ライフサイエンスコーパス: fenestrae

1 ulae the keel is perforated by open windows (fenestrae).

2 arly signs of endothelial injury and loss of fenestrae.

3 networks were reduced and their SECs lacked fenestrae.

4 organelles and the diaphragms of endothelial fenestrae.

5 morphology and ontogeny of the portulae and fenestrae.

6 cytoplasmic protrusions through endothelial fenestrae.

7 associated with a change in the diameter of fenestrae and can be interpreted as a component of the "

8 rocess leading to the formation of apertured fenestrae and channels are also described in fenestrated

9 Notch1(lox/lox) mice had reduced endothelial fenestrae and developed portal hypertension and hepatic

10 ndothelial swelling with loss of endothelial fenestrae and occlusion of the capillary lumens.

11 ecognizes specifically the diaphragms of the fenestrae and the stomatal diaphragms of caveolae and tr

12 Loss of diaphragms did not affect the fenestrae and transendothelial channels formation but di

13 ion of the diaphragms of caveolae as well as fenestrae and transendothelial channels.

14 the ultrastructure of glomerular endothelial fenestrae appeared entirely normal.

15 Second, fenestrae are absent from cis cisternae, but are present

16 When all of the fenestrae are closed, the new cell wall is complete.

17 Fenestrae are dynamic structures that can contract or di

18 We demonstrate that glomerular fenestrae are filled with dense negatively charged polys

19 Fenestrae are small pores in the endothelium of renal gl

20 e two structures separate to lie in distinct fenestrae as a bipolar spindle forms.

21 agms, and swollen endothelial cells with few fenestrae as revealed by transmission electron microscop

22 though decades of studies have characterized fenestrae at the ultrastructural level, little is known

23 Through metaphase, the SPBs remain in their fenestrae, bound to the polar ends of spindle MTs; at ab

24 Contraction of fenestrae by serotonin was inhibited by chelation of ext

25 As anaphase proceeds, the nuclear fenestrae close, and the SPBs are extruded back into the

26 , little is known on the mechanisms by which fenestrae form.

27 eolae formation, glomerular endothelial cell fenestrae formation in vivo does not require caveolin-1,

28 The small sizes of the fenestrae have so far prohibited any functional analysis

29 d gain, particularly in relation to bars and fenestrae, have led to a variety of hypotheses concernin

30 examined by scanning electron microscopy for fenestrae in sieve plates.

31 architecture and the ordered arrangement of fenestrae in sieve plates.

32 Previous reports indicate that endothelial fenestrae in vitro can form by fusion of caveolae or cav

33 her formation of glomerular endothelial cell fenestrae in vivo similarly involves caveolae and caveol

34 These results suggest that contraction of fenestrae is associated with Ca2+ influx.

35 of VEGF and TGF-beta resulted in the loss of fenestrae on CP vasculature and thickening of the otherw

36 out the previous hypothesis that endothelial fenestrae represent fused caveolae, at least for glomeru

37 ull, including the antorbital and mandibular fenestrae, serrated teeth, and closed lower temporal bar

38 The number and distribution of the fenestrae suggest that they form at the edges of the med

39 mall upper and ventrally open lower temporal fenestrae, supporting the hypothesis of diapsid affiniti

40 cytosol, ions traverse one of four external fenestrae to enter the rotundal vestibule and then cross

41 ached MTs arise de novo over remaining large fenestrae to focus local growth to these regions.

42 The response of fenestrae to serotonin was mimicked by addition of a Ca2

43 anelles implicated in vascular permeability: fenestrae, transendothelial channels, and caveolae.

44 lae and transendothelial channels as well as fenestrae upon PMA treatment.

45 The reduction in the number of fenestrae was associated with a change in the diameter o

46 Liver endothelial cells possess fenestrae, which are pores supported by a cytoskeleton r

47 tinct diapsids comprise an open framework of fenestrae (window-like openings) separated by bony strut

48 thelial cells, had CD31 immunoreactivity and fenestrae with diaphragms, but they had a branched pheno