ライフサイエンスコーパス: fetal growth restriction

1 atory cytokines that have been implicated in fetal growth restriction.

2 IFN-gamma levels at birth may be related to fetal growth restriction.

3 rst half of pregnancy and is associated with fetal growth restriction.

4 mpletely prevented the high altitude-induced fetal growth restriction.

5 a strong predictor of both preterm birth and fetal growth restriction.

6 rates of preterm birth, low birth weight, or fetal growth restriction.

7 esting mechanisms underlying hypoxia-related fetal growth restriction.

8 acental inflammation and was associated with fetal growth restriction.

9 the placenta, followed several days later by fetal growth restriction.

10 impact on stillbirth, preterm delivery, and fetal growth restriction.

11 d amino acid transporter activity and causes fetal growth restriction.

12 omplications, uteroplacental dysfunction, or fetal growth restriction.

13 logical conditions, including stillbirth and fetal growth restriction.

14 ies and preterm newborns without evidence of fetal growth restriction.

15 ype in response to hypoxia, a major cause of fetal growth restriction.

16 , and impaired spiral artery remodeling with fetal growth restriction.

17 hi women, especially those at higher risk of fetal growth restriction.

18 out resulted in reduced placental weight and fetal growth restriction.

19 estational hypoxia such as pre-eclampsia and fetal growth restriction.

20 ances from maternal blood and contributes to fetal growth restriction.

21 presence or absence of ultrasonic markers of fetal growth restriction.

22 o trigger delivery in mothers of babies with fetal growth restriction.

23 complicated with intervillositis, can cause fetal growth restriction.

24 sed to play a role in the pathophysiology of fetal growth restriction.

25 ), several are of pathological relevance for fetal growth restriction.

26 ancies worldwide and is often complicated by fetal growth restriction.

27 oteinuria, and edema) and, in some patients, fetal growth restriction.

28 egnancy disorders including preeclampsia and fetal growth restriction.

29 sk of major birth defects, preterm birth, or fetal growth restriction.

30 ot the PCE area, was associated with LBW and fetal growth restriction.

31 ncluding late miscarriage, preeclampsia, and fetal growth restriction.

32 except for early preterm birth and possibly fetal growth restriction.

33 nce of preeclampsia, placental abruption, or fetal growth restriction.

34 are associated with maternal infections and fetal growth restriction.

35 f gestation; 1.16, 1.01-1.34; I(2)=64%), and fetal growth restriction (1.26, 1.20-1.33; I(2)=1%).

36 An atmosphere of 13% oxygen induced fetal growth restriction (1182 +/- 9 mg, n = 90 vs. 1044

37 The link between fetal growth restriction and abruption suggests that the

38 uld have broad implications for the study of fetal growth restriction and birth weight, and for the p

39 d with incident CHF, atrial arrhythmias, and fetal growth restriction and complex CHD was associated

40 After controlling for fetal growth restriction and early delivery, the high ri

41 s common environmental risk factors for both fetal growth restriction and high blood pressure or whet

42 nd JZ+D at GD14 and GD18 in association with fetal growth restriction and higher blood pressure.

43 his may be due to conditions associated with fetal growth restriction and iatrogenic preterm birth.

44 pregnant dams during early pregnancy led to fetal growth restriction and infection of the fetal brai

45 Here we show that both fetal growth restriction and over-growth are associated

46 Placental hypoxia is causally implicated in fetal growth restriction and preeclampsia, with both occ

47 tions and by the likelihood of recurrence of fetal growth restriction and preterm birth.

48 also occurred in other pregnancy disorders (fetal growth restriction and recurrent miscarriage), ind

49 ry condition of the placenta associated with fetal growth restriction and stillbirth.

50 posure is a significant mechanism underlying fetal growth restriction and the programming of adverse

51 tudies have suggested an association between fetal growth restriction and the risk of spontaneous pre

52 ove pregnancy outcomes in severe early-onset fetal growth restriction and therefore it should not be

53 he hypothesis that shared factors cause both fetal growth restriction and urogenital anomalies was su

54 centrations are associated with proportional fetal growth restriction and with an increased risk of p

55 Here, we developed a mouse model of fetal-growth restriction and placental insufficiency tha

56 he fetus and is associated with fetal death, fetal growth restriction, and a spectrum of central nerv

57 mined low birth weight (LBW), preterm birth, fetal growth restriction, and birth defects among births

58 weight-for-gestational-age, an indicator of fetal growth restriction, and furthermore the authors ob

59 rmation has been described in pre-eclampsia, fetal growth restriction, and miscarriage.

60 weight includes babies born preterm and with fetal growth restriction, and not all these infants have

61 se obstetric outcomes such as pre-eclampsia, fetal growth restriction, and preterm birth.

62 ications of pregnancy, such as preeclampsia, fetal growth restriction, and stillbirth.

63 egnancy complications, such as preeclampsia, fetal growth restriction, and stillbirth.

64 Premature labor, fetal demise, and fetal growth restriction are accompanied by indices of i

65 Increased morbidity and fetal growth restriction are reported in uninfected chil

66 We defined fetal growth restriction as a combination of estimated f

67 evisited the prevailing hypotheses regarding fetal growth restriction as a risk factor for urogenital

68 revious understanding and interpretations of fetal growth restriction as represented by small for ges

69 Fetal growth restriction associates with increased risk

70 Hypoxia-related fetal growth restriction becomes apparent between 25 and

71 late levels in pregnancy are associated with fetal growth restriction, but the underlying mechanisms

72 rnal vitamin D deficiency has been linked to fetal growth restriction, but the underlying mechanisms

73 ro tobacco exposure has been associated with fetal growth restriction, but uncertainty remains about

74 Severe early-onset fetal growth restriction can lead to a range of adverse

75 Epidemiological studies have indicated that fetal growth restriction correlates with later disease,

76 Molecular mechanisms underlying fetal growth restriction due to placental insufficiency

77 e consumption led to placental inefficiency, fetal growth restriction, elevated fetal serum glucose a

78 Later the compensation fails and fetal growth restriction ensues.

79 yncytiotrophoblasts, leading to intrauterine fetal growth restriction, fetal liver hypocellularity, a

80 folate availability causes diseases such as fetal growth restriction, fetal malformations and cancer

81 e availability causes human diseases such as fetal growth restriction, fetal malformations and cancer

82 Fetal growth restriction (FGR) affects >200,000 pregnanc

83 Fetal growth restriction (FGR) affects 5% to 10% of newb

84 Fetal growth restriction (FGR) affects around 5% of preg

85 folate and choline status resulted in severe fetal growth restriction (FGR) and impaired fertility in

86 Fetal growth restriction (FGR) and preeclampsia (PE) are

87 ed with complications of pregnancy including fetal growth restriction (FGR) and preeclampsia.

88 tal exposure to disinfection by-products and fetal growth restriction (FGR) and preterm birth in the

89 eenage motherhood and short birth intervals, fetal growth restriction (FGR) and preterm birth, child

90 vanced maternal age (AMA) are susceptible to fetal growth restriction (FGR) and stillbirth.

91 Preeclampsia (PE) and fetal growth restriction (FGR) are associated with impai

92 The placenta of pregnancies whose outcome is fetal growth restriction (FGR) are characterized by abno

93 Preeclampsia (PE) and fetal growth restriction (FGR) are serious complications

94 nancies complicated by preeclampsia (PE) and fetal growth restriction (FGR) compared with control thi

95 n of placental vessel networks in normal and fetal growth restriction (FGR) complicated pregnancies.

96 Fetal growth restriction (FGR) is associated with global

97 However, placentas from fetal growth restriction (FGR) pregnancies are character

98 Fetal growth restriction (FGR) results from placental in

99 of the most common and preventable causes of fetal growth restriction (FGR), a condition in which a f

100 esponses, both of which were associated with fetal growth restriction (FGR).

101 normal pregnancies and those complicated by fetal growth restriction (FGR).

102 s a major cause of antepartum stillbirth and fetal growth restriction (FGR).

103 putative aetiologies in the pathogenesis of fetal growth restriction (FGR); however, the regulating

104 ry transformation occurs in preeclampsia and fetal growth restriction (FGR); these processes are not

105 Fetal growth restriction (FGR, <0.46 g) was defined as f

106 Fetal growth restriction has also been proposed, althoug

107 6-32 weeks of gestation who had very preterm fetal growth restriction (ie, low abdominal circumferenc

108 Campylobacter rectus infection that induces fetal growth restriction in a mouse model also compromis

109 us infection increases fetal resorptions and fetal growth restriction in a mouse model.

110 Placental sO2 was lower in fetal growth restriction in an angiotensin-converting en

111 cific transcript alone (Igf2P0(+/-)) lead to fetal growth restriction in mice.

112 mplex (OR, 31.8; 95% CI, 4.3-236.3) CHD, for fetal growth restriction in noncomplex (OR, 1.6; 95% CI,

113 ability; Sildenafil does not protect against fetal growth restriction in the chick embryo, supporting

114 ereas, placental growth restriction precedes fetal growth restriction in the Igf2P0(+/-) mouse.

115 cells is supported by Hmox1 and ameliorates fetal-growth restriction in Hmox1 deficiency.

116 Sequelae of fetal growth restriction include metabolic disease as we

117 Markers of fetal growth restriction included biometric ratios, uter

118 uartile, low birth weight increased >5-fold, fetal growth restriction increased >6-fold, and infant b

119 onatal glucose homeostasis and is altered by fetal growth restriction induced by maternal undernutrit

120 Fetal growth restriction is a major determinant of adver

121 Intrauterine fetal growth restriction (IUGR) is often associated with

122 quartiles had offspring with third-trimester fetal growth restriction, leading to a smaller head circ

123 Epidemiological data suggest that fetal growth restriction, maternal factors such as smoki

124 l-recessive disorder characterized by severe fetal growth restriction, microcephaly, a distinct facia

125 Screening procedures for fetal growth restriction need to identify small babies a

126 lve impaired placental function, either with fetal growth restriction or preterm labour, or both.

127 However, it is not clear whether it is fetal growth restriction or the accelerated postnatal gr

128 et count syndrome, intrauterine fetal death, fetal growth restriction, or placental abruption who had

129 gestation without congenital malformations, fetal growth restriction, or severe postnatal morbidity.

130 seizure, medically indicated preterm birth, fetal-growth restriction, or perinatal death.

131 p for trend < 0.001) and a decreased risk of fetal growth restriction (p for trend < 0.05).

132 cause of maternal hypertension in pregnancy, fetal growth restriction, premature birth, and fetal and

133 explored the associations of abruption with fetal growth restriction, preterm delivery, and perinata

134 tin are associated with an increased risk of fetal growth restriction, preterm delivery, and preeclam

135 ing that cardiovascular disease is linked to fetal growth restriction rather than to premature birth.

136 GT/GT) mice died perinatally associated with fetal growth restriction, reduced hepatic glycogen store

137 ow that fetal growth acceleration as well as fetal growth restriction, resulting from between-breed e

138 his correlation to recurrent miscarriage and fetal growth restriction, revealing the common mechanism

139 UtA) blood flow and relative protection from fetal growth restriction seen in altitude-adapted Andean

140 t undernutrition in the aggregate--including fetal growth restriction, stunting, wasting, and deficie

141 We used a mouse model of fetal growth restriction, the placental-specific Igf2 kn

142 e than placental malaria per se, might cause fetal growth restriction, through impaired transplacenta

143 and 6 days' gestation and severe early-onset fetal growth restriction to receive either sildenafil 25

144 eatures with human recurrent miscarriage and fetal growth restriction, we identified tissue factor (T

145 Maternal undernutrition contributes to fetal growth restriction, which increases the risk of ne

146 anomalies, suggesting an effect of relative fetal growth restriction within families.

147 example, maternal smoking (Z) is a cause of fetal growth restriction (X), which subsequently affects