ライフサイエンスコーパス: ferric chloride

1 iron source, but retained the ability to use ferric chloride.

2 ith LDA followed by oxidation with anhydrous ferric chloride.

3 y oxidation with dilute aqueous solutions of ferric chloride.

4 al over a 24-h period following injection of ferric chloride.

5 wing mesenteric arteriolar injury induced by ferric chloride.

6 the growth medium was amended with 25 microM ferric chloride.

7 PAI-1-deficient mice (PAI-1 -/-; n=11) with ferric chloride.

8 ts investigated were: (1) 10% citric acid/3% ferric chloride, (2) 10% maleic acid, (3) 2.5% nitric ac

9 ere compared to particles formed from dosing ferric chloride, a common water treatment coagulant.

10 asein fractions, even at the lowest level of ferric chloride addition (5mM).

11 sulting solutions from Fe(VI) self-decay and ferric chloride addition in borate- and phosphate-buffer

12 ed to a carotid artery injury assay in which ferric chloride administration induces de-endothelializa

13 Reaction of carbaporphyrin 4a with aqueous ferric chloride afforded the corresponding 21-chloro der

14 e exteriorized mesentery was superfused with ferric chloride and the accumulation of fluorescently la

15 d acenaphthylene ring was also oxidized with ferric chloride and this produced a ketal derivative wit

16 erated, but animal growth was stunted in the ferric chloride animals compared with the control group.

17 either intra-arterial thrombin injection or ferric chloride application followed by measurement of c

18 Sprague-Dawley rats (n = 7) underwent ferric chloride application on the femoral vein to trigg

19 travenous infusion and induced thrombosis by ferric chloride application to the carotid artery (high

20 sein-iron precipitates were formed by adding ferric chloride at >/=10mM to sodium caseinate solutions

21 The effects of ferric chloride, at two concentration levels (100 and 50

22 HBC is achieved in a one-pot reaction using ferric chloride both as a Lewis acid catalyst and as an

23 demonstrated antithrombotic effects in both ferric chloride carotid artery and laser-induced microva

24 (n=10) formed stable occlusive thrombi after ferric chloride carotid artery injury, whereas the major

25 isomeric purities by two catalytic methods, ferric chloride-catalyzed addition of acid anhydrides to

26 Up to 20mM ferric chloride could be added to sodium caseinate solut

27 to the iron chelator 2,2'-dipyridyl, but not ferric chloride, demonstrated an increase in fumarase ac

28 n few-layer graphene (FLG) intercalated with ferric chloride (FeCl(3)) have an outstandingly low shee

29 e necessity to use powerful oxidants such as ferric chloride (FeCl(3)) or DDQ/H(+) for Scholl reactio

30 Application of ferric chloride (FeCl(3)) to exposed blood vessels is wi

31 Ferric chloride (FeCl3) injury was used to induce platel

32 chanism of action of the widely used in vivo ferric chloride (FeCl3) thrombosis model remains poorly

33 et al demonstrate that thrombus formation in ferric chloride (FeCl3) thrombosis models relies on phys

34 By using a ferric chloride (FeCl3)-induced carotid artery injury th

35 Furthermore, using intravital microscopy to ferric chloride (FeCl3)-injured mesenteric arterioles an

36 sensitive and heat labile, and could utilize ferric chloride, ferric citrate, and human holotransferr

37 strains acquired iron from ferrous chloride, ferric chloride, ferrous sulfate, ferric ammonium citrat

38 H(2)Cl(2), followed by oxidation with DDQ or ferric chloride, gave excellent yields of corannulenopor

39 , and subsequent oxidation with 0.2% aqueous ferric chloride generated a series of fully conjugated n

40 e oxidation of bacteriopyropheophorbide with ferric chloride hexahydrate or its anhydrous form produc

41 roxide was less effective than freshly dosed ferric chloride in accelerating Fe(VI) decomposition.

42 nd to undergo regioselective oxidations with ferric chloride in methanol, ethanol, isopropyl alcohol,

43 ere investigated by ultrasound in a model of ferric chloride induced non-occlusive carotid artery thr

44 Further evaluation of mutant mice by the ferric chloride-induced arterial injury model suggests t

45 However, using a ferric chloride-induced arterial thrombosis model, the f

46 fXII-deficient mice are resistant to ferric chloride-induced arterial thrombosis, and this re

47 ) mice but not TFPI(LysM) mice had increased ferric chloride-induced arterial thrombosis.

48 mers and offered systemic protection against ferric chloride-induced arterial thrombosis.

49 We found that ferric chloride-induced arterial thrombus formation was

50 owed severely impaired thrombus formation on ferric chloride-induced carotid artery injury.

51 of NAC on larger vessels, we also performed ferric chloride-induced carotid artery thrombosis.

52 ced injury (0.3-3 mg/kg, iv), and guinea pig ferric chloride-induced injury (0.3-1 mg/kg, iv).

53 red the time to vessel occlusion (TTO) after ferric chloride-induced injury.

54 Furthermore, 3F8 prevented ferric chloride-induced occlusive arterial thrombogenesi

55 t that Par3(-/-) mice were protected against ferric chloride-induced thrombosis of mesenteric arterio

56 ling in mouse platelets and protects against ferric chloride-induced thrombosis of mouse mesenteric a

57 ant to both pulmonary thromboembolism and to ferric chloride-induced thrombosis of the carotid artery

58 y, arrestin2(-/-) mice are less sensitive to ferric chloride-induced thrombosis than WT mice, suggest

59 % less seizure activity than vehicle-treated ferric chloride-injected animals, suggesting that lipoic

60 o observe the formation of platelet plugs in ferric chloride-injured arterioles of live mice.

61 Using gene-targeted mice, we show that in ferric chloride-injured veins platelet adhesion to suben

62 elets form in occluding murine thrombi after ferric chloride injury and are attenuated with megakaryo

63 ht) prevented carotid artery occlusion after ferric chloride injury in a plasminogen-dependent proces

64 ombus-mediated flow reduction in response to ferric chloride injury of the carotid arteries was signi

65 rpin prevents thrombus formation provoked by ferric chloride injury of the carotid artery and increas

66 Ferric chloride injury of the midportion of the common c

67 n carotid arteries of C57Bl6 mice in vivo by ferric chloride injury were then assessed with ultrasoun

68 At 21 days after ferric chloride injury, neointima formation in P2Y(12)(-

69 ls of arterial injury, namely ballooning and ferric chloride injury.

70 ombus formation in an in vivo carotid artery ferric chloride-injury model is significantly impaired.

71 itrile-functionalized ionic precursors and a ferric chloride mediator.

72 A ferric chloride model of thrombosis was used to investig

73 in VI (GPVI) and integrin alpha2beta1 in our ferric chloride murine thrombosis model.

74 y bioavailable to Trichodesmium, relative to ferric chloride or citrate-associated iron.

75 Iron, supplied as ferrous sulfate, as ferric chloride, or as holo-transferrin, was able to neg

76 dase-mediated reduction of iron in ferritin, ferric chloride, or ferric ADP.

77 The addition of ferric chloride restore HER formation.

78 degrees C, followed by addition of anhydrous ferric chloride, resulted in an efficient tandem halogen

79 zed materials synthesized in the presence of ferric chloride showed higher activity and stability in

80 The presence of ferric chloride significantly (P<0.05) affected the oxid

81 method involves an extraction with an acidic ferric chloride solution, to quantitatively convert EDTA

82 give, following oxidation with 0.1% aqueous ferric chloride solutions, a series of tropiporphyrins 9

83 ipyrrane, followed by oxidation with aqueous ferric chloride solutions, afforded moderate yields of a

84 f freshly precipitated ferric hydroxide from ferric chloride solutions.

85 he reaction mixture was stirred with aqueous ferric chloride solutions.

86 Exogenous ferric chloride suppressed these deficiencies in the apb

87 stable brown color in the presence of acidic ferric chloride that can be quantitated spectrophotometr

88 Compound 30d showed efficacy in the rat ferric chloride thrombosis model when administered intra

89 When Cbl-b(-/-) mice were tested in the ferric chloride thrombosis model, occlusion time was inc

90 rtery occlusion times on the rose bengal and ferric chloride thrombosis models.

91 onbiomimetic polyene cyclization mediated by ferric chloride to generate the generic celastroid penta

92 As adding >5mM ferric chloride to sodium caseinate solutions results in

93 By addition of ferric chloride to the reaction mixture, a selective aro

94 presence of HCl, followed by oxidation with ferric chloride, to give a modest yield of an azuliporph

95 presence of TFA, followed by oxidation with ferric chloride, to give a tropone-fused carbaporphyrin

96 ted group (P = 0.057), and 62.5 mg/d for the ferric chloride-treated group (P < 0.002).

97 Platelet adhesion to ferric chloride-treated mesenteric arterioles in IL4Ralp

98 rmed in vivo by characterizing blood flow in ferric chloride-treated mouse carotid arteries.

99 Here we demonstrate that exposure to ferric chloride triggers rugose biofilm formation by the

100 In the ferric-chloride vasculature injury model, Abcc4 KO mice

101 Ferric chloride was used to induce carotid artery injury

102 In the large-vessel model, ferric chloride was used to injure the carotid artery, a

103 hesia, unilateral intracortical infusions of ferric chloride were performed stereotaxically.

104 oncentration was overcome by the addition of ferric chloride which also contributed to the color stab