ライフサイエンスコーパス: fat-free mass

1 kcal/d; P = 0.02 or 0.04 when expressed per fat-free mass).

2 t in men and women whether related to REE or fat free mass.

3 x DeltaFFM, g/d), FM is fat mass, and FFM is fat-free mass.

4 ns and tissues, and the cellular fraction of fat-free mass.

5 that reported previously between height and fat-free mass.

6 eived health but not bone mineral density or fat-free mass.

7 th increased protein utilization and loss of fat-free mass.

8 promote gains in performance, strength, and fat-free mass.

9 and late pregnancy GWGs were associated with fat-free mass.

10 y expenditure in tissue other than muscle or fat-free mass.

11 index, and waist circumference, but not for fat-free mass.

12 al impedance analysis for the calculation of fat-free mass.

13 composition variables-females: REE = 0.101 x fat-free mass + 0.025 x fat mass + 0.293 x height(3) - 0

14 - 0.185 x race + 1.643; males: REE = 0.078 x fat-free mass + 0.026 x fat mass - 2.646 x 1/height(2) -

15 %), but changes by DXA were not significant (fat-free mass, 0.2 +/- 1.2 kg; fat mass, 1.0 +/- 3.9 kg;

16 after adjustment for age, sex, fat mass, and fat-free mass (1,998+/-45 vs. 1,824+/-45 kcal/24 hours).

17 lementation plus physical activity increased fat-free mass (1.7-kg gain, P < 0.001), relative skeleta

18 with the high-PA (0.0005 +/- 0.0001 mg . kg fat-free mass(-1) . min(-1)) diet.

19 .2 +/- 2.5 mg/dL, 16.3 +/- 1.2 micromol . kg fat-free mass(-1) . min(-1)) IHTG content.

20 .1 +/- 1.2 mg/dL, 16.2 +/- 1.1 micromol . kg fat-free mass(-1) . min(-1)) or high (89.2 +/- 2.5 mg/dL

21 t protein balance was -0.50 +/- 0.07 mg x kg fat-free mass(-1) x min(-1) when the patients did not re

22 not receive rhGH and -0.39 +/- 0.04 mg x kg fat-free mass(-1) x min(-1) when the patients received r

23 of glucose rate of appearance [micromol x kg fat-free mass(-1) x min(-1)] x insulin [mU/L]) was great

24 sponse to isoproterenol (6, 12 and 24 ng (kg fat-free mass)(1) min(1): %EE 11 +/- 2, 14 +/- 3, 23 +/-

25 normoxia (225 +/- 23 vs. 128 +/- 30 nmol (kg fat free mass)(-)(1) pmol l(-)(1) min(-)(1); P =0.03), a

26 oterenol (isoprenaline): 6, 12 and 24 ng (kg fat-free mass)-1 min-1) in 25 sedentary (11 males; 51+/-

27 xidant, ascorbic acid (vitamin C; 0.04 g (kg fat-free mass)-1).

28 percentage body fat, 2.8 +/- 4.7%) and UWW (fat-free mass, -1.1 +/- 2.5 kg; fat mass, 2.1 +/- 3.6 kg

29 significant (P </= 0.02) as assessed by MC (fat-free mass, -1.5 +/- 3.7 kg; fat mass, 2.3 +/- 4.1 kg

30 Total body fat-free mass (175 +/- 96 vs 84 +/- 71, P < 0.001) and s

31 sed fat mass (24.0 g; 95% CI: 17.4, 30.5 g), fat-free mass (34.0 g; 95% CI: 21.4, 46.6 g), and percen

32 body mass (31 versus 36 mL/kg per minute) or fat-free mass (44 versus 51 mL/kg fat-free mass per minu

33 y mass (43 versus 31 mL. kg(-1). min(-1)) or fat-free mass (50 versus 43 mL/kg fat-free mass per minu

34 100% (14% versus 28%), with little change in fat-free mass (66 versus 72 kg).

35 onatal fat mass (5.2 g; 95% CI: 3.5, 6.9 g), fat-free mass (7.7 g; 95% CI: 4.5, 10.9 g), and percenta

36 loss of body mass, a 0.69 kg greater loss of fat-free mass, a 1.29% greater loss in percentage body f

37 etween HIV-infected and uninfected subjects, fat-free mass accounted for 51% in men but only 18% in w

38 were no differences in age, BMI Z score, or fat-free mass across tertiles.

39 y protein (4CL-TBPro)] and another that used fat-free mass, age, and sex [Wang equation-derived prote

40 0.02 mg center dot min(-1) center dot kg(-1) fat-free mass Ala92 homozygotes vs. 0.44 plus minus 0.02

41 The glucose infusion rate (adjusted for fat free mass and circulating insulin concentration) req

42 Fat-free mass and abdominal visceral fat were the primar

43 In the present study, body composition (fat-free mass and body cell mass) was determined by usin

44 obese children, as a result of their greater fat-free mass and body fat, than in lean children.

45 ted with the corresponding reductions in leg fat-free mass and estimated leg oxygen consumption (both

46 re adjusted with the use of fat-free mass or fat-free mass and fat mass as covariates.

47 nificantly, despite significant increases in fat-free mass and fat mass between the visits.

48 dition to serial measures of body weight and fat-free mass and fat mass by dual-energy X-ray absorpti

49 increased cardiac output is related to both fat-free mass and fat mass in obesity.

50 Fat-free mass and fat mass were measured by dual-energy

51 Adjusted for weight or fat-free mass and fat mass, energy requirements still di

52 ge in body composition, specifically loss of fat-free mass and gain in fat mass, in older adults is a

53 r kilogram of body weight or per kilogram of fat-free mass and in all quartiles of self-reported leis

54 he complex associations between fat mass and fat-free mass and Max(dur) in a population setting.

55 Whole-body protein synthesis per fat-free mass and muscle protein fractional synthesis ra

56 in healthy men; (2) reductions in both limb fat-free mass and oxygen consumption are related to the

57 BK(2)R expression is associated with reduced fat-free mass and quadriceps strength in COPD.

58 BK(2)R gene polymorphism is a determinant of fat-free mass and quadriceps strength in patients with C

59 Evidence suggests that fat-free mass and resting metabolic rate (RMR), but not

60 taining diet contributed to greater gains in fat-free mass and skeletal muscle mass with RT in older

61 teady-state glucose utilization adjusted for fat-free mass and steady-state insulin concentration [M/

62 th age-appropriate exercise, not only boosts fat-free mass and strength but also enhances other aspec

63 ontinue to question whether the decreases in fat-free mass and total body water observed in all subje

64 he Max(dur) was 1.3 s longer per kilogram of fat-free mass and was 0.5 s shorter per kilogram of fat-

65 he Max(dur) was 2.7 s longer per kilogram of fat-free mass and was 2.8 s shorter per kilogram of fat-

66 onents (SM, residual mass, AT-free mass, and fat-free mass), and liver scaled to height with powers o

67 -obese women after controlling for fat mass, fat-free mass, and age (P < 0.0017).

68 Sex-specific, mixed-effects models with REE, fat-free mass, and age as fixed effects were used to tes

69 y was to examine the scaling of weight, fat, fat-free mass, and bone mineral content to height.

70 346) that included related estimates of fat, fat-free mass, and bone mineral mass.

71 Dairy intake was related to sex, fat-free mass, and daily intakes of energy, protein, and

72 2), P = 0.02 for RMR; adjusted for age, sex, fat-free mass, and fat mass).

73 with CF was impaired on the basis of height, fat-free mass, and fat mass, when observed longitudinall

74 outcome measures were adult body mass index, fat-free mass, and fat mass.

75 e was associated with adult body mass index, fat-free mass, and fat mass.

76 However, body mass index also reflects fat-free mass, and few studies have examined the relatio

77 ceived testosterone had a slight increase in fat-free mass, and men in both treatment groups had an i

78 ween body mass index (in kg/m(2)), fat mass, fat-free mass, and RMR with acute (1 meal) and daily (24

79 ody mass index, fat mass, relative fat mass, fat-free mass, and waist circumference at 17 y (P < 0.00

80 h hormone secretion, significantly increased fat-free mass, and was generally well tolerated.

81 x, and change in fat mass, visceral fat, and fat-free mass; and was similar in children at low, mediu

82 o age 15 y (P < 0.001), and AEE adjusted for fat-free mass appeared to decrease over the same interva

83 Fat-free mass as well as intraabdominal fat correlated t

84 of weight gained in the etanercept group was fat-free mass, as compared with only 14% in the methotre

85 The fat-free mass, as measured by whole-body potassium scann

86 ceps maximum voluntary contraction force and fat-free mass assessed by bioelectrical impedance analys

87 he boys with and without CF was observed for fat-free mass assessed by skinfold-thickness measurement

88 stress were related to lower infant percent fat-free mass at 5 months of age, particularly in offspr

89 ocity was positively associated with fat and fat-free mass at age 3 y (all P < 0.0001), whereas a lat

90 associated with higher neonatal fat mass and fat-free mass but not percentage of body fat relative to

91 ly associated with adult body mass index and fat-free mass but not with measures of adult fat mass.

92 , designed primarily to assess increments in fat-free mass by a deuterium dilution technique and chan

93 by computed tomography scan and body fat and fat-free mass by dual-energy X-ray absorptiometry.

94 VD mortality than did fit men in all fat and fat-free mass categories.

95 rth change was more strongly associated with fat-free mass change (r(2) = 0.22, P < 0.01) than with f

96 diet vs. -4.8 kg with the low-fat diet) than fat-free mass (change, -3.3 kg vs. -2.4 kg, respectively

97 vels of resting energy expenditure and lower fat-free mass compared to healthy controls.

98 d feeding efficiency, but reduced length and fat-free mass compared with MC3R(hWT/hWT).

99 Mean fat-free mass decreased in the placebo group but increas

100 consistency of the relation between BCM and fat-free mass decreases with increasing weight loss, the

101 V-associated weight loss, the weight lost as fat-free mass depends on the initial percentage of body

102 Body mass index and fat-free mass did not differ significantly between genot

103 Increased fat-free mass did not result in changes in strength or f

104 copenia) was defined as fat-free mass index (fat-free mass divided by the square of height) <17.5 (me

105 tein at levels exceeding the RDA may protect fat-free mass during short-term weight loss.

106 Fat-free mass, energy intake, and sex accounted for 86%

107 When the model relating fat-free mass, energy intake, and sex to energy expendit

108 Fat-free mass explained the sex difference in RMR, but b

109 ndent variables such as sex, weight, height, fat-free mass, fat mass, age, and hemoglobin concentrati

110 In the NT group, changes in fat-free mass, fat mass, and percentage body fat were si

111 es used for validation, model predictions of fat-free mass, fat mass, and total body water matched ac

112 After adjustment for fat-free mass, fat mass, height, overweight status, race

113 for human studies that reported the outcomes fat-free mass, fat mass, or the percentage of fat mass i

114 are tailored to metabolic variables, such as fat-free mass, fat mass, resting metabolic rate (RMR), a

115 better than single predictions at predicting fat-free mass, fat mass, total body water, and RMR.

116 l study that measured gestational-changes in fat-free mass, fat mass, total body water, and total ene

117 ency results in an immediate decrease in the fat free mass (FFM).

118 1.0 for serotonin levels, and.45 and.76 for fat free mass (FFM).

119 ndependent of changes in body weight (BW) or fat free mass (FFM).

120 and BMI (adjusted P < 0.001) or fat mass and fat-free mass (FFM) (+3.3%; adjusted P < 0.001)].

121 (PAEE. kg(-1). d(-1)), 3) PAEE adjusted for fat-free mass (FFM) (PAEE. kg FFM(-1). d(-1)), and 4) th

122 Relevant data adjusted for fat-free mass (FFM) also were extracted.

123 Fat-free mass (FFM) and fat mass (FM) (4-compartment mod

124 e natural longitudinal patterns of change in fat-free mass (FFM) and fat mass (FM) in older adults an

125 Fat-free mass (FFM) and fat mass (FM) were estimated by

126 Fat-free mass (FFM) and fat mass (FM) were estimated wit

127 Fat-free mass (FFM) and fat mass (FM) were measured by b

128 Up to age 52 y, fat-free mass (FFM) and fat mass (FM) were positively as

129 Before and after this period, fat-free mass (FFM) and fat mass were assessed by using

130 ronic obstructive pulmonary disease, loss of fat-free mass (FFM) and loss of bone mineral density (BM

131 ive equations for total body water (TBW) and fat-free mass (FFM) and to refit the best-performing mod

132 ) and activity-related EE (AEE) adjusted for fat-free mass (FFM) and total body fat, mothers' and fat

133 d the hypothesis that both fat mass (FM) and fat-free mass (FFM) are proportionately lower in childre

134 sociated with decreases in fat mass (FM) and fat-free mass (FFM) by 72.0% and 28.0%, respectively.

135 e subject to error arising from variation in fat-free mass (FFM) composition.

136 These show that fat-free mass (FFM) declines with age.

137 which they correspond with fat mass (FM) or fat-free mass (FFM) during infancy.This study aimed to e

138 provide reference data for fat mass (FM) and fat-free mass (FFM) from birth to the age of 6 mo from a

139 MI), total body fat (BF), percentage BF, and fat-free mass (FFM) from underwater weighing of 102 men

140 igher total and regional adiposity and lower fat-free mass (FFM) in healthy women across the adult ag

141 ge body fat (PBF), total body fat (TBF), and fat-free mass (FFM) in the adult population in the Unite

142 Fat-free mass (FFM) loss differed significantly by sex (

143 All studies agree that variation in fat-free mass (FFM) plays a major role, but effects of f

144 higher fat mass (FM) percentage and a lower fat-free mass (FFM) than do term infants at the time of

145 resting energy expenditure (REE) relative to fat-free mass (FFM) than do whites.

146 Fat-free mass (FFM) was assessed by deuterium dilution t

147 water (TBW) was not significantly different, fat-free mass (FFM) was significantly lower (P < 0.05),

148 Fat mass (FM) and fat-free mass (FFM) were assessed by bioimpedance.

149 d by magnetic resonance imaging, and fat and fat-free mass (FFM) were measured by dual-energy X-ray a

150 Changes in weight, fat mass (FM), and fat-free mass (FFM) were used to estimate change in ener

151 control for differences in fat mass (FM) and fat-free mass (FFM) when comparing RMR values.

152 1.3 vs. 29 +/- 3.3 ml min(-1) 100 g forearm fat-free mass (FFM)(-1) and 21.7 +/- 1.2 vs. 33.6 +/- 4.

153 %, from 38.8 +/- 1.2 to 46.0 +/- 1.0 ml x kg fat-free mass (FFM)(-1) x min(-1) (P < 0.05).

154 llowing doses: 0, 2.5, 5, and 10 micro g. kg fat-free mass (FFM)(-1).h(-1).

155 went two 180 min hyperinsulinaemic (2 mU (kg fat-free mass (FFM))(-1) min(-1)), hypoglycaemic (3.33 m

156 height, and body composition [fat mass (FM), fat-free mass (FFM), and %BF] were measured with dual-en

157 weight, waist circumference, fat mass (FM), fat-free mass (FFM), and appendicular mass by dual-energ

158 etween birth weight and later fat mass (FM), fat-free mass (FFM), and fat distribution.

159 The impact of body size, fat-free mass (FFM), and fat mass (FM) on cardiorespirat

160 ifferent effects of total body weight (TBW), fat-free mass (FFM), and fat mass (FM) on left ventricul

161 Total body mass, fat-free mass (FFM), and fat mass (FM) were assessed by

162 d body-composition [including fat mass (FM), fat-free mass (FFM), and percentage body fat (%BF) evalu

163 nce values were generated for fat mass (FM), fat-free mass (FFM), and percentage body fat (PBF) by ge

164 e kinetics under fasting and fed conditions, fat-free mass (FFM), and serum insulin were determined o

165 Fat mass (FM), fat-free mass (FFM), and weight were measured by DXA and

166 on, body mass index (BMI) z score, fat mass, fat-free mass (FFM), arm muscle circumference (AMC), for

167 eadths and measures of total body fat (TBF), fat-free mass (FFM), bone mineral content (BMC), and bon

168 nt models (2CMs) to assess fat mass (FM) and fat-free mass (FFM), but to our knowledge no study has u

169 bottom 15th percentiles of BMR, adjusted for fat-free mass (FFM), fat mass, age, and sex, were identi

170 Data from the outcome variables fat-free mass (FFM), fat mass, type I and II muscle fibe

171 ion between the weight-for-age z score (WZ), fat-free mass (FFM), percentage of body fat (%BF), and m

172 When expressed relative to fat-free mass (FFM), the MFO was 10.8 +/- 3.2 mg . min(-

173 Adaptive thermogenesis (AT) is the fat-free mass (FFM)-independent reduction of resting ene

174 riods in infancy and later fat mass (FM) and fat-free mass (FFM).

175 (D(FFM)) and hydration fraction (HF(FFM)) of fat-free mass (FFM).

176 eight changes because of concurrent gains in fat-free mass (FFM).

177 EE was standardized by fat-free mass (FFM).

178 0.12 mmol/L) and mitigation of reductions in fat-free mass (FFM; 0.43 kg; 95% CI: 0.09, 0.78 kg) and

179 with WL (39.2 +/- 0.8 vs. 39.8 +/- 1.1 ml x fat-free mass [FFM](-1) x min(-1)).

180 y (6-min-walk distance [6MWD]), muscle mass (fat-free mass [FFM]), and systemic inflammation (fibrino

181 glucose disposal >8 mg x min(-1) x kg(-1) of fat-free mass [FFM], n = 58; impaired = glucose disposal

182 - 0.7 vs. 22.9 +/- 1.1 ml x min(-1) x kg(-1) fat-free mass [FFM]; P = 0.011) and insulin sensitivity

183 to ART initiation were BMI; height adjusted fat free mass (FFMI); height adjusted fat mass (FMI), an

184 nalyses adjusted for age, sex, fat mass, and fat-free mass, free T(3) was a positive predictor of SMR

185 The mean fat-free mass gained by the dutasteride groups was 0.6 k

186 The mean fat-free mass gained by the placebo groups was 0.8 kg (9

187 Recently, fat-free mass has been considered to be more strongly co

188 ass in both men and women and independent of fat-free mass, height, race, television watching, physic

189 orrespond with increased adiposity and lower fat-free mass in early infancy.

190 he leg-to-leg BIA system accurately assessed fat-free mass in obese and nonobese women, and changes i

191 Fat-free mass in obese people contributed little to the

192 We observed similar results for fat and fat-free mass in relation to mortality.

193 The assessment of fat mass and fat-free mass in relation to the symptom-limited maximal

194 Changes in fat-free mass in response to graded testosterone doses d

195 e mass and was 2.8 s shorter per kilogram of fat-free mass in the highest fat-mass quartile.

196 e mass and was 0.5 s shorter per kilogram of fat-free mass in the highest fat-mass quartile.

197 e initial percentage of body fat and loss of fat-free mass in the women.

198 protein); and 3) maintenance or accretion of fat-free mass--in some individuals, a moderately higher

199 at decreased (-2.2 +/- 0.7 kg; P = 0.02) and fat-free mass increased (2.5 +/- 0.6 kg; P = 0.01).

200 In the ET group, fat-free mass increased significantly as assessed by DXA

201 ith training, whereas total body density and fat-free mass increased.

202 There was no difference in fat-free mass increment in WF or WF-L compared with CSB+

203 mass (0.91 [95% CI: 0.87 to 0.96]) but not a fat-free mass index (0.97 [95% CI: 0.92 to 1.03]).

204 Low muscle mass (sarcopenia) was defined as fat-free mass index (fat-free mass divided by the square

205 ects without taking fat mass index (FMI) and fat-free mass index (FFMI) into account.

206 At age 6 y, fat mass index (FMI) and fat-free mass index (FFMI) were measured with dual-energ

207 for height to give fat mass index (FMI) and fat-free mass index (FFMI), respectively.

208 The mean (+/-SD) fat-free mass index (in kg/m(2)) was significantly lower

209 Thirty-three patients had a low fat-free mass index (kg/m(2)), 17 of whom had a normal b

210 lower body cell mass index (P = 0.0001) and fat-free mass index (P = 0.003) than did matched control

211 The same outcomes were generated for the fat-free mass index and fat mass index.

212 d with a lower fat mass, fat mass index, and fat-free mass index at age 3 y (all P < 0.001).

213 The fat-free mass index tended to be lower in Asians than in

214 A high fat-free mass index was associated with only a decreased

215 mass, percentage of fat, fat mass index, and fat-free mass index were assessed.

216 we further measured fat mass index (FMI) and fat-free mass index with dual-energy X-ray absorptiometr

217 lations of body composition (fat mass index, fat-free mass index) and adiposity (body mass index, wai

218 By contrast, comparisons of fat-free mass indicated lower concentrations than the co

219 ivided by height in meters squared); fat and fat-free mass indices and percentage of body fat from bi

220 have normal-to-high body fat stores, loss of fat-free mass is independent of the initial percentage o

221 Leg blood flow normalized for leg fat-free mass is preserved with age in women taking chro

222 Body fatness, not fat-free mass, is associated with cardiovascular disease

223 MRp (MJ/d) = 0.024 x fat mass (kg) + 0.102 x fat-free mass (kg) + 0.85.

224 percentage of body fat compared with loss of fat-free mass (kg) suggested a nonlinear relation over t

225 on was included, TDEE (kcal/d) = 454 + 38.7 (fat-free mass, kg) - 5.4 (fat mass, kg) + 4.7 (age in y)

226 , BP control, anemia, sodium intake, income, fat-free mass, left ventricular mass index, and ejection

227 We propose that RMR (largely determined by fat-free mass) may be a marker of energy intake and coul

228 Body weight, fat mass, and fat-free mass (measured by dual-energy x-ray absorptiome

229 Changes in fat and fat-free mass, measured by dual-energy X-ray absorptiome

230 Repeated measures included weight, fat mass, fat-free mass, midupper arm circumference, triceps skinf

231 t-free mass min); 95% CI: 0.4, 0.5 mumol/(kg fat-free mass min); 3 mo-mean 1.4 mumol/(kg fat-free mas

232 ed nearly 3-fold [before-mean: 0.5 mumol/(kg fat-free mass min); 95% CI: 0.4, 0.5 mumol/(kg fat-free

233 fat-free mass min); 3 mo-mean 1.4 mumol/(kg fat-free mass min); 95% CI: 0.8, 1.9 mumol/(kg fat-free

234 t-free mass min); 95% CI: 0.8, 1.9 mumol/(kg fat-free mass min); P = 0.002], and time to peak was muc

235 .4 +/- 1.2 vs. 8.1 +/- 0.8 10(-2) mg. kg(-1) fat-free mass. min(-1). microU(-1). ml(-1)).

236 (6.64 +/- 0.52 vs. 8.45 +/- 0.54 mg. kg(-1) fat-free mass. min(-1); P < 0.05 vs. FH(-)).

237 e-body protein synthesis (5.05 +/- 1.3 mg/kg fat-free mass/min versus 3.22 +/- 0.3 mg/kg fat-free mas

238 0.041 +/- 0.005 vs. 0.051 +/- 0.007 mumol/kg fat-free mass/min) (P < 0.05 for all).

239 fat-free mass/min versus 3.22 +/- 0.3 mg/kg fat-free mass/min; P < 0.05).

240 the variance of measured REE per kilogram of fat-free mass (model R(2) = 0.66, P < 0.0001).

241 ometry, based on a 2-component (fat mass and fat-free mass) model.

242 comitant ingestion of whey protein (0.6 g/kg fat-free mass; n = 11) or leucine that matched the amoun

243 d with LMI and, after adjustment for sex and fat-free mass, negatively associated with FMI but not wi

244 24.3 +/- 4.2 vs. 59.6 +/- 10.0 micro mol. kg fat-free mass of the leg(-1). min(-1); P < 0.02) were al

245 ture variables were adjusted with the use of fat-free mass or fat-free mass and fat mass as covariate

246 ables (age, sex, ethnic group, fat mass, and fat-free mass or lean tissue mass) was assessed.

247 were eliminated after adjustment for either fat-free mass or muscle mass.

248 energy intake from dietary records, sex, and fat-free mass (or weight and height).

249 =0.68 for adiposity outcomes and >/=0.80 for fat-free mass outcomes.

250 Changes in body mass, fat mass, and fat-free mass over 1 y were assessed by the 3 methods.

251 s and protein-mineral mass were not changed, fat-free mass (P = 0.004) and total body water (P = 0.01

252 ormal basal metabolic rate when adjusted for fat-free mass, partial hypogonadotropic hypogonadism and

253 . infused with insulin (1.5 milliunits/kg of fat-free mass per min) while clamping glucose, amino aci

254 or high-dose insulin (0.25 and 1.5 mU/kg of fat-free mass per min, respectively), and glucose.

255 and PO (4.43 +/- 0.7 and 5.71 +/- 1.2 mg/kg fat-free mass per min, respectively), compared with a ne

256 ral balance with control (0.25 +/- 0.5 mg/kg fat-free mass per min; P = 0.002 and <0.001 for IDPN ver

257 in(-1)) or fat-free mass (50 versus 43 mL/kg fat-free mass per minute).

258 minute) or fat-free mass (44 versus 51 mL/kg fat-free mass per minute).

259 Leg fat-free mass (r = -0.48) and estimated leg oxygen consu

260 were shown for TBF (r = 0.32, P = 0.035) and fat-free mass (r = 0.34, P = 0.025) between values (kg)

261 reflect prenatal and maternal influences on fat-free mass rather than on fat mass in older people.

262 s: body weight, BMI, fat mass, visceral fat, fat-free mass, resting metabolic rate (RMR), VO2max, lei

263 g/kg were associated with 0.60 kg additional fat-free mass retention compared with diets with protein

264 The primary outcome was change in fat-free mass; secondary outcomes: changes in fat mass,

265 Body weights and fat free mass significantly decreased in the placebo gro

266 ucation, higher body mass index, and greater fat-free mass, social desirability, and dissatisfaction

267 controlled physical activity would increase fat-free mass, strength, physical function, and quality

268 hereas adjustment of remethylation rates for fat-free mass tended to attenuate the sex-related effect

269 weighed more and had more body cell mass and fat-free mass than did control women, although control w

270 ference than for waist circumference and for fat-free mass than for fat mass, which was explained lar

271 included in the model in place of whole-body fat-free mass, the ethnic difference in REE decreased.

272 Adjusted for fat-free mass, the relationship was stronger (linear tre

273 The greatest predictor of ERs was fat-free mass, then ambulatory status.

274 The slope of the line relating fat-free mass to resting metabolic rate was the same in

275 Age, height, body weight, BMI, fat-free mass, visceral fat, energy expenditure, respira

276 Femoral blood flow normalized for leg fat-free mass was 17 % lower (P < 0.01) in the postmenop

277 igher TDEE and RMR than did AA children when fat-free mass was considered.

278 Loss of fat-free mass was dependent on the initial percentage of

279 Fat-free mass was estimated by dual-energy X-ray absorpt

280 In formula-fed infants, fat-free mass was higher at 3-4 mo [mean difference (95%

281 When trunk fat-free mass was included in the model in place of whol

282 oportion of weight loss due to reductions in fat-free mass was lower (P<0.05) and the loss of fat mas

283 y the Max(dur), although the contribution of fat-free mass was positive in thinner people.

284 The hydration of the fat-free mass was significantly higher in the fatter chi

285 Leg fat-free mass was smaller in the postmenopausal groups (

286 Questionnaire), and EI (food buffet or menu).Fat-free mass was the best predictor of acute EI (R(2) =

287 As a percentage of fat-free mass, water decreases throughout infancy wherea

288 HIV infection on weight, body cell mass, and fat-free mass were analyzed by using both unadjusted and

289 linic visits, at which time weight, fat, and fat-free mass were determined.

290 cumference, hip circumference, fat mass, and fat-free mass were linearly related to incident AF.

291 To adjust for body size, fat mass and fat-free mass were normalized for height and expressed a

292 tween the dutasteride and placebo groups for fat-free mass were not significant (P = .18).

293 nd total body water, and 4-component fat and fat-free masses were calculated.

294 e while serum creatinine was correlated with fat-free mass, which is a marker of muscle mass.

295 ying in dietary protein on 2-year changes in fat-free mass, whole body total percentage of fat mass,

296 Fat mass modified the association of fat-free mass with the Max(dur) (2-way interaction P < 0

297 however, when the analysis was adjusted for fat-free mass, women had significantly higher TEE than d

298 TGD) (6.9 +/- 0.7 to 9.2 +/- 0.8 mg x kg(-1) fat-free mass x min(-1)) (all P < 0.001), and decreased

299 - 5.1 vs. 22.9 +/- 2.6 micromol x kg(-1) leg fat-free mass x min(-1), P < 0.05).

300 Percent body fat, fat-free mass z scores, and triceps skinfold z scores de