戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1   The mutant viruses remained susceptible to favipiravir.
2 reatment of monoclonal antibodies (ZMAb) and favipiravir.
3  treated with a combination of ribavirin and favipiravir.
4  in Hartley guinea pigs, we demonstrate that favipiravir, a broad-spectrum antiviral agent and leadin
5                               In the case of favipiravir, a polymerase inhibitor with activity agains
6 d trial, in which all patients would receive favipiravir along with standardized care.
7                                              Favipiravir, an antiviral developed for the treatment of
8   We found a synergistic interaction between favipiravir and ribavirin in vitro and an increased surv
9                   These results suggest that favipiravir can induce norovirus mutagenesis in vivo, wh
10               All participants received oral favipiravir (day 0: 6,000 mg; day 1 to day 9: 2,400 mg/d
11 roviding a proof-of-principle for the use of favipiravir derivatives or mutagenic nucleosides in the
12 upport the further preclinical evaluation of favipiravir for Lassa fever and other VHFs.
13                         One of these, T-705 (favipiravir), has a mechanism of action that is not full
14 lect data on the safety and effectiveness of favipiravir in reducing mortality and viral load in pati
15                       They also suggest that favipiravir monotherapy merits further study in patients
16 ow that the broad-range antiviral nucleoside favipiravir reduces viral load in vivo by exerting antiv
17                                              Favipiravir remained highly effective against lethal LAS
18     In this model, once daily treatment with favipiravir significantly reduced viral titers in tissue
19                                              Favipiravir suppressed Lassa virus replication in cell c
20      We studied the therapeutic potential of favipiravir (T-705) for Lassa fever, both alone and in c
21                            Susceptibility to favipiravir (T-705) was assessed using plaque reduction
22 TAT2 knockout (KO) hamsters is responsive to favipiravir treatment, which protected all animals from
23                                         Oral favipiravir was part of the current standard of care in
24                                              Favipiravir was well tolerated.
25 nificantly different between adults starting favipiravir within <72 h of symptoms compared to others.

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。