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1 ation parameters and maintain patency of the fetal circulation.
2 ity in the way blood is streamed through the fetal circulation.
3 to cells located closely to the maternal and fetal circulation.
4 roxychromans (alpha- and gamma-CEHCs) in the fetal circulation.
5 with levels of interleukin-6 and S100beta in fetal circulation.
6 missing enzyme across the placenta into the fetal circulation.
7 G and mediates transcytosis from maternal to fetal circulation.
8 ring waste products and xenobiotics from the fetal circulation.
9 ing boluses of specific amino acids into the fetal circulation.
10 minants of the amino acid composition in the fetal circulation.
11 Much weaker relations were seen in the fetal circulation.
12 placental uptake of organic anions from the fetal circulation.
13 and/or persistence of maternal cells in the fetal circulation.
14 derived trophoblasts separates maternal from fetal circulation.
15 AA and DHA from the mother and enriches the fetal circulation.
16 ge was observed between the placenta and the fetal circulation.
17 es (anti-SSA/Ro and/or anti-SSB/La) into the fetal circulation.
18 e nitric oxide synthase (iNOS) regulates the fetal circulation.
19 to drive accumulation of substrates from the fetal circulation.
20 chnique for studying the distribution of the fetal circulation.
21 1 and NGM-SZ21 were transported equally into fetal circulation (8.9% vs 8.7%, respectively, P = .58).
22 es aids in understanding the function of the fetal circulation and may be helpful in detecting the hu
23 promised the integration of the maternal and fetal circulation and presumably the transfer of methylT
24 which form the barrier between maternal and fetal circulations and thus govern the cross talk betwee
25 composition of amino acids delivered to the fetal circulation, and impaired placental amino acid sup
26 opment by crossing the placenta and entering fetal circulation, and indirectly through AT(1)-AA-induc
27 at AT(1)-AAs cross the mouse placenta, enter fetal circulation, and lead to small fetuses with organ
28 (Ang II) concentrations in the maternal and fetal circulations are associated with dramatic increase
29 ed cytokines in the amniotic fluid or in the fetal circulation be viewed as a humoral expression and
30 ne, was transferred from the maternal to the fetal circulation by non-exchange mechanisms also (P<0.0
32 Previous techniques for the study of the fetal circulation did not permit assessment of phasic ev
33 erangement of CXC chemokines in maternal and fetal circulation distinguishes VUE from acute chorioamn
35 Very little drug passively diffuses to the fetal circulation during the first trimester, when organ
37 ng maternal IgG across the human placenta to fetal circulation has not been identified, although the
38 s such as meconium aspiration and persistent fetal circulation have fostered clinical trials demonstr
39 We measured flow in the major vessels of the fetal circulation in 40 late-gestation normal human fetu
40 f contrast material between the maternal and fetal circulation in gravid mice through the use of dyna
42 the present review was to compare changes in fetal circulation, in terms of both velocimetry and actu
44 when maternal antibodies gain access to the fetal circulation, just prior to the clinical detection
45 al microchimerism, and maternal cells in the fetal circulation, known as maternal microchimerism.
46 e data suggest that intact beta(2)GPI in the fetal circulation may be a novel cardioprotective factor
47 s of the known vasoconstrictor Ang II in the fetal circulation may indeed play a role in the marked i
48 processes, the high levels of Ang II in the fetal circulation may serve to modulate overall fetoplac
49 ed transport of cholesterol from maternal to fetal circulation might attenuate congenital malformatio
50 ncoding the human factor IX protein into the fetal circulation of immunocompetent hemophiliac and nor
51 n increased metabolite concentrations in the fetal circulation, suggesting increased maternal or feta
52 l boluses of unlabelled amino acids into the fetal circulation to provide substrates for exchange (tr
53 easured in the absence of amino acids in the fetal circulation (transfer by non-exchange mechanisms)
54 late and UMFA concentrations in maternal and fetal circulation warrant additional investigation becau
55 lls in the transport of IgG from maternal to fetal circulation, we studied Fc gamma receptor (Fc gamm
56 exchangers, all amino acids appearing in the fetal circulation were substrates of TAT1, LAT3 or LAT4.
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