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1 ated EOMs did not show evidence of increased fatigability.
2            Treatment had no effect on muscle fatigability.
3 atients showed increased tendency for muscle fatigability.
4 the degree of which is referred to cognitive fatigability.
5 ability, which explains the patient's muscle fatigability.
6 ere NMJs dismantlement, muscle weakness, and fatigability.
7 standing individual differences in cognitive fatigability and developing interventions for clinical c
8 uing exercise protocol were used to quantify fatigability and the local muscle hemodynamic profile.
9 mal forces and showed only a 10% increase in fatigability, and no signs of oxidative damage or apopto
10                    The muscular weakness and fatigability associated with myasthenia gravis are engen
11 roximal muscles, without short-term clinical fatigability but with marked variation in strength over
12 h (Pt) and tetanic (Po) tensions, as well as fatigability during 5 secs of nerve stimulation at 50, 1
13 ation class II-III patients with EI and high fatigability exhibited significantly faster rates of exe
14 ative capacity compared with healthy and low-fatigability HF patients, suggesting that SM metabolism
15 fects in glycolytic metabolism and increased fatigability in dystrophic muscle may be caused in part
16 ies occur and contribute to EI and increased fatigability in HF patients with reduced or preserved ej
17 tal muscle RyR have been linked to increased fatigability in HF.
18 pose of this study was to compare quadriceps fatigability in patients with varying severity of chroni
19 ) transport compromised and locomotor muscle fatigability is exacerbated with a combined net effect o
20 rEF and HFpEF patients with EI and increased fatigability manifest early, rapid exercise-induced decl
21               Immobilization reduced Pt, Po, fatigability, muscle mass, and fiber cross-sectional are
22 ate decline than did HFrEF patients with low fatigability (New York Heart Association class I), despi
23                                    Mice have fatigability of limb muscles, electrophysiological evide
24 he loss of nNOS contributes to the increased fatigability of mdx mice, a model of DMD.
25                                The increased fatigability of skalpha2(-/-) muscles is reproduced in c
26                 Our aim was to determine the fatigability of thenar motor units paralysed chronically
27        An energetic plantar flexion exercise fatigability test and magnetic resonance spectroscopy we
28 chronic paralysis did not limit the range of fatigability typically found for thenar units, only its
29                      Furthermore, quadriceps fatigability was evaluated in 12 patients by measuring u
30 smission and metabolic diseases cause muscle fatigability, which is characterised by failure to susta

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