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4 NV replication were derived from studies of feline calicivirus and rabbit hemorrhagic disease virus,
5 further divided into the following species: Feline calicivirus and Vesicular exanthema of swine viru
6 of the canonical start/stop site in huNV and feline calicivirus but not in rabbit hemorrhagic disease
7 el neutralizing B-cell epitope, derived from feline calicivirus capsid protein, and a well characteri
8 irus strains, MD145-12 (genus Norovirus) and feline calicivirus (FCV) (genus Vesivirus), to investiga
10 solution structures of the VPg proteins from feline calicivirus (FCV) and murine norovirus (MNV), whi
11 located at the 3' end of the genomic RNA of feline calicivirus (FCV) encodes a small (12.2-kDa) mino
12 s approach is demonstrated by the capture of feline calicivirus (FCV) from cell culture media that is
14 Here we show how longitudinal analysis of feline calicivirus (FCV) infection in an animal rescue s
19 study was to identify the active form of the feline calicivirus (FCV) RNA-dependent RNA polymerase (R
21 mid was engineered in which the LC region of feline calicivirus (FCV) was placed under the control of
24 LBC) epitopes in the major capsid protein of feline calicivirus (FCV), an expression library containi
25 qPCR) for feline herpesvirus type 1 (FHV-1), feline calicivirus (FCV), Mycoplasma felis, Chlamydophil
26 ause of infectious acute gastroenteritis and feline calicivirus (FCV), which causes respiratory illne
31 in types of feline coronaviruses (FCoVs) and feline caliciviruses (FCVs), respectively, and are impor
33 gs on the RdRp activity of the norovirus and feline calicivirus Pro(-)Pol enzymes were compared and f
35 death in young cats, and virulent, systemic feline calicivirus (vs-FCV) causes a highly fatal diseas
36 we have been exploiting endemic infection of feline calicivirus within five geographically distinct h
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