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1 l neurologic abnormalities, and intrauterine fetal death).
2 ancy is associated with an increased risk of fetal death.
3 had no significant effects on prematurity or fetal death.
4 low birth weight (<2500 g), prematurity, and fetal death.
5 ntal infection although it did not result in fetal death.
6 different adjuvants fails to protect against fetal death.
7 ed risk of respiratory distress syndrome and fetal death.
8 y or indirectly to abnormal placentation and fetal death.
9 reterm birth but not in inflammation-induced fetal death.
10 pesticides showed no strong association with fetal death.
11 fy the relation between maternal smoking and fetal death.
12 , and, ultimately, abnormal placentation and fetal death.
13 ns during pregnancy can increase the risk of fetal death.
14  in all analyses but was most pronounced for fetal death.
15 rombophilic women were at increased risk for fetal death.
16 ncy was associated with an increased risk of fetal death.
17 and human complement induced placentitis and fetal death.
18 entrations of air pollution to each birth or fetal death.
19 omatic dengue infection during pregnancy and fetal death.
20  causing defective placental development and fetal death.
21 utrophil activation, trophoblast injury, and fetal death.
22 ns, 1.3% in ectopic pregnancies, and 0.5% in fetal deaths.
23 ancies, including 9,402 livebirths and 3,063 fetal deaths.
24  cells, and led to placental hemorrhages and fetal deaths.
25 s and stillbirth, hopefully leading to fewer fetal deaths.
26 s; 18 were spontaneous abortions and 14 were fetal deaths.
27                    There were 3 intrauterine fetal deaths (1 woman had used LMWH); 9 cases of preecla
28 2 terminations of pregnancy and intrauterine fetal deaths, 189 fetopsies were available: 16 (8.5%) di
29 gs are that 1) smoking increased the risk of fetal death; 2) regular supplement use either before or
30 aths (mean [SD] estimated gestational age at fetal death, 26.3 [8.7] weeks) that were collected from
31  patients, were discovered in 3 intrauterine fetal deaths (3.3% [95% CI, 0.68%-9.3%]).
32      There were no maternal deaths and three fetal deaths (4%).
33 rriage (28.6% versus 9.2%; P<0.001) and late fetal death (7.1% versus 0.7%; P=0.016).
34 he following singleton birth categories: 450 fetal deaths; 782 very low birth weight (VLBW, < 1,500 g
35 s spontaneous abortions, neonatal mortality, fetal deaths, admission to the neonatal intensive care u
36                                There were no fetal deaths after 10 weeks' gestation and no evidence o
37 men that were raised by anecdotal reports of fetal deaths after vaccination.
38 ence of maternal smoking; 3) odds ratios for fetal death among smoking women who regularly used suppl
39                                 The risks of fetal death and congenital malformation were not related
40  ionising radiation are at increased risk of fetal death and congenital malformation.
41  significantly reduced fusobacterial-induced fetal death and decidual necrosis without affecting the
42 indicate that TF is an important mediator in fetal death and growth restriction and that statins may
43 l studies, revealed significant increases in fetal death and malformation after Q fever during pregna
44            The potential association between fetal death and residential proximity to agricultural pe
45                Outcomes of include abortion, fetal death and respiratory disease in newborn piglets.
46 evel of TNF-alpha was always associated with fetal death and that the sTNFRs may be important for fet
47 ng pregnancy is a major risk factor for late fetal death and the sudden infant death syndrome, we inv
48 d 259 cases were reviewed after exclusion of fetal deaths and deaths of children who were not residen
49 ed 6% of pregnancy outcomes (primarily early fetal deaths and elective terminations) were lost.
50                 Singleton term (37-42 weeks) fetal deaths and live births, stratified by sex, served
51 villus sampling, (iv) amniocentesis, and (v) fetal deaths and live births.
52 e pregnancy outcomes including 143 000 early fetal deaths and stillbirths, 62 000 neonatal deaths, 44
53 tment during pregnancy only in case of prior fetal death), and women with negative thrombophilia scre
54 ham-vaccinated controls experienced viremia, fetal death, and abortion postchallenge.
55 f delivery, birth defects, preterm delivery, fetal death, and low Apgar score.
56 significant contributor of preterm birth and fetal death, and that these adverse phenotypes are rescu
57 ications (spontaneous abortion, intrauterine-fetal-death, and preterm delivery) and neonatal sequelae
58 reconceptual monitoring to increased risk of fetal death are equivocal and require further investigat
59  (aRR = 5.9; 95% CI, 1.8-19.7), intrauterine fetal death (aRR = 9.0; 95% CI, 1.2-65.5), and maternal
60 eral supplement use might reduce the risk of fetal death associated with maternal smoking.
61 tions before the 10th week of gestation or 1 fetal death at or beyond the 10th week of gestation.
62 tions before the 10th week of gestation or 1 fetal death at or beyond the 10th week of gestation.
63                                  Delivery or fetal death before 35 weeks occurred in 41.5% of pregnan
64    The primary study outcome was delivery or fetal death before 35 weeks of gestation.
65 ave significantly smaller litters because of fetal death between 8.5 and 15.5 days postcoitum.
66 th defects and oral clefts in livebirths and fetal deaths between 1997 and 2000 in seven Texas counti
67  human complement-induced placentitis caused fetal death, but some fetuses were born normal when lowe
68 evere dengue infection increased the risk of fetal death by about five times (4.9, 2.3-10.2).
69  a disruption of placental architecture, and fetal death by E15.5.
70 ural, and erythroid lineages, culminating in fetal death by embryonic day 15.5 (E15.5).
71        Rate of fetal injury deaths, based on fetal death certificates coded with an underlying cause
72             Data were derived from birth and fetal death certificates that were linked for the first
73 acental transmission to the fetus along with fetal death, congenital microcephaly, and/or Central Ner
74                         Using live birth and fetal death data from New Jersey from 1998 to 2004, the
75 y have reduced the risk of influenza-related fetal death during the pandemic.
76 sure to higher incidence of miscarriages and fetal death, even at blood lead elevations ( approximate
77          In 3 states, reported crash-related fetal deaths exceeded that of crash-related infant death
78 eterious to the fetus and is associated with fetal death, fetal growth restriction, and a spectrum of
79 nd low platelet count syndrome, intrauterine fetal death, fetal growth restriction, or placental abru
80    Certificates of 1,449,287 live births and fetal deaths filed in Georgia from 1980 through 1992 wer
81  CHD cases (n=26 598) comprised live births, fetal deaths from 20 weeks gestation, and terminations o
82                            We examined 8,510 fetal deaths (&gt;/=20 weeks' gestation) to estimate their
83 ly, other serious neurologic conditions, and fetal death, health ministries across the region have ad
84                  In addition, 5 intrauterine fetal deaths hosted SCN5A rare nonsynonymous genetic var
85  uncontrolled complement activation leads to fetal death in aPL-treated mice.
86 signaling mediates neutrophil activation and fetal death in APS and that statins may be a good treatm
87 t causes defective placental development and fetal death in DBA/2-mated CBA/J mice.
88 tion of pregnancy did not affect the risk of fetal death in the absence of maternal smoking; 3) odds
89 ational comparison, stillbirth is defined as fetal death in the third trimester (>/=1000 g birthweigh
90 n setting, a region with the highest rate of fetal death in the world.
91 placental diseases-the most common causes of fetal death in this at-risk group.
92                                  The reduced fetal death in TLR4-deficient mice was accompanied by de
93 oup and three in the intervention group) and fetal death in utero (ten in the standard care group and
94 s in key safety outcomes including abortion, fetal death in utero, and congenital anomalies.
95 mplement activation in the placenta leads to fetal death in utero.
96 on on the risk of specific malformations and fetal death is lacking.
97                               There were 163 fetal death/live-birth different-sex twin pairs.
98                               There were 434 fetal death/live-birth same-sex twin pairs.
99 l cleft, low birth weight, preterm delivery, fetal death, low Apgar score, and mode of delivery were
100 l cleft, low birth weight, preterm delivery, fetal death, low Apgar score, and mode of delivery.
101 sure with orofacial cleft, preterm delivery, fetal death, low Apgar score, and mode of delivery.
102 owing outcomes were considered: maternal and fetal death; malformations; preterm delivery; small for
103 nience sample of 91 unexplained intrauterine fetal deaths (mean [SD] estimated gestational age at fet
104 netic evaluation of 91 cases of intrauterine fetal death, missense mutations associated with LQTS sus
105  during pregnancy almost doubled the odds of fetal death (mOR 1.9, 95% CI 1.6-2.2).
106 dies (44 publications) with more than 10,147 fetal deaths, more than 16,274 stillbirths, more than 43
107 mains of reproductive toxicity: intrauterine fetal death, morphologic teratogenicity, growth impairme
108 imary outcome was a composite of intrapartum fetal death, neonatal death, an Apgar score of 3 or less
109 le autoantibodies, Raynaud's phenomenon, and fetal death occurred at 20 weeks gestation.
110 .0% (95% CI, 15.2% to 23.2%) of pregnancies; fetal death occurred in 4%, neonatal death occurred in 1
111 egnancy resulted in p-aHUS, one intrauterine fetal death occurred, and seven pregancies were uneventf
112                                           30 fetal deaths occurred among women assigned multivitamins
113 re often seen in children, and abortions and fetal deaths occurred in pregnant women infected with VE
114                                 Stillbirths (fetal deaths occurring at >/=20 weeks' gestation) are ap
115  of all trauma during pregnancy, with 82% of fetal deaths occurring during these automobile accidents
116 -specific prevalence of cerebral palsy after fetal death of the co-twin is much higher than that repo
117 lete disruption of the N-myc gene results in fetal death on the first day of renal organogenesis.
118 se serious consequences include miscarriage, fetal death or an infant born with birth defects (i.e.,
119           We defined three primary outcomes: fetal death or birth before 34 weeks and 0 days gestatio
120                                 Intrauterine fetal death or stillbirth occurs in approximately 1 out
121 f early preterm birth, gestational diabetes, fetal death or stillbirth, perinatal death, or admission
122 OR = 0.34, 95% CI 0.21-0.54) or mothers with fetal deaths (OR = 0.36, 95% CI 0.19-0.67).
123  are diagnosed with CHD die as a TOFPA, late fetal death, or early neonatal death.
124 failure (anembryonic gestation, embryonic or fetal death, or incomplete or inevitable spontaneous abo
125 32 live-birth controls and 3,328 live-birth, fetal-death, or electively terminated cases had exposure
126 sis utilized 319 cases of selected causes of fetal death other than congenital anomalies and 611 non-
127 ferences between the 2 groups in the risk of fetal death (P = 0.99) or early infant death (P = 0.19).
128 regnancy and assuming no further spontaneous fetal deaths (P=0.28).
129                           A peak rate of 9.3 fetal deaths per 100 000 live births was observed among
130  leading trauma mechanism (82% of cases; 2.3 fetal deaths per 100 000 live births), followed by firea
131 tic fetal injury deaths were identified (3.7 fetal deaths per 100 000 live births).
132 ctive toxicity in five domains (intrauterine fetal death, physical malformations, growth impairment,
133 aborative Research Network Initial Causes of Fetal Death protocol from the Eunice Kennedy Shriver Nat
134             IL-10 attenuated the LPS-induced fetal death rate (to 22%) and growth restriction (P<0.05
135                                          The fetal death rate was 45.6 per 1000 births, the neonatal
136                                 In vivo, the fetal death rate was significantly reduced in TLR4-defic
137               This ecologic study notes that fetal death rates (FDR) during the Washington DC drinkin
138 ough linkage of death records with birth and fetal death records, and 47% (n = 116) through review of
139 ta of reproductive-age women, live birth and fetal death records, and medical examiner records in Mar
140  death, thrombophilic women experienced less fetal death recurrences, less preterm births and preecla
141 41 weeks) at E17.5 had fewer pups, more late fetal deaths, reduced fetal weight, increased placental
142 tor vehicle crashes are the leading cause of fetal deaths related to maternal trauma.
143  0.49, 1.05; P = 0.09] and increased risk of fetal death (RR = 1.58; 95% CI = 0.95, 2.63; P = 0.08),
144 s into consideration to reduce the burden of fetal death, stillbirth, and infant death.
145  that maternal obesity increases the risk of fetal death, stillbirth, and infant death; however, the
146 l BMI were associated with increased risk of fetal death, stillbirth, and neonatal, perinatal, and in
147                                              Fetal death, stillbirth, and neonatal, perinatal, and in
148 ng adjusted relative risk (RR) estimates for fetal death, stillbirth, or infant death by at least 3 c
149 live births of 160 infants (4 twin pairs), 1 fetal death/stillbirth, 11 spontaneous abortions, and 1
150                       Among women with prior fetal death, thrombophilic women experienced less fetal
151 y RR per 5-unit increase in maternal BMI for fetal death was 1.21 (95% CI, 1.09-1.35; I2 = 77.6%; n =
152 clinical diagnosis of influenza, the risk of fetal death was increased (adjusted hazard ratio, 1.91;
153                                  The risk of fetal death was reduced with vaccination during pregnanc
154  CI, 0.91 to 1.11; P=0.87), and the rates of fetal death were 4.3% and 5.0%, respectively (relative r
155                                     Rates of fetal death were 7% in both groups; overall adverse outc
156 0, absolute risks per 10,000 pregnancies for fetal death were 76, 82 (95% CI, 76-88), and 102 (95% CI
157                                    Causes of fetal death were assigned by using the Stillbirth Collab
158                                         When fetal deaths were counted as treatment failures, the per
159  induced abortions, ectopic pregnancies, and fetal deaths were obtained by using data from the Center
160 proof that live vaccination protects against fetal death, whereas immunization using whole-tachyzoite
161 rder to prevent congenital abnormalities and fetal death which can result from both excessive and ins
162 ression models to estimate hazard ratios for fetal death, with the gestational day as the time metric
163 scarriages in the first trimester, and 1 had fetal death, with the macerated stillborn showing diffus

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