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1 MR) spectroscopy of C. difficile, grown with fermentable (13)C substrates, informed dynamic flux bala
2  protein is related to the SNF2 (sucrose non-fermentable 2) family of chromatin remodeling ATPases.
3                          Enterococci provide fermentable amino acids, including leucine and ornithine
4    Although the null mutant can grow on both fermentable and non-fermentable carbon sources at lower
5      The cls1::TRP1 null mutant grew on both fermentable and non-fermentable carbon sources but more
6 a mutant cells when grown in the presence of fermentable and non-fermentable carbon sources, although
7               The consumption of prebiotics (fermentable and nondigestible carbohydrates) has been pr
8 ilize a broad range of substrates, including fermentable biomass-derived sugars.
9 oholic fermentation, the consumption rate of fermentable brewing sugars and dissolved O(2), estimated
10 beneficial probiotic strains, and was highly fermentable by faecal microbiota producing gas and short
11 t that equol excretion may be related to the fermentable carbohydrate content of the diet; additional
12 t be a higher energy density, entailing high fermentable carbohydrate content.
13                Keys advocated a diet high in fermentable carbohydrate for the benefit of general heal
14 es S. pneumoniae with a convenient source of fermentable carbohydrate in vivo.
15 and Yudkin postulated that excessive dietary fermentable carbohydrate intake led-in the absence of de
16 or no changes were observed when the type of fermentable carbohydrate was varied.
17 ver time between acid-producing bacteria and fermentable carbohydrate, and many host factors includin
18 al caries is a cariogenic bacteria-mediated, fermentable carbohydrate-driven dynamic disease.
19 industrial amylases to convert starch into a fermentable carbohydrate.
20    BACKGROUND & AIMS: Dietary restriction of fermentable carbohydrates (a low FODMAP diet) has been r
21             Recently, dietary restriction of fermentable carbohydrates (a low-FODMAP diet) in combina
22 at increased reliance on wild plants rich in fermentable carbohydrates and changes in food processing
23               Current evidence suggests that fermentable carbohydrates and nocebo effects contribute
24 ort-chain fatty acid butyrate, produced from fermentable carbohydrates by gut microbiota in the colon
25                              Poorly absorbed fermentable carbohydrates can provoke irritable bowel sy
26 emical and enzymatic treatments can liberate fermentable carbohydrates from plant biomass, but microb
27 ogical variability-including the presence of fermentable carbohydrates in challenge preparations-limi
28  frequent consumption of plant foods rich in fermentable carbohydrates in food-producing societies.
29          BACKGROUND & AIMS: Poorly digested, fermentable carbohydrates may induce symptoms of irritab
30                             Poorly digested, fermentable carbohydrates may induce symptoms of irritab
31  considering their high content in fiber and fermentable carbohydrates, together with other target bi
32 es shared-as a common cause-a diet of excess fermentable carbohydrates.
33 vary function, and frequency of ingestion of fermentable carbohydrates.
34 ance of dental caries suggest a diet high in fermentable-carbohydrates.
35                          The requirement for fermentable carbon and the branched-chain auxotrophy occ
36 is unable to grow on medium containing a non-fermentable carbon source (YPG), indicating that the enz
37 onse to low glucose or the presence of a non-fermentable carbon source and negatively by two redundan
38 low but detectable level in cells grown on a fermentable carbon source at 25 degreesC, while dihydros
39 ed allele of RRF1, rrf1-L209P, grew on a non-fermentable carbon source at 30 but not at 36 degrees C,
40 tes rapid degradation of mRNA targets in the fermentable carbon source dextrose.
41 but incapable of respiratory growth on a non-fermentable carbon source due to mitochondrial dysfuncti
42 iability but did result in a dependence on a fermentable carbon source for growth, a temperature sens
43 ional lethality, and several preferred a non-fermentable carbon source for growth.
44                         Cells grown in a non-fermentable carbon source had beta-galactosidase levels
45 P5 was able to restore its growth on the non-fermentable carbon source lactate.
46 nt mutant (E194K) on medium containing a non-fermentable carbon source, but fails to rescue a coq7 nu
47  nitrate-dependent anaerobic growth on a non-fermentable carbon source, glycerol.
48 entation of medium containing ethanol, a non-fermentable carbon source, rescued growth in only two of
49 response to nitrogen starvation and abundant fermentable carbon source.
50 nes necessary for growth in the absence of a fermentable carbon source.
51 n ofpykApreventsMtbgrowth in the presence of fermentable carbon sources and has a cidal effect in the
52 a mutant along with its growth defect on non-fermentable carbon sources and hypersensitivity to hydro
53 tuberculosis shortened the delayed growth on fermentable carbon sources and restored growth on nonfer
54  mutant can grow on both fermentable and non-fermentable carbon sources at lower temperatures, it can
55 for their ability to respire and grow on non-fermentable carbon sources at nearly wild-type rates.
56 null mutant grew on both fermentable and non-fermentable carbon sources but more poorly on the latter
57 tant was defective in the utilization of non-fermentable carbon sources but not in oxidative phosphor
58                               Cells grown on fermentable carbon sources do not require mitochondrial
59 rowing by aerobic glycolysis, whereas on non-fermentable carbon sources metabolism shifts towards res
60 g doxycycline increases to senescence in non-fermentable carbon sources or at high temperatures, cond
61  holo-iso-1-cytochromes c grew better on non-fermentable carbon sources than the corresponding rho+ S
62 grown in the presence of fermentable and non-fermentable carbon sources, although the extent of the i
63  of wild-type (rho+) mtDNA in cells grown on fermentable carbon sources, and for efficient recombinat
64 r respiration, for their growth rates on non-fermentable carbon sources, and for their cytochrome c o
65 dent manner upon caloric restriction, on non-fermentable carbon sources, as well as under osmotic and
66                                           On fermentable carbon sources, the Deltamqo mutant exhibite
67 sely correlated with the energy yield of non-fermentable carbon sources, the requirement of ubiquinon
68  for glucose or when grown strictly with non-fermentable carbon sources.
69 ed acceleration in production of acid on non-fermentable carbon sources.
70 e switched from growth in fermentable to non-fermentable carbon sources.
71 ear the promoter, but only in the absence of fermentable carbon sources.
72 an restore growth in glucose, but not in non-fermentable carbon sources.
73 SC1 resulted in markedly lower growth in non-fermentable carbon sources.
74 e involved in the oxidation of different non-fermentable carbon sources.
75 unction as evidenced by normal growth on non-fermentable carbon sources.
76  has shifted to the aerobic oxidation of non-fermentable carbon sources.
77 of the yeast Saccharomyces cerevisiae on non-fermentable carbon sources.
78 rowth of S. cerevisiae in fermentable or non-fermentable carbon sources.
79 hydrogenase null mutant will not grow on non-fermentable carbon sources.
80 h rates and lower molar growth yields on non-fermentable carbon sources.
81 ublished genome-wide screens of multiple non-fermentable carbon sources.
82 galactose-containing medium but not on other fermentable carbon sources.
83                 Moreover, in response to non-fermentable carbon substrates, Rgt2 and Hxt1-RT (RT, Rgt
84 lly starvation for amino acids, nitrogen, or fermentable carbon.
85 lity of significantly increasing the mass of fermentable cell wall components in bioenergy crops.
86                       The switch/sucrose non-fermentable chromatin remodeling complex, also known as
87                  SWI/SNF (switch/sucrose non-fermentable) chromatin remodelers possess unique functio
88                  SWI/SNF (SWItch/Sucrose Non-Fermentable) chromatin remodeling complexes play signifi
89 tations in other SWI/SNF (SWItch/Sucrose Non-Fermentable) chromatin remodeling subunits are also obse
90 dentify PBRM1, a SWI/SNF (SWItch/Sucrose Non-Fermentable) complex subunit gene, as a significantly mu
91                 This study revealed that the fermentable components of AFFF can stimulate TCE dechlor
92 therefore an indicator for the proportion of fermentable DF in grain fractions and wheat-based foods
93 , where it obtains nutrients and metabolizes fermentable dietary carbohydrates.
94 oyed the same knockout and DSS models adding fermentable dietary fiber (0 or 2.5% inulin for 8 wks).
95 upplementation of HFD-fed male mice with the fermentable dietary fiber fructooligosaccharides (FOS) w
96    Inulin-type fructans (ITFs) are a type of fermentable dietary fiber that can confer beneficial hea
97                                              Fermentable dietary fibers promote the growth of benefic
98 y rich in (1,3;1,4)-beta-glucan, a source of fermentable dietary fibre that protects against various
99 ion favoured utilisation of less reduced and fermentable DOC while carbon-limited environments favour
100 e gut microbiota during infancy by acting as fermentable energy source.
101 n plasticity most frequently between the two fermentable environments, with mutations causing signifi
102  Bottle conditioning occurs when yeast and a fermentable extract are added to beer prior to packaging
103 Members of the Swi2/Snf2 (switch/sucrose non-fermentable) family depend on their ATPase activity to m
104 ot study, 30 adults (ages 50-75 y), received fermentable fiber (33 g/d soluble corn fiber; SCF) plus
105 huris muris to investigate the effect of the fermentable fiber inulin on host responses to infection
106 sed to HCC when diets were enriched with the fermentable fiber inulin.
107                Thus, the positive effects of fermentable fiber on gut inflammation appear to be conte
108 the synergistic effects of fish oil (FO) and fermentable fiber on the gut transcriptional profiles an
109                                        Thus, fermentable fiber protects against metabolic syndrome by
110                 Dietary supplementation with fermentable fiber suppresses adiposity and the associate
111                               Interestingly, fermentable fiber when combined with fish oil containing
112 ctulose, a nonabsorbable sugar), psyllium (a fermentable fiber), or methylcellulose (a nonfermentable
113 assigned to receive a supplement of a highly fermentable fiber, gum arabic (50 g/d), or a placebo (1
114 3 polyunsaturated fatty acids, curcumin, and fermentable fiber, have been proposed to exert chemoprot
115 iver injury and HCC, particularly when fed a fermentable fiber-enriched diet.
116  prone to developing this disease when fed a fermentable fiber-enriched diet.
117  study establishes that a single nonabsorbed fermentable food product can alter the IM in both a regi
118 wever, altered transit in mice fed a diet of fermentable fructooligosaccharide indicates that diet ca
119  aim of rapid conversion of glucose into non-fermentable gluconic acid.
120 dic linkages, beta-glucosidases produce free fermentable glucose and alleviate the inhibition of othe
121                MLG is also a major source of fermentable glucose for the human gut microbiota (HGM).
122          Following transfer from medium with fermentable glucose to medium with nonfermentable acetat
123  expression plasticity were observed between fermentable (glucose or galactose) and nonfermentable (g
124 he predominant localization in the IBM under fermentable growth conditions is prevented by inhibiting
125                          We found that under fermentable growth conditions, Oxa1 is enriched in the I
126 and RCF3 affects cellular survival under non-fermentable growth conditions, suggesting an overlapping
127                                           On fermentable growth medium, the mutant cells were prone t
128 oth the Ras-adenylate cyclase as well as the fermentable growth medium-induced pathways, and our resu
129                           We find that while fermentable (inulin), but not insoluble (cellulose), fib
130 he former consists of limiting the supply of fermentable material to the colonic bacteria.
131 M2-null cells arrest after 25 generations on fermentable media.
132 sses the growth arrest of RPM2-null cells on fermentable media.
133 l matrix results in growth impairment on non-fermentable medium caused by decreased levels of CcO.
134  of PIC2 grew poorly on copper-deficient non-fermentable medium supplemented with silver and under re
135 proach improved the extraction efficiency of fermentable monosaccharides, particularly glucose and xy
136 mponents of the mammalian switch/sucrose non-fermentable (mSWI/SNF or BAF) chromatin remodeling compl
137 atin remodelers mammalian SWItch/Sucrose Non-Fermentable (mSWI/SNF) are mutated, deleted, or amplifie
138 osome remodeler mammalian SWItch/Sucrose Non-Fermentable (mSWI/SNF) by the transcription factors RUNX
139 activity of the mammalian switch/sucrose non-fermentable (mSWI/SNF) chromatin remodeling complex.
140 erturbations to mammalian switch/sucrose non-fermentable (mSWI/SNF) chromatin remodeling complexes ha
141 emonstrate that mammalian SWItch/Sucrose Non-Fermentable (mSWI/SNF) chromatin remodeling complexes, s
142                 Mammalian switch/sucrose non-fermentable (mSWI/SNF) complexes are multi-component mac
143 fit to examine the impact of other potential fermentable nutrients and their products on IBD outcomes
144 ultiple food groups, avoid gluten, or reduce fermentable oligo-, di-, and mono-saccharides and polyol
145 ports dietary modifications, such as the low-fermentable oligo-, di-, and monosaccharides and polyols
146 e specific carbohydrate diet and diet low in fermentable oligo-, di-, and monosaccharides and polyols
147 ointestinal disorders, due to high levels of fermentable oligo-, di-, monosaccharides and polyols (FO
148                        The efficacy of a low fermentable oligo-, di-, monosaccharides and polyols (FO
149 ith gluten often contain fructans, a type of fermentable oligo-, di-, monosaccharides and polyols.
150 (IBS) has been associated with diets rich in fermentable oligo-, di-, monosaccharides, and polyols (F
151 nt of fermentable short-chain carbohydrates (fermentable oligo-, di-, monosaccharides, and polyols [F
152  poorly absorbed, short-chain carbohydrates (fermentable, oligo-, di-, monosaccharides, and polyols [
153                                A diet low in fermentable oligosaccharides, disaccharides, monosacchar
154  with IBS who choose to follow a diet low in fermentable oligosaccharides, disaccharides, monosacchar
155 There is limited evidence that a diet low in fermentable oligosaccharides, disaccharides, monosacchar
156                                A diet low in fermentable oligosaccharides, disaccharides, monosacchar
157                                        A low fermentable oligosaccharides, disaccharides, monosacchar
158                                        A low fermentable oligosaccharides, disaccharides, monosacchar
159     Assessing the potential effects of a low fermentable oligosaccharides, disaccharides, monosacchar
160 ed to treat patients with IBS, a diet low in fermentable oligosaccharides, disaccharides, monosacchar
161                                              Fermentable oligosaccharides, disaccharides, monosacchar
162          Gluten challenge should be FODMAPS (fermentable oligosaccharides, disaccharides, monosacchar
163             However, the mechanisms by which fermentable oligosaccharides, disaccharides, monosacchar
164 th the cumbersome and resource-intensive low fermentable oligosaccharides, disaccharides, monosacchar
165                                Foods high in fermentable oligosaccharides, disaccharides, monosacchar
166  to investigate associations with content of fermentable oligosaccharides, disaccharides, monosacchar
167                                A diet low in fermentable oligosaccharides, disaccharides, monosacchar
168  gut bacteria cannot ferment compared to the fermentable ones: mice colonized with B. thetaiotaomicro
169 essential for the growth of S. cerevisiae in fermentable or non-fermentable carbon sources.
170 substrates to the colonic microbiota by more fermentable particulate fiber.
171                       Here, we show that non-fermentable pentoses inhibit growth and end-product form
172 effects of gluten after dietary reduction of fermentable, poorly absorbed, short-chain carbohydrates
173 -3p suppresses expression of the sucrose non-fermentable-related serine/threonine-protein kinase SNRK
174 ofuel feed stocks that harbor easy-to-access fermentable saccharides by incorporating self-destructin
175               A diet with reduced content of fermentable short-chain carbohydrates (fermentable oligo
176                      Here, we have taken key fermentable solubilized polysaccharides of plant cell wa
177                                     When the fermentable source (glucose) is depleted, cells switch t
178     Such diets are high in dietary fiber and fermentable substrate (ie, nondigestible or undigested c
179                              The delivery of fermentable substrate(s) to subsurface environments stim
180 robial electrolysis cell (MEC) is fed with a fermentable substrate, such as glucose, a significant fr
181 gene expression in yeast switched from a non-fermentable substrate, to glucose, in the presence and a
182 ix loop is incompatible with survival on non-fermentable substrate, whereas the L200W variant is func
183 ith lactate or acetate and a small amount of fermentable substrate.
184  electrolysis cells (MECs), but studies with fermentable substrates and set potentials are lacking.
185 hlorination is stimulated by the addition of fermentable substrates to generate a reducing environmen
186                            Thus, addition of fermentable substrates to stimulate syntrophic relations
187 s that generate acetyl-CoA and NADH from non-fermentable substrates.
188 ively dynamic profile, with large amounts of fermentable substrates.
189  conversion of lignocellulosic feedstocks to fermentable sugar for biofuel production is inefficient,
190 r exogenous enzyme supplements that improved fermentable sugar generation by ~ 20 g/L.
191 t yield penalties, offsetting their gains in fermentable sugar yield.
192          In the absence of glucose (or other fermentable sugar), individual cells adopted a nonaxial
193                            The production of fermentable sugars (FS) in gluten-free (GF) brewing is h
194 id transport and fermentation of a number of fermentable sugars (including galactose and maltose, not
195  high costs of processing this material into fermentable sugars (saccharification).
196 roorganisms that can both convert biomass to fermentable sugars and ferment the resultant sugars to e
197  an increased biomass, higher levels of both fermentable sugars and hydrolyzed cellulose and altered
198 hat are retained on the dentition accumulate fermentable sugars and short-chain carboxylic acids (SCC
199  unique narrow niche) when glucose and other fermentable sugars are not available.
200 to efficiently degrade cellulosic biomass to fermentable sugars at large, commercially relevant scale
201  (OFIC) was performed for extracting maximum fermentable sugars by optimizing process parameters usin
202  saccharification of cellulosic biomass into fermentable sugars can enable production of bioproducts
203  the atmosphere, which can be converted into fermentable sugars for production into ethanol.
204    At present, plant biomass is converted to fermentable sugars for the production of biofuels using
205 ars, making it of key interest for producing fermentable sugars from biomass for biofuel production.
206  but is challenged by the costs of producing fermentable sugars from these materials.
207 t saccharification released nearly 200% more fermentable sugars from transgenic lines than controls,
208                                        Total fermentable sugars in the particles of high-starch foods
209 lysaccharides cellulose and hemicellulose to fermentable sugars is a research priority for the transi
210              Degradation of plant biomass to fermentable sugars is of critical importance for the use
211 e variations in pyridoxine, beta-glucans and fermentable sugars levels were observed both for small a
212 chnologies that can convert plant biomass to fermentable sugars must occur.
213 en in the growth media or supplementation of fermentable sugars such as glucose not only alleviated a
214  impedes breakdown of polysaccharides to the fermentable sugars that are used in biofuel production.
215 D/H)Is] representing the contribution of the fermentable sugars to (D/H)I.
216 fficient saccharification of this biomass to fermentable sugars will be a key technology in future bi
217  enabled the complete hydrolysis of MeGXn to fermentable sugars with the help of a single accessory e
218 1,4-glycosidic bonds of cellulose to produce fermentable sugars would greatly facilitate the engineer
219                          Nitrogen compounds, fermentable sugars, total phenolic content and antioxida
220  they can convert lignocellulosic biomass to fermentable sugars.
221 rolyzes plant cell wall polysaccharides into fermentable sugars.
222 ial for deconstruction of plant biomass into fermentable sugars.
223 r conversion of lignocellulosic biomass into fermentable sugars.
224 he hydrolysis of plant cell walls to release fermentable sugars.
225 rance of walls to efficient degradation into fermentable sugars.
226 , suggesting some specificity for nonreadily fermentable sugars.
227 eting cellulose hydrolyses into higher-yield fermentable sugars.
228  the efficient breakdown of the biomass into fermentable sugars.
229 c conversion of lignocellulosic biomass into fermentable sugars.
230 ulic acid, thus limiting depolymerization to fermentable sugars.
231 a-xylosidase cleaves the oligosaccharides to fermentable sugars.
232 t high-value components before hydrolysis to fermentable sugars.
233 oduction, due to their high concentration of fermentable sugars.
234 bio-conversion of lignocellulosic biomass to fermentable sugars.
235  of conversion of lignocellulosic biomass to fermentable sugars; however, many questions remain about
236  renewable cellulosic biomass to inexpensive fermentable sugars; new and more efficient fermentation
237 via complexes such as the switch-sucrose non-fermentable (SWI-SNF) chromatin remodeling complex.
238 RT, but requires residual SWItch/Sucrose Non-Fermentable (SWI/SNF) activity mediated by BRG1/SMARCA4.
239  evolutionarily conserved SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeler complex in pla
240 on factor SS18:SSX alters SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling and global me
241                       The SWItch/Sucrose non-fermentable (SWI/SNF) chromatin remodeling complex has a
242                       The SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling complex was a
243                           SWitch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling complexes are
244                 Switch defective/sucrose non-fermentable (SWI/SNF) chromatin remodeling complexes are
245                           SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling complexes dis
246 , a core component of the Switch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling complexes.
247  is incorporated into the switch/sucrose non-fermentable (SWI/SNF) chromatin-remodeling complexes and
248                       The switch/sucrose non-fermentable (SWI/SNF) complex has a crucial role in chro
249 the chromatin-remodelling switch/sucrose non-fermentable (SWI/SNF) complex) are susceptible to infect
250                 Mammalian switch/sucrose non-fermentable (SWI/SNF) complexes (also known as Brg1/Brg-
251             The mammalian SWItch/Sucrose Non-Fermentable (SWI/SNF) helicase SMARCA4 is frequently mut
252 omatin remodeling complex switch/sucrose non-fermentable (SWI/SNF) in intestinal epithelial cells.
253 have a defect in growth when switched from a fermentable to a nonfermentable carbon source that is co
254 yces cerevisiae to an abrupt transfer from a fermentable to a nonfermentable carbon source was charac
255 nner, when cells are switched from growth in fermentable to non-fermentable carbon sources.
256 pecialized metabolism provides an avenue for fermentable, unicellular organisms such as Saccharomyces
257 nsit largely depends on the amount and type (fermentable vs nonfermentable) of polysaccharides presen
258 y the transplastomic plant-derived Xyl10B to fermentable xylose and xylobiose.
259  of aldouronates and xylooligosaccharides to fermentable xylose.

 
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