ライフサイエンスコーパス: female sex

1 c hospital resources, late presentation, and female sex.

2 renal disease (ESRD) patients, especially of female sex.

3 ed with an increased risk of CP were age and female sex.

4 omy use, number of stents, hypertension, and female sex.

5 ongest predictors were continued smoking and female sex.

6 h increased age, later age of AMD onset, and female sex.

7 xicity included obesity, increasing age, and female sex.

8 recurrent ACS over 12 months, independent of female sex.

9 decrease in VSF; 95% CI, -0.66 to -0.43) and female sex (-0.25 logit decrease in VSF; 95% CI, -0.45 t

10 ; >/=80 years: 7.57 [5.71-10.04], P < .001), female sex (1.10 [1.01-1.20], P = .047), nonwhite race/e

11 face area 2.2 versus 1.8: 1.44 [1.35-1.53]), female sex (1.38 [1.33-1.43]), immunosuppression (1.38 [

12 e (quantified as odds ratios [95% CIs]) were female sex (1.97 [1.64-2.37]), immigrant background (1.4

13 ed (data reported as odds ratio [95% CI]) by female sex (2.4 [2.3-2.5]), entering Army service at 25

14 per 100,000 person-years) were predicted by female sex (2.8 [2.0-4.1]), entering Army service at 25

15 Variables correlated with TdP were (1) female sex, 2) 500-mug dose, (3) reduced ejection fracti

16 ), 60 to 69 (-5), 70 to 79 (-6), >/=80 (-7); female sex (4); arterial hypertension (-4); positive fam

17 .8 years at the time of the index diagnosis; female sex, 52%) received a diagnosis of QAV between Jan

18 The only independent predictors were female sex (54.4% vs 27.2%; P = .018) and the 3-month BC

19 age at diagnosis (mean, 49 vs 57 years) and female sex (9% vs 21% for males; P < .001).

20 Female sex, a low maternal level of education, and less

21 with time to spontaneous clearance included female sex (adjusted hazards ratio [AHR]: 2.16; 95% CI:

22 Female sex (adjusted odds ratio [OR] 0.69; 95% CI, 0.59-

23 n in pts with initially successful CA for AF female sex, AF type, in-hospital AF relapse and comorbid

24 Older age, female sex, African American race and Hispanic ethnicity

25 derivation cohort included the variables of female sex, age of 13 years or older, physician-diagnose

26 idence interval: 1.117 to 2.407; p = 0.012), female sex, age, diabetes, smoking, heart failure, previ

27 .2, a mean CHADS2VASC (CHADS2 in addition to female sex, ages 65-75, as well as double impact of age

28 9 studies identified an association between female sex and a greater willingness to undergo bariatri

29 In pCIS, female sex and a multifocal onset were risk factors for

30 The multivariate analysis revealed female sex and AF type prior to the procedure as predict

31 emonstrated there was no association between female sex and attrition at our institution.

32 Female sex and black race were associated with higher an

33 variate analysis showed that increasing age, female sex and d4T exposure were associated with increas

34 ndently associated with major bleeding risk; female sex and DBP <90 mm Hg were associated with a decr

35 rge cohort of patients with TTR amyloidosis, female sex and decreased VA were associated with ocular

36 Female sex and device infections are associated with hig

37 Independent predictors of complications were female sex and device infections.

38 Recent studies have focused on the role of female sex and estradiol (E2) in pulmonary arterial hype

39 aring the major genetic risk factors for MS (female sex and HLA risk haplotypes).

40 nhanced platelet activation correlating with female sex and microvascular and oxidative damages.

41 For all recipients, female sex and primary sclerosing cholangitis were assoc

42 Female sex and receipt of antibiotics during the hospita

43 Female sex and shockable rhythms were associated with hi

44 Female sex and the presence of both risk alleles of CFH-

45 Female sex and the presence of comorbidities were associ

46 high nevus count, low risk for melanoma, and female sex) and 1 melanoma-related factor (in situ melan

47 f anesthesia assistance included black race, female sex, and a nonscreening indication; anesthesia as

48 bserved in 68.9% of patients, with AMD type, female sex, and age being independent risk factors.

49 of proximal large polyps increased with age, female sex, and black race.

50 Risk factors for recurrence were older age, female sex, and comorbidity.

51 ptor antagonists at presentation, older age, female sex, and history of heart failure.

52 verse outcome were younger age at diagnosis, female sex, and increased left atrial dimension.

53 Younger age, female sex, and lower income were also independently ass

54 ssion included remote history of depression, female sex, and more symptomatic angina based on Canadia

55 ransplantation included older recipient age, female sex, and percentage weight gain within 12 months

56 requency was inversely related to older age, female sex, and smoking.

57 literacy level, a higher educational level, female sex, and the absence of diabetes mellitus.

58 increasing age, clinical stage III disease, female sex, and the presence of medical comorbidities (a

59 ants were significantly associated with age, female sex, and valvulopathy.

60 entricular hypertrophy included systolic BP, female sex, anemia, and use of other antihypertensive me

61 race/ethnicity (aOR 3.3; 95% CI, 2.3, 4.7), female sex (aOR 2.0; 95% CI, 1.4, 2.9), age >/= 35 years

62 Female sex (AOR: 3.96, 95% CI: 1.55-10.09), higher grade

63 A resistance in MDR MTBC was associated with female sex (aPR, 1.25; 95% CI, 1.08-1.46) and previous t

64 e, smoking, plasma HDL cholesterol, BMI, and female sex are associated with AMD.

65 t to iliofemoral minimal artery diameter and female sex are associated with vascular complications re

66 Younger age and female sex are both associated with greater mental stres

67 rotype; for 5 and 6 serotypes, respectively, female sex (around 2-fold increased odds) and cigarette

68 an elevated alkaline phosphatase level, and female sex as independent risk factors.

69 nts, logistic regression analysis identified female sex as protective for all-cause mortality (odds r

70 alth Stroke Scale at admission, hemineglect, female sex, atrial fibrillation, and no history of strok

71 iate analysis indicated that use of the BVS, female sex, balloon-artery ratio >1.25, expansion index

72 or resection were age younger than 50 years, female sex, being married, higher tumor grade, and prese

73 6; 95% confidence interval [CI], 0.96-2.76), female sex (beta=1.22; 95% CI [0.22-2.24]), minority rac

74 ors were nonmodifiable and included age <60, female sex, black race, higher comorbidity burden, previ

75 eiving testing was inversely associated with female sex, black race, other/unknown race, or having pr

76 6-month unplanned readmissions included age, female sex, black/Hispanic race, prior amputation, Charl

77 Mean age was 57 years, 25% female sex, BMI was 28, CHA2DS2-VASc score was 0 to 1 in

78 with baseline characteristics that included female sex, body mass index >/=35, fasting glucose >5.5

79 Female sex, body mass index >50 kg/m, type 2 diabetes, h

80 ardium scarred to clinical information (age, female sex, body mass index, history of hypertension, di

81 ecies are gonochoristic (i.e., have male and female sexes), but self-fertile hermaphroditic species a

82 ependent on renal function, age, weight, and female sex, but not ethnicity, geographic region, ASA us

83 lymphocyte count, history of depression, and female sex, but not nadir neutrophil count, were associa

84 Female sex, cardiovascular disease, higher body mass ind

85 tes and became partitioned to either male or female sex cells later in evolution.

86 ic background (i.e., lacking the ancestral W female sex chromosome), and is hemizygous.

87 stem by targeting sequences exclusive to the female sex chromosome.

88 tion (XCI) ensure the selective silencing of female sex chromosomes during mouse embryogenesis.

89 Female sex, chronic kidney disease, enrollment date, and

90 aseline clinical characteristics (older age, female sex, chronic obstructive pulmonary disease; P<0.0

91 The EuroSCORE risk factors of female sex, chronic pulmonary disease, neurological dise

92 ic regression analysis included younger age, female sex, comorbid angina, chronic obstructive pulmona

93 associated symptoms with increasing age and female sex; detection of respiratory syncytial virus (RS

94 o is enhanced by expression of the canonical female sex determinant Dsx(F), indicating that chinmo ac

95 ent with this finding, ectopic expression of female sex determinants in the adult testis disrupts tis

96 g from activating genes normally involved in female sex determination and ovarian development and sho

97 We demonstrate that the female sex determination gene, Sex-lethal (Sxl), functio

98 that the Wolbachia insert is now acting as a female sex-determining region in pillbugs, and that the

99 Female sex, diabetic end-stage renal disease, history of

100 lier year of transplantation, nonwhite race, female sex, diagnosis other than dilated cardiomyopathy,

101 ll-cause and attributable mortality included female sex, elevated bilirubin, lymphopenia, and mechani

102 er of antiepileptic drugs before withdrawal, female sex, family history of epilepsy, number of seizur

103 s of cancer in the model included older age, female sex, family history of lung cancer, emphysema, la

104 Female sex, favorable IL28B genotype, and HCV genotype 1

105 the success of genets through their male and female sex functions.

106 ctors influencing longer wait times included female sex, greater age, being employed, less social sup

107 cal features of AF-TR included advanced age, female sex, greater right atrial than left atrial enlarg

108 ore, only prior stroke, age >/=75 years, and female sex had a stronger association with incident stro

109 Female sex had an inverse relationship with PHE growth,

110 R is the opposite of that in SAVR, for which female sex has been shown to be independently associated

111 Male and female sexes have evolved repeatedly in eukaryotes but t

112 Risk factors for reflux recurrence included female sex (hazard ratio [HR], 1.57 [95% CI, 1.29-1.90];

113 Female sex (hazard ratio [HR]: 2.06; 95% confidence inte

114 d ratio, 1.67; 95% CI, 1.24-2.24; P < .001), female sex (hazard ratio, 1.34; 95% CI, 1.03-1.73; P = .

115 ine ADAU was negatively associated with age, female sex, height, and body mass index, and these varia

116 Hepatocellular injury, female sex, high levels of TBL, and a high ratio of aspa

117 ntly associated with the outcome: older age, female sex, higher baseline serum creatinine value, albu

118 Younger age, female sex, higher metabolic equivalents of task achieve

119 s with patients about the use of HS included female sex, higher self-reported knowledge, prior educat

120 res associated with risk included older age, female sex, history of smoking, history of hypertension,

121 st and SMC markers, expression of VEGF-D and female sex hormone receptors, reduced autophagy, and met

122 tween migraine and estrogen, the predominant female sex hormone, with a focus on studies published in

123 for RANK in lung cancer and may explain why female sex hormones accelerate lung cancer development.

124 cal and experimental evidence indicates that female sex hormones and hormonal contraceptives regulate

125 model of anaphylaxis and explore the role of female sex hormones and the mechanisms responsible.

126 There is evidence implicating the role of female sex hormones as a major factor in determining mig

127 tween male and female mice, and we show that female sex hormones can promote lung cancer progression

128 Female sex hormones have been hypothesized to play a rol

129 riectomized mice, we highlighted the role of female sex hormones in the phenotype.

130 as to assess mechanisms behind the impact of female sex hormones on host immune responses to P. aerug

131 Because actions of female sex hormones on normal renal tissue might protect

132 tations throughout the body, mediated by the female sex hormones progesterone and estrogen.

133 The estrogens are female sex hormones that are involved in a variety of ph

134 smoking, epidemiological studies have linked female sex hormones to lung cancer in women; however, th

135 Together, these results indicate that female sex hormones, estrogens, and X chromosome complem

136 e lung IL2Cs, uterine ILC2s are regulated by female sex hormones, which may specialize them for speci

137 menopausal women, which may be attributed to female sex hormones.

138 aling and also was related to the effects of female sex hormones.

139 s age <45 years] = 1.79; 95% CI, 1.03-3.11), female sex (HR = 1.61; 95% CI, 1.04-2.49), and Hispanic

140 education (HR, 0.67; 95% CI, 0.54-0.85) and female sex (HR, 0.65; 95% CI, 0.52-0.80) were associated

141 31 to 1.45]) versus a single-chamber device, female sex (HR, 1.16 [CI, 1.12 to 1.21]), and black race

142 (HR, 1.154; 95% CI, 1.051-1.267; P = 0.003), female sex (HR, 1.263; 95% CI, 1.027-1.553; P = 0.027),

143 wing: age (hazard ratio [HR]=1.04; P=0.039), female sex (HR=2.33; P=0.043), New York Heart Associatio

144 Female sex, hypertension, diabetes mellitus, higher body

145 Most animals have male and female sexes, implying that sex is ancient and beneficia

146 cribed in association with disability in 50, female sex in 40, older age in 34, depression in 36, anx

147 Female sex increased odds by 34%.

148 , vascular disease, aged 65 to 74 years, and female sex) increased from 0/1 to 6+ points, the inciden

149 e presence of underlying medical conditions, female sex, increased mucosal IL-6 level, and longer dur

150 Genetic risk score, older age, and female sex independently correlated with each outcome.

151 Female sex is a known risk factor for complications duri

152 ABSTRACT: Female sex is a risk factor for inherited and acquired l

153 s well as vascular and stroke complications, female sex is an independent predictor of late survival

154 Female sex is an independent risk factor for death or st

155 Female sex is associated with poorer outcomes after surg

156 ion with ulcer or gangrene, age >/=65 years, female sex, large hospital size, teaching hospital statu

157 After multivariable adjustment, older age, female sex, left bundle branch block, worsened heart fai

158 nd presents negative relationships with age, female sex, left ventricular ejection fraction, and body

159 Female sex, longer unpaced QRS duration, and smaller bas

160 Risk factors for caregiver burden include female sex, low educational attainment, residence with t

161 DMRT1 expression in the ovary silenced the female sex-maintenance gene Foxl2 and reprogrammed juven

162 Female sex, maternal age, and high maternal educational

163 -PCI predictors of readmission included age, female sex, Medicare or State insurance, congestive hear

164 On univariate and multivariate analyses, female sex (men to women risk of readmission odds ratio

165 Female sex, more comorbidities, longer symptom duration,

166 g waitlisted individuals included older age, female sex, more years on dialysis before waitlisting, t

167 Female sex, multiple comorbidities, concomitant therapy

168 Female sex,nodular and unclassified or other histologic

169 olbachia insert shows perfect linkage to the female sex, occurs in a male genetic background (i.e., l

170 In multivariate analysis, female sex (odds ratio = 7.05; P = .002) and TDF+emtrici

171 Other predictive factors included female sex (odds ratio [OR] = 1.73), visits 1 year prior

172 Female sex (odds ratio [OR] range, 1.82-2.22), inpatient

173 lly significant associations were found with female sex (odds ratio [OR], 1.68; 95% confidence interv

174 ratio, 4.06; 95% CI, 3.87-4.23; p < 0.001), female sex (odds ratio, 1.17; 95% CI, 1.14-1.20; p < 0.0

175 Female sex (odds ratio, 2.2; 95% confidence interval, 1.

176 death and RHF (121 total events) identified female sex (odds ratio=2.0 [95% confidence interval, 1.2

177 HTLV-1 seropositivity was associated with female sex, older age, and black and Asian race/ethnicit

178 Persons of white race, female sex, older age, and lower BMI were at increased r

179 with emphysema, decreasing body mass index, female sex, older age, and lower percentage change in pr

180 HTLV-2 seropositivity was associated with female sex, older age, nonwhite race/ethnicity, lower ed

181 ing clinical evidence as to the influence of female sex on outcomes after transcatheter aortic valve

182 d (odds ratio [OR] 11.9 [95% CI, 3.4-41.8]), female sex (OR 2.0 [95% CI, 1.1-3.6]), and childhood hay

183 th a minimum loss of 5 degrees over 2 years: female sex (OR = 1.59, P = 0.03), history of stroke (OR

184 fidence interval [CI], .94-.99; P < .01) and female sex (OR, 0.35; 95% CI, .14-.75; P < .01) were les

185 , 1.03; 95% CI, 1.01-1.04; P < .01), whereas female sex (OR, 0.46; 95% CI, .33-.66; P < .01) and hist

186 older age (odds ratio [OR], 1.03; P < .001), female sex (OR, 1.25; P < .001), black race (vs white; O

187 3.4; 95% confidence interval [CI], 2.6-4.4), female sex (OR, 2.0; 95% CI, 1.3-3.2), and presence of r

188 Female sex (OR, 3.9; 95% CI, 1.7 to 9.5; P < .01) and ch

189 ement to bed (OR: 0.49; 95% CI: 0.44, 0.55), female sex (OR: 0.53; 95% CI: 0.5, 0.56), younger age (O

190 dds ratio [OR]: 1.05; 95% CI: 1.02 to 1.08), female sex (OR: 2.1; 95% CI: 1.1 to 4.0), heart failure

191 factors in questionnaires were older age and female sex ( P < .01).

192 Risk of deficiency was increased 2.0-fold by female sex (P < .001) and 1.4-fold by winter (P < .05).

193 were history of prior radiation (P = .022), female sex (P < .001), age </= 65 years (P = .005), cyto

194 ade hemorrhage was more likely to occur with female sex (P = .001), older age (P = .003), emphysema (

195 a self-limiting AHC that was associated with female sex (P = .028), older age (P = .018), alanine ami

196 a lower CD4(+) T-cell count (P < .001), and female sex (P = .047).

197 major vascular complication (p = 0.04), and female sex (p = 0.04).

198 igher baseline NMS score was associated with female sex (p=0.008), higher baseline MDS-UPDRS Part II

199 < .001) and male (21 for male sex and 19 for female sex, P < .001) patients from high-volume institut

200 le urinary tract infections (UTIs) and their female sex partners, 6 strains (all UTI causing) were sh

201 ners and inversely associated with number of female sex partners.

202 Predictors of baseline MWS scores include female sex, personal history of melanoma, and higher Hos

203 impose selection pressure on the long-range female sex pheromone channel and consequently affect the

204 females (more 5,9-C27:2 and less 7,11-C27:2, female sex pheromone isomers).

205 In moths, more long-range female sex pheromones have been identified than in any o

206 ors using volatile semiochemicals that mimic female sex pheromones.

207 On univariate analysis, older age, female sex, postgraduate year, training in a university

208 Independent predictors for SVCs were female sex, pre-operative New York Heart Association fun

209 Older age, female sex, preference for English, more education, heal

210 tiating RCVS-SAH from cSAH were younger age, female sex, prior hypertension, chronic headache disorde

211 Our findings suggest that female sex protects against systemic inflammation-induce

212 Female sex, public insurance, medical comorbidities, lon

213 subsets; 95% CI, 0.705-0.746): younger age; female sex; racial or ethnic minority; no history of hyp

214 Our data suggest an ancient female sex ratio bias that predates the loss of adults,

215 The male-female sex ratio was 1.17.

216 ne attacks occurring in 37 patients (male-to-female sex ratio, 1.05; mean +/- SD age, 51 +/- 11.4 yr)

217 f one of these genes, DMRT1, lead to male-to-female sex reversal and are associated with development

218 or DMRT1 actively prevents postnatal male-to-female sex reversal by blocking the activation of retino

219 d syndrome, craniosynostosis with XY male-to-female sex reversal or CSR.

220 ryos by RNA interference resulted in male to female sex reversal, characterized by obvious feminizati

221 h range from hypospadias to complete male-to-female sex reversal.

222 macrophages cultured from male, female, and female sex-reversed embryos.

223 leted, but germline stem cells remain, adult females sex-revert to sperm-producing males, indicating

224 Poisson regression modeling identified female sex (RR, 1.30 [95% CI, 1.02-1.65]), assault-relat

225 Female sex (RR, 1.9; 95% CI, 1.3 to 2.6; P < .001) and t

226 stroke, vascular disease, age 65 to 74, and female sex) score might improve stroke prediction in pat

227 for vascular disease, age 65 to 74 years, or female sex) score were 2.8 +/- 1.2 and 4.4 +/- 1.7, resp

228 the reference population included older age, female sex, single cohabitation status, basic educationa

229 Predictors of symptoms included young age, female sex, smoking, and experiencing symptoms before RY

230 Female sex, stage IV disease, and well-differentiated hi

231 N silencing occurs through the action of the female sex-steroid progesterone.

232 Our results suggest that female sex steroids, most likely estrogen, have importan

233 preprocedural analgesia was associated with female sex, term birth, high illness severity, tracheal

234 In multivariable analysis, younger age, female sex, thickness of 0.76 mm or larger, increasing C

235 rtical opacity incidence was associated with female sex, Ultraviolet-B exposure, and a history of dia

236 Younger age, female sex, unemployment, alcohol misuse, and greater op

237 nger age, treatment at an academic facility, female sex, urban population, higher income, lower Charl

238 ined independently predicted by younger age, female sex, urban population, lower Charlson score, smal

239 Independent predictors of UC included female sex, use of Chinese herbal medicine, and transpla

240 Female sex was also a significant risk factor (P = 0.03)

241 Female sex was also associated with a lower risk of LTD

242 cation density, and aortic annulus diameter, female sex was an independent risk factor for higher fib

243 Female sex was associated significantly with a decrease

244 In multivariable analyses, female sex was associated with a greater risk of complic

245 Female sex was associated with a higher rate of 30-day r

246 In a propensity-matched analysis, female sex was associated with a higher rate of vascular

247 After adjusting for >20 risk factors, female sex was associated with a significant risk of dea

248 Female sex was associated with all and uncomplicated eve

249 Adjusting for age, female sex was associated with exudative AMD (OR, 2.10;

250 A competing risk analysis demonstrated that female sex was independently associated with a 10% (conf

251 6.5% vs. 6.5% [p = 0.93], respectively), but female sex was independently associated with improved su

252 Female sex was not associated with outcomes post-ACS.

253 On multivariate analysis, only female sex was significantly associated with serious tho

254 In contrast, female sex was significantly protective for time to deat

255 determinants of DeltaVO2/DeltaWR flattening; female sex was strongly associated (odds ratio, 6.10; co

256 Female sex was the strongest predictor of tongue cleanin

257 Tobacco smoking and female sex were associated with higher levels of plasmin

258 ng from a Muslim family, height for age, and female sex were the socioeconomic risk factors associate

259 group were SAH, trimethylamine, choline, and female sex, whereas plasma phosphatidylcholine was a neg

260 Predictors of ASC were age >75 years, female sex, white race, no prior PVI, nonfemoral arteria

261 This study was designed to determine if female sex work is associated with changes in the mucosa

262 els with robust standard errors clustered on female sex worker (FSW) were used to explore social and

263 al lavage and plasma from 122 HIV-uninfected female sex workers (FSW) and 44 HIV-uninfected low-risk

264 ology and other correlates, we recruited 350 female sex workers (FSW) who were 18 to 50 years old in

265 HIV transmission in the general population, female sex workers (FSW), and men who have sex with men

266 ollected from HIV-seropositive (HIV+) Kenyan female sex workers (FSWs) (n = 20), HIV-seronegative (HI

267 In China, female sex workers (FSWs) are at high risk of syphilis i

268 Project, which offered both interventions to female sex workers (FSWs) at 2 urban clinic sites in Sou

269 Female sex workers (FSWs) bear a disproportionately larg

270 nisms compared to the standard of care among female sex workers (FSWs) in Zambia.

271 -testing may thus be particularly useful for female sex workers (FSWs), who should test frequently bu

272 hey had experience of STD infection, and for female sex workers (FSWs; n = 1,083) when they had high

273 Female sex workers accounted for a large proportion of n

274 vention, that of changing risk behaviours in female sex workers and high-risk men who have sex with m

275 e used to reproduce HIV prevalence trends in female sex workers and their clients, men who have sex w

276 Higher-risk populations included female sex workers and their clients, men who have sex w

277 acquisition in a prospective cohort study of female sex workers and their intimate (noncommercial) ma

278 82 (81%) of 101 female sex workers distributed more than one self-test t

279 n post-partum care, and 41 (14%) of 298 from female sex workers had positive results.

280 ss-sectional data collected from 1,814 adult female sex workers in Karnataka, India, between August 2

281 Female sex workers in Mombasa, Kenya, completed a monthl

282 of their risk of infection, and to high-risk female sex workers only, are $65 160 (95% credible inter

283 infection averted when providing PrEP to all female sex workers regardless of their risk of infection

284 erapy is included; however, PrEP for MSM and female sex workers would be included only at much higher

285 natal care, 117 in post-partum care, and 102 female sex workers); follow-up interviews were completed

286 vention programs for sex workers, especially female sex workers, are cost-effective in several contex

287 IV transmission in specific key populations (female sex workers, male sex workers, and men who have s

288 91 from post-partum care, and 53 (83%) of 64 female sex workers.

289 ther men who have sex with men (MSM) or even female sex workers.

290 eatest HIV burdens continue to be in African female sex workers.

291 te risk for HIV compared with natal male and female sex workers.

292 ork are associated with HIV prevalence among female sex workers.

293 post-partum clinics and a drop-in centre for female sex workers.

294 106 in post-partum care, and 64 (75%) of 85 female sex workers.

295 gs here essentially pertain to prevalence in female sex workers.

296 n: two in the post-partum care group and two female sex workers.

297 terms of a hypothetical protective effect of female sex, yet little research has tested this hypothes

298 ciated with increased odds of mortality were female sex, young age, very severe pneumonia, clinically

299 inoma, with the highest risk associated with female sex, younger age (<60 years), and lighter smoking

300 all cost-related nonadherence measures were female sex, younger age, lower income (<$30000), self-re