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1 tion of proteins and signal transduction via fibroblast growth-factor receptor.
2 rmal growth factor receptor, Ins-Ralpha, and fibroblast growth factor receptors.
3  changes associated with focal adhesions and fibroblast growth factor receptors.
4 RBB2 and ERBB3; and trace phosphorylation of fibroblast growth factor receptors.
5 ition of EGFRs but not by the stimulation of fibroblast growth factor receptors.
6 rrelations of diseases caused by variants in Fibroblast Growth Factor Receptor 1 ( FGFR1) and report
7 f Drosophila Torso (alpha(Tor)) or the mouse fibroblast growth factor receptor 1 (alpha(FR)).
8 revious studies have individually implicated fibroblast growth factor receptor 1 (FGFR1) and canonica
9         Recently, we found that mice lacking fibroblast growth factor receptor 1 (Fgfr1) and Fgfr2 (K
10              Expression of the gene encoding fibroblast growth factor receptor 1 (FGFR1) and subseque
11  post-implantation lethality associated with fibroblast growth factor receptor 1 (FGFR1) deficiency,
12 L in cell motility control via regulation of fibroblast growth factor receptor 1 (FGFR1) endocytosis.
13                      These cells require the fibroblast growth factor receptor 1 (Fgfr1) gene for the
14 on and differentiation via expression of the fibroblast growth factor receptor 1 (FGFR1) gene is posi
15 atively spliced alpha-exon of the endogenous fibroblast growth factor receptor 1 (FGFR1) gene, which
16             Previous studies have implicated fibroblast growth factor receptor 1 (FGFR1) in limb deve
17                                              Fibroblast growth factor receptor 1 (FGFR1) is a transme
18                                          The fibroblast growth factor receptor 1 (FGFR1) is ectopical
19                                              Fibroblast growth factor receptor 1 (Fgfr1) is expressed
20                                              Fibroblast growth factor receptor 1 (FGFR1) is frequentl
21                                              Fibroblast growth factor receptor 1 (FGFR1) is the predo
22                             Amplification of fibroblast growth factor receptor 1 (FGFR1) occurs in ap
23             CRISPR/Cas9-mediated deletion of fibroblast growth factor receptor 1 (FGFR1) or pretreatm
24                                              Fibroblast growth factor receptor 1 (Fgfr1) plays pleiot
25 lpha-exon by alternative RNA splicing of the fibroblast growth factor receptor 1 (FGFR1) primary tran
26                                    Activated fibroblast growth factor receptor 1 (FGFR1) propagates F
27 d growth factor receptor alpha (PDGFRA), and fibroblast growth factor receptor 1 (FGFR1) to cell prol
28 tivated with other tyrosine kinases, such as fibroblast growth factor receptor 1 (FGFR1), concomitant
29 nescence resonance energy transfer analysis, fibroblast growth factor receptor 1 (FGFR1)-5-hydroxytry
30 express a wide range of receptors, including fibroblast growth factor receptor 1 (FGFR1).
31 mal growth factor receptor (HEGFR) and human fibroblast growth factor receptor 1 (HFGFR1) mRNAs in th
32 hypothalamic amenorrhea: two variants in the fibroblast growth factor receptor 1 gene FGFR1 (G260E an
33                                Inhibition of fibroblast growth factor receptor 1 in Tsc1-null mice su
34 et-derived growth factor receptor beta and a fibroblast growth factor receptor 1 kinase inhibitor) an
35 cible transcript-3.0 (wit3.0), also known as fibroblast growth factor receptor 1 oncogene partner 2 (
36                                       Fgfr1 (fibroblast growth factor receptor 1) was identified as a
37  as inhibited the mitogenic activity of both fibroblast growth factor receptor 1-expressing BaF3 cell
38 elial growth factor receptor 2 (VEGFR-2) and fibroblast growth factor receptor 1.
39 n-activated protein kinase and overexpressed fibroblast growth factor receptor 1.
40 thelial growth factor receptors 1, 2, and 3, fibroblast growth factor receptors 1 through 4, platelet
41  the second half of Ig-like domain 3 (D3) of fibroblast growth factor receptors 1-3 (FGFR1 to -3) gen
42        Activating mutations in the genes for fibroblast growth factor receptors 1-3 (FGFR1-3) are res
43 s of hypertrophy, including type X collagen, fibroblast growth factor receptors 1-3, parathyroid horm
44              Conditional derivatives of both fibroblast growth factor receptor-1 (F36VFGFR1) and the
45 owever, DNA synthesis and phosphorylation of fibroblast growth factor receptor-1 (FGFR-1) induced by
46  contrast, hypoxic mice carrying a disrupted fibroblast growth factor receptor-1 (Fgfr1) gene in GFAP
47                       In integrative nuclear fibroblast growth factor receptor-1 (FGFR1) signaling a
48                A mouse model of human ZNF198-fibroblast growth factor receptor-1 (FGFR1) stem cell le
49 on-cleavage screen, including Hsp70, Notch1, fibroblast growth factor receptor-1 (FGFR1), poly-A-bind
50 ell-surface proteins cytokeratin-7 (CK7) and fibroblast growth factor receptor-1 (FGFR1), which are t
51  combined with the cytoplasmic domain of the fibroblast growth factor receptor-1 (FGFR1).
52 , we discovered that activation of inducible fibroblast growth factor receptor-1 (iFGFR1) in the mamm
53 vitro, this differentiation was inhibited by fibroblast growth factor receptor-1 Ab, demonstrating a
54 r-1 Ab, demonstrating a role of the CD56 and fibroblast growth factor receptor-1 interaction in this
55 lar endothelial growth factor receptor-2 and fibroblast growth factor receptor-1 that are two key gro
56 ss in self-renewal occurs via alterations in fibroblast growth factor receptor-1, p38alpha and p38bet
57 lar endothelial growth factor receptor-2 and fibroblast growth factor receptor-1.
58 volves fibroblast growth factor-2 as well as fibroblast growth factor receptor-1.
59                      Conditional ablation of Fibroblast Growth Factor receptors-1 and -2 (FGFR1/2) in
60                      Fgf23 mRNA in bones and fibroblast growth factor receptors 1c and 3c and Klotho
61                                  Splicing of fibroblast growth factor receptor 2 (FGFR2) alternative
62                                 Mutations in fibroblast growth factor receptor 2 (FGFR2) and its liga
63 single nucleotide polymorphisms (SNP) within fibroblast growth factor receptor 2 (FGFR2) as one of th
64                 Pax3cre-mediated deletion of fibroblast growth factor receptor 2 (Fgfr2) broadly in r
65                      Activating mutations in fibroblast growth factor receptor 2 (FGFR2) cause severa
66 report that mice lacking the IIIb isoform of fibroblast growth factor receptor 2 (Fgfr2) exhibit seve
67               Mutually exclusive splicing of fibroblast growth factor receptor 2 (FGFR2) exons IIIb a
68                                          The fibroblast growth factor receptor 2 (FGFR2) gene exons I
69 ntify substitutions at nucleotide 755 of the fibroblast growth factor receptor 2 (FGFR2) gene in sper
70               Alternative splice variants of fibroblast growth factor receptor 2 (FGFR2) IIIb, design
71  have recently shown activating mutations in fibroblast growth factor receptor 2 (FGFR2) in 16% of en
72 etic evidence that En1 functions upstream of fibroblast growth factor receptor 2 (Fgfr2) in regulatin
73            Mice with conditional deletion of fibroblast growth factor receptor 2 (Fgfr2) in the urete
74                                              Fibroblast growth factor receptor 2 (FGFR2) is a crucial
75 on of epithelial and mesenchymal isoforms of Fibroblast Growth Factor Receptor 2 (FGFR2) is achieved
76                    The ligand specificity of fibroblast growth factor receptor 2 (FGFR2) is determine
77    The regulatory mechanism of one such RTK, fibroblast growth factor receptor 2 (FGFR2) kinase, is s
78                                          The fibroblast growth factor receptor 2 (FGFR2) locus has be
79 e recognition sites such as exon IIIb of the fibroblast growth factor receptor 2 (FGFR2) mRNA.
80 ome (ABS), which has also been attributed to fibroblast growth factor receptor 2 (FGFR2) mutations.
81                                              Fibroblast growth factor receptor 2 (FGFR2) promotes ost
82 or receptor-bound protein 2 (Grb2), controls fibroblast growth factor receptor 2 (FGFR2) signaling by
83 In epithelial cells, alternative splicing of fibroblast growth factor receptor 2 (FGFR2) transcripts
84                      Alternative splicing of fibroblast growth factor receptor 2 (FGFR2) transcripts
85                 Previous studies demonstrate fibroblast growth factor receptor 2 (FGFR2) triggers Nan
86  Here, we show that somatic mutations of the fibroblast growth factor receptor 2 (FGFR2) tyrosine kin
87     In a panel of gastric cancer cell lines, fibroblast growth factor receptor 2 (FGFR2) was overexpr
88 g from activating or neomorphic mutations in fibroblast growth factor receptor 2 (FGFR2), underscore
89 s of Pten deletion, we deleted one allele of fibroblast growth factor receptor 2 (FGFR2).
90 otype associated with syndromic mutations in fibroblast growth factor receptor 2 (Fgfr2).
91 ding to amino acid changes S252W or P253R in fibroblast growth factor receptor 2 (FGFR2).
92 cgamma1 compete for the same binding site on fibroblast growth factor receptor 2 (FGFR2).
93 ffected individuals have a S252W mutation in fibroblast growth factor receptor 2 (FGFR2).
94                    The epithelial isoform of fibroblast growth factor receptor 2 (Fgfr2b) is essentia
95 hnRNP F proteins as being novel silencers of fibroblast growth factor receptor 2 exon IIIc.
96                                  SNPs in the fibroblast growth factor receptor 2 gene (FGFR2) and the
97 s to study renal development in mice lacking fibroblast growth factor receptor 2 in the ureteric bud
98 n breast cancer prevention, and suggest that fibroblast growth factor receptor 2 signalling has an im
99 XA1, GATA3 and PTTG1 as master regulators of fibroblast growth factor receptor 2 signalling, and show
100  and show that ERalpha occupancy responds to fibroblast growth factor receptor 2 signalling.
101                          In addition, FGFR2 (fibroblast growth factor receptor 2) depletion in HSulf-
102 thelial growth factor receptor 2, TIE-2, and fibroblast growth factor receptor 2).
103 es expression of bone morphogenic protein 7, fibroblast growth factor receptor 2, insulin-like growth
104                               Methylation at fibroblast growth factor receptor 2, methionine adenosyl
105                                          The fibroblast growth factor receptor 2-IIIb (FGFR2b) and th
106                      Alternative splicing of fibroblast growth factor receptor-2 (FGFR2) mutually exc
107 FR-2, showed very modest selectivity against fibroblast growth factor receptor-2 (IC(50) = 0.21 micro
108  suggested that FGF10, acting mainly through fibroblast growth factor receptor 2b (FGFR2b), is crucia
109  lacks a Src-binding site, using shedding of fibroblast growth factor receptor 2iiib variant as an as
110 an autosomal-dominant activating mutation in fibroblast growth factor receptor 3 (FGFR3(P250R) ).
111 ice with the K644E kinase domain mutation in fibroblast growth factor receptor 3 (Fgfr3) (EIIa;Fgfr3(
112 have the same point mutation in the gene for fibroblast growth factor receptor 3 (FGFR3) and more tha
113 n that it involves 2 potential target genes: fibroblast growth factor receptor 3 (FGFR3) and multiple
114                                 Mutations to fibroblast growth factor receptor 3 (FGFR3) and phosphat
115 athymic nude mice, due to down regulation of fibroblast growth factor receptor 3 (FGFR3) and PI3K/Akt
116             Although activating mutations of fibroblast growth factor receptor 3 (FGFR3) are frequent
117                                 Mutations of fibroblast growth factor receptor 3 (FGFR3) are responsi
118                        Ectopic expression of fibroblast growth factor receptor 3 (FGFR3) associated w
119                                              Fibroblast growth factor receptor 3 (FGFR3) belongs to a
120  have previously shown that dysregulation of fibroblast growth factor receptor 3 (FGFR3) by the t(4;1
121                                 Mutations in fibroblast growth factor receptor 3 (FGFR3) cause the mo
122 380R mutation in the transmembrane domain of fibroblast growth factor receptor 3 (FGFR3) causes achon
123                                              Fibroblast growth factor receptor 3 (FGFR3) coimmunoprec
124 ently reported that receptor tyrosine kinase fibroblast growth factor receptor 3 (FGFR3) directly tyr
125                                    Recently, fibroblast growth factor receptor 3 (FGFR3) has also bee
126 380R mutation in the transmembrane domain of fibroblast growth factor receptor 3 (FGFR3) in >97% of t
127                   We recently found that the fibroblast growth factor receptor 3 (FGFR3) interacts wi
128                                              Fibroblast growth factor receptor 3 (FGFR3) is a key reg
129                                              Fibroblast growth factor receptor 3 (FGFR3) is a major n
130                                              Fibroblast growth factor receptor 3 (FGFR3) is a recepto
131                        Ectopic activation of fibroblast growth factor receptor 3 (FGFR3) is associate
132                                              Fibroblast growth factor receptor 3 (FGFR3) is expressed
133 ly, we found that the enhanced activation of fibroblast growth factor receptor 3 (FGFR3) is linked to
134                        The A391E mutation in fibroblast growth factor receptor 3 (FGFR3) is the genet
135                                              Fibroblast growth factor receptor 3 (FGFR3) mutations ar
136                        Missense mutations of fibroblast growth factor receptor 3 (FGFR3) occur in up
137  Gly380 --> Arg mutation in the TM domain of fibroblast growth factor receptor 3 (FGFR3) of the RTK f
138                                              Fibroblast growth factor receptor 3 (FGFR3) plays a crit
139 ysregulation of the receptor tyrosine kinase fibroblast growth factor receptor 3 (FGFR3) plays a path
140                                              Fibroblast growth factor receptor 3 (FGFR3) plays an imp
141                                     Aberrant fibroblast growth factor receptor 3 (FGFR3) signaling di
142                  Research on the mutation in Fibroblast Growth Factor Receptor 3 (FGFR3) that causes
143 is associated with ectopic overexpression of fibroblast growth factor receptor 3 (FGFR3) that sometim
144 y of dimer formation of the G380R mutants of fibroblast growth factor receptor 3 (FGFR3) transmembran
145  for heterodimer formation between wild-type fibroblast growth factor receptor 3 (FGFR3) transmembran
146 hogenic mutations (V664E in Neu and A391E in fibroblast growth factor receptor 3 (FGFR3)) on receptor
147                  Activating mutations within fibroblast growth factor receptor 3 (FGFR3), a receptor
148                  Activating mutations within fibroblast growth factor receptor 3 (FGFR3), a receptor
149 ons that exaggerate the signal output of the fibroblast growth factor receptor 3 (FGFR3), a receptor
150 estigated whether the TM domains of one RTK, fibroblast growth factor receptor 3 (FGFR3), dimerize in
151 nic mutation is the Ala391-->Glu mutation in fibroblast growth factor receptor 3 (FGFR3), linked to C
152 erozygous Pro250Arg substitution mutation in fibroblast growth factor receptor 3 (FGFR3), which incre
153  is caused by a gain-of function mutation in fibroblast growth factor receptor 3 (FGFR3).
154 inomas contain activating point mutations of fibroblast growth factor receptor 3 (FGFR3).
155 titution of an amino acid with a cysteine in fibroblast growth factor receptor 3 (FGFR3).
156 tions on dimerization of TM domains of human fibroblast growth factor receptor 3 (FGFR3).
157 rmation induced by the constitutively active fibroblast growth factor receptor 3 (K/E-FR3) (Oncogene,
158 g 11q13 (cyclin D1), 6p21 (cyclin D3), 4p16 (fibroblast growth factor receptor 3 [FGFR3] and multiple
159 E TM domain mutation on the stability of the fibroblast growth factor receptor 3 dimer in the presenc
160 these results to the case of the isolated TM fibroblast growth factor receptor 3 domains.
161 ge and the frequencies of sperm with DFI and fibroblast growth factor receptor 3 gene (FGFR3) mutatio
162 ude HER2, epidermal growth factor receptors, fibroblast growth factor receptor 3, mTOR and others.
163  caused by gain-of-function mutations in the fibroblast growth factor receptor 3-encoding (FGFR3-enco
164 ctivity, including: CHIR-258, which inhibits fibroblast growth factor receptor 3; NVP-ADW742, which i
165 lasia or achondroplasia, in the TM domain of fibroblast growth factor receptors 3 (FGFR3).
166 rm of human achondrodysplasia, linked to the fibroblast-growth-factor receptor 3 locus, not linked to
167 sia caused by a gain-of-function mutation in fibroblast-growth-factor-receptor 3 (FGFR3).
168                                              Fibroblast growth factor receptor-3 (FGFR-3) expression
169 thelial cells, secretoneurin also stimulated fibroblast growth factor receptor-3 and insulin-like gro
170  receptor, bone morphogenetic protein-7, and fibroblast growth factor receptor-3 expression increased
171  observed stimulation of VEGF receptor-1 and fibroblast growth factor receptor-3.
172 mulates further growth factor receptors like fibroblast growth factor receptor-3.
173 vascular endothelial growth factor (VEGF) or fibroblast growth factor receptor-3.
174 l approach involving antisense inhibition of fibroblast growth factor receptor 4 (FGFR4) in periphera
175                                  The role of fibroblast growth factor receptor 4 (FGFR4) in regulatin
176  KLB rs17618244 with 3 nonsynonymous SNPs of fibroblast growth factor receptor 4 (FGFR4) revealed tha
177 ocal acetylcholine release, synergizing with fibroblast growth factor receptor activation to increase
178 Y240 phosphorylation can be mediated by both fibroblast growth factor receptors and SRC family kinase
179 ing platelet-derived growth factor receptor, fibroblast growth factor receptor, and Met, leading to i
180                                 Mutations in fibroblast growth factor receptors are known as the gene
181 t to a LOX-PP target at or near the level of fibroblast growth factor receptor binding or activation.
182 epithelial growth factor receptor (EGFR) and fibroblast growth factor receptor/breathless (FGFR, Btl)
183 transition promoted by the oncogenic Met and fibroblast growth factor receptors, but not by progester
184 the Notch ligands Jagged1 and Delta1 induced fibroblast growth factor receptor-dependent cell transfo
185  growth factor-A (PDGF-A), dominant-negative fibroblast growth factor receptor (DN-FGFR), or DN-Sprou
186      Signaling by the Caenorhabditis elegans fibroblast growth factor receptor EGL-15 is activated by
187 f both populations, whereas argonaute2 and a fibroblast growth factor receptor-encoding gene are requ
188  Interestingly, FAK knockdown did not modify fibroblast growth factor receptor expression induced by
189 p11;q12) results in expression of the ZNF198-fibroblast growth factor receptor (FGFR) 1 fusion tyrosi
190 oncogenes including erbB-2/HER-2, C-MYC, and fibroblast growth factor receptor (FGFR) 2.
191 iency results in cell-autonomous loss of the fibroblast growth factor receptor (Fgfr) 2b, which is re
192 ithelial cells resulted in a higher level of fibroblast growth factor receptor (FGFR) activation and
193  to mediate cell repulsion is antagonized by fibroblast growth factor receptor (FGFR) activation that
194                                    Levels of fibroblast growth factor receptor (FGFR) and cSrc were 4
195 athway analysis of these 69 genes implicated fibroblast growth factor receptor (FGFR) as a key regula
196 F3 cell line expresses only a single type of fibroblast growth factor receptor (FGFR) but produces ne
197                            Activation of the fibroblast growth factor receptor (FGFR) by neural cell
198                The current working model for fibroblast growth factor receptor (FGFR) dimerization an
199 n of Frs2alpha (Six2creFrs2alphaKO), a major fibroblast growth factor receptor (Fgfr) docking protein
200 esses paralleled the dorsoventral changes in fibroblast growth factor receptor (FGFR) expressions.
201                             The prototypical fibroblast growth factor receptor (FGFR) extracellular d
202 ivation specific for the PTKs in the Src and fibroblast growth factor receptor (FGFR) families, key e
203   FGFRL1 is a newly identified member of the fibroblast growth factor receptor (FGFR) family expresse
204                                          The fibroblast growth factor receptor (FGFR) family of recep
205 c pathways mediated by the VEGFR family, the fibroblast growth factor receptor (FGFR) family, the pla
206 red receptor tyrosine kinase families is the fibroblast growth factor receptor (FGFR) family, with am
207  Fgf ligands and heparin, is a member of the fibroblast growth factor receptor (Fgfr) family.
208                                              Fibroblast growth factor receptor (FGFR) gene products (
209 -frame the tyrosine kinase coding domains of fibroblast growth factor receptor (FGFR) genes (FGFR1 or
210                        The expression of the fibroblast growth factor receptor (FGFR) heartless and i
211             Here, we show that the selective fibroblast growth factor receptor (FGFR) inhibitor PD173
212 s, an effect abolished by treatment with the fibroblast growth factor receptor (FGFR) inhibitor SU540
213 e rates have been low for biomarker-directed fibroblast growth factor receptor (FGFR) inhibitor thera
214 ng mutation, and the combination of EGFR and fibroblast growth factor receptor (FGFR) inhibitors was
215  Epidermal Growth Factor Receptor (EGFR) and Fibroblast Growth Factor Receptor (FGFR) is required for
216 e the dimer stabilities of three full-length fibroblast growth factor receptor (FGFR) mutants harbori
217  identified cross-talk between the Hippo and fibroblast growth factor receptor (FGFR) oncogenic signa
218 d enhanced Ihh and Pthrp expression, whereas fibroblast growth factor receptor (FGFR) or mitogen-acti
219 nto the eye field, whereas activation of the fibroblast growth factor receptor (FGFR) represses these
220                                              Fibroblast growth factor receptor (FGFR) signaling deter
221 tivating activity, controls ERK1/2-dependent fibroblast growth factor receptor (FGFR) signaling in ch
222        Here, we aim to determine if 1) FGF-2/fibroblast growth factor receptor (FGFR) signaling is su
223        Aberrant activations of the Notch and fibroblast growth factor receptor (FGFR) signaling pathw
224 ce that this alternative pathway may involve fibroblast growth factor receptor (FGFR) signaling.
225 n fibroblasts, RCC broadly induced low-level fibroblast growth factor receptor (FGFR) signaling.
226 HNSCC) cell lines exhibit dominant autocrine fibroblast growth factor receptor (FGFR) signaling.
227 n substrate for two proteins involved in the fibroblast growth factor receptor (FGFR) signalling path
228 th factor receptor (KGFR) is a member of the fibroblast growth factor receptor (FGFR) superfamily.
229 for the work described here, we selected the fibroblast growth factor receptor (FGFR) tyrosine kinase
230   JNJ-42756493 is an orally administered pan-fibroblast growth factor receptor (FGFR) tyrosine kinase
231                            N-cadherin caused fibroblast growth factor receptor (FGFR) upmodulation, r
232             We show here that the C. elegans fibroblast growth factor receptor (FGFR), EGL-15, affect
233                   The Caenorhabditis elegans fibroblast growth factor receptor (FGFR), EGL-15, regula
234 ), insulin growth factor 2 receptor (IGF2R), fibroblast growth factor receptor (FGFR), etc.
235 ed significant inhibitory activities against fibroblast growth factor receptor (FGFR), platelet-deriv
236 of diverse transmembrane receptors including fibroblast growth factor receptor (FGFR), platelet-deriv
237                            The expression of fibroblast growth factor receptor (FGFR)-1 correlates wi
238 en induced cell growth, MM134 cells required fibroblast growth factor receptor (FGFR)-1 signaling to
239 or (A(2A)R) and the receptor tyrosine kinase fibroblast growth factor receptor (FGFR).
240 on of Frizzled (Fz) serpentine receptors and fibroblast growth factor receptor (FGFR).
241 activation mechanism of the kinase domain of Fibroblast Growth Factor Receptor (FGFR).
242                                              Fibroblast growth factor receptors (Fgfr) comprise a wid
243                       Aberrant activation of fibroblast growth factor receptors (FGFR) contributes to
244                                              Fibroblast growth factor receptors (FGFR) play key roles
245  study the hetero-interactions between three fibroblast growth factor receptors-FGFR1, FGFR2, and FGF
246                                          The fibroblast growth factor receptor FGFR2 is overexpressed
247 ct gain-of-function mutations in five genes (fibroblast growth factor receptors FGFR2 and FGFR3, tyro
248 th factor receptor alpha (PDGFRA) and type 3 fibroblast growth factor receptor (FGFR3), receptor tyro
249                            Activation of the fibroblast growth factor receptor FGFR4 by FGF19 drives
250 rs351855) in the transmembrane domain of the fibroblast growth factor receptor (FGFR4) is associated
251                      Conditional deletion of fibroblast growth factor receptors (Fgfrs) 1 and 2 in th
252               To determine the importance of fibroblast growth factor receptors (fgfrs) 1 and 2 in th
253                                 Mutations in fibroblast growth factor receptors (Fgfrs) 1-3 cause ske
254              Endothelial cells (ECs) express fibroblast growth factor receptors (FGFRs) and are exqui
255 ingly, Prox1 also controls the expression of fibroblast growth factor receptors (FGFRs) and can bind
256                                              Fibroblast growth factor receptors (FGFRs) are a family
257                                              Fibroblast growth factor receptors (FGFRs) are activated
258                                              Fibroblast growth factor receptors (FGFRs) are frequentl
259                                              Fibroblast growth factor receptors (FGFRs) are involved
260          We show here that in mouse embryos, Fibroblast growth factor receptors (FGFRs) are required
261                                              Fibroblast growth factor receptors (FGFRs) can act as dr
262                                 Mutations in fibroblast growth factor receptors (FGFRs) cause human b
263         We show that loss of function of all fibroblast growth factor receptors (FgfRs) expressed at
264                       Somatic alterations of fibroblast growth factor receptors (FGFRs) have been des
265           To understand the role of specific fibroblast growth factor receptors (FGFRs) in cortical d
266                            Signaling through fibroblast growth factor receptors (FGFRs) is essential
267                                              Fibroblast growth factor receptors (FGFRs) play key role
268                                The family of fibroblast growth factor receptors (FGFRs) plays an impo
269 olid tumors harboring genetic alterations in fibroblast growth factor receptors (FGFRs) to determine
270                                              Fibroblast growth factors receptors (FGFRs) are thought
271 rogen receptor gene (ESR1), PIK3CA gene, and fibroblast growth factor receptor gene (FGFR2), among ot
272               In addition, we identified the fibroblast growth factor receptor gene, Fgfr2, as one oc
273 ell-based models of fibroblast growth factor/fibroblast growth factor receptor, glial cell line-deriv
274  mediated by the Forkhead TFs regulating the fibroblast growth factor receptor Heartless (Htl) and th
275                 Furthermore, inactivation of fibroblast growth factor receptor in PyVmT-N-cadherin ce
276 domain, FGF23-21c was still able to activate fibroblast growth factor receptors in the presence of al
277                   After injury and prolonged fibroblast growth factor receptor inhibition to arrest c
278 sensitivity to phosphoinositide 3-kinase and fibroblast growth factor receptor inhibition.
279 alpha), a key docking protein that transmits fibroblast growth factor receptor intracellular signalin
280 bited the highest kinase selectivity against fibroblast growth factor receptor kinase, platelet-deriv
281 se schistosome neoblast-like cells express a fibroblast growth factor receptor orthologue.
282 og; OSX, osterix transcription factor; FGFR, fibroblast growth factor receptor; PMN, polymorphonuclea
283     In vitro binding assays with recombinant fibroblast growth factor receptor protein revealed that
284 in unanswered, such as the identification of fibroblast growth factor receptor responsible for glucos
285 s differential responses to EGF receptor vs. fibroblast growth factor receptor signaling as well as t
286                         Lastly, we show that fibroblast growth factor receptor signaling likely media
287 tes the epidermal growth factor receptor and fibroblast growth factor receptor signaling pathways inv
288 ed lymphatic endothelial cells by activating fibroblast growth factor receptor signaling.
289 , a protein that moderates glycosylation and fibroblast growth factor receptor signaling.
290 tor cells present in the eye, independent of fibroblast growth factor receptor signaling.
291                             As determined by fibroblast growth factor receptor substrate 2 (FRS2) pho
292 ing pathways through their interactions with fibroblast growth factor receptor substrate 2 (FRS2).
293           We report that the docking protein fibroblast growth factor receptor substrate 2 (FRS2alpha
294 ion of a pan-FGF receptor adaptor Frs2alpha (fibroblast growth factor receptor substrate 2 alpha) in
295 ching defects; however, ureteric deletion of fibroblast growth factor receptor substrate 2alpha (Frs2
296 t either the neurotrophic signaling effector fibroblast growth factor receptor substrate 2alpha or th
297 cular endothelial growth factor receptor and fibroblast growth factor receptor, to cetuximab has show
298 f a plasmid expressing the nucleus-targeting fibroblast growth factor receptor type 1 resulted in sig
299  as platelet-derived growth factor receptor, fibroblast growth factor receptor, VEGF-R1, and VEGF-R3,
300 tinib, a potent tyrosine-kinase inhibitor of fibroblast growth factor receptors, VEGF receptors, PDGF

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