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1 CTH secretion (alone or with CRH) from human fetal cells.
2 a male fetus or evidence of persistent male fetal cells.
3 sheathed more host axons per donor cell than fetal cells.
4 ions of neurotrophic factors and implants of fetal cells.
5 se attributable to transplacentally acquired fetal cells.
6 uclear transfer-derived sheep generated from fetal cells.
7 ty requires cytogenetic analysis of amniotic fetal cells.
8 n cell lines established from adults than in fetal cells.
9 ation process examined in this review, these fetal cells acquire properties of leukocytes and endothe
12 rved from yeast to human cells, expressed in fetal cells and most adult tissues, and acting synergist
13 IGFBP2 gene, which is normally expressed in fetal cells and turned off in adult cells, becomes react
14 r is not continuously exposed to circulating fetal cells and, in fact, has the capacity to eliminate
17 tion known to induce iNOS maximally in human fetal cells, and the expression of iNOS was determined b
22 ed in neonatal RBC, will direct parasites to fetal cells, as well as F cells, and less to the approxi
24 native strategy, which suggested that intact fetal cells can be found in maternal plasma by use of si
27 s in humans have shown that during pregnancy fetal cells can enter the maternal circulation and persi
28 lure, our objective was to determine whether fetal cells can migrate to the maternal heart and differ
34 significantly higher than the number of male fetal-cell DNA equivalents detected in 46,XY fetuses (P
39 ing of the interactions between maternal and fetal cells during early placentation and highlights nov
43 postpartum, the result of an encounter with fetal cells expressing the appropriate MHC/peptide compl
44 er the mechanism by which two populations of fetal cells (fetal cortex derived Gli1-expressing cells
48 is method of prenatal diagnosis by enriching fetal cells from maternal blood by magnetic cell sorting
49 nosis of single gene disorders by recovering fetal cells from maternal circulation appears to be a fe
50 iptome analyses of primary cultures of human fetal cells from pregnancies affected with trisomy 21 (t
53 recent report that showed calves cloned from fetal cells have longer telomeres than their age-matched
55 Sc remains to be elucidated, the presence of fetal cells in internal organs suggests that they could
56 dies have confirmed the residual presence of fetal cells in maternal blood and tissues following preg
58 ckgrounds (e.g., circulating tumor cells and fetal cells in maternal blood), detection of cells/parti
59 s possible that persistent microchimerism of fetal cells in maternal circulation may, for some mother
62 l microchimerism (FCM) is the persistence of fetal cells in the maternal circulation and organs follo
64 ion-independent manner; approximately 40% of fetal cells in the maternal heart express Caudal-related
65 fficking results in long-term persistence of fetal cells in the mother and maternal cells in her offs
66 Proposed hypotheses about the function of fetal cells in the pathogenesis of maternal cancer inclu
67 pes in ways more closely resembling those of fetal cells, including increased LCR-gamma-globin contac
69 maternal cell trafficking, or the passage of fetal cells into the maternal circulation, is now a well
71 lex (MHC) expressed by the trophoblasts, the fetal cells invading the maternal decidua during pregnan
73 shows that compared with well-differentiated fetal cells, less differentiated embryonal and undiffere
74 rodents with TLE, but the scarcity of human fetal cells limits applicability in patient populations.
81 interactions with CD1d expressed on invading fetal cells may play an immunoregulatory role at the mat
84 gate whether there is an association between fetal cell microchimerism and thyroid disease in women.
92 ononuclear cytotrophoblasts, the specialized fetal cells of the placenta that invade the uterus, play
95 ound in the maternal circulation and if male fetal cells persist in maternal tissues postdelivery.
97 eatures include extended in vitro culture of fetal cells preceding nuclear transfer, as well as in vi
98 nstrated that after inflammatory stimulation fetal cells secreted more IL-8 (LPS, 8-fold; IL-1beta, 2
102 have been directed towards the enrichment of fetal cells, such as erythroblasts, from maternal blood
103 vasion of the uterine lining by trophoblast, fetal cells that are the major cell type of the placenta
106 ined by mAb FDC6, and expressed in cancer or fetal cells/tissues, but not in normal adult cells/tissu
107 with a reduced capacity of the TCDD-exposed fetal cells to compete with control cells in a mixed com
109 orts the very long-term clinical outcomes of fetal cell transplantation in 2 patients with Parkinson
111 ntact between maternal cells and specialized fetal cells (trophoblasts) in the human placenta: firstl
112 omical interfaces between maternal cells and fetal cells (trophoblasts): (i) the villous region where
115 ellular localization of transcripts in human fetal cells, we show that human TSIX antisense transcrip
116 L against fetal Ags, we found that migrating fetal cells were cleared by the maternal immune system.
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