153 Fat-free mass (FFM) and fat mass (FM) were estimated with the use of dual-energy

154 Changes in weight, fat mass (FM), and fat-free mass (FFM) were used to esti

155 o included body weight, waist circumference, fat mass (FM), fat-free mass (FFM), and appendicular mas

156 ectional reference values were generated for fat mass (FM), fat-free mass (FFM), and percentage body

157 ventricular infusion of AG in mice increased fat mass (FM), in comparison with the saline-infused con

158 ipoprotein (HDL) cholesterol, triglycerides, fat mass (FM), systolic and diastolic blood pressure, fa

159 as a significant decrease in body weight and fat mass for both groups (P < 0.001 for time effect).

160 l body mass index (BMI) on offspring BMI and fat mass from childhood to early adulthood.

161 ions significantly inhibited body weight and fat mass gain compared to animals fed an HFD continuousl

162 energy expenditure (EE) predicts weight and fat mass gain in this population.

163 luence high fat diet-induced body weight and fat mass gain throughout the study.

164 s of physical activity are a risk factor for fat mass gain.

165 oving glucose tolerance and modestly slowing fat mass gain.

166 odestly improves glucose tolerance and slows fat mass gain.

167 jor role of adipocyte ABCG1 in adiposity and fat mass growth and suggests that adipose ABCG1 might re

168 t engage in regular PA exhibited higher BMI, fat mass, HC, and WC with statistical significance (P <

169 s between BMI, hip and waist circumferences, fat mass/height(2), and risk of death were shown.

170 productive function, body temperature, white fat mass, hepatic glucose output, and response to hypogl

171 After adjustment for fat mass, higher PAEE was associated with higher appendi

172 a pivotal role in regulating body weight and fat mass; however, the underlying mechanisms are poorly

173 established; it leads to decreased body and fat mass, improved glucose homeostasis and extended life

174 isms underlying the marked increase in total fat mass in 6-month-old mutants.

175 association of uMg with fasting insulin and fat mass in a general population.

176 Prenatal phthalate exposures and childhood fat mass in a New York City cohort.

177 n prenatal phthalate exposures and childhood fat mass in a prospective cohort study.

178 lower BMI z-score, waist circumference, and fat mass in boys during early childhood.

179 er, the data show that the increase in total fat mass in Cc1(-/-) mice is mainly attributed to hyperp

180 l DEHP exposure may be associated with lower fat mass in childhood.

181 Reduced fat mass in FL-PGC-1alpha(-/-) mice was closely associat

182 varian reserve and increased intra-abdominal fat mass in granddaughters, accompanied by accelerated a

183 s was positively correlated with the percent fat mass in infants (r = 0.475; P < 0.05).

184 reversed loss of body weight, lean mass, and fat mass in juvenile SIV-infected rhesus macaques.

185 was associated with the offspring's relative fat mass in late (P-trend = 0.006) but not early (P-tren

186 ght and ultimately resulted in a doubling of fat mass in males and females.

187 Accumulation of fat mass in obesity may result from hypertrophy and/or h

188 The expansion of fat mass in the obese state is due to increased adipocyt

189 Mean +/- SD weight and absolute and relative fat mass in the offspring at birth were 3769 +/- 542 g,

190 Analysis of body mass index z score and fat mass in the same cohort highlighted inconsistent est

191 in late gestation is associated with a lower fat mass in their offspring at birth.

192 A across adulthood was associated with lower fat mass (in women only) and higher appendicular lean ma

193 e associations between flavonoid intakes and fat mass.In a study of 2734 healthy, female twins aged 1

194 of dual-energy X-ray absorptiometry-derived fat mass included the limb-to-trunk fat mass ratio (FMR)

195 , that MC3R(hDM/hDM) have greater weight and fat mass, increased energy intake and feeding efficiency

196 MR and LR decreased body and fat mass, increased food intake, elevated lipid cycling

197 proanthocyanidins, are associated with lower fat mass independent of shared genetic and common enviro

198 ied homeostat for body weight regulates body fat mass independently of fat-derived leptin, revealing

199 ancy because it is unknown whether abdominal fat mass, independently of body size, affects GWG and br

200 tality risk in older subjects without taking fat mass index (FMI) and fat-free mass index (FFMI) into

201 At age 6 y, fat mass index (FMI) and fat-free mass index (FFMI) were

202 At 6 y of age, we further measured fat mass index (FMI) and fat-free mass index with dual-e

203 The fat mass index (FMI) and lean mass index (LMI) were surr

204 index z-scores (BMIZ) at 5 and 7 years, and fat mass index (FMI), percent body fat (%BF), and waist

205 The primary outcome was fat mass index (FMI), whereas the secondary outcomes wer

206 ity was defined as BMI (in kg/m(2)) >/=30 or fat mass index (FMI; fat mass divided by the square of h

207 ar skeletal muscle index (ASMI; kg/m(2)) and fat mass index (FMI; kg/m(2)) were developed.

208 a higher percentage of fat (P = 0.0001) and fat mass index (P = 0.0001) and a significantly lower bo

209 ntly higher percentage of fat (P = 0.03) and fat mass index (P = 0.04) and significantly lower body c

210 = 0.3 kg (95% CI: -0.6, 1.6 kg);P= 0.341] or fat mass index [Delta = 0.4 kg/m(2)(95% CI: -0.3, 1.1 kg

211 energy X-ray absorptiometry [DXA] determined fat mass index [FMI]) in a MR approach.

212 /m(2) (95% CI: -0.02, 0.36) higher DXA total fat mass index in mid-childhood.

213 ficantly lower BMI, percentage body fat, and fat mass index than did those born AGA, with a dose-resp

214 A high fat mass index was associated with a higher Rint (Z scor

215 uded the limb-to-trunk fat mass ratio (FMR), fat mass index, and central fat mass index.In cross-sect

216 t peak was associated with a lower fat mass, fat mass index, and fat-free mass index at age 3 y (all

217 , weight, body cell mass, percentage of fat, fat mass index, and fat-free mass index were assessed.

218 ry pattern with lower body mass index, lower fat mass index, and lower risk of being overweight, but

219 obesity risk, body fat mass, fat percentage, fat mass index, and waist circumference, but not for fat

220 was associated with lower blood pressure and fat mass index, and with more physical activity.

221 6178 children aged 6 years, we measured BMI, fat mass index, android/gynoid ratio, and preperitoneal

222 rdiometabolic risk factors (body mass index, fat mass index, blood pressure, physical activity, smoki

223 ass ratio (FMR), fat mass index, and central fat mass index.In cross-sectional multivariable analyses

224 ith dual-energy X-ray absorptiometry-derived fat mass indexes of >/=13 kg/m(2) for women (n = 15) and

225 l/d) = 454 + 38.7 (fat-free mass, kg) - 5.4 (fat mass, kg) + 4.7 (age in y) + 103 (sex: 1 = female, 0

226 bioelectrical impedance-derived measures of fat mass, lean body mass, and fat percentage.

227 scores (SDS) for weight, length/height, BMI, fat mass, lean mass, and percentage of body fat at birth

228 Body composition, including fat mass, lean mass, bone mineral content, and bone mine

229 /-) mice fed chow had decreased body weight, fat mass, leptin levels, insulin levels, and adipocyte n

230 rans-retinoic acid (ATRA) on body weight and fat mass, lipid metabolism, and retinoic acid signaling

231 show that a reduced BCAA diet promotes rapid fat mass loss without calorie restriction in obese mice.

232 feeding studies showed greater weight loss, fat mass loss, and preservation of lean mass after highe

233 .5 kg [95% CI, -5.6 to -1.4 kg]; P = 0.002), fat mass (mean difference in change, -1.5% [CI, -2.6% to

234 netic risk peers, and they gained weight and fat mass more rapidly during follow-up.

235 y weight (p160: 410 g), >5-fold less central fat mass, normal hepatic glucose efflux, and >70% reduce

236 ted higher value in the offspring's relative fat mass of 2.1% (95% CI: 0.6%, 3.7%), which corresponde

237 tio, 2.99; 99% CI, 1.35-6.59; P = .0004) and fat mass of the 80th percentile or greater at day 2 (adj

238 Moreover, BW gain and total fat masses of usually superobese ob/ob mice were signifi

239 rence, the Adipoq-LPL mice did not gain more fat mass on HFD than control mice and did not have incre

240 5a ablation or overexpression did not affect fat mass or adipocyte size.

241 Fenugreek did not alter body weight, fat mass, or food intake in either group, but did transi

242 as no effect was seen on cortical bone mass, fat mass, or thymus weight.

243 I, dual-energy X-ray absorptiometry-assessed fat mass, overweight, or obesity (International Obesity

244 ipose interleukin-6 messenger RNA levels and fat mass (p < 0.001; R = 0.64 and 0.89).

245 in LNFPI was associated with a 0.79-g lower fat mass (P = 0.02), whereas disialyl-lacto-N-tetraose a

246 P = 0.334; SED: 0.2), and loss of percentage fat mass (P = 0.179; SED: 0.2) did not differ between th

247 ere eradicated when normalised to whole-body fat mass (P = 0.416).

248 ed exclusively by associations with visceral fat mass (P=0.002), with no association seen between Del

249 :gynoid (hip) fat ratio (mean differences in fat mass per 1-standard deviation increase in PAEE were

250 iated with a tendency for a higher abdominal fat mass percentage (quartile 4 compared with quartile 1

251 offspring and a tendency for a higher total fat mass percentage among male offspring (quartile 4 com

252 easured childhood body mass index (BMI), the fat mass percentage, and the android:gynoid fat ratio wi

253 Fat mass, percentage of fat, and all thigh fat volumes d

254 Total body fat and regional fat mass percentages of the child were assessed with dua

255 ponse may be an early defect with increasing fat mass, potentially dependent on altered anabolic sign

256 , which is likely due to alterations of both fat mass quantity and qualitative changes, including a r

257 confidence interval(CI): 0.424~0.687), total fat mass (r = 0.492, 95%CI: 0.407~0.570), body mass inde

258 ong correlation between HOMA-IR and visceral fat mass (r = 0.570, 95% confidence interval(CI): 0.424~

259 with reduced levels of BCAAs lost weight and fat mass rapidly until regaining a normal weight.

260 -derived fat mass included the limb-to-trunk fat mass ratio (FMR), fat mass index, and central fat ma

261 dy fat percentage and a lower android:gynoid fat mass ratio (P< 0.05) but not with childhood BMI and

262 A high android/gynoid fat mass ratio was associated with a lower Feno (Sym% [9

263 od total-body fat percentage, android:gynoid fat mass ratio, and abdominal preperitoneal fat mass are

264 or, reduced circulating activin A, preserved fat mass, reduced lipotoxicity, and increased insulin se

265 r is critical for weight loss, anorexia, and fat mass reduction induced by central GLP-1R activation.

266 wo independent negative feedback systems for fat mass regulation.

267 ain, weight, the percentage of body fat, and fat mass remained significantly reduced from baseline th

268 2-g (P = 0.02) and 0.42-g (P = 0.02) greater fat mass, respectively.

269 ke accompanied with significant reduction in fat mass, resulting in reduced body weight and improved

270 centage of body fat (rhoG = 0.28, P = 0.04), fat mass (rhoG = 0.34, P = 0.02), waist circumference (r

271 ed on the C57/BL6J genetic background, total fat mass rises significantly with age, and glucose intol

272 ted with a reduced total BW and overall body fat mass, smaller adipocytes, and reduced leptin levels.

273 e individuals are proportional to whole-body fat mass, suggesting a compensatory down-regulation, pre

274 en consumption and decreased body weight and fat mass, suggesting an increased energy cost of cold ad

275 sequence variants (C17A+G241A) have greater fat mass than controls.

276 The PRO group had a greater loss of fat mass than did the CON group (PRO: -4.8 +/- 1.6 kg; C

277 sed to 0.8 ppm ozone had lower lean mass and fat mass than pooled control offspring.

278 ll as the uterine weight, whereas it reduced fat mass, thymus weight, and the growth plate height in

279 Body weight, BMI, fat mass, total cholesterol, LDL-C and triglyceride conc

280 alate metabolite concentrations with percent fat mass using linear mixed-effects regression models wi

281 Fat mass was 3.06% (95% CI: -5.99, -0.09%) lower among c

282 Abdominal fat mass was also higher.

283 16.3 to -3.4]), whereas a high preperitoneal fat mass was associated with a higher Feno (Sym% [95% CI

284 n in delivery to and retention of TG in WAT, fat mass was largely preserved by a compensatory increas

285 Although whole-body fat mass was not affected, visceral adipose tissue mass

286 Subcutaneous fat mass was not associated with any respiratory outcome

287 on between carbohydrate intake and offspring fat mass was observed, but the associations became signi

288 0.01) following the confinement, whole body fat mass was only reduced in the Exercise group (-1.5 kg

289 Fat mass was quantified with computed tomography imaging

290 Although the increase in body weight and fat mass was the same in both groups, hepatic, skeletal

291 Relative fat mass (%) was the primary outcome.

292 The odds of gaining >3% of fat mass were between 1.8 and 3.8 times as high for indi

293 urinary phthalate concentrations and percent fat mass were modified by child's sex.

294 in promoting increases in LBM and losses of fat mass when combined with a high volume of resistance

295 xpectedly, AhR(AdQ) mice exhibited increased fat mass when fed a standard LF or high fat (HF) diet.

296 onfounded by differences in lean mass versus fat mass when modeled on weight.

297 circumference and for fat-free mass than for fat mass, which was explained largely by height.

298 equently deliver large babies with increased fat mass, who are susceptible to perinatal complications

299 nd lower arm (P < 0.001) and leg (P = 0.001) fat masses with respect to control subjects.

300 pentapeptide inhibited weight gain, reduced fat mass without change in energy intake, and increased