ライフサイエンスコーパス: fetal stage

1 nhibit upstream Vgamma rearrangements at the fetal stage.

2 ssion of the gamma-gene was recruited to the fetal stage.

3 results in hematopoietic defects during the fetal stages.

4 ng in the development of the prostate during fetal stages.

5 tinct stages, marking the entire CCS by late fetal stages.

6 mRNA content of IGF system components during fetal stages.

7 a in the early embryonic CNS compared to the fetal stages.

8 ells persisted on in vivo differentiation to fetal stages.

9 maternal allele in various mouse tissues at fetal stages.

10 In contrast, at late fetal stages (18.5 dpc), cells that were Shh-responsive

11 hybridization studies in human embryonic and fetal stages (35 days post-ovulation to 9 weeks post-con

12 icate that ciliogenesis is initiated in late fetal stages after neuroblast migration, when the mother

13 s retain their undifferentiated state during fetal stage and become adult Leydig cells in post-pubert

14 s subjected to severe food restriction since fetal stage and controls were transferred to a moderatel

15 findings on white matter development at the fetal stage and in infancy as well as DTI applications f

16 the family manifests with early onset at the fetal stage and is associated with neonatal sudden death

17 gamma2 gene segment predominates in the late fetal stages and beyond, in cells destined for the secon

18 To address the function of Pax3 in later fetal stages and in specific adult tissues, we generated

19 ich expressed Myf5 for the first time at the fetal stage ( approximately E15).

20 he development and growth of prostate during fetal stages as well as during prostate carcinogenesis a

21 ollagen content increased from early to late fetal stages but was subsequently unchanged, whereas ela

22 ld cells that typically show the behavior of fetal stage cells.

23 In contrast to embryonic and fetal stages, deletion of the DRR resulted in a pronounc

24 During mid to late fetal stages (embryonic day 15.5-18.5), epithelial diffe

25 The abundance of glutamate at fetal stages, especially in the ventricular zone, is con

26 tor element, inhibit inappropriate embryonic/fetal stage expression of the beta-globin gene.

27 yte-derived signals are required during late fetal stages for continued development and maintenance o

28 In the mid-to-late fetal stage, hepatocytes have few metabolic functions as

29 At later embryonic and fetal stages, however, no defect in MyoD expression was

30 Thymic hypoplasia at later fetal stages in compound mutants was associated with inc

31 monocytes arrive in the lungs during a late fetal stage, maturing to alveolar macrophages through a

32 ternal allele becomes hypermethylated during fetal stages, methylation of the maternal allele begins

33 ly from cells present in the brain since the fetal stage of development, or if there is further input

34 the control of gamma-gene expression in the fetal stage of development.

35 rom embryonic stem cells mimics an embryonic/fetal stage of hematopoiesis.

36 We show here that circumventing the fetal stage of hematopoietic stem cell (HSC) development

37 lly expressed at high levels only during the fetal stage of human development.

38 ose expression is normally restricted to the fetal stage of ventricular development.

39 bud precursors, and heart tube, but by late fetal stages of development, MNF is down-regulated withi

40 tion and cell proliferation at embryonic and fetal stages of heart development.

41 e nucleus represents the early embryonic and fetal stages of lens development, we infer that BHMT exp

42 ired for normal progression through the late fetal stages of lung development that culminate in alveo

43 id progenitor expansion during adult but not fetal stages of murine development.

44 the myogenic determination gene Myf5 during fetal stages of myogenesis.

45 Specifically, new fetal stage-specific contacts were uncovered between a r

46 f DS fetal progenitors, revealing a complex, fetal stage-specific regulatory network.

47 opment, and that they retain this state from fetal stages through meiosis and gametogenesis.

48 orebrain and hindbrain during early and late fetal stages, through an exogenous source of 5-HT which

49 alyze changes in pelvic morphology from late fetal stages to adulthood in a known-age/known-sex foren

50 ha and NRXN1-beta, in prefrontal cortex from fetal stages to aging.

51 systems begin to develop, and finally to the fetal stage where growth and physiological maturation oc

52 in the most caudal part of the hindbrain at fetal stages, where it is maintained until adulthood.

53 tream Vgamma3 and Vgamma4 genes in the early fetal stage, which switches to a preference for rearrang