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1 ntify phenotypes in defecatory disorders and fecal incontinence.
2 to guide the initial management of men with fecal incontinence.
3 Some patients reported symptoms of fecal incontinence.
4 colon, but 3 were subsequently reversed for fecal incontinence.
5 erformed in 28 women (aged 27-74 years) with fecal incontinence.
6 No patient developed fecal incontinence.
7 s and management of defecatory disorders and fecal incontinence.
8 ledge about the long-term outcome of SNS for fecal incontinence.
9 younger group developed ischemic colitis and fecal incontinence.
10 pproach might be used to treat patients with fecal incontinence.
11 uropathy) are not included within functional fecal incontinence.
12 function ranging from fecal urgency to frank fecal incontinence.
13 an cause urge-related or diarrhea-associated fecal incontinence.
14 pplies to the experience of individuals with fecal incontinence.
15 s to measure severity and quality of life in fecal incontinence.
16 al sphincter muscle as a method for treating fecal incontinence.
17 s outcome variables in therapeutic trials of fecal incontinence.
18 risk factors common to both constipation and fecal incontinence.
19 and ileal pouch reconstruction can result in fecal incontinence.
20 and constipation are known risk factors for fecal incontinence.
21 o, more sucralfate-treated patients reported fecal incontinence (16% v 34%, respectively; P =.04) and
22 us bowel movements/week and </= 1 episode of fecal incontinence/2 weeks, from study weeks 5-8 (respon
23 55.1% in the placebo group had a history of fecal incontinence; 60.4% and 55.1% in the prucalopride
27 dverse events included vomiting, urinary and fecal incontinence, agitation, combativeness, a labile l
28 ables determination of the anatomic cause of fecal incontinence, allowing the surgeon to select patie
29 tify ways to counteract the social stigma of fecal incontinence and assist physicians in providing pa
30 has been advocated as therapy for refractory fecal incontinence and for anorectal reconstruction to a
31 d that target anal canal resting pressure in fecal incontinence and hypersensitivity to distention in
32 on of quality-of-life issues associated with fecal incontinence and improved assessment and communica
34 and advice to patients around the world with fecal incontinence and other gastrointestinal disorders,
36 % of women (95% CI, 7.3%-10.7%) experiencing fecal incontinence, and 2.9% of women (95% CI, 2.1%-3.7%
37 was the greatest risk factor for developing fecal incontinence, and fecal incontinence was the great
38 enced swallowing difficulties, tinnitus, and fecal incontinence, and he had undergone cataract surger
39 elvic floor disorders (urinary incontinence, fecal incontinence, and pelvic organ prolapse) affect ma
41 diverticulitis, enhanced recovery protocols, fecal incontinence, and single incision laparoscopic sur
42 sorption, anal sphincter dysfunction causing fecal incontinence, and the irritable bowel syndrome.
43 e and/or innervation insufficient to explain fecal incontinence, and/or (3) normal or disordered bowe
44 agnosing functional anorectal disorders (ie, fecal incontinence, anorectal pain, and disorders of def
47 dure for patients with refractory, end-stage fecal incontinence as well as for patients who require a
48 ting of strategies for primary prevention of fecal incontinence associated with childbirth, and furth
49 utcome of sacral nerve stimulation (SNS) for fecal incontinence at 5 years after implantation and to
50 x, constituting moderate to severe leakage), fecal incontinence (at least monthly leakage of solid, l
51 It is recommended that outcome measures for fecal incontinence be more clearly defined, that future
53 t (biofeedback) has been reported to improve fecal incontinence but has not been compared with standa
54 oaches may benefit a subset of patients with fecal incontinence, but more controlled studies are need
55 encopresis with or without constipation and fecal incontinence caused by anatomic or organic disease
56 creased stool frequency or fewer episodes of fecal incontinence compared to handsewn IPAA, which exci
57 71%, 50%, and 66% for patients with acquired fecal incontinence, congenital incontinence, and total a
58 reviewed recent publications in the areas of fecal incontinence, constipation, single incision and ro
60 gested that psychological symptoms can cause fecal incontinence, data are lacking to support a causat
61 mpounds offering new treatment approaches to fecal incontinence, development and testing of strategie
62 ogen levels can contribute to development of fecal incontinence (FI) in women after menopause by alte
66 alence of different types and frequencies of fecal incontinence (FI), describe demographic factors, a
67 nd management of common anorectal disorders: fecal incontinence (FI), functional anorectal pain and f
74 c floor retraining by biofeedback therapy in fecal incontinence; however, the predictive value of imp
76 ses the diagnosis and treatment of pediatric fecal incontinence in 4 main categories: (1) Functional
82 Salvage options for patients with refractory fecal incontinence include passive or electrically stimu
90 he most important tool in the "treatment" of fecal incontinence is its prevention, which should be th
92 mpered by heterogeneity in the definition of fecal incontinence, lack of consensus on what constitute
97 , and amount of stool loss and the impact of fecal incontinence on coping mechanisms and lifestyle/be
99 ofeedback retraining include the duration of fecal incontinence, pudendal nerve damage, patient age,
101 All patients were evaluated with the Wexner Fecal Incontinence Questionnaire after stoma closure.
106 s of the multifaceted mechanisms maintaining fecal incontinence should be incorporated as outcome var
107 physiologic factors that predict response to fecal incontinence therapy would be helpful in choosing
108 of furthering research activities related to fecal incontinence through the National Institutes of He
111 actor for developing fecal incontinence, and fecal incontinence was the greatest risk factor for deve
113 n January 1995 and January 1998, 37 men with fecal incontinence were evaluated in the John Radcliffe
115 Before surgery, 22 patients (32%) reported fecal incontinence, which improved after surgery in 15 c
116 of toileting assistance on the frequency of fecal incontinence, while significant, are less dramatic
117 s by far the most prominent association with fecal incontinence, with a prevalence approaching 50%.
118 recent surgical options for the treatment of fecal incontinence within the context of established the
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