ライフサイエンスコーパス: femoral head

1 ion in the rate of avascular necrosis of the femoral head.

2 ith a medial and posterior slip of the right femoral head.

3 (larger marrow volume fraction) than in the femoral head.

4 ion, and excessive osteoclastogenesis in the femoral head.

5 aphy in the measurement of sphericity of the femoral head.

6 were upregulated in chondrocytes in ischemic femoral heads.

7 trol subjects in all proximal femur regions (femoral head, 8.51-8.73 GPa vs 9.32-9.67 GPa; P = .04; f

8 This study shows that sterilization of femoral head allografts contaminated with S. aureus and

9 Femoral head allografts were contaminated with Staphyloc

10 The activity concentration in the femoral head and neck (mean and s.d. = 0.04 +/- 0.02 %ID

11 jects were assessed for shape changes in the femoral head and neck before, during, and after the deve

12 stic modulus as a measure of strength in the femoral head and neck, Ward triangle, greater trochanter

13 iographic changes include deformation of the femoral head and osteophyte growth, which are usually me

14 rticular chondrocytes were isolated from the femoral head and tibial plateau of patients undergoing k

15 of trabecular bone were taken from both the femoral heads and humeral epiphyses of a 51-y-old male s

16 imarily results from ischemia/hypoxia to the femoral head, and one of the cellular manifestations is

17 illated and nonfibrillated sites of 11 human femoral heads, and extracted in buffer containing 8M ure

18 to other surgical options in men with large femoral heads, and inferior implant survivorship in othe

19 sses anterior, posterior, and lateral to the femoral head; and recesses anterior, posterior, and medi

20 In this study, we researched mouse femoral head articular cartilage explants and knee chond

21 nt between the proximal femoral neck and the femoral head at the level of the open physis, with biome

22 Human cartilage was obtained from femoral heads at joint replacement for OA or following f

23 amples of human cartilage were obtained from femoral heads at the time of joint replacement surgery f

24 ugh radial and tibial length and biiliac and femoral head breadth show signs of responses to directio

25 We studied mature femoral head cap cartilage explants and immature primary

26 in abundance between wild-type and knock-out femoral head cartilage by capillary HPLC tandem mass spe

27 ical analyses were performed to characterize femoral head cartilage from 7 patients with OA and 4 pat

28 In the ex vivo culture of femoral head cartilage from mesenchymal cell-specific Re

29 Serine proteinase gene expression in femoral head cartilage obtained from either patients wit

30 uman articular chondrocytes derived from the femoral head cartilage of patients with a fracture of th

31 parable to arthrography for demonstration of femoral head containment and congruency of the articular

32 ontainment, femoroacetabular congruency, and femoral head deformity.

33 lengths and estimated body masses (based on femoral head diameters).

34 A posterior displacement of the femoral head epiphysis with a physeal step was seen on t

35 s unable to induce aggrecan release from the femoral head explants obtained from Chloe mice that resi

36 We used ex vivo mouse femoral head explants to determine how mMCP-6 and its hu

37 ted radiographic changes in the hip, showing femoral head flattening and secondary degenerative arthr

38 ver, MR imaging failed to depict one case of femoral head flattening.

39 Bone samples of femoral heads from five embalmed donors and five fresh-f

40 wed that the trabecular bone proximal to the femoral head growth plate developed at an earlier time i

41 Lesions occupying more than 30% of the femoral head have high likelihood of joint deterioration

42 urfacing, with various sizes to fit over the femoral head, have been explored.

43 en collected from two implanted instrumented femoral head hemiprostheses.

44 rata was supported by gross pathology of the femoral heads, histologic grading of cartilage slices, a

45 (ACTH) protects against osteonecrosis of the femoral head induced by depot methylprednisolone acetate

46 ent for younger patients with less extensive femoral head involvement.

47 sults of this study indicate that AVN of the femoral head is a frequent complication in children with

48 n risk of fractures and osteonecrosis of the femoral head is less understood.

49 -null mice at 9 weeks, whereas the wild-type femoral head is still composed of hypertrophic chondroct

50 vier infants compared with dimensions of the femoral head (n = 7) and ankle (n = 7) than what is foun

51 r identified on a short-axis MR image at the femoral head-neck junction correlates with surgical find

52 Short-axis MR images of the femoral head-neck junction were reformatted with multipl

53 ement was performed to assess anterosuperior femoral head-neck morphology.

54 ity visible beyond a best-fit circle and (b) femoral head-neck offset angles.

55 modulus were measured in cartilage from the femoral heads of Prg4(-/-) and WT mice ages 2, 4, 10, an

56 cular invasion and ossification start in the femoral heads of TSP3-null mice at 9 weeks, whereas the

57 Osteonecrosis of the femoral head (ONFH) primarily results from ischemia/hypo

58 ply results in ischemic osteonecrosis of the femoral head (ONFH).

59 ll lead to effective measures for saving the femoral head or, better yet, preventing osteonecrosis.

60 Forty-six (92%) of 50 patients with femoral head osteonecrosis at both examinations were pla

61 actors predicting clinical joint outcomes of femoral head osteonecrosis in pediatric patients with le

62 Juvenile femoral head osteonecrosis is due to disruption of blood

63 on of MR imaging earlier in the diagnosis of femoral head osteonecrosis, as well as its more widespre

64 pared to the control group in a pig model of femoral head osteonecrosis.

65 ps in 92 patients with clinical suspicion of femoral head osteonecrosis.

66 associated with impaired blood supply to the femoral head resulting in bone necrosis and collapse.

67 In men with smaller femoral heads, resurfacing resulted in poor implant surv

68 months (range, 5 to 114 months), AVN of the femoral head(s) developed in 49 patients (9%).

69 femoral neck ratio as an interval measure of femoral head shape, and the femoral neck shaft angle.

70 We extracted bone cylinders from human femoral heads, simulated an injury using a drill-hole de

71 +/- 2) to assess the presence of AVN of the femoral head; six children had metabolic renal disease,

72 t in steroid-associated osteonecrosis of the femoral head (SONFH).

73 d artifacts due to simulated implants in the femoral head, sternum, and spine (P = 0.01, 0.01, and 0.

74 a significant reduction of artifacts in the femoral head, sternum, and spine.

75 en in the subchondral zone of osteoarthritic femoral heads, supporting a greater proportion of osteoi

76 spherical, indeterminate, or spherical), the femoral head-to-femoral neck ratio as an interval measur

77 The risk of hip OA increased as the femoral head-to-femoral neck ratio decreased (P for tren

78 4.64-10.41]), and the prevalence of abnormal femoral head-to-femoral neck ratio in at least 1 hip was

79 nilateral hip OA, the prevalence of abnormal femoral head-to-femoral neck ratio in the unaffected hip

80 angle, impingement angle, acetabular slope, femoral head-to-femoral neck ratio, and the crossover si

81 with lesions occupying more than 30% of the femoral head volume; 80% of hips with these lesions coll

82 in the appropriate quartile of percentage of femoral head weight-bearing surface involvement by both

83 The percentage of involvement of the femoral head weight-bearing surface was evaluated subseq

84 Histologic sections of each femoral head were stained and graded.

85 Femoral heads were collected from normal-weight or over-

86 of the right proximal physis below the right femoral head, with a medial and posterior slip of the ri

87 etabolism is increased within osteoarthritic femoral heads, with the greatest changes occurring withi