ライフサイエンスコーパス: fetal heart

1 ay indicate novel adaptive strategies in the fetal heart.

2 genes decreased to the same levels as in the fetal heart.

3 were higher in the nonfailing heart than the fetal heart.

4 es in the nonfailing heart compared with the fetal heart.

5 s preferentially expressed in both adult and fetal heart.

6 l capillary-level adaptations in the in vivo fetal heart.

7 rs via the umbilical arterial route into the fetal heart.

8 glucocorticoid exposure in the Hsd11b2(-/-) fetal heart.

9 indow for the structural organization of the fetal heart.

10 f fiber architecture in the developing human fetal heart.

11 y binding to apoptotic cardiomyocytes in the fetal heart.

12 he sex differences in SP1 methylation in the fetal heart.

13 ssion of the alpha1D Ca channel in the human fetal heart.

14 ells contribute to normal development of the fetal heart.

15 a result most likely due to a defect in the fetal heart.

16 lette, an actin-binding protein expressed in fetal heart.

17 n of chamber-specific genes was defective in fetal hearts.

18 tion and decreased coronary artery volume in fetal hearts.

19 ndance for highly expressed miRNAs in HF and fetal hearts.

20 KDR and PDGFRalpha in first-trimester human fetal hearts.

21 onatal mouse hearts compared with 16-day-old fetal hearts.

22 decreases in PKCepsilon protein and mRNA in fetal hearts.

23 was up-regulated (P<0.05) in OB maternal and fetal hearts.

24 xpression of the alpha1D Ca channel in human fetal heart, (2) inhibition of alpha1D I(Ca-L) by positi

25 Among ongoing pregnancies with fetal heart activity, the multiple gestation rate with l

26 imers flanking exon 4 and mRNA from 22 human fetal hearts (age 11-25 weeks) and 3 adult hearts.

27 In fetal hearts ages 22-25 weeks and adult heart, the 52bet

28 the examiner to identify normal and complex fetal heart anatomy during the early second to the late

29 Sildenafil protects the fetal heart and circulation directly in hypoxic developm

30 ial effects of sildenafil transcend onto the fetal heart and circulation in complicated development i

31 nrichment in DNase I hypersensitive sites in fetal heart and lung.

32 the hypothesis that oxidative stress in the fetal heart and vasculature underlies the molecular basi

33 RNA and protein to control levels in vivo in fetal hearts and in vitro in embryonic myocyte cells.

34 on and phenotype in fibroblasts derived from fetal hearts and lungs was investigated by Affymetrix ar

35 ned in ex vivo hypoxic treatment of isolated fetal hearts and rat embryonic ventricular myocyte cell

36 oxide bioavailability (54.7 +/- 6.1%) in the fetal heart, and promoted peripheral endothelial dysfunc

37 ide bioavailability (115.6 +/- 22.3%) in the fetal heart, and restored endothelial function in the pe

38 cts of their cognate antibodies on the human fetal heart are unknown.

39 e of the electromechanical properties of the fetal heart as well as the mechanisms of arrhythmia to f

40 -1) at Ser-307 were increased (P<0.05) in OB fetal heart associated with lower downstream PI3K-Akt ac

41 sgene, IGF-1B mRNA was not detectable in the fetal heart at the end of gestation, was present in mode

42 ally, ICA cells could be identified in human fetal hearts at a developmental stage before sympathetic

43 of both the left and right ventricles of the fetal heart by day 78 of gestation.

44 We report that the fetal heart contains a reservoir of stem/progenitor cell

45 Abnormal fetal heart development was not previously described.

46 ulation of endocardial and epicardial EMT in fetal heart development, and we summarize key literature

47 differentiation and tissue formation during fetal heart development.

48 ds were administered to pregnant rats during fetal heart development.

49 ion and gene environment interactions during fetal heart development.

50 ibited a normal contractile function vs. CON fetal hearts during basal perfusion, they developed an i

51 the parallel development of the placenta and fetal heart early in pregnancy, very few studies suggest

52 Although OB fetal hearts exhibited a normal contractile function vs.

53 The fetal heart expresses the slow skeletal TnI (ssTnI) isof

54 Both in vitro and in fetal hearts, EZH2 interacted with cardiac transcription

55 Age-matched fetal hearts from voluntary terminations expressed neith

56 alent to about 2% of adult or 25% of 16-week fetal heart function) in a modified working heart prepar

57 d long-term consequences of these changes in fetal heart gene expression and induction of specific ho

58 Transcripts representing most members of the fetal heart gene program remained elevated from fetal to

59 Fetal heart growth occurs through cardiomyocyte prolifer

60 as performed on formalin-fixed sections of 4 fetal hearts identified in utero as having CHB or isolat

61 cture and function of the rapidly developing fetal heart is unknown.

62 (Flk-1) was used as a bait to screen a human fetal heart library in the yeast two-hybrid system.

63 e transcriptase protein (hTERT) during human fetal heart, liver, and kidney development.

64 Bhmt2 is expressed in fetal heart, lung, liver, kidney and eye.

65 Understanding hypoxia adaptation in the fetal heart may allow development of strategies to prote

66 ude new guidelines for the interpretation of fetal heart monitoring, advances in intrapartum fetal pu

67 itial use of Doppler US for the detection of fetal heart motion and the eventual use of pulsed and co

68 onsible for delivering oxygen to the anaemic fetal heart muscle using contrast-enhanced echocardiogra

69 Human fetal hearts (n = 5) between 10-19 weeks of gestation we

70 es, -346 and -268, were demonstrated in both fetal hearts of maternal hypoxia and H9c2 cells treated

71 and cross-reactive (laminin) autoantigens in fetal hearts of varied ages and in adult hearts.

72 es may be involved in the examination of the fetal heart (pediatric cardiologists, obstetricians, mat

73 ete atrioventricular (AV) block in the human fetal heart perfused by the Langendorff technique and in

74 ed a novel Langendorff, biventricular, ovine fetal heart preparation to investigate the effects of ad

75 notype explained the majority of variance in fetal heart rate (-10 beats per minute per added mutatio

76 the separate indications of a nonreassuring fetal heart rate (7.1% and 7.9%, respectively; P=0.30) a

77 l cord occlusion on the normal maturation of fetal heart rate (FHR) and mean arterial pressure (MAP)

78 rees AVB; 3) reactive ventricular and atrial fetal heart rate (FHR) tracings at ventricular rates >56

79 to fetal mean arterial blood pressure (MAP), fetal heart rate (FHR), and fetal baro- and chemoreflexe

80 pnic hypoxia (Pa,O2, 12 +/- 0.6 mmHg) on the fetal heart rate (FHR), mean systemic arterial blood pre

81 Our purpose was to characterize the fetal heart rate (FHR)/gestational age (GA) profile of f

82 and by the blockade of reflex reductions in fetal heart rate after intravenous injection of phenylep

83 idenced by a reduction in the variability of fetal heart rate and by the blockade of reflex reduction

84 Strong correlations between fetal heart rate and neonatal heart rate (r=0.700; P<0.0

85 Labor complicated by a nonreassuring fetal heart rate before randomization was documented for

86 2 LQT1 founder populations, third trimester fetal heart rate discriminated between fetal genotypes a

87 This finding strengthens the role of fetal heart rate in the early detection and risk stratif

88 Electronic monitoring of the fetal heart rate is commonly performed, in part to detec

89 .3%), and preterm labor (52.3%) and abnormal fetal heart rate monitoring (22.2%) were more common in

90 QTS, although the most common LQTS rhythm, a fetal heart rate of less than third percentile for gesta

91 no association between the highest or lowest fetal heart rate recorded for each child and the risk of

92 nitoring strategies: reduced cardiotocograph fetal heart rate STV (CTG STV), early DV changes (pulsat

93 bgroup of 2168 women in whom a nonreassuring fetal heart rate was detected before randomization.

94 During hypoglycaemia, an initial rise in fetal heart rate was followed by a slower fall.

95 mal findings on electronic monitoring of the fetal heart rate were associated with an increased risk

96 lar block, but sinus bradycardia, defined as fetal heart rate<3% for gestational age, is most common.

97 is revealed significant associations between fetal heart rate, genotype, and phenotype; mean third tr

98 We investigate third trimester fetal heart rate, routinely recorded within public mater

99 allopurinol led to significant increases in fetal heart rate, umbilical blood flow and umbilical vas

100 aphy versus intermittent auscultation of the fetal heart rate.

101 The masked system functioned as a normal fetal heart-rate monitor.

102 junct to conventional intrapartum electronic fetal heart-rate monitoring did not improve perinatal ou

103 junct to conventional intrapartum electronic fetal heart-rate monitoring modifies intrapartum and neo

104 dditional information for use when uncertain fetal heart-rate patterns were detected.

105 and phenotype; mean third trimester prelabor fetal heart rates obtained from obstetric records (gesta

106 The human fetal heart remains highly isotropic until 14-19 weeks,

107 f Fallot (TOF) without a 22q11.2 deletion, 3 fetal heart samples, and 8 normally developing infants.

108 nism by which these heart defects may arise, fetal heart structure and function in these transgenic a

109 onin affect the functional properties of the fetal heart that lead to infantile cardiomyopathy.

110 ctors that may have an adverse effect on the fetal heart, there is a growing body of epidemiological

111 In fetal hearts, there was significantly higher abundance o

112 immunohistologic evaluation of CHB-affected fetal heart tissue and by determination of erythropoieti

113 odel by transplanting second-trimester human fetal heart tissues s.c. into the ear pinna of a SCID mo

114 cells were delivered into these functioning fetal heart tissues: in contrast to traditional murine h

115 ination of the structure and function of the fetal heart together with the evaluation of other parame

116 ned by bradycardia, deceleration, or lack of fetal heart tones (FHTs).

117 xpression levels and with novel exons in the fetal heart transcriptome are known to play central role

118 window to study electrical properties of the fetal heart, unlike what has been available to date.

119 The fetal heart was visualized outside the chest through a d

120 At 10-14 weeks the fetal hearts were highly isotropic and few tracts could

121 Following exsanguination under anaesthesia, fetal hearts were mounted in the Langendorff preparation

122 m days 15 to 20 of gestational age, and term fetal hearts were studied.

123 In tissues such as fibroblasts and fetal hearts, where IGF1 levels are high, we found eithe

124 interacted with the SP1 binding sites in the fetal heart, which may explain the sex differences in SP

125 of changes in structure and function in the fetal heart with the focus on congenital heart disease m

126 by late gestation exhibit markedly enlarged fetal hearts with increased myocardial trabeculation and