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1 ed the survival of CLL cells on a macrophage feeder layer.
2 oth in the presence and absence of the glial feeder layer.
3 n serum-containing medium using a fibroblast feeder layer.
4  pure neuronal cultures deprived of the glia feeder layer.
5 c cultures in the presence or absence of MSC feeder layers.
6 ed in media with or without serum and/or 3T3 feeder layers.
7 in an undifferentiated state by culturing on feeder layers.
8 animal-derived 'serum replacements' on mouse feeder layers.
9 oly-L-lysine-coated glass substrates without feeder layers.
10 t mechanisms when neurons are grown on glial feeder layers.
11 use yolk sac-derived endothelial cell (C166) feeder layers.
12 n blot; SC clonal growth was measured on 3T3 feeder layers.
13                    While culture of HMECs on feeder layers abrogates M0 and p16(INK4A) inactivation,
14                        A cardiac mesenchymal feeder layer allows renewal of the isolated progenitor c
15 lips, which are suspended above an astrocyte feeder layer and maintained in serum-free medium.
16 showed vivid clonal growth on 3T3 fibroblast feeder layers and complete epithelial-mesenchymal transi
17 in the germ line has allowed a separation of feeder layers and contaminating testis somatic cells fro
18 er different conditions were seeded onto 3T3 feeder layers and cultured for 12 days.
19  episomal or integrated HPV-16, but required feeder-layer and growth-factor support, were consistentl
20                        In vitro selection of feeder-layer and growth-factor-independent variants even
21 ioned medium from mouse embryonic fibroblast feeder layers, and (ii) direct cell differentiation.
22  cells and their progeny to animal and human feeder layers, and thus the risk of contamination with p
23 ells cultured on bone marrow-derived stromal feeder layers are more resistant to chemotherapy, increa
24 ltivating the telomerase-expressing cells on feeder layers avoids the growth arrest associated with i
25 ) cell lines were derived 25 years ago using feeder-layer-based blastocyst cultures, subsequent effor
26                                    Without a feeder layer but in the presence of hepatocyte growth fa
27   By culturing MGE progenitors on a cortical feeder layer, cell fate analyses suggest that Shh signal
28      Once cell colonies were established REF feeder layers could be replaced with REF conditioned med
29 e of the role of HA in early development and feeder layer cultures of hESCs and the controllability o
30                Cells grown on inserts with a feeder layer developed into confluent monolayers consist
31    Here we show that primary HMECs, grown on feeder layers, do not undergo this growth arrest and can
32 agated indefinitely when placed onto stromal feeder layers engineered to produce IL-7.
33 gation of hESCs on mouse fibroblast or human feeder layers, enzymatic cell removal, and spontaneous d
34 ures from mice lacking nNOS grown on a glial feeder layer fail to respond to neurotrophin-mediated en
35 bal specimens and clonally expanded on a 3T3 feeder layer, followed by subcultivation of holoclones o
36  and obviates the requirement for serum or a feeder layer for maintenance.
37  primary human skin fibroblasts were used as feeder layers for cloning lymphoid cells by limiting dil
38 ore, mouse and human amniocytes can serve as feeder layers for iPS cells and for mouse and human embr
39  limbal stroma when seeded on 3T3 fibroblast feeder layers from P1 to P3.
40                         Mitotically inactive feeder layers have been used previously to support the g
41 kely to require using human serum with human feeder layers, ideally starting with fresh HESC that hav
42 on by culture on a mitomycin C treated STON+ feeder layer in a hepatoblast culture medium consisting
43 ation) were cultured on mouse STO fibroblast feeder layers in the presence of human recombinant leuke
44  in Matrigel in the absence of a mesenchymal feeder layer, individual cells divided and formed self-o
45 ead angiogenesis assay experiment, FCSC cell feeder layer induced HUVECs to form significantly shorte
46 e primary neuronal cultures grown on a glial feeder layer is significantly less than that of neurons
47   In vitro colony assays with rUCMS cells as feeder layers markedly reduced Mat B III colony size and
48 ouse-derived primed hESCs on mouse embryonic feeder layer (MEF) to a naive state within 5-6 days in n
49 pluripotent stem cells requires the use of a feeder layer of cells.
50  cultures by using a combination of L1 and a feeder layer of human hair follicle-derived mesenchymal
51 gen-coated culture inserts suspended above a feeder layer of myofibroblasts.
52 cosal tissues (ANM cells) were plated onto a feeder layer of newborn rat cortical glia (astrocytes) i
53  Blastocysts were cultured individually on a feeder layer of rat embryonic fibroblasts (REFs) in fibr
54                                              Feeder layers of cerebellar granule cells derived from w
55 bone marrow microenvironment and cultured on feeder layers of CSF-1, DBP-MAF, or DBP-MAF/CSF-1 for co
56 By contrast, mouse EBs cultured alone, or on feeder layers of mouse embryonic fibroblasts or avian no
57 nic stem (ES) cells are commonly cultured on feeder layers of primary murine embryonic fibroblasts (M
58 rmatogonial behavior similar to that seen on feeder layers of SNL fibroblasts.
59 tent cells without the need for coculture on feeder layers or cell sorting to obtain a highly enriche
60 (hESCs) are routinely cultured on fibroblast feeder layers or in fibroblast-conditioned medium (CM).
61 f these iPS cells on mitotically inactivated feeder layers prepared from the same amniocytes.
62 ddition of neuregulin-1 to cultures on MSC-1 feeder layers resulted in spermatogonial behavior simila
63 enescence on plastic (S-P) and senescence on feeder layers (S-FL) in HMECs, as it does also in cultur
64 ns cultivated in the absence of an astrocyte feeder layer showed abundant AbetaO binding to dendritic
65                   Serially passaged with 3T3 feeder layer support, the keratinocytes were examined fo
66 nsional collagen gel matrix and a fibroblast feeder layer that supports hepatocytic differentiation f
67 ficient, serum-free culture medium without a feeder layer, that could be used for intra-oral grafting
68                        When cultured without feeder layers, the early lineage markers disappear, and
69 hen cortical neurons are cultured on a glial feeder layer, they do not reach nearly as mature a pheno
70 nd cell mixtures failed to produce a stromal feeder layer to support marrow cell growth in vitro.
71 rly hepatic lineage markers when cultured on feeder layers; under these conditions, neither mature he
72           The presence of a mesenchymal cell feeder layer was essential for supporting hepatocytic di
73 ch passage, clonogenicity on 3T3 fibroblasts feeder layers was compared among progenitor cells remove
74 nic stem cells (hESCs) differentiated on S17 feeder layer were transplanted by intraperitoneal inject
75 e system using fetal bone stromal cells as a feeder layer, which facilitates the survival and growth
76 experiments use primary embryonic fibroblast feeder layers, which assist in the maintenance of the pl

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