ライフサイエンスコーパス: feeding behavior

1 importance in the higher-order regulation of feeding behavior.

2 hypothalamus as mediating rapid control over feeding behavior.

3 ention and reward processing and may promote feeding behavior.

4 mic Agrp neurons are known to be crucial for feeding behavior.

5 retory cells and could control locomotor and feeding behavior.

6 ed energy expenditure without alterations in feeding behavior.

7 extrusion in controlling MC4R signaling and feeding behavior.

8 othalamic populations that are important for feeding behavior.

9 hypothalamus to the midbrain responsible for feeding behavior.

10 sting hypotheses about craniodental form and feeding behavior.

11 cilia on hypothalamic neurons in regulating feeding behavior.

12 directly controls the movements involved in feeding behavior.

13 affects learned and motivational aspects of feeding behavior.

14 ptor expressing neurons had no effect on the feeding behavior.

15 controlling synaptic plasticity, memory, and feeding behavior.

16 with both microbiome composition and insect feeding behavior.

17 re part of an extended circuit that mediates feeding behavior.

18 granule interneuron production depending on feeding behavior.

19 in myc234 drastically modified S. littoralis feeding behavior.

20 cuate nucleus (ARH), a center that regulates feeding behavior.

21 omponent of the neural circuitry controlling feeding behavior.

22 the PVT itself may be involved in mediating feeding behavior.

23 pocyte to the central nervous system to time feeding behavior.

24 development of neural circuits that regulate feeding behavior.

25 ical manipulation of opioid receptors alters feeding behavior.

26 ressed in the hypothalamus where it controls feeding behavior.

27 rcuits that are critical for leptin-mediated feeding behavior.

28 rlipidemic rats without affecting the normal feeding behavior.

29 ate the state of satiety into alterations in feeding behavior.

30 hat vector infection by a plant virus alters feeding behavior.

31 e brain sites controlling multiple levels of feeding behavior.

32 na fide mechanoreceptor channel for nematode feeding behavior.

33 tion in the hypothalamus, a key structure in feeding behavior.

34 important hormone in the central control of feeding behavior.

35 impacts other brain structures that regulate feeding behavior.

36 of diet provides more nuanced insights into feeding behavior.

37 d the entire body, most likely by regulating feeding behavior.

38 ioning and lipid metabolism independent from feeding behavior.

39 and taste bud numbers as part of a change in feeding behavior.

40 regulating higher-order cognitive aspects of feeding behavior.

41 GABA(B) receptors in the tLH act to suppress feeding behavior.

42 s essential for normal cognitive arousal and feeding behavior.

43 clock in the Me5 serves in regulating daily feeding behavior.

44 there is an effect of estrogen and leptin on feeding behavior.

45 unctional significance of these peptides for feeding behavior.

46 ard behavior and the motivational aspects of feeding behavior.

47 BA) neurons in mice to compare their role in feeding behavior.

48 in the regulation of sleep architecture and feeding behavior.

49 t and evidence for their role in controlling feeding behavior.

50 oid receptors, both of which are involved in feeding behavior.

51 f the LHa does not have a reliable effect on feeding behavior.

52 ialist birds known for their touch-dependent feeding behavior.

53 n influence distinct components of motivated feeding behavior.

54 he hypothalamus are critical for homeostatic feeding behavior.

55 pallial amygdala that has been implicated in feeding behavior.

56 elate to its loss of a radula and its unique feeding behavior.

57 g between tanycytes and AN neurons, altering feeding behavior.

58 uate their effects on sucrose perception and feeding behavior.

59 regional heterogeneity in frontal control of feeding behavior.

60 eptides, is a crucial process that regulates feeding behavior.

61 important molecular mechanism that regulates feeding behavior.

62 that integrates nutrient signals to control feeding behavior.

63 cally modulate ARC(AgRP) neuron activity and feeding behavior.

64 s cell cluster that suppress mating, but not feeding behavior.

65 re both necessary and sufficient for driving feeding behavior.

66 organ, regulating digestion, metabolism, and feeding behavior.

67 gans integrate internal and external cues in feeding behavior.

68 tivity minutes), parenting style, and parent feeding behaviors.

69 ies with sediment-associated food chains and feeding behaviors.

70 rate a causal link between cue responses and feeding behaviors.

71 ists of GPR103 could play a role in managing feeding behaviors.

72 ysical activity, parenting style, and parent feeding behaviors.

73 ls play critical roles in food selection and feeding behaviors.

74 nxiety, mood, and drug abuse, in addition to feeding behaviors.

75 vous system, thereby disrupting swimming and feeding behaviors.

76 ircuits may trigger deviations from adaptive feeding behaviors.

77 antitative trait locus (QTL) analysis of key feeding behaviors.

78 uces body weight, food intake, and motivated feeding behaviors.

79 t be reaching parents and influencing infant feeding behaviors.

80 the brain structures involved in reward and feeding behaviors.

81 from D. wrightii elicited normal flight and feeding behaviors.

82 isms of the LHA that contribute to motivated feeding behaviors.

83 is involved in the regulation of stress and feeding behaviors.

84 echnique can be applied to validate reported feeding behaviors.

85 xtracellular ATP signaling in the control of feeding behaviors.

86 are involved in compulsive and perseverative feeding behaviors.

87 chicken taste buds in association with their feeding behaviors.

88 ird ventricle (3V) affected Siberian hamster feeding behaviors; (2) whether food deprivation (FD) co-

89 geoning literature focusing on addictive and feeding behaviors across multiple domains and levels of

90 c AgRP and POMC neurons in the regulation of feeding behaviors across multiple timescales.

91 cture was analyzed to identify components of feeding behavior affected by Ex4.

92 intervention successfully modified parental feeding behaviors, affected children's diets positively,

93 Changes in feeding behavior after acute intraperitoneal administrat

94 Changes in feeding behavior after intracerebroventricular injection

95 examining the effects of leptin repletion on feeding behavior after weight loss.

96 ently from the sea lion but displays similar feeding behavior, also has all three Tas1rs inactivated,

97 cause Mchr1 is involved in the regulation of feeding behavior and BBS is associated with hyperphagia-

98 in the central nervous system (CNS) affects feeding behavior and body energy stores, the metabolism

99 into the molecular and neural components of feeding behavior and body weight regulation.

100 aria endemicity reflect, in part, changes in feeding behavior and climate adaptation of mosquito vect

101 he hypothalamus and has a positive impact on feeding behavior and energy balance.

102 ronal circuits involved in the regulation of feeding behavior and energy expenditure are soft-wired,

103 l energy balance and adiposity by regulating feeding behavior and energy expenditure, the roles for i

104 ggested that FTO plays a role in controlling feeding behavior and energy expenditure.

105 st century regarding the neuronal control of feeding behavior and energy expenditure.

106 molecular and neuronal mechanisms of complex feeding behavior and energy expenditure.

107 iates affect neural outputs that modify both feeding behavior and energy expenditure.

108 in the CNS of all vertebrates that regulates feeding behavior and energy homeostasis via interaction

109 ns, including the involvement of the MC4R in feeding behavior and energy homeostasis, making this sys

110 4 receptor is involved in the control of the feeding behavior and energy homeostasis.

111 s a stomach hormone normally associated with feeding behavior and energy homeostasis.

112 Other mechanisms that govern feeding behavior and food reward may also underlie the i

113 the ARC to the PVN are pivotal for balancing feeding behavior and glucose metabolism, we investigated

114 egulated balance between nutrient resources, feeding behavior and growth rate.

115 ophila brain houses the circuitry underlying feeding behavior and is involved in many other aspects o

116 has focused attention on the role of MC4R in feeding behavior and macronutrient intake.

117 ent did not activate c-Fos expression in key feeding behavior and metabolic centers in ZDF rat brain

118 ehavior, hypersensitivity to stress, altered feeding behavior and metabolism, and cardiovascular abno

119 As Mchr1 mediates feeding behavior and metabolism, our results implicate c

120 gulation of physiological responses, such as feeding behavior and mood, and has been implicated in th

121 t the major fat metabolism pathway regulates feeding behavior and NRs could be the mediators to link

122 lucose sensing is involved in the control of feeding behavior and peripheral glucose homeostasis, and

123 phum padi), by examining aphid life history, feeding behavior and plant physiology and biochemistry.

124 t a critical role for VTA Lepr in regulating feeding behavior and provide functional evidence for dir

125 suggest that molecular pathways controlling feeding behavior and reproduction in solitary insects ar

126 nal state to forebrain regions implicated in feeding behavior and responses to immune challenge, and

127 uronal circuits in the brain help to control feeding behavior and systemic metabolism in response to

128 r, FOXO/4E-BP signaling in muscles decreases feeding behavior and the release of insulin from produci

129 erphagic conditions plays important roles in feeding behavior and thermogenesis by modulating neurona

130 pothalamus (DMH) has long been implicated in feeding behavior and thermogenesis.

131 ry to assess shark residency in the pass and feeding behavior and used bioenergetics models to unders

132 ed measures of subjective "automaticity" for feeding behaviors and a brief child food-frequency measu

133 Taste compounds elicit innate feeding behaviors and act as rewards or punishments to e

134 a temporal association between limited child feeding behaviors and risk for HHV-8 infection.

135 Few studies have examined observed maternal feeding behaviors and their potential association with c

136 to identify factors associated with maternal feeding behaviors and to test the hypothesis that more m

137 ell function, adipocyte differentiation, and feeding behavior) and presented chemical screening data

138 components, plant growth patterns and insect feeding behavior) and revealed that leaf amino acid cont

139 s an important role in modulating analgesia, feeding behavior, and a range of autonomic functions.

140 ous system sites of direct ghrelin action on feeding behavior, and as inspiration for future studies

141 The brain ultimately determines feeding behavior, and here we review the mechanisms by w

142 r is involved in obesity, energy metabolism, feeding behavior, and viability.

143 roduction; and modulating sperm competition, feeding behaviors, and mating plug formation.

144 ted with changes in parental automaticity of feeding behaviors, and program acceptability was high.

145 Importantly, these effects on feeding behavior are observed in the absence of any meas

146 The effects of these two pathways on feeding behavior are unaltered by conditioning.

147 of child obesity is whether and how parental feeding behaviors are associated with the food intake an

148 Accordingly, neurons that contribute to feeding behaviors are localized to central, peripheral,

149 Our observations identify enhanced feeding behavior as a novel component of the Drosophila

150 ite of integration for control mechanisms of feeding behavior as it has extensive reciprocal connecti

151 For all parental feeding behaviors, automaticity increased more in the in

152 dered passive feeding as compared with other feeding behaviors because the whales do not swim forward

153 originate in the nervous system and regulate feeding behavior but also peripheral fat regulation thro

154 are known to regulate energy homeostasis and feeding behavior, but how these circuits are established

155 Therefore, NPY also increased appetitive feeding behaviors, but its consummatory effects were lim

156 exposure to palatable foods can drive future feeding behavior by "rewiring" mesolimbic dopamine neuro

157 Analysis of feeding behavior by using lickometer cages revealed that

158 omeostatic activities, including metabolism, feeding behaviors, cardiovascular functions and reproduc

159 oreover, they displayed a novel head-lifting feeding behavior characterized by holding the vertical p

160 ) received training on habit formation for 3 feeding behaviors; control participants (n = 68) were as

161 irgin females mimics the effect of mating on feeding behavior, demonstrating that SP is the main agen

162 Animals change feeding behavior depending on their metabolic status; st

163 oreover, these neurons orchestrate different feeding behaviors depending on the magnitude of the stim

164 Here we show that this feeding behavior depends on fat metabolism mediated by t

165 f increasing the homeostatic drive to eat on feeding behavior during appetite suppressing conditions

166 Hypothalamic functions that affect feeding behavior, endocrine function, and circadian rhyt

167 eural circuits relevant to the regulation of feeding behavior, energy expenditure, and glucose homeos

168 d circuits that underscore the regulation of feeding behavior, energy expenditure, glucose homeostasi

169 nction due to the role of these receptors in feeding behavior, energy homeostasis, sexual function, e

170 colepsy and that has also been implicated in feeding behavior, energy homeostasis, thermoregulation,

171 Feeding behavior engages multiple somatic and visceral t

172 ator generates approximately 24-h rhythms in feeding behavior, even under constant environmental cond

173 However, these differences in ecology and feeding behavior failed to explain the differences in to

174 al functions, including glucose homeostasis, feeding behavior, fat deposition, bone remodeling, and v

175 to create a compendium of genes relevant to feeding behavior (FB) and/or body weight (BW) regulation

176 rtions of frontostriatal systems may release feeding behaviors from regulatory control, thereby perpe

177 ne plasma levels while stimulating grooming, feeding behaviors, gastric transit and acid secretion, w

178 erations in circulating hormones involved in feeding behavior, glucose metabolism, hunger, and appeti

179 interaction between opioids and adenosine on feeding behaviors has received less attention.

180 Leptin plays a central role in regulating feeding behavior, homeostasis and reproduction.

181 thought to be involved in the regulation of feeding behavior, hormone secretion, and reproduction.

182 licated the cholinergic system in modulating feeding behavior; however, its specific function remains

183 Here, we explore the genetic architecture of feeding behavior in a herbivorous insect that has become

184 Taste memories allow animals to modulate feeding behavior in accordance with past experience and

185 limits several starvation-induced changes in feeding behavior in adult Drosophila, including increase

186 properties of SNc mDA neurons and impact on feeding behavior in adult mice.

187 ; it also regulates fatty acid oxidation and feeding behavior in animals.

188 lenges we searched for compounds that affect feeding behavior in C. elegans and sought to identify th

189 the evolution of a specific trait--divergent feeding behavior in D. sechellia.

190 s in brain expression of genes implicated in feeding behavior in Drosophila melanogaster.

191 Here, we showed that DEET suppressed feeding behavior in Drosophila, and this effect was medi

192 ilarity in gustatory system organization and feeding behavior in flies and mammals, as well as diurna

193 An automated analysis of feeding behavior in freely moving flies shows that IR60b

194 Studies of feeding behavior in genetically tractable invertebrate m

195 essential regulators of centrally regulated feeding behavior in invertebrates, the role of this prim

196 This review examines human feeding behavior in light of psychological motivational

197 entrating hormone (MCH) is implicated in the feeding behavior in mammals affording a potential target

198 , Wu et al. show that these neurons modulate feeding behavior in mice by providing GABAergic input to

199 Accordingly, we detected compulsive-like feeding behavior in obese but not lean rats, measured as

200 Our results suggest that feeding behavior in pea aphids is neither simple nor hig

201 Here, we examine the modulation of feeding behavior in the fruit fly, Drosophila melanogast

202 As a model, we examine the hovering flower-feeding behavior in the hawkmoth Manduca sexta In the la

203 otor neurons, B7 and B8, which contribute to feeding behavior in the marine mollusk Aplysia californi

204 tightly linked genes) has a major effect on feeding behavior in the pea aphid.

205 genes and/or pathways controlling anemia and feeding behavior in the trypanotolerant N'Dama, coat col

206 To address this question, we examined two feeding behaviors in the marine mollusk Aplysia californ

207 They also exhibit a distinctive feeding behavior, in which the premaxilla is cyclically

208 RYGBP-induced altered feeding behavior, including reduced appetite and changes

209 uninfected males, with the frequency of all feeding behaviors increasing by up to threefold, thus in

210 Electrical monitoring of the GPA feeding behavior indicates that the GPA stylets found si

211 he hypothesis that capacity to modify vector feeding behavior is a conserved trait among plant- and a

212 Feeding behavior is a highly complex process with multip

213 that bitter-compound-mediated inhibition on feeding behavior is alleviated by acids.

214 Feeding behavior is heavily influenced by hippocampal-de

215 Feeding behavior is influenced primarily by two factors:

216 Rather, feeding behavior is modulated by various contextual fact

217 Feeding behavior is orchestrated by neural circuits prim

218 Altered "nocturnal" activity and feeding behavior is present from a very early age and do

219 role of IC cue-related activity in mediating feeding behaviors is poorly understood.

220 orexin system in regulating wakefulness and feeding behavior, little is known about the downstream s

221 The oligogenetic basis of variation in feeding behavior may facilitate host shifts, providing o

222 between signals of homeostasis and motivated feeding behavior may inspire new treatment options for e

223 across levels of BMI and varying aspects of feeding behavior may promote the identification of novel

224 ut innervation in INT-BDNF(-/-) mice altered feeding behavior, meal pattern and microstructural analy

225 Feeding behavior, metabolism and circadian clocks are in

226 factors may carry enduring consequences for feeding behavior, metabolism, and obesity risk.

227 nervous system and has a role in regulating feeding behavior, obesity, energy homeostasis, male erec

228 on from foraging (feeding) to wandering (non-feeding) behavior occurs prior to pupariation and metamo

229 quences of future ocean acidification on the feeding behavior of a deep-sea echinoid, the sea urchin,

230 Feeding behavior of Aplysia provides a useful model syst

231 use in chimpanzees [3] and in the vocal and feeding behavior of cetaceans [4, 5].

232 BLM and TRA frameworks are confounded by the feeding behavior of D. magna where the ingestion of AgNP

233 Little is known about the feeding behavior of hematophagous insects that require p

234 ecting genus in the Bunyaviridae, alters the feeding behavior of its thrips vector, Frankliniella occ

235 We investigated the feeding behavior of mice carrying a non-functional 5-hyd

236 r during phytoplankton blooms and the filter-feeding behavior of the blue mussel.

237 eed consumed per day, but did not affect the feeding behavior of the dominant and subordinate fish.

238 faunivory, provide crucial data to infer the feeding behavior of the first dinosaurs.

239 e-driven changes in leaf metabolomics on the feeding behavior of Trichopulsia ni larvae.

240 lutionary relationships, as well as diet and feeding behavior, of ancient wolves.

241 This inferred feeding behavior offers a generalized view of dinosaur f

242 ks, and potential impacts of those roles and feeding behavior on associated extinctions.

243 the SCN and the number of influences such as feeding behavior on circadian rhythm.

244 ranscript by RNA interference disturbs aphid feeding behavior on fava beans measured by the electrica

245 is whether to engage in active movement and feeding behavior or to become quiescent.

246 at regulate insulin-like peptide expression, feeding behavior, or both.

247 ing meal frequency, daytime versus nighttime feeding behavior, or locomotor activity.

248 agal afferents are involved in regulation of feeding behavior, particularly meal size, and have been

249 lob may serve a novel regulatory function in feeding behavior, possibly by influencing the excitabili

250 ation for the onset of peak circadian insect feeding behavior, providing evidence for the underlying

251 ersatile regulator of energy expenditure and feeding behavior, rapidly binds neurons in the vicinity

252 Feeding behavior rarely occurs in direct response to met

253 ccount for the previously reported decreased feeding behavior, reduced growth rates and aborted devel

254 MCT1 expression in tanycytes plays a role in feeding behavior regulation.

255 r perceived food quality into alterations in feeding behavior remains poorly understood.

256 uality and signal to fat metabolism, growth, feeding behavior, reproduction, and life span.

257 Feeding behaviors require intricately coordinated activa

258 n (POMC) positively and negatively influence feeding behavior, respectively, possibly by reciprocally

259 Electrical monitoring of aphid feeding behavior revealed that PAD4 modulates a phloem-b

260 tal modulation of two distinct components of feeding behavior: reward valuation based upon taste perc

261 BMI and feeding-behavior scales were transformed to SD scores.

262 thesis is that the transition to maladaptive feeding behavior seen in eating disorders or obesity may

263 ion promoting habit formation for 3 parental feeding behaviors: serving fruit/vegetables, serving hea

264 eras), permit in situ observations of shrimp feeding behavior, shrimp size and internal anatomy, and

265 le for MeCP2 in the regulation of social and feeding behaviors since the Mecp2 conditional knockout (

266 ominantly anchovies, demonstrated a range of feeding behaviors such as oblique, vertical, and lateral

267 erences in a number of phenotypes, including feeding behavior, such as filter feeding in the Mysticet

268 ulate energy stores, free glucose levels, or feeding behavior suggesting the sleep phenotype of trsn

269 neurons disrupts conditioned, but not naive, feeding behavior, suggesting these neurons are selective

270 halamus (LH) has long been known to regulate feeding behavior, taste processing in LH remains relativ

271 mbionts (i.e. yeast-like symbionts, YLS) and feeding behavior that can interact to affect the spread

272 e loopers (Trichoplusia ni) display rhythmic feeding behavior that is sustained under constant condit

273 restriction leads to an altered anticipatory feeding behavior that temporarily abrogates the anorecti

274 Leptin appears to modulate feeding behavior through these circuits, suggesting ther

275 rons and motoneurons, and, in one case, link feeding behavior to gut peristalsis and locomotion.

276 r and NRs could be the mediators to link the feeding behavior to the metabolic changes.

277 fts or sleep deprivation, it markedly alters feeding behaviors ultimately promoting obesity and insul

278 ulation of the NPY/AgRP neurons that promote feeding behavior until satiety signals kick in.

279 of color vision on reproductive success and feeding behavior using nine years of morphological, demo

280 h suggests that colon-derived SCFAs modulate feeding behavior via central mechanisms.

281 teral hypothalamus (LHA) regulates motivated feeding behavior via GABAergic LHA neurons.

282 rvation and participate in the expression of feeding behavior was comparable in OEA-treated WT and HD

283 This suspected feeding behavior was less appreciable in the presence of

284 found that the only compound that stimulated feeding behavior was morphine.

285 Neuropeptide expression and feeding behavior were measured in MCT1-inhibited animals

286 dy distribution, and (iii) effect on isopods feeding behavior were observed regardless of whether the

287 experimental enclosure where observations of feeding behavior were performed.

288 Stool consistency and color and feeding behavior were recorded.

289 Insect settling and feeding behavior were unaffected on the mpl1 mutant.

290 monstrated a reduced longevity and a reduced feeding behavior when the animals were left unperturbed.

291 et a background motivational tone regulating feeding behavior, whereas beta-endorphin underlies orose

292 Activation of TPNs influences innate feeding behavior, whereas inhibition has little effect,

293 , but we demonstrate here that rippling is a feeding behavior which occurs when M. xanthus cells make

294 pseudogenized, consistent with their unique feeding behavior, which entails swallowing food whole wi

295 NPF acutely increases sleep without altering feeding behavior, which it affects only on a much longer

296 ted the hypothesis that the effect of MOR on feeding behavior will be attenuated in the absence of th

297 circuitries that regulate hedonic aspects of feeding behavior will be reviewed.

298 These included reduced blood feeding behavior, with almost 100% of insects infected w

299 nal nutrient sensors play important roles in feeding behavior, yet their molecular structure and mech

300 and nutrient additions influenced herbivore feeding behavior, yet while sea urchins preferred nutrie