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1 logy, were not found within the transplanted fetal tissue.
2 gs on chimeric maternal cells that reside in fetal tissue.
3 at myelin disorders is limited at present to fetal tissue.
4 ocortex in paraformaldehyde-fixed postmortem fetal tissue.
5 fection in lymphocytes, placenta, and bovine fetal tissue.
6 in response to increased invasiveness of ape fetal tissue.
7 variants of at least 500 kb in placental or fetal tissue.
8 own's syndrome and control post-mortem human fetal tissue.
9 uman neoplasms because they are derived from fetal tissue.
10 nd mTORC2 signaling in multiple maternal and fetal tissues.
11 blood leukocytes, placenta, lung, aorta, and fetal tissues.
12 f mRNA transcripts in a variety of adult and fetal tissues.
13 tantial reactions were also found in certain fetal tissues.
14 observed in lung, pancreas and spleen and in fetal tissues.
15 ripts by northern-blot analysis of adult and fetal tissues.
16 han those seen in first and second trimester fetal tissues.
17 ceeded those observed in the placenta and in fetal tissues.
18 LCK isoform(s) in both human adult and human fetal tissues.
19 w splice variants of Bright were observed in fetal tissues.
20 is ubiquitously expressed in both adult and fetal tissues.
21 subunit, was investigated in human adult and fetal tissues.
22 ntion of a maternal immune attack on foreign fetal tissues.
23 and dentate gyrus in the adult brain and to fetal tissues.
24 xpression and is greatly enhanced in various fetal tissues.
25 ze all-trans-retinol and steroid alcohols in fetal tissues.
26 ent of these cells in second trimester human fetal tissues.
27 els of viral RNA in maternal, placental, and fetal tissues.
28 ity and increases infection of placental and fetal tissues.
29 r ACD cells, contribute to the allantois and fetal tissues.
30 the control of metabolism and maturation of fetal tissues.
31 t for BH(4) compared with maternal and other fetal tissues.
32 dinated interactions between endometrial and fetal tissues.
33 es the loss of Rb in both extraembryonic and fetal tissues.
34 d more rapid uptake and clearance than other fetal tissues.
35 d on embryonic stem cells derived from human fetal tissues.
36 r mechanisms of the immune response to human fetal tissues.
37 and has a tendency for higher expression in fetal tissues.
38 region that is differentially methylated in fetal tissues.
39 delicate balance of active hormone levels in fetal tissues?
40 the fetoplacental interface and in numerous fetal tissues, 2) ex vivo, the placenta released high am
43 7me3) was lower in the Zfp423 promoter of OB fetal tissue, accompanied by reduced binding of enhancer
46 A number of exploratory studies using human fetal tissue allografts have suggested that transplantat
47 f-1L have similar distributions in adult and fetal tissues, although apaf-1L transcripts are more abu
48 gnant women may result in acute infection of fetal tissue and brain tissue, causing microcephaly and
49 ry cilium in developing podocytes from human fetal tissue and in undifferentiated cultured podocytes.
50 in adult skeletal muscle but very low in the fetal tissue and in undifferentiated skeletal muscle cel
51 human samples, including 17 adult tissues, 7 fetal tissues and 6 purified primary haematopoietic cell
54 ntigen-presenting cells can be identified in fetal tissues and are related to adult populations of an
55 and KCNQ1OT1, with monoallelic expression in fetal tissues and biallelic expression in adult samples.
59 P-C in reproductive, placental, and selected fetal tissues and fetal fluid, PrP-Sc was detected only
62 ion is based upon histopathologic changes in fetal tissues and identification of tissue parasites by
64 ase isozyme M2 (hPKM2) is expressed in early fetal tissues and is progressively replaced by the other
66 endogenous CEA promoter; CEA is expressed in fetal tissues and normal gastrointestinal tissues, and C
67 ion of PCOLCE RNA in various human adult and fetal tissues and of Pcolce RNA at various stages of mou
69 tic domains, are expressed in neoplastic and fetal tissues and potentially have a role in embryonic d
70 The even distribution of PDE7A mRNA among fetal tissues and the relative abundance of its two mRNA
72 decidual T cells proliferated in response to fetal tissue, and depletion of T regulatory cells led to
74 Gadolinium chelate levels in the placenta, fetal tissues, and amniotic fluid were obtained by using
77 n thymus, bone marrow, fetal liver and other fetal tissues, and in all proliferating cell lines teste
80 eptor are widely expressed in both adult and fetal tissues, and recent gene-targeting and disruption
81 progenitor B (pro-B), pre-B, and B cells in fetal tissues, and thus supports the idea of a multifoca
82 transcripts were detected in human adult and fetal tissues, and very high transcript levels were foun
83 Despite profound placental abnormalities, fetal tissues appeared remarkably normal, suggesting tha
88 for the first time its distribution in human fetal tissues, as well as its expression in adult kidney
89 sion of prolactin receptors (PRLRs) in human fetal tissues at 7.5-14 wk of gestation and in tissues o
91 c expression patterns; they are expressed in fetal tissues, breast, and prostate, and in many cancers
92 n mRNA-binding protein that is abundant in a fetal tissue but either absent from or scarce in adult t
93 l tissues, and Nox4 is expressed in not only fetal tissues, but also kidney, placenta and glioblastom
94 ts are expressed in brain and lung tumor and fetal tissues, but are generally absent from normal adul
96 ocytosed nonspecifically and then carried to fetal tissues by a pH gradient from acidic endosomes to
97 By contrast, early in gestation, injured fetal tissues can be completely recreated, without fibro
99 d CCR5 on T cells and their progenitors from fetal tissue, cord blood, SCID-hu Thy/Liv mice, and adul
100 roducts were observed in matching normal and fetal tissues; deleted cDNA sequence revealed canonical
101 nt mice and ICP-MS analyses of placental and fetal tissue demonstrated undetectably low transplacenta
102 s higher than that previously observed using fetal-tissue-derived OPCs, and no tumors from these graf
104 s is developmentally lethal, indicating that fetal tissue development is tightly controlled by miRNAs
107 spite intimate juxtaposition of maternal and fetal tissues during mammalian pregnancy, reciprocal mig
110 family members, ADAMTS9 is expressed in all fetal tissues examined as well as some adult tissues.
118 ed because it might be more practicable than fetal tissue for the purpose of transplantation to prima
123 udy, time-dependent (3 h-11 days) changes in fetal tissue gene expression in a rat model of in utero
124 within the female reproductive tract and in fetal tissues harvested from CWD experimentally and natu
125 isease and the difficulty in obtaining human fetal tissue has generated interest in finding correspon
126 cultures of NPCs derived from human iPSCs or fetal tissue have similar characteristics, although they
127 e in a neonatal spinal environment or within fetal tissue implants, especially because previous work
133 cript of PKDL is expressed at high levels in fetal tissues, including kidney and liver, and down-regu
134 n of survivin in several apoptosis-regulated fetal tissues, including the stem cell layer of stratifi
135 ndings were obtained in 12- to 18-week human fetal tissue, indicating a highly restricted pattern of
139 ic expression in female hybrid placental and fetal tissues is negatively correlated with the other X-
140 NMHC II-A and II-B, the mRNA level in human fetal tissues is substantially lower than in adult tissu
141 be ubiquitously expressed in human adult and fetal tissues, is highly related to the DRPLA gene, in w
144 med expression in both human adult and human fetal tissues (lung, liver, brain, and kidney) and in hu
145 aternal unresponsiveness to HLA-G-expressing fetal tissues may be shaped in the thymus by a previousl
146 cell polarity (PCP) pathway is required for fetal tissue morphogenesis as well as for maintenance of
150 mprinting tumors was also observed in normal fetal tissues of 6 12 weeks gestation, suggesting a simi
152 olated from the placenta, amniotic fluid, or fetal tissues only from rats infected prior to breeding
153 n embryos (human embryonic stem cells), from fetal tissue or from adult sources (bone marrow, fat, sk
155 n embryos (human embryonic stem cells), from fetal tissue, or from adult sources (bone marrow, fat, s
156 certain extrahepatic tissues (in addition to fetal tissues), our data raise the possibility that CYP3
157 on analysis revealed two active promoters in fetal tissues, P2 and P3, whereas P3 was the only promot
159 ive CSF-1 crossed the placenta and peaked in fetal tissue, placenta, and serum 10 minutes after injec
162 ve tract and target Neu5Gc-positive sperm or fetal tissues, reducing reproductive compatibility.
163 quantitative distribution of VHL mRNA within fetal tissues reflected that seen by in situ hybridizati
164 iptional regulatory genes that might mediate fetal tissue regeneration, we surveyed homeobox gene exp
165 HOXB13 expression was decreased in wounded fetal tissue relative to unwounded fetal controls or wou
168 hern hybridization analysis revealed that in fetal tissue RTVP-1 RNA was detected only in the kidney,
169 amples, 188 chorionic villus samples, and 37 fetal tissue samples were tested; the amplification fail
170 titutively in extrahepatic organs, including fetal tissue samples, and is highly inducible in various
171 eir expression, with exception of testis and fetal tissues, seems to be restricted to tumor cells onl
175 mRNA broadly distributed in normal adult and fetal tissues, structurally distinct from the 1.9-kb Sur
177 both IGF2 and H19 was observed in all other fetal tissues, suggesting a tissue-specific LOI in the c
178 on in carcinomas as well as normal adult and fetal tissues suggests an important functional role for
180 on of the sedlin gene in all human adult and fetal tissues tested, with the highest levels in kidney,
186 ng LCM of epithelial cells from fresh frozen fetal tissue that enables quantitative analyses of micro
187 ressed ubiquitously in the limited number of fetal tissues that were tested, but is selectively expre
189 t epithelial (RPE) cells isolated from human fetal tissue, three separate studies failed with various
191 els in the general population, children, and fetal tissue to establish background levels and the risk
192 programs remarkably similar to those of the fetal tissue to organize into cerebral cortex-like regio
193 es conducted directly in human embryonic and fetal tissue to provide an overview of what is known abo
195 rovide evidence for the ability of different fetal tissues to support the development of gammadelta T
197 genes that are unmethylated in 11 different fetal tissue types, we show that increases in DNA methyl
202 e maternal plasma samples and homogenates of fetal tissues were determined by high-performance liquid
203 opism towards reproductive and embryonic and fetal tissues where exposure to a single alpha particle
204 wed monoallelic expression in both adult and fetal tissue, whereas PHLDA2, SLC22A18, and SLC22A18AS w
205 erfamily are expressed in tumors, testis and fetal tissues, which has been described as a cancer/test
206 els of viral RNA in maternal, placental, and fetal tissues, which resulted in protection against plac
207 have now extended these studies by replacing fetal tissue with neonatal pig thymic and hematopoietic
208 ed in a variety of human and mouse adult and fetal tissues with substantial expression levels in the
210 hest expression levels of FBN3 were found in fetal tissues, with only low levels in postnatal tissues
211 nces in transcript abundance among adult and fetal tissues, with predominant expression in liver, kid
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