ライフサイエンスコーパス: feather bud

1 lized growth zone (LoGZ) to regulate growth (feather buds).

2 pressed in the developing chick limb bud and feather bud.

3 ablished; the BDNF in dermis is localized to feather buds.

4 HH, NCAM, Tenascin-C) were characteristic of feather buds.

5 ectoderm results in the ectopic formation of feather buds.

6 nvolved in establishing the A-P asymmetry of feather buds.

7 o cartilage rudiments and then in developing feather buds.

8 (without feathers), FGFs can also induce new feather buds.

9 ion and a region with an increased number of feather buds.

10 donor dermis induces host epidermis to form feather buds according to the spatial pattern and timeta

11 s characteristic of the middle and posterior feather buds and suggest that P-D elongation of vertebra

12 In the embryonic chicken skin, feather buds and the intervening interbud tissue form in

13 ng parallel of molecular profiles in the A-P feather buds and the ventral-dorsal (V-D) Drosophila app

14 n the dermis and epidermis of the developing feather buds and their expression is induced in embryoni

15 In addition, feather buds are often observed on some of the scuta sca

16 Chick embryonic feather buds arise in a distinct spatial and temporal pa

17 appendage induction using developing chicken feather buds as a model.

18 ional insights into pattern formation in the feather bud can be inferred from the effects of altered

19 propose a dual role for Delta-1 in promoting feather bud development and in lateral inhibition.

20 e interfered with Shh signaling during early feather bud development and observed a dramatic change i

21 e molecule(s) responsible by using the chick feather bud development as a model.

22 elopment and is expressed in early stages of feather bud development though its role has not been def

23 In the early stages of feather bud development, Delta-1 and Notch-1 are localiz

24 ic mechanism plays a primary role in hair or feather bud development, we are beginning to discover th

25 er morphogenic signalling systems to control feather bud development.

26 genic and morphogenetic processes throughout feather bud development.

27 dynamic pattern from the earliest stages of feather bud development.

28 After this procedure, existing feather buds disappeared and new buds were regenerated.

29 protein kinase C is suppressed in the normal feather bud domain.

30 r buds, representing FGFs' ability to expand feather bud domains in developing skin.

31 rs such as the FGF receptor, was enriched in feather bud domains.

32 he ectoderm of the forming hair follicle and feather bud during normal development.

33 from entering the epidermis and the core of feather buds during development in vivo.

34 irectional cell rearrangements and abolishes feather bud elongation.

35 ts message is predominantly expressed in the feather bud epithelium, and the protein is enriched in t

36 f interbud fate and increased acquisition of feather bud fate.

37 Feather buds form sequentially in a hexagonal array.

38 an inhibitor of tyrosine kinase, suppressed feather bud formation and the effect of FGF.

39 entiate, the forced expression of Shh causes feather bud formation.

40 in induction of Notch-1 and-2 and a loss of feather buds from the embryo in either large or small pa

41 he chick skin leads to both feather loss and feather bud fusions, suggesting that DLX proteins play a

42 d gene expression, with a concurrent loss of feather bud gene expression and morphology.

43 The spacing of feather buds in a tract is thought to arise from the int

44 blishment of the periodic pattern of hair or feather buds in the developing skin.

45 rphogenetic protein (BMP) signaling from the feather bud inhibits bud formation in the adjacent inter

46 d tenascin, molecules that are important for feather bud initiation as well as bud outgrowth and morp

47 in the skin in a localized pattern prior to feather bud initiation.

48 The development of feather buds is a highly ordered process involving epith

49 rizing activity," localized in the posterior feather bud, is necessary and sufficient to mediate the

50 ted in the growth and differentiation of the feather buds, little is known about how the discrete pat

51 ce new Hox C6 and NCAM microgradients in the feather bud mesenchyme.

52 Delta-1 expressing cells differentiate into feather buds more quickly than normal and inhibit their

53 ceptor impair the epithelial contribution to feather bud morphogenesis, while the dermal contribution

54 inhibiting this transcription factor alters feather bud number and size in a stage-specific manner.

55 pment by the localized molecular reversal of feather bud polarity.

56 he midline, FGFs led to fusion of developing feather buds, representing FGFs' ability to expand feath

57 At the short feather bud stage, the localized growth zones shifted to

58 -mesenchymal signaling interactions generate feather buds that are neatly arrayed in space and time.

59 Like normal feather buds, the newly induced buds express Shh.

60 ure system to interfere with EphA4 levels in feather buds using anti-sense oligonucleotides, demonstr

61 In feather tracts, short, wide, and curled feather buds with abnormal morphology and random orienta

62 In apteric and scale-producing regions, new feather buds with normal-appearing follicle sheaths, der