ライフサイエンスコーパス: fibrinoid

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1 ndovascular trophoblasts (vEVTs) embedded in fibrinoid.

2 t also reemphasizes the relationship between fibrinoid and miliary aneurysm formation.

3 Rejection with fibrinoid arterial necrosis (4% of biopsies) did not res

4 Plasmin produced fibrinoid changes in the blood vessels at 24 h, coincidi

5 ive abnormalities such as lipohyalinosis and fibrinoid changes within penetrating artery branches vis

6 Associated damage included extensive fibrinoid deposits, fibrosis, avascular villi, and edema

7 eks, wild-type (WT) mice developed extensive fibrinoid hepatic venous thrombi and biochemical evidenc

8 uscle cells (VSMCs) are lost and replaced by fibrinoid, incorporating extravillous trophoblast (EVT)

9 stabilizing uteroplacental attachment at the fibrinoid layer found at the fetal-maternal junction.

10 trophy and became fibrotic; some also showed fibrinoid necrosis and a mixed inflammatory cell infiltr

11 ity of CAA-associated vasculopathic changes (fibrinoid necrosis and concentric splitting of the wall)

12 nucleated giant cells (MGCs), MGC death, and fibrinoid necrosis and fibrosis.

13 5% versus 9%; severe ATI, 75% versus 9%; and fibrinoid necrosis in glomeruli, 20% versus 0%, or arter

14 s versus 40% (2 of 5) for those who also had fibrinoid necrosis of arteries.

15 leads to increased vascular permeability and fibrinoid necrosis of blood vessel walls.

16 (3) Type III: arterial fibrinoid necrosis or transmural inflammation with or wi

17 ograft, intimal edema, matrix deposition and fibrinoid necrosis were specifically presented in the xe

18 c capillary involvement) and AHR 2 (arterial fibrinoid necrosis).

19 us, specific arterial lesions (endarteritis, fibrinoid necrosis, activated endothelial cells, mononuc

20 nuated renal injury (albuminuria, glomerular fibrinoid necrosis, and crescent formation), but mAbs to

21 o endothelial cells, endothelial activation, fibrinoid necrosis, foam cells, and intimal fibrosis.

22 arcts, microhaemorrhage, larger haemorrhage, fibrinoid necrosis, microaneurysms, perivascular space d

23 within the wall of an arteriole at a site of fibrinoid necrosis.

24 ealed leukocytoclastic vasculitis with focal fibrinoid necrosis.

25 eritis, 28% with endarteritis, and 100% with fibrinoid necrosis.

26 A fibrinoid substance of unclear origin often covered the

27 Histopathology showed fibrinoid vascular necrosis, rare amastigote nests withi

28 Large fibrinoids with avascular villi, edema, and inflammation

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