ライフサイエンスコーパス: fetal membrane

1 omprises the maternal decidua, placenta, and fetal membranes.

2 s (MMPs) and prostaglandin (PG) synthesis in fetal membranes.

3 is of preterm labor and premature rupture of fetal membranes.

4 inases (MMPs) and prostaglandin synthesis in fetal membranes.

5 of pregnant women with premature rupture of fetal membranes.

6 chlamydial inclusions in the endometrium and fetal membranes.

7 E (sPE) on the smooth chorion portion of the fetal membranes.

8 gulation of proinflammatory cytokines in the fetal membranes.

9 We examined the contribution of the fetal membranes, amnion and chorion, to human embryonic

10 spiked samples was between 95% and 103% for fetal membranes and between 99% and 114% for placenta.

11 skeletal muscle, and, to a lesser degree, in fetal membranes and other tissues of infected fetuses.

12 for placenta and 1.19 + or - 0.68 pmol/g for fetal membranes, and 0.93 + or - 0.28, 0.88 + or - 0.33,

13 Pghs2, and Mpges1 expression in placenta and fetal membranes, and it elevated amniotic fluid IL-1beta

14 Our results suggested that fetal membranes are the probable source of AF sIL-6R and

15 drostenedione induced maternal endocrine and fetal membrane biochemical changes, and alteration of my

16 vity but did not produce preterm delivery or fetal membrane changes.

17 e model, we found that small ruptures of the fetal membrane closed within 72 h whereas healing of lar

18 aternal blood, amniotic fluid, placenta, and fetal membranes collected during Caesarean section (n=14

19 ed both survivin and Bcl-xL, indicating that fetal membranes could foster persistent viral infection.

20 labor and with preterm premature rupture of fetal membranes did not achieve statistical significance

21 These alterations occur in normal fetal membranes during late pregnancy, in preparation fo

22 d significantly in the placenta, uterus, and fetal membranes during PGN+poly(I:C)-induced preterm lab

23 iabetes in pregnancy compromise maternal and fetal membrane essential fatty acids (FAs).

24 Chorioamnionitis, premature rupture of fetal membranes (FMs), and subsequent preterm birth are

25 PGE2 content was increased significantly in fetal membranes from thrombin-injected animals.

26 We investigated premature senescence of fetal membranes in women with pPROM and spontaneous PTB

27 ibrillar collagens imparting strength to the fetal membranes, is expressed by leukocytes and chorioni

28 strate significant effects of the mother and fetal membranes on pregnancy outcome, with possible impl

29 CI): 1.1, 4.9), preterm premature rupture of fetal membranes (OR = 3.6, 95% CI: 1.5, 8.7), and cervic

30 At term, fetal membranes overlying the cervix, the future site of

31 ntrations were found in placenta compared to fetal membranes (P<0.0001), in umbilical vein compared w

32 Fetal membranes possess distinctive properties that can

33 Preterm, prelabour rupture of the fetal membranes (pPROM) is the commonest antecedent of p

34 has linked preterm premature rupture of the fetal membranes (PPROM) to placental abruption.

35 ies were 42 and 36% for the placenta and the fetal membranes, respectively.

36 The mechanisms of normal and pathologic fetal membrane rupture are not well understood.

37 Thus, fetal membrane rupture is likely to be the result of bio

38 Premature fetal membrane rupture occurs not infrequently and is as

39 hemical and endocrine changes accompanied by fetal membrane rupture, cervical dilatation and live del

40 th, second trimester loss, preterm premature fetal membrane rupture, or a history of a cervical proce

41 V-1 from mother to infant increases when the fetal membranes rupture more than four hours before deli

42 t membranes (<34 weeks) and the inducibility fetal membrane senescence phenotype by oxidative stress

43 Comparison with a prior cohort of fetal membranes shows that acute inflammation only takes

44 Interstitial collagen gives fetal membranes tensile strength, and membrane rupture h

45 unknown role for the gene in the yolk sac, a fetal membrane that is the site of hematopoiesis and is

46 of the cervix and biochemical changes in the fetal membranes that lead to rupture.

47 In the placenta and fetal membranes, the rs2280964 major allele homozygotes

48 Fetal membranes usually rupture during the process of la

49 ry cytokine genes Il1b, Il6, Tnf and Il10 in fetal membranes was suppressed by (+)-naloxone, and cyto

50 Term fetal membranes were exposed to cigarette smoke extract

51 lli, which are bathed in maternal blood, and fetal membranes, which encapsulate the amniotic cavity.