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1 ce in their 5' leaders, Gtx itself and FGF2 (fibroblast growth factor 2).
2 ors (serum, insulin-like growth factor I, or fibroblast growth factor-2).
3 ls by vascular endothelial growth factor and fibroblast growth factor 2.
4 and invasion, in the presence or absence of fibroblast growth factor 2.
5 owth; Igf2 does so in a manner additive with fibroblast growth factor 2.
6 idermal growth factor-like growth factor and fibroblast growth factor 2.
7 uring cortical explants in medium containing fibroblast growth factor 2.
8 conventional secretion shared by HIV-Tat and fibroblast growth factor 2.
9 ll as intracellular and secreted isoforms of fibroblast growth factor-2.
10 process is mediated at least in part through fibroblast growth factor-2.
11 (EC(50) = 88 pmol/L) induced by 500 ng/ml of fibroblast growth factor-2.
12 matid separation, and regulates secretion of fibroblast growth factor-2.
13 ompanied by an increase in astrocyte-derived fibroblast growth factor-2.
14 An opposite pattern was noted for basic fibroblast growth factor-2.
15 y and expression of glutamine synthetase and fibroblast growth factor-2.
16 mation, and VEGFR-2 signaling in response to fibroblast growth factor-2.
17 cant increases in expression of collagen and fibroblast growth factor (2.8 and 3.4 fold, p<0.05).
18 e or presence of different concentrations of fibroblast growth factor 2, a known regulator of cortica
19 is is through the regulation of secretion of fibroblast growth factor-2, a guidance factor for migrat
20 ontaining vascular endothelial growth factor/fibroblast growth factor-2 achieved normoglycemia at a h
22 rough binding to cell-surface tropomyosin in fibroblast growth factor-2-activated endothelial cells (
23 onstrate here that the continued presence of fibroblast growth factor 2 along with N-CAM or brain-der
24 retinoic acid, insulin-like growth factor-1, fibroblast growth factor-2, alpha-thrombin, and interleu
26 ed only in cultures grown in the presence of fibroblast growth factor 2 and either N-CAM or brain-der
27 ound, however, that direct administration of fibroblast growth factor 2 and epidermal growth factor i
28 l growth factor, fibroblast growth factor 7, fibroblast growth factor 2 and hepatocyte growth factor
29 other pro-angiogenic growth factors, namely fibroblast growth factor 2 and hepatocyte growth factor.
30 were differentiated by sequential culture in fibroblast growth factor 2 and human activin-A, hepatocy
31 ectodomain of the core protein and required fibroblast growth factor 2 and stroma-derived factor 1.
32 We have discovered that a combination of fibroblast growth factor 2 and transforming growth facto
34 n expression in cardiomyocytes is induced by fibroblast growth factor-2 and accumulates in response t
36 our murine retinal angiogenesis model, both fibroblast growth factor-2 and interleukin-6 administrat
37 elial dysfunction, characterized by abnormal fibroblast growth factor-2 and interleukin-6 signaling,
38 nsforming growth factor-beta, in contrast to fibroblast growth factor-2 and interleukin-6, promotes h
39 everal other angiogenesis factors, including fibroblast growth factor-2 and the receptors Flt-1 and F
40 into gene expression cascades revealed that fibroblast growth factor-2 and transforming growth facto
43 del for two biomedically important proteins, fibroblast growth factor-2 and vascular endothelial grow
44 larly; however, when CECs are modulated with fibroblast growth factor-2 and/or corneal endothelium mo
45 of tumor necrosis factor alpha, IL-8, IL-10, fibroblast growth factor 2, and IL-7 remained higher.
46 es, associated with binding to antithrombin, fibroblast growth factor-2, and herpes simplex virus env
47 ulating proteins, including cyclinD1, c-Myc, fibroblast growth factor-2, and ornithine decarboxylase,
48 in-6 and monocyte chemoattractant protein-1, fibroblast growth factor-2, and the potent vasoconstrict
50 enic signaling molecules, including VEGF and fibroblast growth factor-2, are mainly expressed by non-
51 d vascular endothelial growth factor but not fibroblast growth factor-2 as mediators associated with
52 equires O-sulfation in heparin, and involves fibroblast growth factor-2 as well as fibroblast growth
53 (a) vascular endothelial growth factor; (b) fibroblast growth factor 2/basic fibroblast growth facto
54 5(S)-HETE induced the expression of basic fibroblast growth factor 2 (bFGF-2) in a Jak-2- and PI3-
56 ylation, global protein synthesis, and basic fibroblast growth factor-2 (bFGF-2) expression in VSMC.
57 ke growth factor 1 (IGF-1), as well as basic fibroblast growth factor 2 (bFGF2), reportedly astrocyte
60 (95% confidence interval [CI], 34%-42%) and fibroblast growth factor-2 by 64% (95% CI, 44-85%; P<0.0
63 -11 also attenuated glutamine synthetase and fibroblast growth factor-2 expression, but did not rever
64 ice is attributable, in part, to upregulated fibroblast growth factor-2 expression, which is inhibite
65 e mitogens epidermal growth factor (EGF) and fibroblast growth factor 2 (FGF 2), and then by serum.
68 ran sulfate to participate in a complex with fibroblast growth factor 2 (FGF-2) and its receptor tyro
69 ave shown previously that fibrin(ogen) binds fibroblast growth factor 2 (FGF-2) and potentiates stimu
71 pathway involves the release of pericellular fibroblast growth factor 2 (FGF-2) from the articular ca
72 ntagonizes interleukin-1 beta (IL-1beta) and fibroblast growth factor 2 (FGF-2) in proteoglycan metab
77 Here we show that the mechanism by which fibroblast growth factor 2 (FGF-2) protects small cell l
79 in(ogen) binding potentiates the capacity of fibroblast growth factor 2 (FGF-2) to stimulate endothel
80 we describe the cloning of the regulator of fibroblast growth factor 2 (FGF-2) transcription (RFT) u
81 ced apoptosis was associated with suppressed fibroblast growth factor 2 (FGF-2) transcription, as ass
83 e the safety and effectiveness of 3 doses of fibroblast growth factor 2 (FGF-2) when combined with a
86 of a number of HS-binding proteins including fibroblast growth factor 2 (FGF-2), and the chemokines C
87 -lysine and mouse laminin in the presence of fibroblast growth factor 2 (FGF-2), nerve growth factor
88 eased from PMNs induced de novo synthesis of fibroblast growth factor 2 (FGF-2), which in turn become
89 t pool of the heparin-binding growth factor, fibroblast growth factor 2 (FGF-2), which is bound to th
91 esponse to epidermal growth factor (EGF) and fibroblast growth factor 2 (FGF-2), while retaining the
92 aft tumor models as well as suppresses basic fibroblast growth factor 2 (FGF-2)-induced neovasculariz
97 in rabbit corneal fibroblasts; in contrast, fibroblast growth factor-2 (FGF) and heparin led to a de
98 th platelet-derived growth factor-BB (PDGF), fibroblast growth factor-2 (FGF), transforming growth fa
99 vented when the cells were treated with both fibroblast growth factor-2 (FGF-2) and antibodies agains
100 viously reported that growth factors such as fibroblast growth factor-2 (FGF-2) and bone morphogeneti
101 d binds and activates growth factors such as fibroblast growth factor-2 (FGF-2) and FGF-7, which are
102 a detailed study of the intercompetition of fibroblast growth factor-2 (FGF-2) and heparin-binding e
104 /-) mice were not as invasive in response to fibroblast growth factor-2 (FGF-2) as cancer cells isola
105 inase B gene (matrix metalloproteinase-9) by fibroblast growth factor-2 (FGF-2) during angiogenesis,
107 ocytes treated with U0126 in the presence of fibroblast growth factor-2 (FGF-2) express normal post-i
109 mined ciliary neurotrophic factor (CNTF) and fibroblast growth factor-2 (FGF-2) expression, because b
110 The resultant cultures were treated with fibroblast growth factor-2 (FGF-2) for a week, with conc
111 angiotensin II (AII) receptors activate the fibroblast growth factor-2 (FGF-2) gene through a unique
112 nded or high molecular weight (HMW) forms of fibroblast growth factor-2 (FGF-2) has been shown to aff
114 ddition, 14,15-EET induced the expression of fibroblast growth factor-2 (FGF-2) in Src- and PI3K-Akt-
115 we used this model to determine the role of fibroblast growth factor-2 (FGF-2) in the remodeling res
117 rbol 13-acetate (TPA), specifically inhibits fibroblast growth factor-2 (FGF-2) induced proliferation
121 inical studies report that the expression of fibroblast growth factor-2 (FGF-2) is decreased in the p
125 al cell (HUVEC) tube formation stimulated by fibroblast growth factor-2 (FGF-2) or vascular endotheli
126 Other endothelial growth factors, such as fibroblast growth factor-2 (FGF-2) or VEGF-A, may also c
128 Da) and low (18-kDa) molecular mass forms of fibroblast growth factor-2 (FGF-2) regulate cell prolife
129 human neuroblastoma cell line, SK-N-MC, with fibroblast growth factor-2 (FGF-2) results in induction
132 study to compare the neurotrophic actions of fibroblast growth factor-2 (FGF-2) with the better chara
133 Endothelial cell growth is stimulated by fibroblast growth factor-2 (FGF-2), and both adhesion an
134 -growth factor (NGF), neurotrophin-3 (NT-3), fibroblast growth factor-2 (FGF-2), and insulin-like gro
135 ivered insulin-like growth factor I (IGF-I), fibroblast growth factor-2 (FGF-2), and/or transforming
136 lar, heparin-binding growth factors, such as fibroblast growth factor-2 (FGF-2), bind to heparan sulf
137 ivator of transcription (Tat), combined with fibroblast growth factor-2 (FGF-2), can induce the dedif
138 h had been genetically engineered to produce fibroblast growth factor-2 (FGF-2), can protect nigrostr
139 o well-characterized lymphangiogenic factors fibroblast growth factor-2 (FGF-2), hepatocyte growth fa
142 ), TNF-alpha, interleukin-1 beta (IL-1beta), fibroblast growth factor-2 (FGF-2), transforming growth
143 n that the gamma chain serves as a depot for fibroblast growth factor-2 (FGF-2), which is likely to p
144 -2 (COX-2), interleukin-1beta (IL-1beta) and fibroblast growth factor-2 (FGF-2), which led to a marke
145 ial dysfunction, is an important mediator of fibroblast growth factor-2 (FGF-2)-induced angiogenesis.
153 ect axonal plasticity, adenoviruses encoding fibroblast growth factor-2 (FGF-2/Adts), nerve growth fa
156 rowth factors (kit ligand [KL], FLT3 ligand, fibroblast growth factor-2 [FGF-2], vascular endothelial
157 this study, we identified the gene encoding fibroblast growth factor 2 (Fgf2 or basic fibroblast gro
158 that bone morphogenetic proteins (BMPs) and fibroblast growth factor 2 (FGF2) act antagonistically t
159 increases in signaling on coactivation with fibroblast growth factor 2 (FGF2) and a 5-HT1A agonist,
161 commonly overexpressed in pancreatic cancer: fibroblast growth factor 2 (FGF2) and heparin-binding EG
163 proteoglycans (neoPGs) with affinity for the fibroblast growth factor 2 (FGF2) and introduced them in
164 r heparan and chondroitin sulfate, including fibroblast growth factor 2 (FGF2) and its receptor FGFR1
167 line-derived neurotrophic factor (GDNF) and fibroblast growth factor 2 (FGF2) are critical for roden
168 al concentrations of the neurotrophic factor fibroblast growth factor 2 (FGF2) are negatively associa
169 el of candidate growth factors we identified fibroblast growth factor 2 (FGF2) as a potent regulator
170 alpha (TNFalpha) as an ototoxic molecule and fibroblast growth factor 2 (FGF2) as an otoprotective mo
171 We also found that increased production of fibroblast growth factor 2 (Fgf2) by HoxA10-overexpressi
174 peptidase E, CPE), concomitant with enhanced fibroblast growth factor 2 (FGF2) expression, and an inc
176 doderm cultured in the presence of exogenous fibroblast growth factor 2 (FGF2) fails to initiate expr
181 reported that targeted overexpression of the fibroblast growth factor 2 (FGF2) high molecular weight
182 e transcriptional response of osteocalcin to fibroblast growth factor 2 (FGF2) in MC3T3 osteoblasts.
183 ascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2) in the development of
191 H3K9me3 at Glucocorticoid Receptor (GR) and Fibroblast growth Factor 2 (FGF2) promoters differ recip
192 oenvironmental proteins FLT3 ligand (FL) and fibroblast growth factor 2 (FGF2) protect FLT3-ITD+ MOLM
193 ling pathway, which is upregulated, owing to fibroblast growth factor 2 (FGF2) secretion or increased
194 endent association between apelin (APLN) and fibroblast growth factor 2 (FGF2) signaling in pulmonary
197 onstrate that loss of NG2 glial secretion of fibroblast growth factor 2 (FGF2) suffices to induce the
198 ted neutrophils expressed substantially more fibroblast growth factor 2 (FGF2) than naive neutrophils
202 her show that beta1-integrin cooperates with fibroblast growth factor 2 (Fgf2), a potent growth facto
203 ere, we focus on unconventional secretion of fibroblast growth factor 2 (FGF2), a secretory mechanism
204 otentiating the action of the growth factors fibroblast growth factor 2 (FGF2), hepatocyte growth fac
206 wth was associated with increased binding of fibroblast growth factor 2 (FGF2), phosphorylation of ex
207 lar to what has been reported previously for fibroblast growth factor 2 (FGF2), providing strong evid
208 racking in fibroblast pericellular matrix of fibroblast growth factor 2 (FGF2), stoichiometrically la
222 ced by intraocular injections of insulin and fibroblast growth factor-2 (FGF2) but not by ciliary neu
223 an postmortem studies have demonstrated that fibroblast growth factor-2 (FGF2) expression is decrease
227 Single-bolus intracoronary administration of fibroblast growth factor-2 (FGF2) improved symptoms and
231 Pharmacological studies have suggested that fibroblast growth factor-2 (FGF2) is involved in cardiop
235 ary mammary epithelial cells stimulated with fibroblast growth factor-2 (FGF2) to model mammary branc
236 heregulin-1beta (a ligand for ERBB2/ERBB3), fibroblast growth factor-2 (FGF2), and activin A support
237 r, we used recombinant adenovirus to express fibroblast growth factor-2 (FGF2), nerve growth factor (
242 we established a role for ASMC-derived basic fibroblast growth factor 2 (FGF2b) and FGF receptor (FGF
243 cans and COL2, -9, -10, and -11), receptors [fibroblast growth factor 2 (FGFR2) and parathyroid hormo
244 eta, IL-6, IL-10, IL-12p40, IL-12p70, IL-15, fibroblast growth factor 2, flt-3 ligand, tumor necrosis
245 n initial angiogenic stimulus is supplied by fibroblast growth factor-2, followed by a subsequent and
246 ne morphogenetic protein 7/human recombinant fibroblast growth factor 2 for 24 hours before induction
247 ry of vascular endothelial growth factor and fibroblast growth factor-2 from HBPA scaffolds significa
248 heparan sulfate 6-O-endosulfatases (Sulfs), fibroblast growth factor 2-, heparin binding epidermal g
249 gamma-induced protein 10, IL-4, IL-9, IL-10, fibroblast growth factor 2, IL-7, IL-15, and transformin
250 e stimulating hormone, stem cell factor, and fibroblast growth factor-2 in skin of mice lacking RXRal
251 nuated increases in glutamine synthetase and fibroblast growth factor-2 in the reactive astrocytes.
252 nhibit the binding and mitogenic activity of fibroblast growth factor-2 in vascular smooth muscle cel
253 tion of platelet-derived growth factor-B and fibroblast growth factor-2 in VEGF-treated wounds, which
254 ndothelial cell basement membrane-associated fibroblast growth factor-2 increased linearly with cultu
257 acid docosahexaenoic acid, inhibit VEGF- and fibroblast growth factor 2-induced angiogenesis in vivo,
258 angiogenesis model and found that it reduced fibroblast growth factor-2-induced angiogenesis by 85% (
263 d the effects of three serum growth factors, fibroblast growth factor-2, insulin, and platelet-derive
264 7A receptors, matrix proteins, CCN proteins, fibroblast growth factor 2, interleukin 13 receptor comp
266 laque and vessel wall, through inhibition of fibroblast growth factor-2, leading to reduced plaque gr
267 Dendrimer glucosamine 6-sulfate blocked fibroblast growth factor-2 mediated endothelial cell pro
268 vascular endothelial growth factor and basic fibroblast growth factor-2 mRNA transcripts in ischemic
270 DF differentiation, via multiple cAMP and/or fibroblast growth factor 2 or basic FGF (FGF2)-dependent
271 ort that intranasal administration of either fibroblast growth factor-2 or heparin-binding epidermal
272 egulated kinase pathway after treatment with fibroblast growth factor-2 or heparin-binding epidermal
273 ntation and >/=50 ng/ml (P < 0.01) of either fibroblast growth factor-2 or vascular endothelial growt
274 and migration (hepatocyte growth factor and fibroblast growth factor 2) or transplantation on materi
275 loss of heparanase 2 led to upregulation of fibroblast growth factor 2/phosphorylated extracellular
276 ession patterns of hepatocyte growth factor, fibroblast growth factor 2, platelet-derived growth fact
278 instead related to enhanced endothelial cell fibroblast growth factor-2 release and permeability.
281 that neprilysin proteolytically inactivates fibroblast growth factor-2, resulting in negative regula
283 effects of the administration of recombinant fibroblast growth factor-2 (rFGF-2) protein on myocardia
284 al stem cell cultures, which is required for fibroblast growth factor 2's (FGF-2) mitogenic activity
285 in-1, matrix metallopeptidase 9 (MMP-9), and fibroblast growth factor 2 serum levels when compared wi
286 s showed abnormal HS composition and altered fibroblast growth factor 2 signaling, which was rescued
287 ic protein-4 and inhibitors of activin A and fibroblast growth factor-2 signaling (BAP treatment).
288 diated by alpha4beta1, whereas antagonism of fibroblast growth factor-2-stimulated chemotaxis is not
289 variation, we observed changes in netrin 4, fibroblast growth factor 2, tenascin C, collagen 1, mepr
290 nesis (vascular endothelial growth factor-A, fibroblast growth factor-2), thrombosis (D-dimer, von Wi
291 oss of cleavage and increased the potency of fibroblast growth factor-2 to induce capillary array for
292 lysis of vascular endothelial growth factor, fibroblast growth factor 2, transforming growth factor b
293 demonstrated increased expression levels of fibroblast growth factor-2, transforming growth factor-b
294 free medium with epidermal growth factor and fibroblast growth factor 2, varied in the level of CD133
296 lucose-induced increase in basement membrane fibroblast growth factor-2 was instead related to enhanc
297 stimulation by EGF and TGF-alpha, but not by fibroblast growth factor 2, was almost completely blocke
300 ed but is then rescued by bath-applied FGF2 (fibroblast growth factor 2), which can activate the hear
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