ライフサイエンスコーパス: fertility

1 on is sufficient to induce gonadogenesis and fertility.

2 to prevent complications and preserve future fertility.

3 f three PEGs reduced starch content and seed fertility.

4 ial for mitosis, meiosis, adult survival, or fertility.

5 lted in delayed estrous cycles and decreased fertility.

6 s avoided excessive branching because of low fertility.

7 ly in male life may alter sperm function and fertility.

8 arated tropical forests which differ in soil fertility.

9 quired for Cu delivery to anthers and pollen fertility.

10 atment with mTOR inhibitors preserved normal fertility.

11 igorous measure of the ovarian reserve), and fertility.

12 mes, even while imposing tremendous costs to fertility.

13 dditional SNPs significantly associated with fertility.

14 penoids pristimerin and lupeol, affect sperm fertility.

15 onsumption of lactose or low-fat dairy harms fertility.

16 elusive hypothalamic pulse generator driving fertility.

17 follicle development, resulting in impaired fertility.

18 or human development policies that may lower fertility.

19 ian reserve were not associated with reduced fertility.

20 is essential for both oocyte maturation and fertility.

21 RNAs, ensuring transposon silencing and male fertility.

22 hyh mutation) in folliculogenesis and female fertility.

23 n important role in spermatogenesis and male fertility.

24 dentical age-specific selective pressures to fertility.

25 r with key roles in the regulation of female fertility.

26 enomic regions strongly associated with bull fertility.

27 leading to increased appetite and suppressed fertility.

28 ome and autosomes that is essential for male fertility.

29 flect long-term Ca sources that sustain soil fertility.

30 , females time reproduction to coincide with fertility.

31 of meiotic transcription and affects female fertility.

32 al reproductive tract anomalies could impair fertility.

33 cal changes to achieve sexual maturation and fertility.

34 nts and biological processes underlying sire fertility.

35 can be considered an option for safeguarding fertility.

36 fferentiation, events that are necessary for fertility.

37 d disturbed pollen tube guidance and reduced fertility.

38 a or increased adiposity, and exhibit normal fertility.

39 any species, including sex determination and fertility.

40 fulness of WHR as an indicator of health and fertility.

41 similar to petroleum coke) and enhanced soil fertility.

42 crease soil carbon content and maintain soil fertility.

43 icular vacuolation associated with decreased fertility.

44 nd proliferation of GCs and is essential for fertility.

45 behaviour, impacting normal development and fertility.

46 e evidence that it is impaired in human male fertility.

47 ulogenesis is essential for mammalian female fertility.

48 ality of clinician-patient discussions about fertility.

49 of serum with haemoglobin fully restored egg fertility.

50 ption factor FOXJ3 in the regulation of bull fertility.

51 ential for sperm hyperactivated motility and fertility.

52 essential for transposon silencing and male fertility.

53 s, the acrosome, which is essential for male fertility.

54 d them over a sustained period and monitored fertility.

55 izing hormone (LH) is critical for mammalian fertility.

56 unt for increased length and improvements in fertility.

57 autosomal retrogenes and their role in male fertility.

58 ment, especially in women who wish to retain fertility.

59 to its influence on soil functions and soil fertility.

60 iopsies and orchidectomies, and can maintain fertility.

61 sufficient for puberty onset and subsequent fertility.

62 ng in species composition, rainfall and soil fertility.

63 , but show developmental defects and reduced fertility.

64 as some compounds typically linked to worker fertility.

65 sed germ cell apoptosis and compromised male fertility.

66 Moreover, these connections are required for fertility [1, 2].

67 hers traded offspring survival for increased fertility, achieving greater reproductive success despit

68 l, where complex interrelations between soil fertility, agricultural productivity, and human settleme

69 ysiological variation confers differences in fertility among males remains to be established.

70 DT predicts unilinear change toward very low fertility and a diversity of union and family types.

71 obial and invertebrate-based soil food webs, fertility and above-ground plant productivity at 289 sit

72 at morc-1 is essential for transgenerational fertility and acts as an effector of endo-siRNAs.

73 denogenesis) and function, are essential for fertility and are controlled by estrogens and other regu

74 urons produce the central output controlling fertility and are regulated by steroid feedback.

75 dairy cows are common after calving, reduce fertility and cause substantial economic losses.

76 omosome sequence, critical for studying male fertility and dispersal, is enriched in repeats and pali

77 Impaired fertility and fecundity in fish following chronic exposu

78 ositive associations among soil biodiversity-fertility and fertility-plant productivity are limited t

79 of services such as climate regulation, soil fertility and food and fibre production by terrestrial e

80 sidered, to monitor, recover or sustain soil fertility and function, and to enhance the ecosystem ser

81 ng 94 SNPs was significantly associated with fertility and functional analysis confirmed that sperm f

82 ce complex (Arc-ME) controls energy balance, fertility and growth through molecularly distinct cell t

83 Obesity reduces fertility and increases time taken to conceive, and obes

84 after biomarkers of endometrial receptivity, fertility and infertility.

85 Abortion can help women to control their fertility and is an important component of health care f

86 nd follicular atresia is critical for female fertility and is strictly regulated by a complex network

87 To investigate PRDM9 binding and its role in fertility and meiotic recombination, we humanized the DN

88 Projections, which uses similar methods for fertility and mortality but deterministic migration proj

89 e's population has one of the world's lowest fertility and mortality rates, and the elderly populatio

90 alg-5 is required for optimal fertility and mutations in alg-5 lead to a precocious tr

91 Nonetheless, both age-controlled fertility and overall reproductive success were positive

92 Soil biodiversity, fertility and plant productivity are strongly positively

93 ack relationships between soil biodiversity, fertility and plant productivity are universal.

94 the relationships between soil biodiversity, fertility and plant productivity weaken considerably, pr

95 s have been conducted to evaluate effects on fertility and pregnancy outcomes.

96 cling exerts significant influence upon soil fertility and productivity - processes largely controlle

97 in regulation of adult uterine function and fertility and provide original evidence that uterine gla

98 onesty in this system is maintained by queen fertility and queen signal production being under shared

99 idence supports early orchidopexy to improve fertility and reduce malignancy in boys with undescended

100 Fat metabolism has been linked to fertility and reproductive adaptation in animals and hum

101 per oocyte development is crucial for female fertility and requires timely and accurate control of ge

102 may cumulatively and permanently reduce the fertility and shorten the lifespan of adult females.

103 nsequences of plant-pathogen interactions on fertility and the evolution of intercellular communicati

104 sed on the shared endocrine control of queen fertility and the production of specific signals.

105 t species X chromosomes would suffer reduced fertility and viability compared with females with two p

106 ic backgrounds to test predictions about the fertility and viability of each cross type.

107 sertions, which usually did not affect plant fertility and were equally well transmitted through male

108 ly break the links between soil biodiversity-fertility and/or fertility-plant productivity, which can

109 ctive hormone levels, estrous cyclicity, and fertility) and metabolic parameters (body weight changes

110 riance between fitness (as measured by total fertility) and other traits changed over time?

111 and 2010s, environmental (i.e. soil salinity/fertility) and societal (i.e. policy/techniques) factors

112 linked to neurological disease, cancer, and fertility, and a recent report correlated a potential lo

113 and delivery to anthers, thereby influencing fertility, and highlight the relationship between Cu hom

114 and Nanos (Nos), which control development, fertility, and neuronal functions.

115 Obesity is associated with reduced fertility, and pregnancies complicated by maternal obesi

116 Reproductive issues including contraception, fertility, and pregnancy are important components of the

117 rtance of surface soil biodiversity for soil fertility, and suggest that any loss of surface soil cou

118 pport the involvement of MAT genes in sexual fertility, and the influence of mating type on the sever

119 ned the association between dairy intake and fertility, and they have had conflicting findings.

120 ate immune system, matrix deposition, female fertility, and vascular biology.

121 a Lotka style equation in which survival and fertility are averaged over a phenotypic distribution th

122 number of germline genes essential for male fertility are coordinately activated.

123 SIGNIFICANCE STATEMENT Sexual maturation and fertility are dispensable at the individual level but cr

124 The fundamental controls on magma metal fertility are tectonic setting, the nature of source roc

125 ariation in soil water availability and soil fertility as well as by utilizing annual climate project

126 floral organs, silique development and plant fertility, as well as directly regulates AS2 expression.

127 ghly prevalent human disease and the reduced fertility associated with endometriosis.

128 rgence of the Homo lineage, due to a partial fertility barrier.

129 his gene cluster has important roles in male fertility based on phenotypic defects of Rhox-mutant mic

130 ox9 and Sox8 are required for postnatal male fertility, but their role in the adult testis has not be

131 eas F1 males recovered normal GC numbers and fertility by adulthood.

132 at a deletion of laminin alpha2 impedes male fertility by disrupting ectoplasmic specialization (ES;

133 emonstrate that KAT8 is essential for female fertility by regulating antioxidant gene expression and

134 xpressed at the BTB and is required for male fertility by supporting the functional integrity of the

135 ciated with body mass index (BMI) and female fertility; c) is partially heritable.

136 een 2005 and 2014, a total of 329 men from a fertility clinic in Boston, Massachusetts, provided 768

137 ent among couples recruited from an academic fertility clinic.

138 Among C2 females, fertility clinical nurse specialist consultation was ass

139 provides resources, clinician education, and fertility clinical nurse specialist consultation.

140 ate of assisted reproductive technologies in fertility clinics.

141 Food security and soil fertility could be significantly improved by greater gra

142 idated genetic variants associated with bull fertility could prove useful for improving reproductive

143 and participation in society, for voluntary fertility declines and ending world population growth, f

144 Therefore, as fertility decreased over time, female lifespan increased

145 (wh, no progeny), characterized by a severe fertility defect and affecting expression of a protein (

146 Uterine Fst-cKO mice demonstrate severe fertility defects and deliver only 2% of the number of p

147 ation and potential new angles to understand fertility defects in humans carrying Aire mutations.

148 We find that Y hybrids show no obvious fertility defects, and no substantial reduction in male

149 This implies that women's fertility depends on the longevity of cohesin proteins t

150 oxidase II, which specifically impairs male fertility due to defects in sperm development and functi

151 le inhibitors preserves ovarian function and fertility during chemotherapy.

152 ta treatments reduced reproductive capacity (fertility, fecundity) by 7.3-57.4% in adult medaka breed

153 ation in female reproductive traits, such as fertility, fecundity, and fecundability, is heritable in

154 reatening cancer treatment to preserve their fertility for a life after cancer.

155 timullerian hormone levels to assess natural fertility for women with these characteristics.

156 We found that the site with the lowest fertility (French Guiana) exhibited greater rates of Rda

157 ity of IRE1 was required for protecting male fertility from ET.

158 uito population, and gene drive disrupting a fertility gene and thereby achieving population suppress

159 ecies in 10 forest plots spanning a regional fertility gradient in Panama.

160 Cold, low fertility growing conditions are often associated with r

161 s have investigated cow fertility while bull fertility has received much less consideration.

162 For other phenotypes (egg-hatching and male fertility), however, one gene shows full rescue, but thr

163 sociated with severe pelvic pain and reduced fertility; however, its molecular mechanisms remain larg

164 ns in the genome in 24 bulls of high and low fertility identified 484 additional SNPs significantly a

165 both husbands and wives have slightly lower fertility; (ii) offspring suffer from inbreeding depress

166 TFT, by showing that introduction of a male fertility-impairing mtDNA haplotype into replicated popu

167 of mutant alleles of AURKC that affect human fertility in a mammalian meiotic system.

168 brane protein, HAP2/GCS1, has been linked to fertility in a wide range of taxa and is hypothesized to

169 nce may therefore suppress puberty onset and fertility in an individual.

170 Kidney transplantation (KT) may restore fertility in chronic kidney disease (CKD).

171 vel the genomic architecture underlying sire fertility in Holstein dairy cattle.

172 ations in the same gene also display reduced fertility in male animals.

173 phic adenoma gene 1 (PLAG1) leads to reduced fertility in male mice, but the mechanism by which PLAG1

174 ute directly to ovarian function and natural fertility in mammals.

175 erfamily protein) plays a major role in male fertility in mice.

176 ed for uterine development as well as female fertility in mice.

177 reader that is essential for male and female fertility in mice.

178 nteract to control flowering time and floret fertility in response to ambient temperature in barley.

179 n spermatocytes restores spermatogenesis and fertility in Rpl10l-deficient mice.

180 enerational epigenetic information to ensure fertility in subsequent generations.

181 segment of D. mauritiana drastically reduced fertility in the presence of Ysim, whereas the fertility

182 epal opening, elongated pistils, and reduced fertility in the primary inflorescence stem.

183 ies that may be used to extend the window of fertility in these patients.

184 y of AgRP neurons is sufficient to attenuate fertility in vivo, we artificially activated them over a

185 Drosophila melanogaster, for instance, male fertility increases with age, and older males are known

186 spermatogenesis, as well as its role in male fertility indicates that avian and mammalian HSPA2 may e

187 but it has not been investigated in natural fertility, indigenous, more energy constrained populatio

188 the metabolic consequences of poverty, high fertility, infectious diseases, and rapid changes in nut

189 ed ovulation rate, and a dramatic decline in fertility is observed in alpha-SNAP-mutant females.

190 rtility in the presence of Ysim, whereas the fertility is partially rescued by the Y chromosome of D.

191 olecular mechanism by which BT-IgSF supports fertility is unclear.

192 to be important for P. berghei female gamete fertility, is shown to serve a different function in Pla

193 lthough GPR64 has an important role for male fertility, its physiological roles in the female reprodu

194 experiencing naturally varying mortality and fertility levels.

195 e, resulting in rapid genetic improvement in fertility, lifespan, and health in a breed where these t

196 s are connected to) has been associated with fertility, longevity, disease and information transmissi

197 mortality (parental investment) or increased fertility (mating effort).

198 This suggests that reduced male fertility may be a general feature of mixtures of human

199 ycles, with visits scheduled with the use of fertility monitors.

200 part of reproductive strategies that enhance fertility more than offspring well-being.

201 We test this hypothesis by investigating fertility, mortality, health, and overall reproductive s

202 The restoration of fertility occurs in spite of an autosomal and X-linked g

203 nt level, longitudinal data on viability and fertility of female worms from the single most comprehen

204 Remarkably, fertility of heterochromatin mutants could be partially

205 Relative fertility of lateral spikelets within each cluster leads

206 Furthermore, Hormad2 deletion rescued the fertility of oocytes containing a synapsis-proficient, D

207 athogens in acute epididymitis; however, the fertility of patients can be permanently impaired.

208 ic use in pregnancy could potentially affect fertility of resulting daughters and grand-daughters.

209 e production of germ cells to the growth and fertility of the adult organisms - using diverse approac

210 iformity.The VRS genes of barley control the fertility of the lateral spikelets on the barley inflore

211 n prices when long-term improvements in soil fertility offset biochar production costs.

212 uence) and RS (mating success, wife quality, fertility, offspring mortality, and number of surviving

213 s is generally used as the proxy to estimate fertility or gain insight into the underlying reasons fo

214 this outcome was not uniformly reported and fertility or rate of pregnancy was not the primary outco

215 eater consumption of high-fat dairy improves fertility or that a greater consumption of lactose or lo

216 ntly affect embryonic and brain development, fertility, or locomotor performance of mutant flies or t

217 ogether, has no detectable effect on growth, fertility, or sensitivity to DNA damage.

218 h the risk of some cancers, hence decreasing fertility patterns contribute to secular increases in th

219 in small-scale societies: here, age-specific fertility patterns.

220 Furthermore, their fertility phenotype was similar to that of the AS2 loss-

221 pplication of WES in AI bulls with divergent fertility phenotypes has identified a novel role for the

222 ations among soil biodiversity-fertility and fertility-plant productivity are limited to the upper so

223 s between soil biodiversity-fertility and/or fertility-plant productivity, which can negatively impac

224 imately affect programmed cell death, female fertility, platelet function, vasculature inflammation,

225 pacted by weathered oil while improving soil fertility, potentially enhancing revegetation.

226 ussing treatment-related fertility risks and fertility preservation (FP) options with patients and in

227 Notable needs also include fertility preservation and navigation through the multip

228 Nevertheless, consideration of fertility preservation before cancer treatment remains i

229 readily accessible pharmacologic approach to fertility preservation during conventional chemotherapy.

230 The field of oncofertility, or fertility preservation for patients facing a cancer diag

231 communication, and developmental factors to fertility preservation outcomes.

232 reezing and vitrification of whole ovary for fertility preservation purposes, in an ewe model.

233 al support, nutritional, rehabilitative, and fertility preservation services; programme value, includ

234 immediate revascularization, ensuring better fertility preservation, but the best cryopreservation me

235 A cancer and fertility program was established at a large cancer cent

236 ation received demonstrate the potential for fertility programs in cancer care settings to improve th

237 increased from 68.1% to 75.6%, and the total fertility rate decreased from 3.0 children per woman to

238 We found strong climate signals in the fertility rates of three species.

239 banisation, women's education, water supply, fertility rates, and child nutrition from the annual nat

240 demographic transition to low mortality and fertility rates.

241 l pH is not an independent regulator of soil fertility-rather, it is ultimately controlled by environ

242 A similar fertility reduction was elicited in the wild-type (WT) C

243 dual strategy of targeted sequencing (TS) of fertility-related beta-defensin genes and whole exome se

244 As a man's fertility relies on the quantity and quality of his sper

245 Bovine fertility remains a critical issue underpinning the sust

246 Faithful meiotic chromosome segregation and fertility require meiotic recombination between homologo

247 richment of H3K36me3, suggesting that robust fertility requires repression of MET-1 activity at nucle

248 For male fertility restoration, transgenic rice plants carrying B

249 ular grafting after cancer healing permitted fertility restoration.

250 suggest that the customized male-sterility & fertility-restoration system can be exploited for qualit

251 e have developed a unique male-sterility and fertility-restoration system in rice by combining Brassi

252 ses that promote internal harmony and higher fertility, resulting in "mutually beneficial exchanges"

253 NA suppresses ovarian development and female fertility, resulting in females producing few viable off

254 gnificantly greater in C2 for information on fertility risks (chi(2) = 62.1, P < .001), FP options (c

255 t clinicians in discussing treatment-related fertility risks and fertility preservation (FP) options

256 For males, satisfaction with information on fertility risks was high in both cohorts but significant

257 treatment or who do not wish to retain their fertility should be considered for surgical management.

258 n to cuticular hydrocarbons which can act as fertility signals emitted by the principal reproductive(

259 lp to maintain the honesty of such queen and fertility signals, however, remain poorly studied.

260 ic induction of flowering occurring at lower fertility sites.

261 n a native mature eucalypt woodland with low fertility soils in southeast Australia.

262 ociated with tree species persistence on low fertility soils.

263 odel revealed adolescent consultation with a fertility specialist (OR, 29.96; 95% CI, 2.48 to 361.41;

264 with a model in which PGF2alpha communicates fertility status via Ptgfr to circuits in the brain that

265 genetic elements and result in reduced male fertility, sterility or inviability.

266 s emerging health applications, such as male fertility testing and wearable diagnostics.

267 Reduced sexual fertility that accompanied the shift to asexual reproduc

268 criteria including 41,910 women who received fertility therapy and 1,400,202 women who did not.

269 t, or randomized designs with 3) exposure to fertility therapy and 4) cardiovascular outcomes clearly

270 The longer term cardiovascular effects of fertility therapy are unknown.

271 ts for studies addressing the question "does fertility therapy increase the risk of longer term cardi

272 of this study was to summarize data linking fertility therapy with subsequent cardiovascular outcome

273 betes mellitus, comparing women who received fertility therapy with those who did not.

274 hs that allow adults and children undergoing fertility-threatening cancer treatment to preserve their

275 l.SIGNIFICANCE STATEMENT The brain regulates fertility through gonadotropin-releasing hormone (GnRH)

276 PL restores normal growth, mitosis, and some fertility to ctf7 plants.

277 in the Drosophila male germline can restore fertility to flies deficient in boule, the Drosophila or

278 We show that Ms1 completely restores fertility to ms1d, and encodes a glycosylphosphatidylino

279 Loss of maternal LIN-15B restores fertility to nos-1nos-2 mutants.

280 sted for association across a broad range of fertility traits in women.

281 Among women undergoing fertility treatment in the Netherlands between 1980 and

282 ial receptivity and offer a novel target for fertility treatment.

283 95 (IVF group) and 5950 women starting other fertility treatments between 1980 and 1995 (non-IVF grou

284 n situ breast cancers in women who underwent fertility treatments was obtained through linkage with t

285 mation on ovarian stimulation for IVF, other fertility treatments, and potential confounders was coll

286 at less than 12 weeks' gestation, not using fertility treatments, fluent in English or Spanish, and

287 oil-Order level, expressing broad changes in fertility, underpins major shifts in foliar phosphorus (

288 , and in efforts to augment human and animal fertility using stem cell approaches.

289 can provide opportunities for improving bull fertility via marker-assisted selection.

290 Fertility was not affected when females were exposed to

291 10% of women and is associated with reduced fertility, we find raised levels of Reactive Oxygen Spec

292 Ovarian function and fertility were reported after 2, 3, 4, and 5 to 7 years

293 Follicle rupture and fertility were significantly impaired in the absence of

294 ristics of semen biology that correlate with fertility when it is mishandled.

295 tion was intentionally performed to preserve fertility, which may be decreased after conventional UFE

296 How Cu affects fertility, which reproductive structures require Cu, and

297 Most studies have investigated cow fertility while bull fertility has received much less co

298 in the mitochondrial genome that impair male fertility while having no effect on females.

299 a result of a reduction in biodiversity and fertility with depth.

300 its (IFU) induced a significant reduction in fertility, with a mean number of embryos/mouse of 4.4 +/