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1 In one mass, repeat LCNB findings showed a fibroadenoma.
2 .91, 1.00) for triple-negative cancer versus fibroadenoma.
3 s between triple-negative cancers and benign fibroadenomas.
4 med to noninvasively coagulate benign breast fibroadenomas.
5 f 5 of 11 infiltrating carcinomas and 2 of 6 fibroadenomas.
6 ypes with mean ADCs above the threshold were fibroadenoma ([1.94 +/- 0.38 {standard deviation}] x 10(
7 y or aspiration of all lesions, revealing 15 fibroadenomas, 12 carcinomas, six fibrocystic nodules, a
8 odel was used to correctly classify 10 of 38 fibroadenomas (26%) and three of seven stromal fibroses
10 were identified within nine of 14 enhancing fibroadenomas (64%) and appeared to correlate with colla
15 ly observed MED12 and RARA mutations in both fibroadenomas and phyllodes tumors, emphasizing the impo
16 ED12 mutations have been frequently found in fibroadenomas and phyllodes tumors, the landscapes of ge
17 apillomas, sclerosing adenosis, radial scar, fibroadenoma, and areas of atypical ductal hyperplasia.
18 ish normal tissue, fibrocystic change (FCC), fibroadenoma, and breast cancer, in the absence and pres
20 n breast tissue (normal, fibrocystic change, fibroadenoma, and infiltrating carcinoma) from 58 patien
25 four of six benign papillomas and one of two fibroadenomas as circumscribed, enhancing subareolar mas
27 ions include lobular and ductal hyperplasia, fibroadenoma, cystic expansions, and papillary adenomas.
30 vasive breast cancers, 7 mastopathias, and 2 fibroadenomas demonstrates that a highly sensitive, spec
31 patients compared with that in patients with fibroadenoma (FIBma) or healthy individuals, and the pos
32 ted for age, menopausal status, soy protein, fibroadenoma history, family breast cancer, physical act
34 iation of PTEN mutation and cancer or breast fibroadenoma in any given CS, BRR or BRR/CS overlap fami
40 pear to reflect intrinsic growth patterns of fibroadenomas, may provide a more reliable basis for dis
42 capture microdissection, we show that MED12 fibroadenoma mutations are present in stromal but not ep
44 ich produced neoplastic phenotype, including fibroadenomas of the breast and salivary gland adenomas.
45 iated mesenchymal tumors, including lipomas, fibroadenomas of the breast, salivary gland adenomas, an
48 o specific categories of normal tissue, FCC, fibroadenoma, or breast cancer (with and without microca
50 were statistically significantly darker than fibroadenomas (P < .005) and substantially larger on the
51 Power Doppler US depicted no vessels in 21 fibroadenomas, peripheral vessels in 15, and penetrating
52 epared from human mammary adenocarcinoma and fibroadenoma predominantly catalyze the metabolic 4-hydr
54 ion profiling of MED12-mutated and wild-type fibroadenomas revealed that MED12 mutations are associat
56 age findings of normal breast tissue, benign fibroadenomas (six of six lesions), and malignant lesion
59 6%) of the 44 masses were cysts; three (7%), fibroadenomas; two (4%), infiltrating ductal carcinomas;
66 tinguishable from normal tissue in all three fibroadenomas, were indistinguishable from normal findin
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