ライフサイエンスコーパス: fibroblast growth factor

1 oss of kinase activity upon stimulation with fibroblast growth factor.

2 larization of the graft bed by agarose-basic fibroblast growth factor.

3 ulated autophagy through IL6, IL8, and basic fibroblast growth factor.

4 esterol levels under normoxia might regulate fibroblast growth factor 1 (FGF-1) gene expression which

5 Recently, we have identified fibroblast growth factor 1 (FGF1) as a target of nuclear

6 The fibroblast growth factor 1 (FGF1) gene is expressed prim

7 otein (marker of mature lipofibroblasts) and fibroblast growth factor 10 (previously shown to identif

8 Importantly, fibroblast growth factor-10 (FGF-10) was markedly suppre

9 miR-504 resides within the fibroblast growth factor 13 (FGF13) gene, which is overe

10 t phosphorylation at these two sites impairs fibroblast growth factor 13 (FGF13)-dependent regulation

11 ary protein of voltage-gated Na(+) channels, fibroblast growth factor 13 (Fgf13).

12 Recent data shows that fibroblast growth factor 14 (FGF14) binds to and control

13 by a number of accessory proteins including fibroblast growth factor 14 (FGF14), a member of the int

14 Here, we show that fibroblast growth factor 14 (FGF14), previously describe

15 ations in FGF14, which encodes intracellular fibroblast growth factor 14 (iFGF14), have been linked t

16 st fexaramine (Fex) robustly induces enteric fibroblast growth factor 15 (FGF15), leading to alterati

17 ctivation would be sufficient to restore the fibroblast growth factor 15 (FGF15)/cholesterol-7alpha-h

18 ed expression of FXR target genes, including fibroblast growth factor 15, and unexpectedly Tgr5 and p

19 atic BA synthesis, potentially through ileal fibroblast growth factor 15- and Fxr-mediated inhibition

20 igh content cell-based screen, we identified fibroblast growth factor 16 (FGF16) as a potent inducer

21 Fibroblast growth factor 19 (FGF19) is an important post

22 Fibroblast Growth Factor 19 (FGF19) is one of the most f

23 ting bile acid metabolism by the gut hormone fibroblast growth factor 19 (FGF19) may represent a nove

24 Effects of fibroblast growth factor 19 (FGF19) on hepatic transport

25 eptor and TGR5, the BA-induced gut hormones, fibroblast growth factor 19 and glucagon-like peptide 1,

26 Fibroblast growth factor-19 (FGF19) is an intestinal hor

27 e the safety and effectiveness of 3 doses of fibroblast growth factor 2 (FGF-2) when combined with a

28 of a number of HS-binding proteins including fibroblast growth factor 2 (FGF-2), and the chemokines C

29 Human fibroblast growth factor 2 (FGF2) and insulin-like growt

30 al concentrations of the neurotrophic factor fibroblast growth factor 2 (FGF2) are negatively associa

31 peptidase E, CPE), concomitant with enhanced fibroblast growth factor 2 (FGF2) expression, and an inc

32 Fibroblast growth factor 2 (FGF2) has been shown to inte

33 Fibroblast growth factor 2 (FGF2) has previously been im

34 Fibroblast growth factor 2 (FGF2) is a key signaling mol

35 Fibroblast growth factor 2 (FGF2) is a potent mitogen pr

36 Fibroblast growth factor 2 (FGF2) is crucial for the dev

37 oenvironmental proteins FLT3 ligand (FL) and fibroblast growth factor 2 (FGF2) protect FLT3-ITD+ MOLM

38 onstrate that loss of NG2 glial secretion of fibroblast growth factor 2 (FGF2) suffices to induce the

39 ted neutrophils expressed substantially more fibroblast growth factor 2 (FGF2) than naive neutrophils

40 Despite strong evidence linking fibroblast growth factor 2 (FGF2) with anxiety and depre

41 her show that beta1-integrin cooperates with fibroblast growth factor 2 (Fgf2), a potent growth facto

42 ere, we focus on unconventional secretion of fibroblast growth factor 2 (FGF2), a secretory mechanism

43 In turn, CAFs produced high levels of fibroblast growth factor 2 (FGF2), initiating a signalin

44 e Na/K-ATPase in unconventional secretion of fibroblast growth factor 2 (FGF2).

45 ascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2).

46 we established a role for ASMC-derived basic fibroblast growth factor 2 (FGF2b) and FGF receptor (FGF

47 ne morphogenetic protein 7/human recombinant fibroblast growth factor 2 for 24 hours before induction

48 DF differentiation, via multiple cAMP and/or fibroblast growth factor 2 or basic FGF (FGF2)-dependent

49 s showed abnormal HS composition and altered fibroblast growth factor 2 signaling, which was rescued

50 of tumor necrosis factor alpha, IL-8, IL-10, fibroblast growth factor 2, and IL-7 remained higher.

51 gamma-induced protein 10, IL-4, IL-9, IL-10, fibroblast growth factor 2, IL-7, IL-15, and transformin

52 variation, we observed changes in netrin 4, fibroblast growth factor 2, tenascin C, collagen 1, mepr

53 viously reported that growth factors such as fibroblast growth factor-2 (FGF-2) and bone morphogeneti

54 -2 (COX-2), interleukin-1beta (IL-1beta) and fibroblast growth factor-2 (FGF-2), which led to a marke

55 In this study, DPCs were treated with fibroblast growth factor-2 (FGF2) for 5-6 consecutive se

56 fference in VAT activity was associated with fibroblast growth factor-2 (FGF2) levels.

57 ic protein-4 and inhibitors of activin A and fibroblast growth factor-2 signaling (BAP treatment).

58 in-6 and monocyte chemoattractant protein-1, fibroblast growth factor-2, and the potent vasoconstrict

59 demonstrated increased expression levels of fibroblast growth factor-2, transforming growth factor-b

60 ) activity and reduced hepatic PPARalpha and fibroblast growth factor 21 (FGF21) expression and lower

61 lve activation of ER stress and secretion of fibroblast growth factor 21 (FGF21) from skeletal muscle

62 Fibroblast growth factor 21 (FGF21) has been identified

63 Fibroblast growth factor 21 (Fgf21) has emerged as a pot

64 Fibroblast growth factor 21 (FGF21) has emerged as an im

65 1), but required expression of liver-derived fibroblast growth factor 21 (FGF21) in both lean and obe

66 of the lipid- and glucose-modulating hormone fibroblast growth factor 21 (FGF21) in the molecular mec

67 Fibroblast growth factor 21 (FGF21) is a hepatokine that

68 Fibroblast growth factor 21 (FGF21) is a peptide hormone

69 Fibroblast growth factor 21 (FGF21) is an important regu

70 Fibroblast growth factor 21 (FGF21) was shown to improve

71 virus expressing short hairpin RNA targeting fibroblast growth factor 21 (FGF21) were used to determi

72 show that the prolongevity ketogenic hormone fibroblast growth factor 21 (FGF21), a member of the end

73 Fibroblast growth factor 21 (FGF21), a peptide hormone w

74 ine metabolite 3-hydroxyisobutyrate (3-HIB), fibroblast growth factor 21 (FGF21), adiponectin, and no

75 es, markers of inflammation, serum levels of fibroblast growth factor 21 (FGF21), and activation of s

76 ses a series of secreted proteins, including fibroblast growth factor 21 (FGF21), bone morphogenetic

77 nsulin resistance with increased circulating fibroblast growth factor 21 (FGF21), elevated Fgf21 mRNA

78 expressed increased levels of E-cadherin and fibroblast growth factor 21 (FGF21), targets of sirtuin-

79 duced expression and secretion of a myokine, fibroblast growth factor 21 (FGF21), that stimulates ene

80 sis in BAT along with enhanced expression of fibroblast growth factor 21 (FGF21).

81 these effects require the metabolic hormone fibroblast growth factor 21 (FGF21).

82 Human fibroblast growth factor 21 (hFGF21) has been characteri

83 ng hyperglycemia and include augmentation of fibroblast growth factor 21 and glucagon-like peptide 1.

84 glucocorticoid inhibitors, and analogues of fibroblast growth factor 21 are being considered as pote

85 iver weight with fasting and increased liver fibroblast growth factor 21 expression and intact ketoge

86 Interestingly, fasting induction of fibroblast growth factor 21 in liver was attenuated.

87 A low BCAA diet transiently induces FGF21 (fibroblast growth factor 21) and increases energy expend

88 the energy balance regulating hormone FGF21 (fibroblast growth factor 21).

89 ncrease hepatic transcription and release of fibroblast growth factor 21, or improve insulin sensitiv

90 MCAO markedly downregulated fibroblast growth factor-21 (FGF-21) and TubA significan

91 Fibroblast growth factor-21 (FGF21) is closely related t

92 n1 and decreased p62 protein levels, induced fibroblast growth factor-21 expression, and corrected HF

93 e structure crucial for learning and memory, fibroblast growth factor 22 (FGF22) and FGF7 specificall

94 Because fibroblast growth factor 22 (FGF22), a heparan sulfate b

95 Here we show that fibroblast growth factor 22 (FGF22), a target-derived pr

96 st growth factor 23 (iFGF23) into C-terminal fibroblast growth factor 23 (cFGF23), elevated levels of

97 Plasma fibroblast growth factor 23 (FGF-23) concentration, plas

98 roid hormone (PTH), 1,25(OH)2D3 (1,25D), and fibroblast growth factor 23 (FGF-23) maintains mineral h

99 ip between repeated measures of vitamin D or fibroblast growth factor 23 (FGF23) and infectious and c

100 sphatemic states arising from high levels of fibroblast growth factor 23 (FGF23) are also associated

101 Levels of fibroblast growth factor 23 (FGF23) are elevated in chro

102 s of the parathyroid hormone (PTH)-vitamin D-fibroblast growth factor 23 (FGF23) axis, creatinine, an

103 Fibroblast growth factor 23 (FGF23) decreases 1,25(OH)2D

104 The discovery of fibroblast growth factor 23 (FGF23) has provided a more

105 asma levels of the osteocyte-derived hormone fibroblast growth factor 23 (FGF23) have emerged as a po

106 Circulating levels of fibroblast growth factor 23 (FGF23) increase during the

107 Circulating levels of bone-derived fibroblast growth factor 23 (FGF23) increase early durin

108 iations between elevated serum phosphate and fibroblast growth factor 23 (FGF23) levels and risks of

109 Elevated fibroblast growth factor 23 (FGF23) levels, measured at

110 unique basal and parathyroid hormone (PTH)-, fibroblast growth factor 23 (FGF23)-, and 1,25(OH)2D3-me

111 D promotes production and cleavage of intact fibroblast growth factor 23 (iFGF23) into C-terminal fib

112 e blood of cKO mice had an elevated level of fibroblast growth factor 23 and reduced level of phospho

113 Levels of intact fibroblast growth factor 23 decreased rapidly during the

114 rminal pro-B-type natriuretic peptide level, fibroblast growth factor 23 level, estimated glomerular

115 The renal extraction of fibroblast growth factor 23 was, on average, lower than

116 ns of certain molecules, such as phosphates, fibroblast growth factor 23, parathyroid hormone, sclero

117 pass renal clearance of parathyroid hormone, fibroblast growth factor 23, vitamin D metabolites, and

118 calcemia, changes in parathyroid hormone, or fibroblast growth factor 23.

119 The bone-derived hormone fibroblast growth factor-23 (FGF-23) activates complexes

120 hrough cross-talk between RAAS and vitamin D-fibroblast growth factor-23 (FGF-23)-Klotho pathways.

121 n (mKL) and recognized as the coreceptor for fibroblast growth factor-23 (FGF23) and a circulating so

122 low serum vitamin D levels, increased serum fibroblast growth factor-23 (Fgf23), and osteomalacia.

123 baseline (1990-1992) serum levels of intact fibroblast growth factor-23 and incident ESRD in 13,448

124 High-fibroblast growth factor-23 concentration positively cor

125 artery disease, diabetes, dialysis vintage, fibroblast growth factor-23 concentration, and age, wher

126 Serum fibroblast growth factor-23 enhanced (P < 0.01), whereas

127 Fibroblast growth factor-23 is a bone-derived hormone th

128 ad range of kidney function, higher baseline fibroblast growth factor-23 levels were associated with

129 rus levels, circulating phosphaturic hormone fibroblast growth factor-23 levels, and heart mass.

130 diator of pathologic cardiac remodeling, and fibroblast growth factor-23 may contribute to cardiac re

131 d markers of mineral metabolism, the highest fibroblast growth factor-23 quintile (>54.6 pg/ml) compa

132 ake stimulates parathyroid hormone (PTH) and fibroblast growth factor-23 secretion, increasing phosph

133 ighted the effect of phosphate depletion and fibroblast growth factor-23 suppression on the developme

134 Fibroblast growth factor-23, 1,25-(OH)2-vitamin D3, and

135 ion did not affect serum calcium, phosphate, fibroblast growth factor-23, or alkaline phosphatase lev

136 ced intact parathyroid hormone and increased fibroblast growth factor-23, with a trend to increase in

137 imed to assess the effect of paricalcitol on fibroblast growth factor-23/KLOTHO axis in renal transpl

138 Fibroblast growth factor 4 (FGF4) is the key signal driv

139 otein A-II and cortisol levels in plasma and fibroblast growth factor 4, heart-type fatty acid bindin

140 retion of several protumor cytokines such as fibroblast growth factor-4, CX3CL1/fractalkine, neurotro

141 Whereas dexamethasone and recombinant human fibroblast growth factor 7 promoted expansion of Foxn1(+

142 iated by altered interactions between HS and fibroblast growth factor-7 (FGF-7).

143 a precise and dynamic expression pattern of fibroblast growth factor 8 (Fgf8) in the HAA anlage, whi

144 Fibroblast growth factor 8 (FGF8) is a member of a large

145 terior to posterior (AP) patterning, whereas fibroblast growth factor 8 (Fgf8) is produced by the api

146 ly, excess RA signaling negatively regulates fibroblast growth factor 8a (fgf8a) expression and posit

147 Interestingly, while miR-155-5p decreased fibroblast growth factor 9 (FGF9) expression in a lucife

148 ads, sex-determining region Y (SRY) triggers fibroblast growth factor 9 (FGF9) expression in somatic

149 lls, induction of cellular HD5 expression by fibroblast growth factor 9 (FGF9) significantly inhibite

150 Fibroblast growth factor 9 (FGF9), vascular endothelial

151 ssociated with increased glial expression of fibroblast growth factor 9 (FGF9), which we demonstrate

152 Fibroblast growth factor 9, a mesenchyme-targeting growt

153 treated the graft/host interface with acidic fibroblast growth factor (aFGF) and chondroitinase ABC (

154 Acidic fibroblast growth factor (aFGF) has shown the great pote

155 We uncovered 16 signaling pathways (such as fibroblast growth factor and Eph/ephrin pathways).

156 scriptional factors, such as sonic hedgehog, fibroblast growth factors, and wingless-int (Wnt)/beta-c

157 Neurotrophic factors, such as basic fibroblast growth factor (bFGF or FGF2), are necessary f

158 embryonic stem (ES) cells cultured in basic fibroblast growth factor (bFGF) and activin A develop as

159 ined the optimal concentration of both basic fibroblast growth factor (bFGF) and neuregulin-1 beta 1

160 Of all factors tested, the basic fibroblast growth factor (bFGF) had the most significant

161 <0.01) and the angiogenesis induced by basic fibroblast growth factor (bFGF) in a Matrigel plug assay

162 Basic fibroblast growth factor (bFGF) may protect stroke patie

163 By basic fibroblast growth factor (bFGF) or vascular endothelial

164 Basic fibroblast growth factor (bFGF) released from a nanostru

165 Cytokine array analysis showed that basic fibroblast growth factor (bFGF) was downregulated in MDA

166 tor 1alpha (Hif-1alpha), Snail1, Slug, basic fibroblast growth factor (bFgf), and retinaldehyde dehyd

167 e fourth ventricles and, together with basic fibroblast growth factor (bFGF), elicited subsequent neu

168 (JW74), in the presence or absence of basic fibroblast growth factor (bFGF).

169 at muscle fibers secrete and concentrate the fibroblast growth factor binding protein 1 (FGFBP1) at N

170 at muscle fibers secrete and concentrate the fibroblast growth factor binding protein 1 (FGFBP1) at N

171 Fibroblast growth factor-binding protein 1 (FGFBP1) is a

172 id hormone-related protein, Indian hedgehog, fibroblast growth factor, bone morphogenetic protein, an

173 sion, apoptosis, and regulation of Hedgehog, fibroblast growth factor, bone morphogenetic protein, an

174 ietic KDR(+)CD235a(-) mesoderm in a WNT- and fibroblast growth factor-dependent manner.

175 d for a detailed structure-activity study of fibroblast growth factor (FGF) 1 and FGF2 signaling thro

176 he fat-derived adiponectin and ileum-derived fibroblast growth factor (FGF) 15.

177 Fibroblast growth factor (FGF) 23 is a phosphaturic horm

178 uble protein, functions as a co-receptor for Fibroblast Growth Factor (FGF) 23, a known pro-inflammat

179 FE is then ventralized by applying Wnt, BMP, fibroblast growth factor (FGF) and retinoic acid (RA) si

180 dies using animal models have implicated the fibroblast growth factor (FGF) family in affect regulati

181 The fibroblast growth factor (Fgf) family of ligands and rec

182 Members of the fibroblast growth factor (FGF) family play essential and

183 Members of the fibroblast growth factor (FGF) family play essential and

184 iate into lens fibers (LFs) in response to a fibroblast growth factor (FGF) gradient.

185 The fibroblast growth factor (FGF) is a potent angiogenic fa

186 Lapsyn works in concert with the fibroblast growth factor (FGF) pathway to promote branch

187 is necessary for pouch outpocketing via the Fibroblast growth factor (Fgf) pathway.

188 t Wnt, bone morphogenetic protein (BMP), and fibroblast growth factor (FGF) pathways are activated at

189 lopment depends on Decapentaplegic (Dpp) and Fibroblast growth factor (FGF) proteins produced by the

190 Activating mutations in fibroblast growth factor (FGF) receptor 3 and inactivati

191 These treatments lead to activation of the fibroblast growth factor (FGF) receptor, phospholipase C

192 Fibroblast growth factor (Fgf) receptors have a recogniz

193 ing response elicited by activation of brain fibroblast growth factor (FGF) receptors, we explored th

194 neural plate of Ciona embryos in response to fibroblast growth factor (FGF) signaling and a localized

195 ival of NPCs have been identified, including fibroblast growth factor (FGF) signaling and the transcr

196 Although restoring Fibroblast growth factor (FGF) signaling can partially r

197 Fibroblast growth factor (Fgf) signaling governs multipl

198 Fibroblast growth factor (FGF) signaling is an essential

199 Fibroblast growth factor (FGF) signaling is important fo

200 Fibroblast growth factor (FGF) signaling is required for

201 Fibroblast growth factor (FGF) signaling pathways are es

202 Moreover, the Hippo/YAP and the fibroblast growth factor (FGF) signaling pathways formed

203 ransforming growth factor beta (TGFbeta) and fibroblast growth factor (FGF) signaling pathways play i

204 Fibroblast Growth Factor (FGF) signaling promotes self-r

205 Fibroblast growth factor (FGF) signaling regulates a mul

206 cription of glycolytic enzymes downstream of fibroblast growth factor (FGF) signaling.

207 (TS) cells, Esrrb is a downstream target of fibroblast growth factor (Fgf) signalling and is critica

208 Fibroblast growth factor (FGF) signalling in the distal

209 other signals in the hindbrain and show that fibroblast growth factor (FGF) signalling regulates FBM

210 We found that the loss of an intracellular fibroblast growth factor (FGF), FGF13, in the mouse DRG

211 ollicle patterning, identifying a network of fibroblast growth factor (FGF), wingless-related integra

212 ; the human hepatocytes do not recognize the fibroblast growth factor (FGF)-15 produced by mouse inte

213 rticipating in phosphate homeostasis include fibroblast growth factor (FGF)-23, a bone-derived hormon

214 a occurs despite a marked elevation in serum fibroblast growth factor (FGF)-23.

215 In Ciona, matrix adhesion polarizes fibroblast growth factor (FGF)-dependent heart progenito

216 Amongst the best studied are the fibroblast growth factor (FGF)-ERK1/2 pathway, PI3K-AKT,

217 Fibroblast growth factor (FGF)-extracellular signal-regu

218 (AER), whereas an alternative Homeobox (Hox)-Fibroblast growth factor (Fgf)-Wingless type MMTV integr

219 This defect was normalized by addition of fibroblast growth factor (FGF).

220 ockout of FXR were given daily injections of fibroblast growth factor (FGF)19.

221 In addition, fibroblast growth factor (FGF)21 has effects similar to

222 n robustly increased hepatic and circulating fibroblast growth factor (Fgf)21 levels.

223 Here, we show that Fibroblast Growth Factors (FGF) 9 and 20, which are expr

224 Fibroblast growth factor (FGF1 and FGF2), but not vascul

225 An intestinal fibroblast growth factor (FGF19) protein expression was

226 reviously implicated in bHR/bLR differences: fibroblast growth factor (FGF2) and the dopamine D2 rece

227 ons included, but were not limited to, basic fibroblast growth factor (FGF2), heparin-binding EGF-lik

228 trong evidence for the physiological role of fibroblast growth factors (FGFs) and FGF receptors (FGFR

229 Fibroblast growth factors (FGFs) and FGF receptors (FGFR

230 Fibroblast growth factors (FGFs) are a family of essenti

231 Fibroblast growth factors (FGFs) are required to specify

232 Fibroblast growth factors (fgfs) are widely believed to

233 lism, little is understood about the role of fibroblast growth factors (FGFs) in this context.

234 Fibroblast growth factors (FGFs) play a central role in

235 ation but also exploiting macrophage-derived fibroblast growth factors (FGFs).

236 nt relies on sodium channel (Nav)-associated fibroblast growth factor homologous factor (FHF) protein

237 Here we show that mice lacking fibroblast growth factor homologous factor 2 (FHF2) have

238 Fibroblast growth factor homologous factors (FHFs) are a

239 vascular endothelial growth factor and basic fibroblast growth factor in these osteogenic progenitor

240 njection of Marimastat also suppressed basic fibroblast growth factor-induced endothelial-mesenchymal

241 (TNF)-like weak inducer of apoptosis (TWEAK)/fibroblast growth factor-inducible 14 (Fn14) system redu

242 ceptor superfamily member 12a, also known as fibroblast growth factor-inducible 14 (Fn14), by these c

243 poptosis (TWEAK), acts through its receptor, fibroblast growth factor-inducible 14 (Fn14), to mediate

244 Fibroblast growth factor-inducible 14 (Fn14; TNFRSF12A)

245 Fibroblast growth factor inhibition blocks tissue conver

246 ermal growth factor (P = 1.18 x 10(-31)) and fibroblast growth factor (P = 2.47 x 10(-31)) signaling

247 ithelial cells resulted in a higher level of fibroblast growth factor receptor (FGFR) activation and

248 athway analysis of these 69 genes implicated fibroblast growth factor receptor (FGFR) as a key regula

249 n of Frs2alpha (Six2creFrs2alphaKO), a major fibroblast growth factor receptor (Fgfr) docking protein

250 The fibroblast growth factor receptor (FGFR) family of recep

251 e rates have been low for biomarker-directed fibroblast growth factor receptor (FGFR) inhibitor thera

252 e the dimer stabilities of three full-length fibroblast growth factor receptor (FGFR) mutants harbori

253 identified cross-talk between the Hippo and fibroblast growth factor receptor (FGFR) oncogenic signa

254 n fibroblasts, RCC broadly induced low-level fibroblast growth factor receptor (FGFR) signaling.

255 JNJ-42756493 is an orally administered pan-fibroblast growth factor receptor (FGFR) tyrosine kinase

256 ), insulin growth factor 2 receptor (IGF2R), fibroblast growth factor receptor (FGFR), etc.

257 of diverse transmembrane receptors including fibroblast growth factor receptor (FGFR), platelet-deriv

258 activation mechanism of the kinase domain of Fibroblast Growth Factor Receptor (FGFR).

259 rrelations of diseases caused by variants in Fibroblast Growth Factor Receptor 1 ( FGFR1) and report

260 CRISPR/Cas9-mediated deletion of fibroblast growth factor receptor 1 (FGFR1) or pretreatm

261 d growth factor receptor alpha (PDGFRA), and fibroblast growth factor receptor 1 (FGFR1) to cell prol

262 express a wide range of receptors, including fibroblast growth factor receptor 1 (FGFR1).

263 Fgfr1 (fibroblast growth factor receptor 1) was identified as a

264 The regulatory mechanism of one such RTK, fibroblast growth factor receptor 2 (FGFR2) kinase, is s

265 cgamma1 compete for the same binding site on fibroblast growth factor receptor 2 (FGFR2).

266 Mutations to fibroblast growth factor receptor 3 (FGFR3) and phosphat

267 Fibroblast growth factor receptor 3 (FGFR3) coimmunoprec

268 We recently found that the fibroblast growth factor receptor 3 (FGFR3) interacts wi

269 Missense mutations of fibroblast growth factor receptor 3 (FGFR3) occur in up

270 Aberrant fibroblast growth factor receptor 3 (FGFR3) signaling di

271 ons that exaggerate the signal output of the fibroblast growth factor receptor 3 (FGFR3), a receptor

272 is caused by a gain-of function mutation in fibroblast growth factor receptor 3 (FGFR3).

273 caused by gain-of-function mutations in the fibroblast growth factor receptor 3-encoding (FGFR3-enco

274 The fibroblast growth factor receptor FGFR2 is overexpressed

275 Activation of the fibroblast growth factor receptor FGFR4 by FGF19 drives

276 mediated by the Forkhead TFs regulating the fibroblast growth factor receptor Heartless (Htl) and th

277 sensitivity to phosphoinositide 3-kinase and fibroblast growth factor receptor inhibition.

278 tes the epidermal growth factor receptor and fibroblast growth factor receptor signaling pathways inv

279 As determined by fibroblast growth factor receptor substrate 2 (FRS2) pho

280 ing pathways through their interactions with fibroblast growth factor receptor substrate 2 (FRS2).

281 ion of a pan-FGF receptor adaptor Frs2alpha (fibroblast growth factor receptor substrate 2 alpha) in

282 ingly, Prox1 also controls the expression of fibroblast growth factor receptors (FGFRs) and can bind

283 Fibroblast growth factor receptors (FGFRs) are frequentl

284 Fibroblast growth factor receptors (FGFRs) are involved

285 Somatic alterations of fibroblast growth factor receptors (FGFRs) have been des

286 olid tumors harboring genetic alterations in fibroblast growth factor receptors (FGFRs) to determine

287 ct gain-of-function mutations in five genes (fibroblast growth factor receptors FGFR2 and FGFR3, tyro

288 tinib, a potent tyrosine-kinase inhibitor of fibroblast growth factor receptors, VEGF receptors, PDGF

289 study the hetero-interactions between three fibroblast growth factor receptors-FGFR1, FGFR2, and FGF

290 rmal growth factor receptor, Ins-Ralpha, and fibroblast growth factor receptors.

291 Fibroblast growth factors receptors (FGFRs) are thought

292 A fibroblast growth factor signaling and four Forkhead tra

293 Fibroblast growth factor signaling was found to regulate

294 rized, while in DU145 cells rLOX-PP targeted fibroblast growth factor signaling.

295 factor, vascular endothelial growth factor, fibroblast growth factor, stem cell factor, and stromal

296 enerate mMSCs by utilizing hypoxia and basic fibroblast growth factor supplementation.

297 development by producing decapentaplegic and fibroblast growth factor that travel via specific cytone

298 rived bone morphogenetic proteins (BMPs) and fibroblast growth factors that signal to epithelial buds

299 Sonic hedgehog, bone morphogenetic protein, fibroblast growth factor, transforming growth factor bet

300 mple tests to quantify angiogenesis factors (fibroblast growth factor, vascular endothelial growth fa