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1 out life (beyond guiding brain wiring during fetal development).
2 terine environment could also interfere with fetal development.
3 sm through which maternal stress may disrupt fetal development.
4 MeCP2 and interacting proteins during human fetal development.
5 e, investigated leukocyte recruitment during fetal development.
6 Cs, and some remain undifferentiated through fetal development.
7 uitment proceeds in a similar fashion during fetal development.
8 transient organ that is necessary for proper fetal development.
9 growth, revealing a role for MC1R in normal fetal development.
10 epigenetic mechanisms potentially affecting fetal development.
11 st as an underlying cause that starts during fetal development.
12 nct characteristics associated with abnormal fetal development.
13 r these gene promoters epigenetically during fetal development.
14 ion from scarless to fibrotic healing during fetal development.
15 ure NK-cell progeny emerge and expand during fetal development.
16 to reconstruct exposure at specific times in fetal development.
17 utcomes across the lifespan can be traced to fetal development.
18 ity of germ cells to study especially during fetal development.
19 naling is required for eyelid closure during fetal development.
20 ing placental transport of Hcy may impact on fetal development.
21 n) is important for successful completion of fetal development.
22 ood-brain barrier permeability during normal fetal development.
23 ophoblast metabolism and function as well as fetal development.
24 e adult Eln(+/)(-) mouse are defined in late fetal development.
25 e human and mouse brain during embryonic and fetal development.
26 ervous system formation during embryonic and fetal development.
27 ion with increases in plasma cortisol during fetal development.
28 wing to inaccessibility of germ cells during fetal development.
29 immunovascular role during placentation and fetal development.
30 rs through impacts on placental function and fetal development.
31 c-Kit(+)) that enlarged centrifugally during fetal development.
32 s a hematopoietic organ during embryonic and fetal development.
33 changes required for placental formation and fetal development.
34 ther subtler (microscopic) defects in murine fetal development.
35 ehog protein focuses on their role in normal fetal development.
36 te the effects of placental insufficiency on fetal development.
37 mechanisms to balance maternal immunity and fetal development.
38 ered to be benign with regards to effects on fetal development.
39 he control of gamma to beta switching during fetal development.
40 expressed an activated phenotype throughout fetal development.
41 eyelid epithelium morphogenesis during mouse fetal development.
42 lopment of the placenta is crucial to normal fetal development.
43 bin gene coexpression and competition during fetal development.
44 Cholesterol is required for fetal development.
45 that amplifies the maternal blood supply for fetal development.
46 anscript is found in multiple tissues during fetal development.
47 specific marker of endothelial cells during fetal development.
48 , a process that is presumed to begin during fetal development.
49 r regulated IGF bioavailability during early fetal development.
50 derstanding of how they affect pregnancy and fetal development.
51 (DETC), homes to the murine epidermis during fetal development.
52 sue-specific genes normally expressed during fetal development.
53 late homeostatic T-cell proliferation during fetal development.
54 the proper positioning of the testes during fetal development.
55 rnal bariatric surgery may affect subsequent fetal development.
56 nesis proceeds rapidly and faithfully during fetal development.
57 ation of two distinct pathways essential for fetal development.
58 ubtypes of neurons that are generated during fetal development.
59 ailure of elastic fiber assembly during late fetal development.
60 sinus primordial mesenchyme in males during fetal development.
61 mutational events during the late stages of fetal development.
62 , a bHLH factor found in the pancreas during fetal development.
63 ocyte atresia or fewer oocytes formed during fetal development.
64 ient phase of PT cyst formation during early fetal development.
65 indicate abnormal morphogenesis during early fetal development.
66 sed their potential to interfere with neural fetal development.
67 tial for the high erythropoietic rate during fetal development.
68 ts of maternal malnutrition on placental and fetal development.
69 Maternal diet and metabolism impact fetal development.
70 ZIKV's neurotropic effects in the course of fetal development.
71 n the brain and increase in abundance during fetal development.
72 , presenting a heightened risk of perturbing fetal development.
73 Folate is vital for fetal development.
74 rise from abnormalities in germ cells during fetal development.
75 an early, but narrow, window of time during fetal development.
76 layers and map transcriptionally to in vivo fetal development.
77 of developmental delay (DD) originate during fetal development.
78 ncluding in the heart and lung, during human fetal development.
79 ol of gene expression is critical for normal fetal development.
80 e compensatory response that sustains normal fetal development.
81 e myelination of the peripheral axons during fetal development.
82 xposure to air pollutants is associated with fetal development.
83 t in spinal cord and vertebral growth during fetal development.
84 ticosteroids during their first trimester of fetal development.
85 nd muscle functions and important for normal fetal development.
86 gration between the 7(th) and 16(th) week of fetal development.
87 participates in beta cell programming during fetal development.
88 ously identified coexpression modules during fetal development.
89 onocytes in an undifferentiated state during fetal development.
90 expression of mouse embryonic globins during fetal development.
91 ly regulated in the endocrine lineage during fetal development, 237 of which are transcriptional regu
94 rs regulate epidermal differentiation during fetal development, affecting key constituents of both ke
95 It has been postulated that during human fetal development, all cells of the lung epithelium deri
96 and pathologically during critical stages of fetal development alter nervous system function and beha
97 bility of choline during critical periods of fetal development alters hippocampal development and aff
99 ieved to regulate growth of the liver during fetal development and after injury in adults, because th
100 eight percent of such DHSs are active during fetal development and are enriched in variants associate
101 g events take place during gametogenesis and fetal development and are thought to have long-lasting c
102 iving germ cells enter meiosis at the end of fetal development and as a result, the postnatal ovary h
103 sis for our understanding of many aspects of fetal development and behaviour that remain in use in cl
105 unctional in cerebral blood vessels early in fetal development and continue to play a vital role in m
108 atus may be an additional factor influencing fetal development and effects of environmental toxins.
112 tion of biological processes can affect both fetal development and health outcomes that manifest late
113 veal an essential role for the Notch1 TAD in fetal development and identify important cell-autonomous
114 acental amino acid transport is required for fetal development and impaired transport has been associ
116 and important function for SALSA during the fetal development and in the mucosal innate immune defen
117 sion (P<0.0001) in the frontal cortex during fetal development and in the temporal-parietal and sub-c
118 in up-regulation of all genes present during fetal development and increases the cell size of neonata
119 s, exposure to FRalpha autoantibodies during fetal development and infancy could contribute to brain
121 essential for neural differentiation during fetal development and is a cardinal feature of neuroendo
123 ction of several enzymes demonstrated during fetal development and keratinocyte differentiation, but
124 importance of somatic mutations during human fetal development and malignant transformation in childr
127 we provide evidence that genes facilitating fetal development and nutrient transport display converg
128 n, the effect of GWG throughout pregnancy on fetal development and other outcomes has not been extens
129 ; 2) pulmonary vascular disease accompanying fetal development and perinatal life; 3) properties of p
130 a single essential nutrient, choline, during fetal development and point to these pathways as candida
134 ts with the MAP3K1 signaling pathways during fetal development and provide strong empirical evidence
135 embryonic myosin isoform is expressed during fetal development and rapidly down-regulated after birth
136 tein expressed during early to mid-gestation fetal development and re-expressed as a surface Ag by tu
138 ations in fetal lambs may indicate a role in fetal development and suggest that extracellular formate
142 receptors to a role in morphogenesis during fetal development and to a role in the metabolism of pho
143 mother for the metabolic stress presented by fetal development and to ensure appropriate nutrient all
144 expand the functional roles of oxysterols to fetal development and to the detoxification of oxidation
145 ing secretions from the placenta that affect fetal development and whether a mitochondria-targeted an
146 an extracellular matrix protein important in fetal development and wound healing, yet its antimicrobi
148 us muscle phenotype during embryogenesis and fetal development, and adults in the laboratory have gro
149 ssociated antigens are also expressed during fetal development, and it is, thus, not surprising that
151 od-brain permeability early, but not late in fetal development, and pretreatment with glucocorticoids
152 are required for lymph node formation during fetal development, and recent evidence implies a role in
153 h vitamin A deficiency can be induced during fetal development, and reveals new functions for the vit
154 8(+) dT that are permissive of placental and fetal development, and reversal of this dysfunctional st
155 ession pattern of Scrb1 during embryonic and fetal development, and show that Scrb1 expression closel
156 s important for NT, particularly during late fetal development, and that actively dividing G1 cells s
157 ained high in Bcl11a(cko/cko) embryos during fetal development, and this was further augmented in Klf
158 al obesity increases oxidative stress during fetal development, and to determine whether administrati
159 gin to endochondral bones are hypoxic during fetal development, and we demonstrate that Hif-1alpha is
161 ecursors from colonizing distal bowel during fetal development are not completely understood in many
165 ant to cancer are well known to overlap with fetal development, as reflected in reactivation of embry
167 Likewise, any gene that favors embryonic/fetal development at consequent cost to the mother--by a
168 ession and adipogenic differentiation during fetal development, at least partially through reducing D
169 ever, antibodies can access the brain during fetal development before the barrier achieves full integ
173 long bones has been studied in-depth during fetal development but not postnatally in the epiphysis.
174 is a rich source of essential nutrients for fetal development, but in contrast, it is also a well-kn
175 sed in most cancers and leukemias and during fetal development, but not in most normal adult tissues.
176 g mechanisms: a direct detrimental effect on fetal development by crossing the placenta and entering
177 le antiandrogenic phthalates exposure during fetal development can have greater impacts than individu
178 Programming of the immune system during fetal development can influence asthma-related risk fact
179 cal mechanism by which folate acts to affect fetal development can inform appraisal of expected benef
180 at the repression of HMGA2 expression during fetal development could contribute to the specific birth
182 pite normal implantation and early placental/fetal development, deletion of Bmpr2 in the uterine deci
185 xpressed in hypertrophic chondrocytes during fetal development (E14.5-E18.5), with maximal expression
187 ply of the essential nutrient choline during fetal development [embryonic day (E) 11-17] in rats caus
188 s of pregnant Etnk2(-/-) females showed that fetal development failed at the late stage of pregnancy
189 tudies suggest that at critical times during fetal development fetal injury programs the development
190 ressing neurons within the brainstem late in fetal development; gender specificity derives from a tim
191 l and ventrolateral prefrontal cortex during fetal development, genes harboring damaging de novo muta
193 rats and humans, and its availability during fetal development has long-lasting cognitive effects.
194 ercholesterolemia in lesion formation during fetal development has previously been established in rab
195 n the Americas and its devastating impact on fetal development have prompted the World Health Organiz
197 imary motor--somatosensory cortex during mid-fetal development in autism spectrum disorder and the fr
198 Lactation is necessary for both infant and fetal development in eutherians and marsupials, although
199 This murine model may have relevance to fetal development in human populations with inadequate r
202 ified widespread expression primarily during fetal development in myocytes and interstitial cells sug
205 epigenetic profile changes occurring during fetal development in response to in utero environment va
212 ting, we studied gene-expression profiles in fetal development in the relevant tissues and time inter
215 exposures, including those that occur during fetal development in utero, can cause epigenetic effects
217 ic signaling pathways that are active during fetal development, including Hedgehog and Hippo/Yes-asso
218 l functions can have a significant impact on fetal development, including the brain, outcomes that ar
219 t and human cardiac physiology and placental-fetal development indicate a need for models in precocia
220 t and human cardiac physiology and placental-fetal development indicate a need for models in precocia
221 een postulated that pathogenic events during fetal development influence atherosclerosis-related dise
222 onmental influences during the embryonic and fetal development influence the individual's susceptibil
223 is known in humans about how testosterone in fetal development influences later neural sensitivity to
226 In addition, overexpression of ZAC during fetal development is believed to underlie the rare disor
227 show that HSPC engraftment of bone marrow in fetal development is dependent on the guanine-nucleotide
228 rogramming occurs when the normal pattern of fetal development is disrupted by an abnormal stimulus o
230 of maternal 25-hydroxyvitamin D [25(OH)D] in fetal development is uncertain, and findings of observat
233 ly highly expressed in the liver only during fetal development, is reactivated in 60% of HCC tumors a
234 idney does not ascend as it should in normal fetal development, it remains in the pelvic area and is
235 ing the proposal that viral infection during fetal development may play a causal role in the pathogen
236 Maternal nutrient reduction (MNR) during fetal development may predispose offspring to chronic di
237 ell S6K1 signaling, rather than IUGR, during fetal development may underlie reduced beta cell growth
241 ents, lenses did not show obvious changes in fetal development, nor in the differentiation of epithel
242 e mouse germ line have revealed that much of fetal development occurs normally in their absence.
243 amniotic fluid choline levels would enhance fetal development of cerebral inhibition and, as a resul
244 indicate that, in contrast to embryonic and fetal development of clones, the process of NT-ES cell d
249 ded filaments are not needed for normal lens fetal development or fiber cell differentiation, they ap
251 ther risk factors (related to twinning or to fetal development) or other factors (genetic or nongenet
253 ly nursing period are representative for the fetal development period, using serial maternal serum sa
254 proliferation of cortical precursors during fetal development provides a likely environment for soma
255 lucidating the mechanisms by which disrupted fetal development raises the risk of this disorder.
256 changes of pregnancy, and specific needs for fetal development, recommendations were made to assist i
258 een studied extensively during embryonic and fetal development, relatively little is known concerning
260 the hypothesis that pathogenic events during fetal development result in persistent changes in arteri
261 of glucocorticoids during specific times of fetal development results in focal and segmental glomeru
262 ues innervated by vagal motor neurons during fetal development reveal potential sites of HGF-MET inte
264 Hematopoietic transitions that accompany fetal development, such as erythroid globin chain switch
265 these data indicate that ARMC5 is crucial in fetal development, T-cell function and adrenal gland gro
267 teins, were expressed more abundantly during fetal development than during postnatal ages, and their
268 Its levels are highest during a period of fetal development that coincides with the emergence of s
269 the peripheral lymphoid tissues early during fetal development, the adaptive immune system in the fet
272 dent mechanisms whereby they could influence fetal development, these 2 nutrients also have a common
273 ating pathogenic mechanisms initiated during fetal development, this approach may identify genes in m
278 O enhances adipogenic differentiation during fetal development through inducing epigenetic changes in
279 rental disorders and offspring outcomes from fetal development to adolescence in high-income, middle-
282 , serves as a donor of methyl groups used in fetal development to establish the epigenetic DNA and hi
283 gues demonstrate a role for platelets beyond fetal development, to maintaining integrity of the adult
286 ection with P. gingivalis compromises normal fetal development via direct placental invasion and indu
287 somatic mutations during the late stages of fetal development was dependent on both gestational age
288 both angiogenesis and vasculogenesis during fetal development, we investigated the hypothesis that V
291 72 hours of birth but are viable throughout fetal development when dramatic cardiovascular structura
293 umber of murine B-1 progenitors peaks during fetal development whereas B-2 B cell production predomin
294 ere on inflamed yolk sac vessels during late fetal development, whereas at earlier embryonic stages (
295 orting a potential physiological role during fetal development, whereas G(o)alpha1c expression increa
296 of gene expression changes occurring during fetal development which are reversed in early postnatal
297 ntrauterine milieu, resulting in accelerated fetal development with increased risk of macrosomia.
298 Maternal nutrient restriction (NR) affects fetal development with long-term consequences on postnat
299 nvironmental conditions during embryonic and fetal development with risk of diseases later in life.
300 erine conditions that provoke adjustments in fetal development, with long-term consequences for stres
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