ライフサイエンスコーパス: female sterility

1 is in the developing ovary, leading to adult female sterility.

2 Mtrm results in both meiotic catastrophe and female sterility.

3 le integument development arrest, leading to female sterility.

4 resulting in thin and fragile eggshells, and female sterility.

5 y fixed for a recessive mutation that causes female sterility.

6 n interchromosomal epistatic basis to hybrid female sterility.

7 appearance of lethal mutations and dominant female sterility.

8 ific expression of pum partially rescues bem female sterility.

9 leaves and floral organs and causes male and female sterility.

10 lleles support zygotic development but cause female sterility.

11 ard juvenility, flower defects, and male and female sterility.

12 , causes partial male sterility and complete female sterility.

13 of oocytes resulted in shrunken ovaries and female sterility.

14 esis, and the mutants show complete male and female sterility.

15 rity during oocyte development, resulting in female sterility.

16 sterility being up to 23 times greater than female sterility.

17 e of p27 also caused an ovulatory defect and female sterility.

18 s in meristem identity and organ number, and female sterility.

19 egg morphology during oogenesis, leading to female sterility.

20 transgenic overexpression of Datx2 result in female sterility, aberrant sensory bristle morphology, l

21 homozygous effects on hybrid male and hybrid female sterility and inviability.

22 tations in a chicken homolog of VLDL-R cause female sterility and premature atherosclerosis.

23 )] gene of Drosophila melanogaster can cause female sterility and suppress mutations that are inserti

24 Depending on the mutation rate to female sterility and the selective advantage of the fema

25 use hypersensitivity to DNA-damaging agents, female sterility, and defects in repairing double-strand

26 he only detected defect for kelch mutants is female sterility, and kelch protein is localized to the

27 hiffon gene cause thin, fragile chorions and female sterility, and were found to eliminate chorion ge

28 Drosophila hnRNP, Squid (Sqd)/hrp40, causes female sterility as a result of mislocalization of gurke

29 t loss of E2f2 is viable, but causes partial female sterility associated with changes in the mode of

30 produces largely normal behavior but severe female sterility associated with ectopic lov expression

31 e mutation of mouse that results in male and female sterility because of defects in gametogenesis.

32 d, pum null mutations fail to complement bem female sterility, behavioral defects, and neuronal hyper

33 iple mutant phenotypes including male and/or female sterility, bristle defects, and defects in eye de

34 genes of hybrid male sterility and none for female sterility by deficiency mapping.

35 dcp-1 gene, which encodes a caspase, caused female sterility by inhibiting this transfer.

36 tion in which results in small body size and female sterility caused by degeneration of the ovaries.

37 o the null with respect to known phenotypes (female sterility, cell cycle checkpoints, and MMS resist

38 However, the female sterility defect of the Deltapre-1 mat A mutant c

39 A kinase, nhk-1, in Drosophila that leads to female sterility due to defects in the formation of the

40 of the Msh4 gene in mice results in male and female sterility due to meiotic failure.

41 s are involved, including male-sterility and female-sterility factors.

42 es in a rare exception to Haldane's rule for female sterility in field cricket sister species (Teleog

43 s are not sufficient to cause inviability or female sterility in hybrid crosses.

44 sis, we report that Setd1b deficiency causes female sterility in mice.

45 pression of position-effect variegation, and female sterility in which ovaries are underdeveloped and

46 We found that female sterility is due to a requirement for DmBlm in ea

47 trast with the low degree of inviability and female sterility, is far greater than expected from prev

48 ain this transition, some involving male and female sterility mutations linked in a region of suppres

49 to prevent both meiotic catastrophe and the female sterility observed in mtrm/mtrm females.

50 t for the lethality, visible phenotypes, and female sterility observed in these mutants.

51 udying their capacity to rescue the male and female sterility of importin alpha2 null flies.

52 Genetic analysis revealed that the female sterility of mpk3(+/-) mpk6(-/-) plants is a spor

53 Female sterility of Sin- plants is due to abnormal ovule

54 sterility evolves substantially faster than female sterility or hybrid inviability.

55 actors have evolved more rapidly than either female sterility or inviability factors.

56 ations in the eya gene cause loss of ocelli, female sterility, or lethality.

57 1377 and AGAP007280) that confer a recessive female-sterility phenotype upon disruption, and inserted

58 n factors, 44% of which when mutated produce female sterility phenotypes, but most were novel.

59 PRMT5 in early PGCs causes complete male and female sterility, preceded by the upregulation of LINE1

60 Loss of Capu leads to female sterility, presumably because polarity determinan

61 However, rescue of female sterility requires twice as much nm23-H2 expressi

62 Female sterility resulting from oocyte destruction is an

63 aerial hyphae, altered conidiation pattern, female sterility, slow growth rate, lack of hyphal fusio

64 The bem mutant exhibits female sterility, sluggishness, and increased motor neur

65 nes (arg1, nic1), mating type (matA+/matA-), female sterility (ste1), spore-killer (Sk), and a gene g

66 wr-bearing chromosome partially relieves the female sterility, the excisions retain the dominant nodD

67 ad to placental abruption, fetal demise, and female sterility, thereby placing BMPR2 at a central poi

68 Female sterility was also revealed, yet the disparity be

69 Dominant female sterility was not associated with larger deletion

70 ckground, MPK3 is haplo-insufficient, giving female sterility when heterozygous.

71 ed an aberrant DEC-1 proprotein that induced female sterility when introduced into wild-type flies.

72 e size, delayed flowering, and both male and female sterility, whereas etiolated-seedling responses w

73 ockdown in somatic gonadal cells resulted in female sterility, whereas males were fully fertile.

74 utations in RPL27aC result in high levels of female sterility, whereas mutations in RPL27aB have a si

75 nic muscle disruption, reduced viability and female sterility, which could be rescued by hsp70-nautil

76 n in Pten-heterozygous mutants, and leads to female sterility with features that recapitulate the phe