ライフサイエンスコーパス: fat pad

1 from primary tumors grown within the mammary fat pad.

2 orthotopic transplantation into the mammary fat pad.

3 mammary gland on implantation into a mammary fat pad.

4 ter transplantation into the dorsal-cervical fat pad.

5 formed by injection of cells in the mammary fat pad.

6 ardiac inoculation or injection into mammary fat pad.

7 hypomorphic and does not completely fill the fat pad.

8 ve stimulator attached to the epicardial AVN fat pad.

9 n muscle tissue or the contralateral mammary fat pad.

10 an increase in the number of adipocytes per fat pad.

11 lay in the MCaIV tumor, grown in the mammary fat pad.

12 ted from synovial membrane or infra-patellar fat pad.

13 of juvenile beige adipocytes in the inguinal fat pad.

14 an breast tumor cells into the mouse mammary fat pad.

15 wth of MDA-MB-231 cells in the mouse mammary fat pad.

16 fter removal of the main interscapular brown fat pad.

17 were inhibited by removal of the parametrial fat pads.

18 latively selectively without affecting other fat pads.

19 RNA (mRNA) and protein levels in epididymal fat pads.

20 nd injected into epithelium-divested mammary fat pads.

21 lly normal ductal trees in wild-type mammary fat pads.

22 s, using both wild-type and Cav-1-/- mammary fat pads.

23 ntation of the transduced cells into mammary fat pads.

24 adipose tissue, and adipocytes within intact fat pads.

25 proximity to the coronary arteries and thick fat pads.

26 PKA in wild-type but not in caveolin-1 null fat pads.

27 were inoculated into syngeneic mice mammary fat pads.

28 strated recombination in the brown and white fat pads.

29 nd arborization of the gland tree in mammary fat pads.

30 ees within epithelial divested mouse mammary fat-pads.

31 Moreover, the infrapatellar fat pad, a soft tissue that becomes highly fibrotic in t

32 d the presence of peroxisomes in the mammary fat pad adipocytes, and functional Pxmp2 was detected in

33 lled study evaluating the impact of anterior fat pad (AFP) maintenance on postoperative atrial fibril

34 TMD cells were harvested from the mammary fat pad after transfecting MDA-MB-231 breast cancer cell

35 An acrylic plaque electrode on the fat pad allowed AG stimulation at voltages ranging from

36 The architecture of the mammary fat pad allowed for detailed analysis of the cells' inte

37 y glands transplanted into wild-type mammary fat pads also display mammary trees with extensive ducta

38 engrafted on the intra-abdominal epididymal fat pad ameliorated streptozotocin-induced hyperglycemia

39 These grow out through the fat pad and bifurcate to lay down the rudimentary ductal

40 eding, increased inflammation in the mammary fat pad and promoted mammary tumorigenesis.

41 s driving ductal elongation into the mammary fat pad and provide the first evidence that reelin signa

42 rophage content of the transplanted visceral fat pad and reduced plasma monocyte chemoattractant prot

43 om both synovial membrane and infra-patellar fat pad and therefore Anakinra can likely have an inhibi

44 enesis when epithelial ducts extend into the fat pad and undergo branching morphogenesis.

45 tes correlated with hexosamine flux in mouse fat pads and 3T3L1 adipocytes.

46 administration depleted all fat from native fat pads and from fat transplants from +/+ donors but no

47 ctivity of AMPK were increased in transgenic fat pads and in 3T3L1 adipocytes treated with glucosamin

48 onectin KO mice possessed smaller epididymal fat pads and showed reduced body weight compared with WT

49 transductants was injected into the mammary fat pads and tail veins of mice to evaluate tumor growth

50 ectant clones were injected into the mammary fat pads and tail veins, respectively, of athymic nude m

51 evidenced by the altered profile of residual fat pads and the decrease in adiponectin plasma levels i

52 lanted Shh-null mammary anlagen into cleared fat pads and under the renal capsule of wild type host m

53 issue, and impaired alveolar coverage of the fat pad, and do not produce sufficient milk for nourishm

54 subunit mRNA were expressed in the patella, fat pad, and meniscus of the rat knee and in human artic

55 seen at the Mayo Clinic who had bone marrow, fat pad, and solid organ tissue samples typed by liquid

56 ly upregulated cytokine in the obese mammary fat pad, and we observed that TNF-alpha dose-dependently

57 ding low fertility, an absence of epididymal fat pads, and a tendency to develop blepharitis.

58 a volume, and the fluid content of abdominal fat pads, and decreased hematocrit.

59 mouse, decreased the size of the parametrial fat pads, and decreased the thickness of the dermal fat

60 mmary gland development with loss of mammary fat pads, and high bone mass with loss of bone marrow fa

61 -bone thinning of the lower jaws, mandibular fat pads, and isolation of the ear region), probably ena

62 ymphoid and non-lymphoid organs (peritoneum, fat pads, and lung).

63 the intertonsillar, tonsils, parapharyngeal fat pads, and pterygoids widths maintained constant prop

64 materials such as connective tissue, buccal fat pads, and resorbable collagen membranes have been re

65 of adjacent fibrocartilages, a bursa, and a fat-pad, and is functionally much more than a focal inse

66 mal collagen I (Col-I) fibers in the mammary fat pad are axially oriented prior to branching morphoge

67 Obese fat pads are frequently undervascularized and hypoxic, l

68 ads over the next 14-21 days, and that these fat pads are morphologically similar to normal subcutane

69 ostoperatively, significantly more if entire fat pads are removed.

70 organs, including the peritoneal cavity and fat pads, are more resistant to apoptosis than those in

71 id not influence tumor growth in the mammary fat pad as a primary site.

72 lly the weight of the reproductive and renal fat pads as well as the liver.

73 Congo red stain on bone marrow biopsy and fat pad aspirate was negative for amyloid light-chain de

74 adipose tissue mass estimated by epididymal fat pad, associated with iron accumulation in adipocytes

75 hat advances only slowly through the mammary fat pad at puberty.

76 ria with their distal electrode pairs at the fat pad-atrial junctions.

77 in all mice after removal of the epididymal fat pad bearing the islet graft.

78 genetic ablation of sympathetic inputs onto fat pads blocks leptin-stimulated phosphorylation of hor

79 ssion of peroxisomal proteins in the mammary fat pad but not in liver or kidney of transgenic mice.

80 d in peripheral tissues such as the visceral fat pad, but not in the spleen.

81 ems when grafted into wild-type host mammary fat pads, but failed to undergo higher order side-branch

82 (i) decreased the weight of the parametrial fat pads by 35, 62, and 77%, respectively; (ii) decrease

83 num toxin, when injected into the epicardial fat pads can suppress atrial fibrillation inducibility.

84 ed on a high-fat diet were unable to remodel fat pad collagen architecture and display blunted weight

85 he distal electrode pair was adjacent to the fat pad containing autonomic ganglia (AG) at the veno-le

86 rapid turnover of adiponectin with multiple fat pads contributing to the plasma levels of adiponecti

87 ered at the pulmonary vein entrances (within fat pads) could be stimulated without atrial excitation.

88 the in vivo environment of the mouse mammary fat pad cultivates the growth of human breast cancer cel

89 Cosmetic blepharoplasty with fat pad debulking should be performed at least 6 months

90 ium and of whole mammary glands implicated a fat-pad dependent paracrine mechanism in the stunted phe

91 t, as opposed to an increase in all visceral fat pad depots evident after insulin replacement-induced

92 -expressing LM2-4 cells implanted in mammary fat pads developed at a slower rate, contained a lower c

93 ogenesis markers increase in parallel during fat pad development.

94 ets, either intraportal or in the epididymal fat pad, displayed similar glucose tolerance curves.

95 ting MMP-3, and increasing invasion into the fat pad during branching.

96 erminal end buds (TEBs) into the surrounding fat pad during mammary gland ductal morphogenesis.

97 Botulinum toxin injection into epicardial fat pads during coronary artery bypass graft provided su

98 ach fat pad; n=30) injection into epicardial fat pads during surgery.

99 Fat pads dynamically regulate energy storage capacity un

100 ly to components of the EGL of confluent rat fat pad ECs (RFPECs), including proteoglycans and glycos

101 re analyzed from confocal micrographs of rat fat-pad ECs after 5 h of shear stress.

102 Patients with superolateral Hoffa fat pad edema (n = 30) and a control group without edema

103 ents more likely to have superolateral Hoffa fat pad edema (P < .009).

104 oove are associated with superolateral Hoffa fat pad edema at MR imaging.

105 les were associated with superolateral Hoffa fat pad edema in the multivariable models: patellar heig

106 well-vascularized sites like the epididymal fat pad (EFP) improved graft outcomes, but only conforma

107 he confined and well-vascularized epididymal fat pad (EFP) site, a model of the human omentum, as opp

108 e negative aspects of the obesity-associated fat pad expansion.

109 ted from the post-TKA fibrotic infrapatellar fat pad express the IL-1 receptor and on exposure to IL-

110 Glycogen synthase activity in fat pad extracts was inhibited in streptozotocin (STZ)-t

111 Collectively, the mitoNEET-enriched fat pads feature a more vascularized, anti-inflammatory

112 ple to the ductal front distance, or percent fat pad filled to evaluate ductal elongation rate can be

113 parison to wildtype transplants (51% vs. 94% fat pad filled).

114 syngeneic islets implanted in the epididymal fat pad, followed by a subrenal capsular implantation of

115 thotopically transplanted into mouse mammary fat pads, followed by selection for cells that metastasi

116 lectrodes were sutured to the epicardial AVN fat pad for delivery of selective AVN-VS by a subcutaneo

117 of botulinum toxin injection into epicardial fat pads for preventing atrial tachyarrhythmias.

118 The infrapatellar fat pad (FP) and synovial fluid (SF) in the knee serve a

119 ne if parasympathetic nerves in the anterior fat pad (FP) can be stimulated at the time of coronary a

120 ach using autonomic stimulation of selective fat pad (FP) regions of the heart in a young canine mode

121 Mammary fat pads from BALB-neuT and wild-type mice (age, 40-106

122 ubcutaneous (9-fold) and epididymal (4-fold) fat pads from db/db mice treated for 8 days with the PPA

123 As predicted from these findings, fat pads from FTO-4 mice fed a high-fat diet show more n

124 In fat pads from mice deficient for lipin 1 (fld mice) and

125 To determine the mechanism, epididymal fat pads from normal wild-type (+/+) and obese (fa/fa) Z

126 kidneys, bone marrow, skin, and subcutaneous fat pads from these mice showed no abnormalities.

127 euT mice aged 76-106 d, compared with normal fat pads from younger mice and carcinomas from older mic

128 The presence of tumor cells in the mammary fat pad further enhanced the local inflammatory milieu.

129 l mice, but mesentery, femoral, and inguinal fat pads grow disproportionately larger.

130 orbed 3D scaffolds implanted in the inguinal fat pad had enhanced adipose tissue formation, suggestin

131 liver weight, and weight of the reproductive fat pad had especially high heritabilities, whereas hear

132 Adipocyte sizing profiles, fat pad histology, and DNA content show that Pio treatme

133 Acute depletion of T cells from epididymal fat pads improved insulin action in young DIO mice but d

134 epithelial ducts invade the stromal mammary fat pad in a wave of branching morphogenesis to form a c

135 D) reduced inflammation in the obese mammary fat pad in the absence of tumor cells and inhibited Py23

136 cells that were inoculated into the mammary fat pad in vivo, which also significantly reduced tumor

137 tes induced vigorous angiogenesis and formed fat pads in a mouse dorsal skin-fold chamber.

138 r deposits of s.c. fat and larger epididymal fat pads in comparison with wild-type mice.

139 crease in adipocyte number within SPARC-null fat pads in comparison with wild-type pads.

140 rved a reduction in collagen I in SPARC-null fat pads in comparison with wild-type.

141 F10DCIS.com cells were injected into mammary fat pads in immunodeficient mice, and BXL0124 was then a

142 Mammary fat pads in mice aged 80 d bore claudin-4-positive aplas

143 nd grew the cells in humanized mammary mouse fat pads in NOD/SCID mice.

144 expressing fibroblasts were able to generate fat pads in the mice.

145 ry adipocytes isolated from mouse epididymal fat pads, in response to acute activation of lipolysis.

146 Liver, kidney, and multiple fat pads increase in weight.

147 obvious cases of achillobursitis or Kager's fat pad inflammation.

148 tasis to the lungs of mice following mammary fat pad injection.

149 nts in both groups had successful epicardial fat pad injections without complications.

150 t epithelial cells to colonize mouse mammary fat pads is problematic.

151 In fat pads isolated from wild-type mice, SPARC mRNA was as

152 In both native and transplanted +/+ fat pads, large numbers of mitochondria were apparent, a

153 ctures (volume of the tongue, parapharyngeal fat pads, lateral walls, and soft palate) was similar be

154 ted human breast cancer cells in the mammary fat pad leads to statistically significant regression (8

155 o have lower body weight, smaller epididymal fat pads, lower blood levels of nonesterified fatty acid

156 Anterior fat pad maintenance did not reduce POAF incidence (34.8%

157 -173 h), no changes occurred in body weight, fat pad mass (omental and retroperitoneal), food intake,

158 Finally OP rats had decreased relative fat pad mass compared to OR rats.

159 and were associated with increased abdominal fat pad mass in Zg16(-/-) animals.

160 from weaning, exhibited increased perirenal fat pad mass relative to body weight and adipocyte hyper

161 Relative white fat-pad mass of Il18(-/-) mice was approximately 2- to 3

162 ificantly increased body mass and some white fat pad masses, markedly reduced Arc Nissl and neuropept

163 This study indicates that the epididymal fat pad maybe a useful islet transplant site in the mous

164 mammaglobin-A-positive and -negative mammary fat pad (MFP) tumors; and by peritumoral MFP injection o

165 ing miR-155 is a critical factor: in mammary fat pads miR-155 prevents tumor dissemination; whereas i

166 Using orthotopic mammary fat pad model of breast cancer (MDAMB231-Luc) in mice, w

167 thotopic primary tumor growth in the mammary fat-pad model of tumorigenesis.

168 acini in 3D cultures and repopulate mammary fat pads more efficiently than cells harvested from unin

169 30) and a control group without edema of the fat pad (n = 60) were evaluated prospectively with magne

170 or placebo (0.9% normal saline, 1 mL at each fat pad; n=30) injection into epicardial fat pads during

171 in (Xeomin, Merz, Germany; 50 U/1 mL at each fat pad; n=30) or placebo (0.9% normal saline, 1 mL at e

172 -A165b with neutralizing antibody stimulated fat pad neovascularization and restored VEGF receptor ac

173 d budding, reduced gland tree size, and less fat pad occupancy in developing mammary glands after AP-

174 vivo with a tumour implanted in the mammary fat pad of a mouse in a window chamber construct.

175 respectively, were injected into the mammary fat pad of ApoE(-/-) and wild-type (WT) females consumin

176 EO771 cells were implanted into the mammary fat pad of C57BL/6 mice.

177 e beta4-deficient cell line into the mammary fat pad of immunocompromised mice yielded significantly

178 s, cells that grew as a tumor in the mammary fat pad of nude mice (TMD-231), metastatic disease to th

179 equal numbers were injected into the dorsal fat pad of nude mice.

180 d from synovial membranes and infra-patellar fat pad of patients undergoing TKA express high levels o

181 ancer cells were coinjected into the mammary fat pad of SCID mice.

182 tumors grown in the epithelium-free mammary fat pad of severe combined immunodeficient (SCID)/Beige

183 olds were then implanted onto the epididymal fat pad of syngeneic mice with streptozotocin-induced di

184 ion of PyMT+ glands into the cleared mammary fat pad of syngeneic recipient mice, MT1-MMP-deficient t

185 of endothelial nitric oxide synthase in the fat pad of the adult rat mesentery during inhibition of

186 ation of mammary gland explants into cleared fat pad of wild type mouse recipients indicates that the

187 eficient mammary epithelium into the cleared fat pad of wild-type mice did not rescue the aberrant ma

188 ication of XPL to the herniated lower eyelid fat pads of 12 subjects resulted in an average 2-grade d

189 denocarcinomas were grown within the mammary fat pads of 19 female Fischer rats.

190 When injected into the residual fat pads of A-Zip lipodystrophic mice, these cells recon

191 egulated genes were higher in the epididymal fat pads of Adipoq-LPL mice than control mice.

192 ing Pak4 form tumors when implanted into the fat pads of athymic mice.

193 inoculated into the epithelium-free mammary fat pads of athymic nude mice.

194 vated in hyperplastic lesions in the mammary fat pads of BALB-neuT mice aged 76-106 d, compared with

195 Control 4T1 cells implanted into mammary fat pads of BALB/c mice exhibited spontaneous metastasis

196 ere orthotopically injected into the mammary fat pads of BALB/c mice, and animals were wounded locall

197 ving injection of ZR-75-1 cells into mammary fat pads of female nu/nu mice, ZR-75-1 cells expressing

198 A-MB-231 TNBC cell xenografts in the mammary fat pads of female nude mice.

199 sel density by implantation into the mammary fat pads of female severe combined immunodeficient mice.

200 expression was substantially induced in the fat pads of four rodent models of obesity/type II diabet

201 s when transplanted into the cleared mammary fat pads of host mice.

202 vely activated MEK1 into the cleared mammary fat pads of immune-competent hosts rapidly produced tumo

203 osa26R mice and inoculated them into cleared fat pads of immunocompromised females.

204 h TNBC were engrafted into humanized mammary fat pads of immunodeficient mice to create 3 independent

205 of early involution, into the mammary gland fat pads of lactating mice increased ZnT2 and Zn in lyso

206 wth of BRCA1-deficient mammary tumors in the fat pads of male mice.

207 ti-gammaH2AX-TAT accumulation in the mammary fat pads of mice aged 76-106 d, compared with control pr

208 The size of tumors transplanted into fat pads of mice with Ets2 targeted alleles was correlat

209 LCC6-DN cells were injected into the mammary fat pads of mice, and the primary xenograft tumors were

210 eration of hyperplastic lesions in humanized fat pads of NOD (nucleotide-binding oligomerization doma

211 nvolution matrix, were injected into mammary fat pads of nude mice.

212 osity in the visceral, but not subcutaneous, fat pads of ob/ob mice.

213 ansplanted serially into the cleared mammary fat pads of p53 wild-type BALB/c female to develop stabl

214 ing p,p -DDE were implanted into the mammary fat pads of prepubertal female mice.

215 rescued by transplantation into the cleared fat pads of SCID/Beige mice.

216 When injected into the mammary fat pads of severe combined immunodeficient mice, the tu

217 /antiapoptotic cytokines) in the parametrial fat pads of sham-operated control mice were 54- to 83-fo

218 atocytes and cholangiocytes, whereas if into fat pads of streptozocin-induced diabetic mice, results

219 ines are introduced in turn into the mammary fat pads of syngeneic Furth-Wistar rats, there is a sign

220 ry epithelial cells into the cleared mammary fat pads of syngeneic hosts.

221 pressing shRNAs against p63 into the mammary fat pads of syngeneic mice delays tumor growth in vivo.

222 nitiation after transplantation into mammary fat pads of syngeneic mice.

223 ts similarly have expanded structures in the fat pads of their fore- and hindfeet, but for three cent

224 cancer cells were injected into the cleared fat pads of virgin hosts at 24, 52, 80, and 150 days of

225 e tissues were transplanted into the mammary fat pads of wild-type males.

226 ize when grown orthotopically in the mammary fat pads of WT mice.

227 ary tree upon transplantation into a cleared fat-pad of a nu/nu recipient mouse.

228 Cs with non-mammary cells within the mammary fat-pads of recipient mice that had their endogenous epi

229 ade) morphology, including the predigits and fat pad, of extant elephants.

230 for 120 days if no tumor was seen in mammary fat pad or chest wall).

231 lanting placental villi to the fifth mammary fat pads or beneath the kidney capsules of Scid mice.

232 thickness dermal incisions above the mammary fat pads or remotely above the scapula 9 days later.

233 eloped tumorous lesions within their mammary fat pads over the course of 130 d.

234 these cells develop into highly vascularized fat pads over the next 14-21 days, and that these fat pa

235 on, we found that removal of the parametrial fat pads (partial lipectomy) 2 weeks before UVB irradiat

236 Removal of the parametrial fat pads (partial lipectomy) from female SKH-1 mice fed

237 teration of a cervical space, the paraspinal fat pad (PFP), can be used as an indicator at computed t

238 nsplantation into an epithelial-free mammary fat pad, PI-MECs exhibited two important features of mul

239 importantly, Cav-1-/- mammary stromal cells (fat pads) promoted the growth of both normal mammary duc

240 peripheral tissue (peritoneal cavity, lung, fat pads) reacted much less with the apoptotic marker An

241 eased penetration of mammary epithelium into fat pad, reduced epithelial proliferation, and inhibited

242 ), a neuronal blocker, was injected into the fat pad, resulting in the loss of AF inducibility in six

243 leted stem/progenitor cells into the mammary fat pad severely disrupted mammary development, and glan

244 trochlear morphology to superolateral Hoffa fat pad (SHFP) edema on magnetic resonance (MR) images i

245 Lipidomics of Pxmp2(-/-)mammary fat pad showed a decrease in the content of myristic aci

246 a >50% reduction in epididymal and perirenal fat pad size.

247 sed in adipocytes and is abundant in mammary fat pads (stroma), but it remains unknown whether loss o

248 ary buds transplanted to a wild-type mammary fat pad support outgrowth of an epithelial tree that adv

249 Since Atf4-/- mice have smaller fat pads than littermate controls, we investigated wheth

250 turbances of lipid metabolism in the mammary fat pad that in turn may result in abnormal epithelial g

251 AVN-VS was delivered to the epicardial fat pad that projects parasympathetic nerve fibers to th

252 hen the cells were injected into the mammary fat pad, there was no difference in the frequency of tum

253 and inoculated into epithelium-free mammary fat pads, they were redirected to non-carcinogenic cell

254 extravasation than in contralateral mammary fat pad tissue, which is consistent with enhanced permea

255 their ability to metastasize from the breast fat pad to distant lymph nodes for an orthotopic xenogra

256 umor xenographs and cell metastases from the fat pad to the brain were significantly suppressed by NO

257 NA knockdown of Ptprb in the cleared mammary fat pad transplant assay resulted in smaller epithelial

258 However, +/+ fat pads transplanted into fa/fa recipients did not resp

259 static sites relative to the primary mammary fat pad tumor.

260 njection of SUM1315 cells into mouse mammary fat pads, tumor burden of implanted tissues was observed

261 d the blood circulation from primary mammary fat pad tumors or are grown in end-over-end suspension c

262 mice bearing a 4T1-luc2 tumor in the mammary fat pad unambiguously revealed the presence of the tumor

263 e stroma, fully repopulate the mammary gland fat pad, undergo unperturbed ductal outgrowth and termin

264 uced by injecting 4T1 cells into the mammary fat pad was blocked by expression of the shRNA for STAT3

265 rtum, the epithelial invasion of the mammary fat pad was clearly impaired in transgenic animals.

266 The epididymal fat pad was evaluated as a site of islet transplantation

267 Interestingly, the structure of the mammary fat pad was fundamentally altered by the consequences of

268 the second transplant, the islet containing fat pad was removed to reintroduce hyperglycemia.

269 breast cancer cell lines into mouse mammary fat pads, we showed that elevated paracrine Wnt signalin

270 mia and reduction in food intake, epididymal fat pad weight declined 55% in wild-type but only 6% in

271 fter 5-7 days, food intake, body weight, and fat pad weight did not differ between Ad-GPAT1 and Ad-en

272 diet had larger adipocytes and greater total fat pad weight than wild-type (WT) mice.

273 increased maternal adiposity, as assessed by fat pad weight, and circulating maternal leptin, decreas

274 t ERalpha deletion increased adipocyte size, fat pad weight, and tissue expression and circulating le

275 oter, showed a substantial decrease in total fat pad weight.

276 ssociated with reduced whole-animal body and fat-pad weight and an improved lipid accumulation in mac

277 ct the obesity-related outcomes body weight, fat (pad) weight, and circulating and tissue levels of t

278 d less than controls and had reduced gonadal fat pad weights and lower hepatic triacylglycerol conten

279 Genetic correlations were very high among fat pad weights and serum leptin, indicating shared gene

280 on increased intramuscular lipid content and fat pad weights characterized by adipocyte hypertrophy b

281 ssion, CORT levels, body weight and inguinal fat pad weights in P27 pups raised on a 65% carbohydrate

282 r hepatic triglyceride levels and epididymal fat pad weights than in SHR harboring mutant Cd36.

283 Fat pad weights were increased 80, 90, and 500% in p27KO

284 absence of a change in total caloric intake, fat pad weights, and adipose-related measures, suggestin

285 tis (NASH) without changes to body weight or fat pad weights.

286 o(-/-) mice are fertile with normal body and fat pad weights.

287 crease in total caloric intake and dissected fat pad weights; (2) a rise in leptin and the metabolite

288 by magnetic resonance imaging and epididymal fat-pad weights.

289 from SPARC-null versus wild-type epididymal fat pads were 252 +/- 61 and 161 +/- 33 microm (means +/

290 asibility of fat transplantation, epididymal fat pads were harvested from wild-type C57BL/6J mice and

291 her chow or HFS diets, and their post-mortem fat pads were weighed.

292 reased homocysteine levels in the epididymal fat pad, which was associated with decreased mRNA levels

293 c to the lung following injection into mouse fat pads while ectopically expressed CPEB1 prevents meta

294 o factors into mice gives rise to an ectopic fat pad with the morphological and biochemical character

295 Incubation of epididymal fat pads with leptin or its i.v. injection in conscious

296 rt-term elimination of T cells in epididymal fat pad without disturbing the systemic T cell homeostas

297 in breast cancer cells isolated from mammary fat pad xenografts compared with parental cells grown in

298 rate of spontaneous metastases from mammary fat pad xenografts including novel metastases to the bra

299 for tumor growth in vivo assessed by mammary fat pad xenografts.

300 Furthermore, mammary fat pad xenotransplantation of ectopically expressed miR