ライフサイエンスコーパス: fecal

1 ularly respiratory allergies, with increased fecal abundance of R gnavus.

2 Soiling, fecal accidents, rectal sensation, and ability to withho

3 They feed and breed in fecal and decaying organic matter, but the microbiome th

4 Analysis of 319 fecal and environmental samples revealed that the commun

5 Fecal and ileal microbiota were analyzed by pyrosequenci

6 We collected information on the fecal and mucosa-associated microbiota of patients with

7 In analyzing fecal and mucosal microbiota from patients with IBS and

8 We collected fecal and mucosal samples from adult patients who met th

9 Fecal and rectal biopsy-associated microbiota compositio

10 gut microbiota composition, and inflammation.Fecal and serum menaquinone concentrations, fecal microb

11 Here, we measured fecal-associated bacterial, archaeal, and fungal communi

12 dhood had a lower proportion of IgA bound to fecal bacteria at 12 months of age compared with healthy

13 the infection-associated metabolites and the fecal bacteria, suggesting that these biochemical pertur

14 Utilizing chemostat-cultured human fecal bacteria, we identified the PTPS-2-producing bacte

15 eful tool for determining sources of aquatic fecal bacteria.

16 This is the first report to address the fecal bacterial communities in HIV-infected individuals

17 SourceTracker results indicated that fecal bacterial inputs into the Lake Superior estuary we

18 supplementation of GOS selectively increased fecal Bifidobacterium species abundance, but this did no

19 xcretion of bile acids prevented the rise in fecal bile acid excretion, changed the bacterial composi

20 Fecal bile acids decreased 2.8-fold, suggesting enhanced

21 sts, bile flow, biliary bile salt secretion, fecal bile salt loss, and expression of major hepatocell

22 by myeloperoxidase over a 2-mo lag.Of the 3 fecal biomarkers studied, 2 that related to intestinal f

23 y rates of asymptomatic adults with positive fecal blood test results.

24 In a hospital setting, fecal calprotectin added the most diagnostic value to sy

25 CDAI, CRP, fecal calprotectin and VCE Lewis inflammatory score did

26 Blood markers and fecal calprotectin are used in the diagnostic workup for

27 NDC supplementation did not affect fecal calprotectin concentration.

28 polydextrose supplementation did not affect fecal calprotectin concentrations.

29 All blood markers and fecal calprotectin individually significantly improved t

30 Adding fecal calprotectin to the diagnostic workup of pediatric

31 When fecal calprotectin was added to the model, the proportio

32 The best marker-fecal calprotectin-improved the area under the curve of

33 tic exposure is a risk factor for developing fecal carriage that may lead to nosocomial infection.

34 Here we quantified fecal coliforms (FC), carbapenem-resistant Enterobacteri

35 measured in seawater (Enterococcus species, fecal coliforms, total coliforms) were strongly associat

36 ty at ambient temperature, and accuracy of 5 fecal collection methods (no additive, 95% ethanol, RNAl

37 To evaluate fecal collection methods, we determined technical reprod

38 Consistent with predictions, maternal fecal communities shift across pregnancy, a process that

39 e </=30%) for 84.5% of about 9000 endogenous fecal compounds.

40 Findings of this study suggest that fecal concentrations greater than parasite inhibitory co

41 ones were not detected in serum, and neither fecal concentrations of individual menaquinones nor the

42 The total median (IQR) fecal concentrations of menaquinones decreased in the WG

43 in rural Bangladesh to (1) quantify domestic fecal contamination in settings with high on-site sanita

44 T) methods, employed to determine sources of fecal contamination in waterways, use molecular markers

45 points of contact with animal feces; animal fecal contamination of food; cultural behaviors of anima

46 Mixed Gram-negative flora, suggesting fecal contamination was, however, in the majority of cas

47 ator bacteria do not strictly indicate human fecal contamination when animals are present.

48 Animal feces contribute to fecal contamination, and fecal indicator bacteria do not

49 and Enterococcus species, both indicators of fecal contamination, were detected using substrates spec

50 e; (2) determine how domestic animals affect fecal contamination; and (3) assess how each environment

51 also show that DHMA is present in murine gut fecal contents and that its production requires the pres

52 exogenous estrogens in the impairment of the fecal continence mechanism.

53 romise to better control sepsis and maintain fecal continence.

54 tients reported impairment of all aspects of fecal control (P < 0.05), except constipation.

55 ith matched peers, significant impairment of fecal control prevails after TEPT in HD patients during

56 han nonallergic infants (P < .004); the high fecal count of Lachnospiraceae in allergic subjects appe

57 fecal microbiota composition, and plasma and fecal cytokine concentrations were measured in 80 men an

58 Urgent treatment with laxatives and fecal disimpaction is indicated to prevent perforation a

59 V4 16S rRNA gene sequencing of fecal DNA demonstrated minimal shifts in taxonomic repre

60 clude CT colonography every 5 years, the FIT-fecal DNA test every 3 years, and flexible sigmoidoscopy

61 Fecal DNA was extracted and sequenced to determine the r

62 ested our STR capture strategy on P. diadema fecal DNA, and report robust initial results and suggest

63 s safety is thought to depend on appropriate fecal donor screening.

64 We found that the concentration of fecal EhMIF correlated with the level of intestinal infl

65 hate residues are positively correlated with fecal esterase activity and acetate level of human diabe

66 terol catabolism and LDL-derived cholesterol fecal excretion and reduces inflamed atherosclerotic pla

67 Fecal excretion of cholesterol is the last step in the a

68 sults in increased plasma levels, as well as fecal excretion, of bile acids that is accompanied by di

69 r ezetimibe promotes LDL-derived cholesterol fecal excretion.

70 Also, the circulation in the blood and fecal excretions showed higher AgNPs contents in compari

71 teatorrhea and hyperoxaluria were defined as fecal fat >7 g/day and urine oxalate >40 mg/day.

72 61 mg/day; there was no correlation between fecal fat and urine oxalate excretion.

73 mg/day, with a positive correlation between fecal fat and urine oxalate excretions (r = 0.71, P < .0

74 For each 10 g/day increase in fecal fat output, fecal water excretion increased only 4

75 stic that can sensitively and rapidly detect fecal free toxins.

76 ats an HFM and showed that HFM increases rat fecal Gram-negative bacteria, elevates lipopolysaccharid

77 ams, we investigated the association between fecal hemoglobin (fHb) concentrations below the FIT cut-

78 olitis, had increased levels of systemic and fecal IgA, and were highly susceptible to chemical-induc

79 lementary analyses were performed, including fecal immunochemical test (FIT) as an alternative test,

80 lementary analyses were performed, including fecal immunochemical test (FIT) as an alternative test,

81 The fecal immunochemical test (FIT) is commonly used for col

82 Mailed fecal immunochemical test (FIT) outreach is more effecti

83 The fecal immunochemical test (FIT), as a direct measure of

84 Solution, fecal occult blood test cards, and fecal immunochemical test tubes).

85 screening enhances patient participation in fecal immunochemical testing (FIT).

86 Fecal immunochemical testing is the most commonly used m

87 BACKGROUND & AIMS: A variety of fecal immunochemical tests (FITs) for hemoglobin (Hb) ar

88 al cancer (CRC) screening using quantitative fecal immunochemical tests (FITs) is rapidly gaining gro

89 cess involving the colonic wall secondary to fecal impaction with high morbidity and mortality; espec

90 ogen levels can contribute to development of fecal incontinence (FI) in women after menopause by alte

91 enced swallowing difficulties, tinnitus, and fecal incontinence, and he had undergone cataract surger

92 Fecal indicator bacteria (FIB) Escherichia coli (E. coli

93 Elevated fecal indicator bacteria (FIB) in beach sand and pore wa

94 Population structures of fecal indicator bacteria (FIB) isolated from catch basin

95 ventory sources of natural and anthropogenic fecal indicator bacteria (FIB).

96 e natural occurrence and potential growth of Fecal Indicator Bacteria (FIB).

97 In this paper we focus on the removal of fecal indicator bacteria (FIB, a pollutant responsible f

98 feces contribute to fecal contamination, and fecal indicator bacteria do not strictly indicate human

99 Rainstorms increase levels of fecal indicator bacteria in urban coastal waters, but it

100 Fecal indicator bacteria measured in seawater (Enterococ

101 Traditionally, general fecal-indicator bacteria are used; however, they cannot

102 correlations were found between FC levels (a fecal indictor), and CRE (r = 0.924; p = 0.005), blaNDM-

103 ominance of Prevotella versus Bacteroides in fecal innocula, identified into two different enterotype

104 ing for the determination of the presence of fecal inputs and identification of specific sources.

105 ium suggested that measured marker reflected fecal inputs versus growth outside the host.

106 ed effluent (n = 233) was used to generate a fecal library to perform community-based MST.

107 scriminant analysis Q(2)(Y) of 0.728] in the fecal lipidome between participants with normal blood gl

108 s a validated lipidomics platform to map the fecal lipidome, which integrates unique information abou

109 Eventually, the potential of fecal lipidomics was exemplified within a clinical conte

110 Gut microbiota dysbiosis was investigated by fecal lipopolysaccharide (LPS) measurement and metabolom

111 ion was also supported by a higher bacterial fecal load in CX3CR1(+/gfp) compared with CX3CR1(gfp/gfp

112 weeks of feeding, as reflected by increased fecal LPS and bacterial metabolites and concomitant high

113 M diet had improved IBS symptoms and reduced fecal LPS levels.

114 Fecal LPS was higher in IBS patients than in healthy sub

115 nation of food; cultural behaviors of animal fecal management; acute and chronic health risks associa

116 Using batch experiments with human fecal material as substrate, we accumulated n-butyric ac

117 ic-treated mice and found that transplant of fecal material from an untreated mouse abolished the pro

118 Newborn and adult mice fecal material was screened for 6-pyruvoyltetrahydropter

119 imonas and greater bacterial richness in the fecal material, resulting in eradication of Klebsiella p

120 acid via the anaerobic fermentation of human fecal material.

121 dy differentiation between polyps and tagged fecal material.

122 is undetermined.This study aimed to quantify fecal menaquinone concentrations and identify associatio

123 entrations and identify associations between fecal menaquinone concentrations and serum vitamin K con

124 tect C. fetus genomes in 8% of healthy human fecal metagenomes, where the human-associated lineages a

125 Fecal methanogens were measured by quantitative polymera

126 Metabolomic analysis indicated increased fecal mevalonate levels in the TNBS-treated mice, which

127 3.6% and 7.3% Operational Taxonomic Units in fecal microbial communities in healthy and stressed mice

128 t C. difficile colonization in complex human fecal microbial communities, whereas treatment with eith

129 Fecal microbial composition, metabolites, and pH of infa

130 the latest information on therapeutic use of fecal microbial transplantation and propose improved str

131 Fecal microbiome analysis was performed in all groups us

132 eeded to assess the relationship between the fecal microbiome and human health and disease.

133 ciated with a significant contraction of the fecal microbiome and were partially rescued by fecal mic

134 In this study, we examined the fecal microbiome by using a cross-species approach in bo

135 The combined evaluation of metabolites and fecal microbiome can be useful to discriminate between h

136 However, there is a lack of knowledge about fecal microbiome composition in several animals and impe

137 case-control study comparing prevaccination, fecal microbiome compositions between 6-week old, matche

138 results indicated a definitive change in the fecal microbiome of lactose-intolerant individuals, incr

139 Analysis of patient fecal microbiome samples (n = 43, 30 responders, 13 nonr

140 The fecal microbiome was also analyzed from mice that expres

141 We investigated fecal microbiomes of Przewalski's horse (PH; Equus ferus

142 assic approaches, we found no differences in fecal microbiota abundance or composition between patien

143 Fecal microbiota analysis of 3 successful FT recipients

144 amily member 3 alpha gene (REG3A) alters the fecal microbiota and affects development of colitis in m

145 crolimus, and mycophenolate mofetil modified fecal microbiota at the family level in each experimenta

146 Prednisolone disrupted fecal microbiota community structure, decreased Bacteroi

147 oos and hosted a strikingly low diversity of fecal microbiota compared to individuals born in natural

148 .Fecal and serum menaquinone concentrations, fecal microbiota composition, and plasma and fecal cytok

149 eral and adipose tissue insulin sensitivity, fecal microbiota composition, plasma and fecal SCFA, ene

150 ecal microbiota from the mice which received fecal microbiota from patients with constipation also up

151 In this study, fecal microbiota from patients with constipation and hea

152 The mice which received fecal microbiota from patients with constipation present

153 Fecal microbiota from REG3A-TG mice protect non-TG mice

154 Moverover, fecal microbiota from the mice which received fecal micr

155 to determine its effects on symptoms and the fecal microbiota in patients with IBS.

156 ubstituting antibiotics, this study assessed fecal microbiota in pigs from different dietary treatmen

157 A similar shift in the composition of the fecal microbiota occurred after a few months in TG mice

158 The groups had similar fecal microbiota profiles, serum markers of inflammation

159 Fecal microbiota showed covariation with mucosal adheren

160 SIRT1 iKO mice also had altered fecal microbiota starting at 4-6 months of age compared

161 cal microbiome and were partially rescued by fecal microbiota transfer.

162 current Clostridium difficile infection with fecal microbiota transplantation (FMT) at a tertiary ref

163 Fecal microbiota transplantation (FMT) could be a novel

164 We hypothesized that fecal microbiota transplantation (FMT) could be used to

165 Fecal microbiota transplantation (FMT) from cancer patie

166 BACKGROUND & AIMS: Fecal microbiota transplantation (FMT) is a highly effec

167 Fecal microbiota transplantation (FMT) may improve dysbi

168 Fecal microbiota transplantation is an efficacious and i

169 Fecal microbiota transplantation is increasingly used to

170 Fecal microbiota transplantation led to resolution of re

171 Third, we report our use of fecal microbiota transplantation to face severe Clostrid

172 Fecal microbiota transplantation, performed in one patie

173 After fecal microbiota transplantation, the SERT expression in

174 commended laboratory screening practices for fecal microbiota transplantation.

175 ygen species (ROS) in bacterial cultures and fecal microbiota using 2',7'-dichlorofluorescein diaceta

176 Fecal microbiota varied with diet; the concentration of

177 Diversity and composition of fecal microbiota were determined by 16S ribosomal RNA ge

178 fects of interventions and breast-feeding on fecal microbiota were investigated.

179 levels of tryptophan and composition of the fecal microbiota, analyzed by 16S ribosomal DNA amplicon

180 In this setting, fecal microbiota, evaluated by 16S rRNA gene amplicon se

181 on deficiency altered the composition of the fecal microbiota, reduced mucosal barrier function, and

182 We assessed brain activation patterns, fecal microbiota, urine metabolome profiles, serum marke

183 ibed spacer 1-based metabarcoding to compare fecal mycobiomes of 18 healthy volunteers with those of

184 The use of the fecal occult blood test (FOBT) for colorectal cancer (CR

185 (HR, 1.38; 95% CI: 1.31, 1.45) but not with fecal occult blood test (HR, 1.00; 95% CI: 0.91, 1.10) t

186 6.26, 165.19), or having undergone a recent fecal occult blood test (OR, 13.69; 95% CI: 3.66, 51.29)

187 5% ethanol, RNAlater Stabilization Solution, fecal occult blood test cards, and fecal immunochemical

188 of colonoscopy, flexible sigmoidoscopy, and fecal occult blood test were 27.9, 0.6, and 29.5 per 100

189 ed for colorectal cancer (CRC) by the guaiac fecal occult blood test, interval cancers develop in 48%

190 ng appears to be lower than that with guaiac fecal occult blood testing.

191 We did not observe any differences in fecal or fasting plasma SCFA concentrations or in system

192 Fecal-oral pathogens are transmitted through complex, en

193 sion of MERS-like-CoVs mainly occurs via the fecal-oral route.

194 l (GI) tract primarily transmissible via the fecal-oral route; (2) in the modern era, C. trachomatis

195 n by improved sanitation/hygiene and reduced fecal-oral transmission; and (3) the rise in the practic

196 us, whether primary transmission pathway was fecal-oral, temperature, and whether contamination was l

197 hic/MS analysis indicated that the renal and fecal pathways are the main routes for 10-NO2-OA excreti

198 Fe-stressed diatoms and high mesozooplankton fecal pellet production.

199 dmitted to intensive care with sepsis due to fecal peritonitis (n = 117) or community-acquired pneumo

200 and urinary biomarkers in an animal model of fecal peritonitis and recovery.

201 sepsis response signature (SRS) subgroups in fecal peritonitis associated with early mortality (P = 0

202 itated, long-term (3 d) rat model of sepsis (fecal peritonitis) and recovery was used to understand t

203 the transcriptomic response to sepsis due to fecal peritonitis, and to compare these with the same pa

204 on absorption was negatively correlated with fecal pH (P < 0.001) and positively correlated with Lact

205 Additionally, fecal pH and microbial metabolite levels were measured.

206 Iron markers, fecal pH, and bacterial groups were assessed at baseline

207 Environmental waters are monitored for fecal pollution to protect public health and water resou

208 ample DNA extracts, simulating a gradient of fecal pollution.

209 cities (98.6%) when tested against an animal fecal reference library, and crAssphage genetic markers

210 rtheless, it is unlikely that the fossilized fecal residues depict year-round feeding habits.

211 The reduction in the amount of fecal Salmonella bacteria with Lactobacillus treatment w

212 In conclusion, all fecal sample collection methods appear relatively reprod

213 Fecal samples (2,965) solicited from 11 geographically d

214 (110 with IBS and 39 healthy subjects); 232 fecal samples and 59 mucosal biopsy samples were collect

215 ntative DNA extraction protocols on the same fecal samples and quantified differences in observed mic

216 64) were designed and evaluated in reference fecal samples and water matrices.

217 f S. typhimurium in inoculated Starling bird fecal samples and whole milk with detection limits of 10

218 Fifty-two healthy volunteers provided fecal samples at the Mayo Clinic in Rochester, Minnesota

219 ducibility was high, with ICCs for duplicate fecal samples between 0.64 and 1.00.

220 ated from the case patients and from chicken fecal samples collected at the live bird markets near th

221 nriched preparations from serially collected fecal samples from 11 children (cases) who developed ser

222 Sixty-five fecal samples from 16 immunocompromised patients were re

223 patterns between ARGs and microbial taxa in fecal samples from 180 healthy individuals from 11 diffe

224 e global pattern of microbial composition of fecal samples from 196 hospitalized patients with suspec

225 Fecal samples from mice with maternal separation and fro

226 rized the bacterial taxonomic composition of fecal samples from participants with and without a diagn

227 ial phylogeny and virome profile analyses of fecal samples from recipients indicated longitudinal cha

228 Fecal samples from REG3A-TG mice had lower levels of ROS

229 Here, fecal samples from stranded pygmy (Kogia breviceps) and

230 g of microbiota from a diverse collection of fecal samples obtained from 11 types of animals (wild, a

231 Fecal samples submitted to our clinical microbiology lab

232 When compared with fecal samples that were frozen immediately, the ICCs wer

233 Fecal samples were analyzed by 16s ribosomal RNA sequenc

234 Fecal samples were collected and microbiota were analyze

235 Fecal samples were collected at baseline and 16 weeks; b

236 Fecal samples were collected at baseline and after 4 wee

237 Fecal samples were collected beginning immediately after

238 Fecal samples were collected from 89 typically developin

239 Fecal samples were collected from patients before and at

240 Fecal samples were collected immediately before treatmen

241 A total of 1447 of 12772 (11%) fecal samples were GDH positive, 866 of 1447 (60%) conta

242 Microbial richness or diversity in fecal samples were not affected.

243 Bacterial communities in fecal samples were profiled by 16S ribosomal RNA-based p

244 that the community compositions in water and fecal samples were significantly different, allowing for

245 Fecal samples with a positive initial screen for C. diff

246 ts (29 with IBS and 17 healthy subjects); 46 fecal samples, but no mucosal samples, were collected an

247 standardized DNA extraction method for human fecal samples, for which transferability across labs was

248 In fecal samples, levels of primary bile acids increased in

249 were determined from the genomic DNA in 255 fecal samples, minimally representing 110 individuals.

250 two distinct samples groups and apply it to fecal samples.

251 ng of ileum tissues and lysozyme activity in fecal samples.

252 died for fecal shedding, 11 were detected in fecal samples.

253 e low FODMAP diet on microbiota diversity in fecal samples.

254 ty, fecal microbiota composition, plasma and fecal SCFA, energy expenditure and substrate oxidation,

255 Increasing participation in fecal screening tests is a major challenge in countries

256 Fecal, serum, lung, and colon tissue samples were collec

257 ine norovirus (MNoV) infection and regulates fecal shedding and viral titers in tissue.

258 d longitudinal IFN-gamma, ELISA-antibody and fecal shedding experimental sensitivity scores for MAP i

259 Of the 16 viruses studied for fecal shedding, 11 were detected in fecal samples.

260 testinal tissue titers and enhanced reovirus fecal shedding.

261 e of spread beyond the respiratory tract and fecal shedding.

262 ded macronutrient intake, stool diaries, and fecal short-chain fatty acid concentrations.Patients wer

263 Levels of the fecal short-chain fatty acids acetate and caproate were

264 CS disaccharides partially restored total fecal short-chain fatty acids from the level significant

265 Concentrations of fecal short-chain fatty acids were determined by using g

266 s study was to evaluate the current need for fecal sludge management (FSM) in LMICs by region, urban/

267 ts in sanitation technology and treatment of fecal sludge.

268 well-established bacterial human-associated fecal-source-identification approaches.

269 We identified a patient with fecal specimens positive for Campylobacter jejuni (ST45)

270 challenged with 145 clinical isolate-spiked fecal specimens.

271 This analysis confirms that the fecal test is the best marker for disease progression an

272 llow-up of asymptomatic adults with positive fecal test results.

273 emory of exposure in the gut for analysis by fecal testing, we detected tetrathionate in both infecti

274 who are toxigenic strain positive (TS+) but fecal toxin negative (FT-) in transmission of Clostridiu

275 nase (GDH)-positive specimens, regardless of fecal toxin result, from Oxford (April 2012 through Apri

276 ission from symptomatic patients with either fecal toxin status accounted for a low overall proportio

277 (60%) contained toxigenic C. difficile, and fecal toxin was detected in 511 of 866 (59%), representi

278 Fecal-toxin-positive and toxin-negative patients were si

279 contained toxin genes, and 451 (46.4%) were fecal-toxin-positive.

280 a by innovative probiotics, antibiotics, and fecal transplant, in combination with the current pharma

281 Fecal transplantation (FT) is a promising treatment for

282 Further fecal transplantation and culture transplantation experi

283 evance of the gut mycobiome was confirmed in fecal transplantation experiments: adult maternally sepa

284 Fecal transplants from mice reared in specific-pathogen-

285 biome as well as in germ-free mice receiving fecal transplants from responding patients.

286 by depletion of the microbiota, we performed fecal transplants in antibiotic-treated mice and found t

287 Fecal, urinary, and plasma metabolomes were assessed by

288 0) of mock-treated HEV-infected pigs cleared fecal viral shedding at 8 wk postinfection.

289 tiologies, we characterized and compared the fecal viromes from 32 healthy animals, 31 animals with a

290 /10 drug-treated HEV-infected pigs continued fecal virus shedding beyond the acute phase of infection

291 liver enzyme gamma-glutamyl transferase and fecal virus shedding were significantly higher in immuno

292 used; however, they cannot distinguish human fecal waste from other animal pollution sources.

293 sanitation is increasing, safe management of fecal waste is a rapidly growing challenge in low- and m

294 bundant in and closely associated with human fecal waste.

295 ion investment to achieve safe management of fecal wastes and protect public health.

296 quency of pellet expulsion, fecal weight and fecal water content.

297 Fecal water cytotoxicity was not modified by HPDs, but w

298 each 10 g/day increase in fecal fat output, fecal water excretion increased only 46 mL/day.

299 including the frequency of pellet expulsion, fecal weight and fecal water content.

300 arrier functions by the oral Lac:Man and the fecal zonulin tests.