ライフサイエンスコーパス: feedback inhibition

1 (+) inhibits Na(+) transport via ENaC (Na(+) feedback inhibition).

2 synapses, arguing for the importance of the feedback inhibition.

3 hich is consistent with a loss of retrograde feedback inhibition.

4 nhibition, which differs from feedforward or feedback inhibition.

5 feedback loop with a built-in time delay in feedback inhibition.

6 with respect to their sensitivity to serine feedback inhibition.

7 e (KD of 22-43 muM), which indicates product feedback inhibition.

8 iated inflammation by enhancing the negative feedback inhibition.

9 s (FSNs), which then synchronize the PCs via feedback inhibition.

10 compromised CCK-INT-mediated feedforward and feedback inhibition.

11 tion is required for starvation-induced PI3K feedback inhibition.

12 lation and eNOS/Cav-1 binding, that is, eNOS feedback inhibition.

13 d, IKK-mediated p85 phosphorylation and PI3K feedback inhibition.

14 boxylase (ACCase) as the enzymatic target of feedback inhibition.

15 s are hyperactivated to trigger the negative feedback inhibition.

16 proposal that beta-endorphin acts to provide feedback inhibition.

17 rk mechanisms of gamma oscillations based on feedback inhibition.

18 on gene regulation, enzymatic activity, and feedback inhibition.

19 endently from receptors mediating reciprocal feedback inhibition.

20 is not caused by the secondary engagement of feedback inhibition.

21 he transcriptional and biochemical levels by feedback inhibition.

22 ium and glutamate, is regulated by glutamine feedback inhibition.

23 nin secretion by interrupting angiotensin II feedback inhibition.

24 e mTORC1, effectively bypassing S6K-mediated feedback inhibition.

25 ion from pyramidal cells, produce late-onset feedback inhibition.

26 I and L174F enzymes were highly resistant to feedback inhibition.

27 vel KAT that may be susceptible to metabolic feedback inhibition.

28 p in which PERIOD (PER) is rate-limiting for feedback inhibition.

29 system combining feed-forward excitation and feedback inhibition.

30 exhibits the highest reported sensitivity to feedback inhibition.

31 with concomitant protection from end-product feedback inhibition.

32 primarily to differences in the strength of feedback inhibition.

33 neer MK proteins with altered sensitivity to feedback inhibition.

34 acts with and phosphorylates FoxO, providing feedback inhibition.

35 de CA1 and CA2 with feedforward and CA3 with feedback inhibition.

36 n, passing repeatedly through zones of local feedback inhibition.

37 ([Na(+)]i) through a process known as Na(+) feedback inhibition.

38 k circuits, neither receiving nor generating feedback inhibition.

39 he enzyme level by allosteric regulation and feedback inhibition.

40 nt sensitizes nociceptor presynapses to this feedback inhibition.

41 ependent recruitment of OLM interneurons for feedback inhibition.

42 ortical inputs, implying a prevalent role in feedback inhibition.

43 inhibiting channel opening to provide vital feedback inhibition.

44 at these early ages despite the presence of feedback inhibition.

45 imulus, and that does not rely on long-range feedback inhibition.

46 drenal glands by restoring adrenal alpha2-AR feedback inhibition.

47 rd input is sufficient to implement divisive feedback inhibition.

48 rrelation have been proposed, among which is feedback inhibition.

49 mbination within the time required to signal feedback inhibition.

50 of a two-tiered microbial 'food chain' with feedback inhibition, after applying an appropriate dimen

51 nism: endogenously driven gain modulation of feedback inhibition among competing channels.

52 We demonstrate that feedback inhibition among SPNs is strong enough to contr

53 ensitivity of the MtDHDPS2 protein to lysine feedback inhibition and a severely reduced activity of t

54 Furthermore, the sites responsible for feedback inhibition and allosteric activation control fi

55 group also defined roles for leptin receptor feedback inhibition and hypothalamic mTor signaling in m

56 Relaxed feedback inhibition and increased expression of the muta

57 positions previously shown to be involved in feedback inhibition and of residues important for cataly

58 -2 tg mice, both the glucocorticoid negative feedback inhibition and spatial learning and memory were

59 scribe general rules for feed-forward versus feedback inhibition and suggest GGN is potentially capab

60 rtical circuits by providing feedforward and feedback inhibition and synchronizing neuronal activity.

61 a permissible environment for MAPK negative feedback inhibition and thus regulated mammary branching

62 tic regulatory mechanism of KLK6 is negative feedback inhibition, and activation is likely achieved t

63 g data about the regulation of excitability, feedback inhibition, and network oscillations in area CA

64 ufficiently strong recurrent excitation; (3) feedback inhibition; and (4) simple spatial properties o

65 L-IPSCs, confirming that these two types of feedback inhibition are mediated by distinct and converg

66 Feedback inhibition arises from a hydrogen bond network

67 he l-lysine epsilon-ammonium group, abrogate feedback inhibition, as do substitutions of residues wit

68 o exogenous CRH stimulation, inferring rapid feedback inhibition at the anterior pituitary.

69 from the synaptic cleft nor by metabotropic feedback inhibition, because it is resistant to blockade

70 igh baseline levels of phospho-MEK, and lack feedback inhibition between ERK and Raf.

71 rolling a brain size set-point that involves feedback inhibition between wnt11-6/wntA/wnt4a and notum

72 ese findings, an R62A mutation abrogates Gln feedback inhibition but does not affect catalysis.

73 Exogenous expression of BRAF V600E disrupts feedback inhibition but does not sensitize cells to AZD6

74 ior and the impaired glucocorticoid negative feedback inhibition, but not the memory deficit, implyin

75 al aqueous cavity and exhibiting synergistic feedback inhibition by AMP and Fru-6-P.

76 Mathematical modeling indicates that feedback inhibition by BMP ligands acts on the ventral s

77 rns of activity within layer 4 to potentiate feedback inhibition by boosting the strength of FS <-->

78 unusually high Km for ATP, are resistant to feedback inhibition by CoA, and are unable to utilize th

79 i type I PanK, this enzyme is not subject to feedback inhibition by CoASH.

80 d releases SAT from substrate inhibition and feedback inhibition by cysteine, the final product of th

81 y but rendered the enzyme insensitive to the feedback inhibition by cysteine.

82 Apparent negative-feedback inhibition by ferrous ions is documented at nan

83 ociated one may participate in the channel's feedback inhibition by intracellular Na+, a process know

84 plants and micro-organisms and is subject to feedback inhibition by l-arginine.

85 A feature of SAT is its feedback inhibition by L-cysteine.

86 shown that MtIPMS is subject to slow-onset, feedback inhibition by l-leucine, the first instance of

87 the dark period, reducing its sensitivity to feedback inhibition by malate and thus enhancing nocturn

88 ereby ruling out an interaction with channel feedback inhibition by Na(+).

89 Such mutations render PSS1 insensitive to feedback inhibition by PS levels.

90 Feedback inhibition by Sox2 on Wnt signaling and by the

91 -23R might not be a major target of negative feedback inhibition by suppressor of cytokine signaling

92 to that of the mammalian enzyme, except that feedback inhibition by the cofactor requires sequences i

93 provides an explanation for the lack of CoA feedback inhibition by the enzyme.

94 At the network level, modulation of feedback inhibition can result in reduction of variabili

95 The first is end-product feedback inhibition, catalytically similar to that of th

96 ng to inactivation and suggesting a negative feedback inhibition control mechanism.

97 Wnt signaling, suggesting that regulation of feedback inhibition controls the binary head-tail regene

98 ound that the balance between excitation and feedback inhibition depended on stimulus frequency; at s

99 Thus, despite general roles in feedback inhibition, DUSP1 plays a transient, often part

100 s dual regulatory strategies, with classical feedback inhibition enhancing metabolic efficiency and d

101 tion of RNR upon commitment to S phase, dATP feedback inhibition ensures that the dNTP concentration

102 and inhibits BMP-9 thereby providing strong feedback inhibition for BMP-9/ALK1 signaling rather than

103 ogens (typically Geobacter species) relieves feedback inhibition for the fermentative consortia, allo

104 GABAergic feedback inhibition from amacrine cells shapes visual si

105 activity in single granule cells can recruit feedback inhibition from Golgi cells.

106 rom pyramidal cells could drive synchronized feedback inhibition from interneurons.

107 expression suppresses S6K1 but, by relieving feedback inhibition from mTORC1 to PI3K signaling, activ

108 ween -65 and -50 mV is sufficient to produce feedback inhibition from periglomerular neurons.

109 This suggests that feedback inhibition from surviving neurons may skew neur

110 instead, it appears that another mechanism, feedback inhibition from the giant GABAergic neuron, ser

111 These projection neurons receive feedback inhibition from the pyloric CPG interneuron ant

112 This negative feedback inhibition functions to restrain PI3K activity

113 Feedforward and feedback inhibition generated by these circuits is likel

114 This feedback inhibition has prevented scientists from engine

115 or the nature of competition (feedforward or feedback inhibition) has been revealed by experiments.

116 g striatal projection neurons (SPNs) called "feedback inhibition," have been proposed to endow the st

117 factor, parental PTSD, on cortisol negative feedback inhibition in adult offspring of Holocaust surv

118 sible manner, demonstrating the existence of feedback inhibition in an oil-accumulating tissue.

119 beta) accessibility but was still subject to feedback inhibition in DP thymocytes.

120 eta) and DJ(beta) gene segments both enforce feedback inhibition in DP thymocytes.

121 ent of end product accumulation and possible feedback inhibition in isoprene-emitting hybrid aspen (P

122 common and underlies the origins of negative feedback inhibition in many metabolic and signaling path

123 e transcription and effective glucocorticoid feedback inhibition in spite of the increase in AT(1) re

124 atergic synapses on interneurons involved in feedback inhibition in the CA1 region of the hippocampus

125 Our results clarify the source of feedback inhibition in the E. coli PhoQ-PhoP system and

126 that provide either feedforward or recurrent/feedback inhibition in the lobe.

127 al role in providing dynamic feedforward and feedback inhibition in the retina.

128 However, the impact of feedback inhibition in the striatal network has remained

129 analysis shows that regulation of the local feedback inhibition increases both the entropy and the F

130 napses on hippocampal interneurons mediating feedback inhibition is "anti-Hebbian":Itis induced by pr

131 The Wnt5a-induced feedback inhibition is active both during in vitro LPS s

132 Feedback inhibition is believed to be controlled, in par

133 ated Na+ channels, and the spatial extent of feedback inhibition is expanded by gap junction connecti

134 A mechanism for feedback inhibition is proposed in which reduced demand

135 the role of the Glu304 residue in glutamine feedback inhibition is proposed.

136 CCAP) continually present, the impact of the feedback inhibition is reduced, prolonging protraction a

137 EphA2-mediated negative feedback inhibition is required for Raf-induced AKT inhi

138 Allosteric feedback inhibition is the mechanism by which metabolic

139 iments reveal that GABAergic feedforward and feedback inhibition is unaffected by carbamazepine and a

140 cation limits NHX-7 proton transport through feedback inhibition, likely to prevent metabolic acidosi

141 Moreover, pApA is involved in a feedback inhibition loop that limits GdpP-dependent c-di

142 g enough to destabilize visual responses but feedback inhibition maintains stability.

143 Enhanced cortisol negative feedback inhibition may be associated with PTSD because

144 endosymbiotic systems suggest that substrate feedback inhibition may be mechanistically important in

145 lls may act as an inbuilt activation-induced feedback inhibition mechanism against excessive or chron

146 regulated by a TPP-binding riboswitch via a feedback inhibition mechanism.

147 ed astrocyte proliferation via a phospho-ERK feedback inhibition mechanism.

148 by the time course of I(MI-MCN1) buildup and feedback inhibition-mediated decay, respectively, in LG.

149 ata and localization, we propose a substrate feedback inhibition model in which the accumulation of t

150 Feedback inhibition of 1-deoxy-D-xylulose-5-phosphate sy

151 acteria that possess a stringent biochemical feedback inhibition of ACC and malonyl-CoA formation tri

152 on evoked dopamine release, suggesting that feedback inhibition of acetylcholine release was not inv

153 r guanine nucleotide pools by regulating the feedback inhibition of adenosine derivatives on de novo

154 Feedback inhibition of adenylyl cyclase III (ACIII) via

155 ggesting one mechanism through which loss of feedback inhibition of Akt may occur in mTORC1 hyperacti

156 ts that mTORC1 activation is associated with feedback inhibition of Akt, a substrate of mTORC2.

157 essing of 6-TG probably through its negative feedback inhibition of Akt.

158 ants in the de novo pathway that disable the feedback inhibition of AMP and GMP biosynthesis also enh

159 SHP, within a single physiological pathway, feedback inhibition of bile acid biosynthesis, by differ

160 Genetic variation in negative feedback inhibition of bile acid synthesis may affect CD

161 ted transnitrosylation cascade that provides feedback inhibition of bile salt synthesis.

162 Our data reveal feedback inhibition of cellulose synthase by UDP but not

163 ed intestinal factors that mediate bile acid feedback inhibition of cholesterol 7alpha-hydroxylase ge

164 closely related proteins, are essential for feedback inhibition of cholesterol biosynthesis.

165 separation of the loops and facilitating the feedback inhibition of cholesterol synthesis.

166 Numerous studies suggest that feedback inhibition of CLOCK-BMAL1 is mediated by time-d

167 he proinflammatory response through negative-feedback inhibition of cytokine receptors.

168 , prolongs DA transients and facilitates the feedback inhibition of DA and glutamate release from the

169 lective loss of D2 autoreceptors impairs the feedback inhibition of DA release and amplifies the effe

170 erapy-resistance mechanism involving reduced feedback inhibition of de novo purine biosynthesis and c

171 ese data suggest that DUSP5 functions in the feedback inhibition of ERK1/2 signaling in response to T

172 nhibitor (TFPI) produces factor Xa-dependent feedback inhibition of factor VIIa/tissue factor-induced

173 anism presents an opportunity for mitigating feedback inhibition of fatty acid synthesis in crop plan

174 s is in agreement with the stronger in vitro feedback inhibition of free SAT by cysteine compared wit

175 ive (DP) differentiation, proliferation, and feedback inhibition of further Vbeta to DJbeta rearrange

176 rk on multiple pulse depression, reveal that feedback inhibition of GABAergic afferents to hilar moss

177 respiration in naked mole-rat tissues avoids feedback inhibition of glycolysis via phosphofructokinas

178 The resultant loss of feedback inhibition of GNE-epimerase activity by CMP-sia

179 Feedback inhibition of GNE-epimerase activity by CMP-sia

180 Activating BRAF mutants cause feedback inhibition of GTP-bound RAS, are RAS-independen

181 ed in plasma and liver, leading to secondary feedback inhibition of hepatic SREBP2 activity.

182 function of cellular membranes and requires feedback inhibition of HMGR, a rate-limiting enzyme of t

183 Irgm1(-/-) animals, suggesting that negative feedback inhibition of IFN signaling by Irgm1 is necessa

184 However, negative feedback inhibition of IFN-beta transcription was found

185 her show that the kinetics of SOCS1-mediated feedback inhibition of IFNgamma signaling is comparable

186 ibits Akt/mTOR signaling because it relieves feedback inhibition of IGF1R and other receptors and thu

187 Taken together, our findings suggest a feedback inhibition of IL-15-mediated NK cell activity b

188 Feedback inhibition of IlvA is required for the curative

189 O production can be cytotoxic to host cells, feedback inhibition of iNOS transcription would represen

190 gnificant role in the mTOR-mediated negative-feedback inhibition of insulin action and increases the

191 ecause activation of PI3K signaling leads to feedback inhibition of insulin receptor substrate-2 (IRS

192 s consistent with a role for Tyr(317) in the feedback inhibition of Jak2 kinase activity after recept

193 Loss of EPHA2 releases feedback inhibition of KRAS, resulting in activation of

194 We identified a powerful type of feedback inhibition of layer II neurons, mostly generate

195 a, may generate regulatory lipids that cause feedback inhibition of LXRalpha in macrophages.

196 d RNA silencing revealed a transient role in feedback inhibition of MAPKs and inflammatory gene expre

197 h waxes and wanes periodically due to phasic feedback inhibition of MCN1 transmitter release.

198 t the loss of electron transport is inducing feedback inhibition of metabolic capabilities that suppr

199 phate competes with substrate ATP to produce feedback inhibition of mevalonate kinase.

200 or example, by amyloid beta peptides, causes feedback inhibition of MPP, leading ultimately to accumu

201 ing their clearance in the bone marrow, with feedback inhibition of neutrophil production via the IL-

202 data in this study also indicated a negative feedback inhibition of nicotine biosynthesis.

203 trations of H2S in a reaction that may allow feedback inhibition of NO production under conditions of

204 ng chemosensitivity due to microRNA-mediated feedback inhibition of p73.

205 activity-dependent adenosine release exerts feedback inhibition of parallel fibre-Purkinje cell tran

206 hanisms regulating sorting of PDGFRalpha and feedback inhibition of PDGFRalpha signaling at the ciliu

207 nsulin/IGF-1 signaling pathway may result in feedback inhibition of PI3K through IRS1 and reduce APP

208 that myr-Akt overexpression may be inducing feedback inhibition of PI3K, resulting in impaired APP t

209 We also define A2bR/A2aR-mediated autacoid feedback inhibition of proinflammatory/profibrotic cytok

210 The feedback inhibition of PtDXS by IDP and DMADP constitute

211 Physiologic feedback inhibition of RAF/MEK signaling down-regulates

212 Activated BRAF mutants evade feedback inhibition of RAS by either of two mechanisms.

213 vation of Raf-MEK1 signaling causes negative feedback inhibition of Ras through the ephrin receptor E

214 bited ERK phosphorylation, but also relieved feedback inhibition of RAS, resulting in induction of pM

215 nation and internalization of PDGFRalpha for feedback inhibition of receptor signaling.

216 hrough monoallelic initiation and subsequent feedback inhibition of recombinational accessibility.

217 ucleosidase, which alleviates AdoHcy product feedback inhibition of S-adenosylmethionine-dependent me

218 within the dorsal raphe nucleus (DR) induces feedback inhibition of serotonin neuron activity and con

219 s protein domain is also required to mediate feedback inhibition of Set1A and Set1B expression, which

220 promoting Ngn1 expression, and that negative feedback inhibition of Sox2 by Ngn1 is an essential step

221 The dsbA-mediated feedback inhibition of SPI1 transcription is not due to

222 y related proteins that are required for the feedback inhibition of SREBP and HMG-CoA reductase (HMGR

223 chanism in which enterocyte apoptosis breaks feedback inhibition of stem cell division.

224 TRN that promotes the TRN-mediated cortical feedback inhibition of thalamic neurons.

225 Furthermore, the feedback inhibition of the BCR signalosome and most of i

226 Furthermore, feedback inhibition of the Ca2+ current (I(Ca)) by relea

227 Recently, a feedback inhibition of the chloroplastic 1-deoxy-D-xylul

228 osynthesis in plants is tightly regulated by feedback inhibition of the end product on the first enzy

229 Feedback inhibition of the first committed step of a pat

230 levels in failing hearts, with corresponding feedback inhibition of the heme synthetic enzymes and no

231 -Idol pathway is an important contributor to feedback inhibition of the LDLR by sterols and a biologi

232 phosphorylation stabilized Grb10, leading to feedback inhibition of the phosphatidylinositol 3-kinase

233 ) excitation of the pFRG and postinspiratory feedback inhibition of the preBotC, can provide a phase-

234 imulated with IGF-1 and TNFalpha via reduced feedback inhibition of the STAT3 and mTOR pathways.

235 on of Vbeta segment accessibility and normal feedback inhibition of the Vbeta to DJbeta rearrangement

236 that this increase was due to the relief of feedback inhibition of translation as a consequence of m

237 This relieves feedback inhibition of upstream EGFR family kinases, res

238 of a functional TCR beta-chain gene triggers feedback inhibition of V(beta)-to-DJ(beta) recombination

239 Feedback inhibition of V(D)J recombination enforces Ag r

240 r chains expressed from one allele to signal feedback inhibition of V-to-(D)J recombination on the ot

241 Our findings suggest that TCRbeta-mediated feedback inhibition of Vbeta14 rearrangements depends on

242 eveal an essential role for ligand-dependent feedback inhibition of vertebrate HH signaling governed

243 on of SBF and MBF transcription factors, and feedback inhibition of Whi5 by G1-S cyclins.

244 not due to differential mTORC1 activation or feedback inhibition on Akt.

245 y due to its ability to block IL-10-mediated feedback inhibition on cytokine transcription in macroph

246 rowing sink organs and reducing Suc-mediated feedback inhibition on photosynthesis.

247 nucleic acid-induced signaling that provides feedback inhibition on specific TLR9-dependent responses

248 tigated the effect of locally regulating the feedback inhibition on the global dynamics of a large-sc

249 in the outer membrane and the corresponding feedback inhibition on the T3SS.

250 Because of the competition mediated by feedback inhibition, only the most excited DG cells fire

251 ing mice suggested that both feedforward and feedback inhibition onto granules cells were diminished

252 C-triggered gastric mill rhythm is shaped by feedback inhibition onto projection neurons from a CPG n

253 diversity of mechanisms underlie glycinergic feedback inhibition onto RBCs, yet they highlight severa

254 ses are not consistent with the conventional feedback inhibition or 'gain control' models of Golgi ce

255 r delivering FA to inhibit HSL (facilitating feedback inhibition) or affecting multicomponent complex

256 -1 signaling pathway is tightly regulated by feedback inhibition pathways, we hypothesized that myr-A

257 default-mode network, whereas regulation of feedback inhibition prevents this.

258 on activation is crucial to synchronize, via feedback inhibition, pyramidal cells in the gamma freque

259 es, suggesting that tightly coupled synaptic feedback inhibition regulates spike timing in principal

260 Feedback inhibition regulates the strength and speed of

261 c inhibition, whereas PLC-mediated GABAergic feedback inhibition remains responsive to leptin.

262 This feedback inhibition renders the concentration of dNTPs a

263 n is dynamically balanced by feedforward and feedback inhibition, resulting in suppression of dendrit

264 eCBs are retrograde messengers that provide feedback inhibition, resulting in the suppression of neu

265 y, complete loss of PTCH2-, HHIP1- and PTCH1-feedback inhibition results in ectopic specification of

266 Dynamic clamp simulation of feedback inhibition revealed differential effects of thi

267 However, to be effective, feedback inhibition should arise from synchronized pools

268 In principle, dATP feedback inhibition should be sufficient to couple dNTP

269 ." Strong balanced amplification arises when feedback inhibition stabilizes strong recurrent excitati

270 In network models deduced from our data, feedback inhibition supports coexistence of theta-nested

271 These results suggest that feedback inhibition suppresses Kenyon cell activity to m

272 ) provide thalamocortical relay neurons with feedback inhibition that influences sensory processing a

273 osynthesis and is a key control point in the feedback inhibition that regulates this pathway.

274 tory interneurons that mediate strong, local feedback inhibition that scales with excitation.

275 dendritic release of dopamine and subsequent feedback inhibition through activation of D2 receptors o

276 wnt11-6/wntA/wnt4a undergoes feedback inhibition through canonical Wnt signaling but

277 of the cerebellum, providing feedforward and feedback inhibition through mossy fiber and parallel fib

278 tabilization and activation by blocking MDM2 feedback inhibition through ribosomal proteins.

279 erior outgrowth, whereas DSH-1 also provides feedback inhibition to attenuate the signaling to allow

280 ethods to show that locally constraining the feedback inhibition to compensate for the excess of long

281 xpressing ectopic PEPC forms with diminished feedback inhibition to examine the role of PEPC in carbo

282 s (MFs) and grcs and provide feedforward and feedback inhibition to grcs.

283 that both feedforward and to a lesser extent feedback inhibition to MSNs in HD can potentially be sou

284 interneurons (PIIs), which provide powerful feedback inhibition to neuronal networks.

285 single A17 amacrine cells provide reciprocal feedback inhibition to presynaptic bipolar cells via hun

286 2+) influx upon repolarization and increases feedback inhibition to produce subthreshold modulation o

287 transcriptional repression of PDE4D9 acts as feedback inhibition to regulate dopaminergic signaling.

288 ar cells and A17 amacrines provide GABAergic feedback inhibition to rod bipolar cells.

289 receives input from relay cells and supplies feedback inhibition to them in return.

290 Collectively, our data suggest that feedback inhibition to VTA DA neurons, mediated by GIRK

291 atin accessibility and locus conformation to feedback inhibition using two novel TCR alleles.

292 recombinational accessibility is subject to feedback inhibition, we analyzed TCRbeta rearrangements

293 s predicted by the accepted model of TCRbeta feedback inhibition, we found that expression of these p

294 nthesis is regulated by end product negative feedback inhibition where the levels of sterols and oxys

295 r, Nodal expression declines by day 4 due to feedback inhibition, whereas TGFbeta persists.

296 e that use of this capacity is restricted by feedback inhibition, which is relaxed at lower night tem

297 We show that this data-based form of feedback inhibition, which is softer than that of winner

298 current synaptic excitation balanced by fast feedback inhibition, which not only instantiates attract

299 ating disynaptic recurrent, feedforward, and feedback inhibition within and across the two projection

300 spatial exploration, are generated by local feedback inhibition without recurrent excitation, and ha

301 inhibition but reduced total GLP-1 and GIP (feedback inhibition) without affecting the numerical con